Jump to content
RemedySpot.com

Cytokine Storm

Rate this topic


Guest guest

Recommended Posts

Guest guest

This is an article I found. I thought it might be of use to those of

us in the group. I had never heard of this until I found out this is

what is making me so sick and I can not seem to get better from a

simple virus!

http://www.wisegeek.com/what-is-the-cytokine-storm.htm

We think of external microbes as our worst enemy during an outbreak

of influenza or bronchitis, but our own immune systems are

potentially more lethal. When our body detects foreign microorganisms

indicating an infection, it might over-protect our lungs. We race so

many antibodies to the site that they collect in a Cytokine storm,

potentially blocking airways and causing suffocation. Medical

researchers have identified the stages of the Cytokine storm and are

working on treatments, other than flu vaccines, to weaken an

overactive immune response.

At all times, sentries circulate in our bloodstream, called white

blood cells, that are the first to sense if a virus or bacteria has

infiltrated. Immediately, our body sends defenders from the immune

system, T-cells, to the site of the infection. During this stage, our

immunity functions properly, and T-cells attack the microbes so they

do not get too strong a foothold in our lungs.

However, the mere presence of T-cells clustered at one site,

especially the lungs, alerts other T-cells that a full-scale war has

started. In the second stage, even more T-cells, known as cytokines,

flood the lungs. This propagates a Cytokine storm where far too many

immune cells are caught in an endless loop of calling even more. The

Cytokine storm ends up inflaming the tissue of the lungs and crowding

air passages, causing breathing difficulties.

Not only can severe inflammation damage your lungs permanently, but a

prolonged Cytokine storm will eventually shut down your breathing.

Airducts get clogged and cells no longer properly absorb oxygen. This

is what makes the Cytokine storm so deadly in certain epidemic

strains, such as bird flu. Even bronchitis, other varieties of

influenza, pneumonia, and possibly rheumatoid arthritis are

susceptible to triggering a Cytokine storm.

Of course, flu vaccines are usually effective at preventing the flu

during its peak season. But they are no guarantee, especially when

flu strains mutate after the vaccine has been manufactured.

Therefore, researchers are pursuing other methods of preventing the

Cytokine storm by bioengineering a drug that could slow the snowball

effect of antibodies. They hope to force the cytokines to recirculate

in the bloodstream, rather than pool in the lungs. Experts predict

that a major influenza pandemic could kill millions of people

worldwide as it has done in centuries past.

Link to comment
Share on other sites

Guest guest

Toni, I'll attempt to answer your question about this soon, but wanted to

mention now that this article has many errors - maybe this geek isn't so

wise.

Not an MD

I'll tell you where to go!

Mayo Clinic in Rochester

http://www.mayoclinic.org/rochester

s Hopkins Medicine

http://www.hopkinsmedicine.org

[ ] Cytokine Storm

> This is an article I found. I thought it might be of use to those of

> us in the group. I had never heard of this until I found out this is

> what is making me so sick and I can not seem to get better from a

> simple virus!

>

> http://www.wisegeek.com/what-is-the-cytokine-storm.htm

>

> We think of external microbes as our worst enemy during an outbreak

> of influenza or bronchitis, but our own immune systems are

> potentially more lethal. When our body detects foreign microorganisms

> indicating an infection, it might over-protect our lungs. We race so

> many antibodies to the site that they collect in a Cytokine storm,

> potentially blocking airways and causing suffocation. Medical

> researchers have identified the stages of the Cytokine storm and are

> working on treatments, other than flu vaccines, to weaken an

> overactive immune response.

>

> At all times, sentries circulate in our bloodstream, called white

> blood cells, that are the first to sense if a virus or bacteria has

> infiltrated. Immediately, our body sends defenders from the immune

> system, T-cells, to the site of the infection. During this stage, our

> immunity functions properly, and T-cells attack the microbes so they

> do not get too strong a foothold in our lungs.

>

> However, the mere presence of T-cells clustered at one site,

> especially the lungs, alerts other T-cells that a full-scale war has

> started. In the second stage, even more T-cells, known as cytokines,

> flood the lungs. This propagates a Cytokine storm where far too many

> immune cells are caught in an endless loop of calling even more. The

> Cytokine storm ends up inflaming the tissue of the lungs and crowding

> air passages, causing breathing difficulties.

>

> Not only can severe inflammation damage your lungs permanently, but a

> prolonged Cytokine storm will eventually shut down your breathing.

