Re: K, this got me to thinking

[Guest guest]

it seems pretty clear to me that the difference in still haveing your olfactory

system/olfactory nerve/tract in working order would also

go along with what might be considered as a cemical sensitivity that is more

common and to varing degrees, dependant on exposures.

but damage to the olfactory system/nerve/tracts is just a whole other side of

this. so really, I can see where MCS has it's place in medical science,as long

as it's reconized that more damage to the olfactory system is something that

goes beyond brain fog.

it seems to me that alot of research is based around a lack of consideration of

what physical damage can accure to organs and tissues with chronic exposure and

high dose exposure.

it's just like the whole " you cant get a fungal infection unless your

immunocompromized " thing. yes, you can! and it's based on the direct physical

damage. I dont understand how and why medical science has failed to reconize

this, the only possability is that it comes down to pretended ignorance, and the

money trail.

geez, if I can comprehend this with my disfunctional brain, theres really no

excuse whatsoever.

all the many people who have suffered for years, some even loseing their lifes

do to WDB exposures.

what a sad f#ing world we live in.

and I'm here basically because of other's greed.

that makes me angry.

I think we are all really here because of greed, no matter what or how it

happened cause I just cant see any excuse of why the effects of toxin exposure

,no matter what the toxin, no matter if it's from self or non-self, who cares,

it's toxic and it causes harm.

I'm just rather discusted with it all. pretended ignorance and the money trail,

thats all it comes down to and thats why we are all here.

>

> you know that article you posted about MCS, it said that you only react to

what you exposed to that you become reactive to. I just dont know how thats

possable. seems you would become reactive to anything that

> has any properties of/or family of a chemical that you were exposed to.

>

> disorders of taste and smell/olfactory

>

> The chemical compounds that are inspired in the air will pass through the

nasal vault, but only 15% of inspired air passes by the olfactory mucosa with

regular breathing. During a sniff, however, a much higher percentage of

inspired air is exposed to the neuroepithelium. Along the ciliated ends of the

ORNs are the olfactory receptors capable of binding the odorants in the air and

signaling the brain of the odor's presence. There are over 1000 genes for the

odorant receptors, 3% of our genome, but due to chemical similarity, receptor

specificity or unspecificity, concentration, binding affinity, solubility, and

exposure time, we are able to process many more than these 1000 odors. The

odorants must traverse the mucus overlying the epithelium, and hydrophobic

compounds must bind transport proteins to carry them to the olfactory receptors.

The binding of the odorant to the olfactory receptor causes activation of the

G-protein class olfactory receptor, leading to influx of Ca2+ via c-AMP pathways

which results in depolarization of the ORN and neuronal communication. The

common chemosensory pathways are separate from those of CN I, as these are

mediated by the first and second division of CN V. Reflexes such and sneezing,

breath holding in the face of noxious odors, and reaction to ammonia are handled

by this pathway.

>

>

>

> Toxin induced olfactory loss is due to noxious chemicals inhaled that result

in direct damage to the neuroepithelium, alterations in the mucus layer, and

ingrowth of respiratory mucosa. There are many toxins that can lead to

olfactory loss, so precautionary safety measures should be used to protect

against this problem.

>

> http://www8.utsouthwestern.edu/utsw/cda/dept28151/files/289930.html

>

[Guest guest]

Go to Pubmed and search for craniocerebral aspergillosis. There are several

cases studies on immune competent individuals with Aspergillomas of the brain.

The portal of entry is via the paranasal sinuses. You will also find papers of

involvement of the sella and pituitary gland.

The senior author on a paper of 25 cases is Siddiqui

Dr. Dennis has also published a paper on the pituitary involvement of his

patients.

[] Re: K, this got me to thinking

it seems pretty clear to me that the difference in still haveing your

olfactory system/olfactory nerve/tract in working order would also

go along with what might be considered as a cemical sensitivity that is more

common and to varing degrees, dependant on exposures.

but damage to the olfactory system/nerve/tracts is just a whole other side of

this. so really, I can see where MCS has it's place in medical science,as long

as it's reconized that more damage to the olfactory system is something that

goes beyond brain fog.

it seems to me that alot of research is based around a lack of consideration

of what physical damage can accure to organs and tissues with chronic exposure

and high dose exposure.

it's just like the whole " you cant get a fungal infection unless your

immunocompromized " thing. yes, you can! and it's based on the direct physical

damage. I dont understand how and why medical science has failed to reconize

this, the only possability is that it comes down to pretended ignorance, and the

money trail.

geez, if I can comprehend this with my disfunctional brain, theres really no

excuse whatsoever.

all the many people who have suffered for years, some even loseing their lifes

do to WDB exposures.

what a sad f#ing world we live in.

and I'm here basically because of other's greed.

that makes me angry.

I think we are all really here because of greed, no matter what or how it

happened cause I just cant see any excuse of why the effects of toxin exposure

,no matter what the toxin, no matter if it's from self or non-self, who cares,

it's toxic and it causes harm.

I'm just rather discusted with it all. pretended ignorance and the money

trail, thats all it comes down to and thats why we are all here.

[Guest guest]

Is it possible for bone spurs to be caused from this? I'll read. I have a bone

spur on my head I have to look into. I had them on my sinuses as well.

Kathy

From: Jack Thrasher, Ph.D. <toxicologist1@...>

Subject: Re: [] Re: K, this got me to thinking

Date: Saturday, July 10, 2010, 4:45 PM

Go to Pubmed and search for craniocerebral aspergillosis. There are

several cases studies on immune competent individuals with Aspergillomas of the

brain. The portal of entry is via the paranasal sinuses. You will also find

papers of involvement of the sella and pituitary gland.

The senior author on a paper of 25 cases is Siddiqui

Dr. Dennis has also published a paper on the pituitary involvement of his

patients.

[Guest guest]

Thanks for the info. Dr. Thrasher

--- In , " Jack Thrasher, Ph.D. " <toxicologist1@...>

wrote:

>

> Go to Pubmed and search for craniocerebral aspergillosis. There are several

cases studies on immune competent individuals with Aspergillomas of the brain.

The portal of entry is via the paranasal sinuses. You will also find papers of

involvement of the sella and pituitary gland.

>

> The senior author on a paper of 25 cases is Siddiqui

>

> Dr. Dennis has also published a paper on the pituitary involvement of

his patients.