Could my MCS be caused by abnormal mucus membrane IgA response?

[Guest guest]

Excerpt:

Most disease-causing organisms enter your body through the mucous

membranes of your nose, mouth, pulmonary system or your digestive tract – ...

These mucous membranes have their own immune system, called the IgA

immune system. It is a different system from the one activated when a

vaccine is injected into your body.

Your IgA immune system is your body's first line of defense. Its job

is to fight off invading organisms at their entry points, reducing or

even eliminating the need for activation of your body's immune system.

When a virus is injected into your body in a vaccine, and especially

when combined with an immune adjuvant like squalene, your IgA immune

system is bypassed and your body's immune system kicks into high gear

in response to the vaccination.

--

I add:

The bolding above is mine. This sounds like MCS to me.

So, could MCS be that the initial IgA system in the mucous membranes

is turned off?

That my symptom of mold hypersensitivity was pain in my nose for 15 seconds,

and then I could no longer smell or taste food... and the doctor said

that is due

to rapid inflammation in the mucus membrane that pinches the nerve that goes

from the membrane to the brain.

Could I still have 'a' pinched nerve, even though I can taste food.

Or damage to the mucus membrane IgA system?

This would mean that when the exposure substance gets into my blood,

my immune system kicks into high gear, and gives me the range of

symptoms I have.

It makes sense to me, but now I need to determine if this is happening in me.

How does one " test " the mucus membrane for IgA response?

I can think of several other mechanisms involved here, other than one

proposed above.

--

http://articles.mercola.com/sites/articles/archive/2009/08/04/Squalene-The-Swine\

-Flu-Vaccines-Dirty-Little-Secret-Exposed.aspx

Oil-based vaccination adjuvants like squalene have been proved to

generate concentrated, unremitting immune responses over long periods of time

The researchers concluded the study raised questions about the role

of adjuvants in chronic inflammatory diseases