> Airducts get clogged and cells no longer properly absorb oxygen. This

> is what makes the Cytokine storm so deadly in certain epidemic

> strains, such as bird flu. Even bronchitis, other varieties of

> influenza, pneumonia, and possibly rheumatoid arthritis are

> susceptible to triggering a Cytokine storm.

>

> Of course, flu vaccines are usually effective at preventing the flu

> during its peak season. But they are no guarantee, especially when

> flu strains mutate after the vaccine has been manufactured.

> Therefore, researchers are pursuing other methods of preventing the

> Cytokine storm by bioengineering a drug that could slow the snowball

> effect of antibodies. They hope to force the cytokines to recirculate

> in the bloodstream, rather than pool in the lungs. Experts predict

> that a major influenza pandemic could kill millions of people

> worldwide as it has done in centuries past.

Link to comment
Share on other sites

Guest guest

Ok, that is good to know...I was searching for more medical abstracts

and published journal articles but did not fid much..I know you are

the maste and finding correct info. I will be patient and wait for

you find some facts.

Toni

--- In , " " <Matsumura_Clan@...>

wrote:

>

> Toni, I'll attempt to answer your question about this soon, but

wanted to

> mention now that this article has many errors - maybe this geek

isn't so

> wise.

>

>

>

> Not an MD

>

> I'll tell you where to go!

>

> Mayo Clinic in Rochester

> http://www.mayoclinic.org/rochester

>

> s Hopkins Medicine

> http://www.hopkinsmedicine.org

>

>

> [ ] Cytokine Storm

>

>

> > This is an article I found. I thought it might be of use to

those of

> > us in the group. I had never heard of this until I found out

this is

> > what is making me so sick and I can not seem to get better from a

> > simple virus!

> >

> > http://www.wisegeek.com/what-is-the-cytokine-storm.htm

> >

> > We think of external microbes as our worst enemy during an

outbreak

> > of influenza or bronchitis, but our own immune systems are

> > potentially more lethal. When our body detects foreign

microorganisms

> > indicating an infection, it might over-protect our lungs. We race

so

> > many antibodies to the site that they collect in a Cytokine storm,

> > potentially blocking airways and causing suffocation. Medical

> > researchers have identified the stages of the Cytokine storm and

are

> > working on treatments, other than flu vaccines, to weaken an

> > overactive immune response.

> >

> > At all times, sentries circulate in our bloodstream, called white

> > blood cells, that are the first to sense if a virus or bacteria

has

> > infiltrated. Immediately, our body sends defenders from the immune

> > system, T-cells, to the site of the infection. During this stage,

our

> > immunity functions properly, and T-cells attack the microbes so

they

> > do not get too strong a foothold in our lungs.

> >

> > However, the mere presence of T-cells clustered at one site,

> > especially the lungs, alerts other T-cells that a full-scale war

has

> > started. In the second stage, even more T-cells, known as

cytokines,

> > flood the lungs. This propagates a Cytokine storm where far too

many

> > immune cells are caught in an endless loop of calling even more.

The

> > Cytokine storm ends up inflaming the tissue of the lungs and

crowding

> > air passages, causing breathing difficulties.

> >

> > Not only can severe inflammation damage your lungs permanently,

but a

> > prolonged Cytokine storm will eventually shut down your breathing.

> > Airducts get clogged and cells no longer properly absorb oxygen.

This

> > is what makes the Cytokine storm so deadly in certain epidemic

> > strains, such as bird flu. Even bronchitis, other varieties of

> > influenza, pneumonia, and possibly rheumatoid arthritis are

> > susceptible to triggering a Cytokine storm.

> >

> > Of course, flu vaccines are usually effective at preventing the

flu

> > during its peak season. But they are no guarantee, especially when

> > flu strains mutate after the vaccine has been manufactured.

> > Therefore, researchers are pursuing other methods of preventing

the

> > Cytokine storm by bioengineering a drug that could slow the

snowball

> > effect of antibodies. They hope to force the cytokines to

recirculate

> > in the bloodstream, rather than pool in the lungs. Experts predict

> > that a major influenza pandemic could kill millions of people

> > worldwide as it has done in centuries past.

>

Link to comment
Share on other sites

Join the conversation

You are posting as a guest. If you have an account, sign in now to post with your account.
Note: Your post will require moderator approval before it will be visible.

Guest
Reply to this topic...

×   Pasted as rich text.   Paste as plain text instead

  Only 75 emoji are allowed.

×   Your link has been automatically embedded.   Display as a link instead

×   Your previous content has been restored.   Clear editor

×   You cannot paste images directly. Upload or insert images from URL.

Loading...
×
×
  • Create New...