Update from Dr.Shaw - ACETAMINOPHEN AND ITS POSSIBLE CONNECTION TO THE RISE IN AUTISM

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pdate from Dr.Shaw - ACETAMINOPHEN AND ITS POSSIBLE CONNECTION TO THE

RISE IN AUTISM

http://ventography.wordpress.com/2012/03/02/update-from-dr-shaw-acetaminophen-and-autism-connection/

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Back in January, we did a post summarizing what we learned from a webinar

we attended given by Dr. Shaw of Great Plains Lab. He discussed

the connection between autism and acetaminophen. Dr. Shaw did not release

any official information or transcripts after the webinar, despite

tremendous public interest, because he had not yet released his official

study. But just yesterday, we got Dr. Shaw’s e-newsletter, with an

official statement about the autism acetaminophen connection.

Here is a summary of what the update said (in mom-lingo):

“It appears that the marked increase in the rate of autism

throughout much of the world may be largely mediated by the marked

increase in the use of acetaminophen in genetically and/or metabolically

susceptible children and perhaps the use of acetaminophen by pregnant

women.” Because many children with autism have defective sulfation, they

cannot properly detoxify acetaminophen. Therefore, it becomes toxic. This then leads to intestinal Clostridia bacteria overgrowth. Then the Clostridia bacteria cause overproduction of brain dopamine

and reduced concentrations of brain norepinephrine. Too much dopamine leads to obsessive, compulsive, and stereotypical

behaviors. Too little norepinephrine leads to reduced exploratory behavior and

learning in new environments. Because sulfation is often defective for people with autism, the body

cannot use proper pathways to get rid of acetaminophen. Therefore, the

body sends a larger than normal amount of acetaminophen to be detoxified

by a pathway called cytochrome p450 2E1. The problem with this pathway is that it leads to excessive

production of N-acetyl-p-benzoquinone imine (NAPQI), a very toxic

metabolite. NAPQI is dangerous because it depletes glutathione which then reduces

the body’s ability to detoxify a host of toxic chemicals in the

environment. In addition, the increase of NAPQI creates oxidative stress which

leads to protein, lipid, and nucleic acid damage from free radicals. It

also causes an increased rate of damage to mitochondrial and nuclear DNA. NAPQI production has been found to be increased in humans at

recommended dosages of acetaminophen and would be expected to be even

higher in people with diminished sulfation capacity (as is often seen in

autism). BOTTOM LINE: Question your acetaminophen use.

By the way, acetaminophen is not just linked to the increase in autism.

It has also been implicated in the rise in asthma. For more information,

please read the following article

http://www.npr.org/blogs/health/2012/02/20/147002356/does-tylenol-worsen-asthma-for-kids

..

For more information on Dr. Shaw’s original webinar about this topic,

please read:

http://ventography.wordpress.com/2012/01/14/acetaminophen-and-its-possible-connection-to-the-rise-in-autism/

..

If you want to stay updated about news on this topic and future studies,

please visit Dr. Shaw’s facebook page at:

http://www.facebook.com/acetaminophenandautism.

Sheri Nakken, former R.N., MA, Hahnemannian

Homeopath

Vaccination Information & Choice Network, Washington State, USA

Vaccines -

http://vaccinationdangers.wordpress.com/ Homeopathy

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