Terri, Love & Prayers to You

[Guest guest]

Terri, I am so sorry you had to go through that. That insensitive so-

and-so.

Two years ago I left a psychiatrist's office crying, I also am

a " tough cookie " ...went straight to a safe place, the Women's

Outreach Drop-in Centre. Saw my favorite crisis counsellor. She

heard my story and said, " you're one of many women who have come in

here and complained about him " . I told her I wanted to report him to

the College of Physicians and Surgeons. She said that would be

awesome, because he's hurt alot of women. Very emotionally abusive

and power-tripping. So I did. It was a very difficult and lengthy

process. The patient advocate assigned to me warned me that it

probably " wouldn't go anywhere " . I said that's o.k. I'll do it

anyway. He was never made accountable and denied everything that

happened. But he does have a black mark against him. So if he gets a

few more....voila!!

I surprised him by getting a copy of a letter he sent to my family

doctor, making me sound like a real nut case. I included it along

with my complaint forms. Ha, ha!

I'll pray for you, Terri....comforting angels are on their way!

Love & Lots of Hugs,

Sunny

> > >>>>

> > >>>>> Dearest Rogene:

> > >>>>>

> > >>>>> Thank you for posting this information and for

the " contact " .

> I

> > >>>>> still have folders that will not be deleted, but some of the

> > > hard

> > >>>>> copies have been destroyed. It hurts to help me to

> > > destroy

> > >>> all

> > >>>>> of my hard work, but we have no choice. This was not about

> > > money,

> > >>> I

> > >>>>> just knew too much in an Oil Rich province.

> > >>>>>

> > >>>>> Love you honey...Lea

> > >>>>> ~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~``

> > >>>>> Fwd: Immunosciences Lab: Autoimmune

> > >>> Diseases

> > >>>>>

> > >>>>>

> > >>>>>

> > >>>>>

> > >>>>> Autoimmune Diseases

> > >>>>> Page 1 of 2

> > >>>>>

> > >>>>> Autoimmunity, in which the immune system recognizes and

> attacks

> > >>> the

> > >>>>> self's own tissue, is not as simple as it seems. Self-

> > > recognition

> > >>>>> appears to be at the heart of health as well as of certain

> > >>> diseases.

> > >>>>> It is generally assumed that the main job of the immune

system

> > >>> is

> > >>>>> to distinguish between what is " self " and what is " not

self " .

> > >>> Once

> > >>>>> the distinction has been made, " self " is pre-served and " not

> > >>> self "

> > >>>>> is destroyed. At the most general level, of course, this is

> > > true,

> > >>>>> and human beings remain alive and healthy only because it is

> > > so.

> > >>>>> Recently it has become clear, however, that at a finer level

> > > of

> > >>>>> detail the distinction between self and other is not

absolute.

> > >>> One

> > >>>>> of the paths to this insight has been provided by the

> > > autoimmune

> > >>>>> disorders, in which the immune system attacks normal,

healthy

> > >>>>> tissue. Autoimmune disease, which may be crippling or fatal,

> > > can

> > >>>>> strike any tissue or organ. Its victims are often in the

> > > prime of

> > >>>>> life, and for unknown reasons they are more frequently women

> > > than

> > >>> men.

> > >>>>> Research work on a form of autoimmune arthritis shows that

the

> > >>> basis

> > >>>>> of autoimmunity may be a resemblance between a specific

> foreign

> > >>>>> molecule and a molecule of the self. This finding is

> consistent

> > >>> with

> > >>>>> a model of the immune system in which the immune system

> > > receptors

> > >>>>> that perform the work of recognition can themselves be

> > > recognized

> > >>> by

> > >>>>> other receptors. Such " self-recognition, " which was strictly

> > >>>>> outlawed by older models of the immune sys-tem, may form the

> > > basis

> > >>>>> of a network whose equilibrium keeps the body healthy. When

it

> > > is

> > >>>>> disrupted, as it is in autoimmunity, disease results.

> > >>>>> This new picture, in which self and world are no longer

> > > absolutely

> > >>>>> distinct, has already begun to yield practical benefits in

the

> > >>> form

> > >>>>> of vaccines that may ultimately ease the substantial

suffering

> > >>>>> caused by autoimmune diseases. The list of autoimmune

diseases

> > > is

> > >>>>> both long and disturbing. It includes multiple sclerosis, in

> > > which

> > >>>>> the tissue attacked is myelin (a sub-stance that sheathes

> > > nerves

> > >>> in

> > >>>>> the central nervous system); myasthenia gravis, in which the

> > >>> target

> > >>>>> is a receptor molecule for the important neurotransmitter

> > >>>>> acetylcholine; rheumatoid arthritis, whose target is the

> > >>> peripheral

> > >>>>> joint; type I (juvenile) diabetes mellitus, in which the

cells

> > >>>>> producing insulin are destroyed, and systemic lupus

> > > erythematosus,

> > >>>>> in which DNA, blood vessels, skin and kidneys are attacked.

In

> > >>>>> contrast to AIDS, which is marked by an in activation of key

> > > cells

> > >>>>> in the immune system, in all these diseases the

immunological

> > >>>>> response is strong and well focused; it is, however,

directed

> > > at

> > >>>>> some essential component of the body. These immunological

> > > attacks

> > >>>>> are detected in clinical laboratory by the measurement of

> > >>>>> tissue-specific and tissue non-specific antibodies.

> > >>>>>

> > >>>>> Autoimmune Diseases

> > >>>>> Page 2 of 2

> > >>>>>

> > >>>>> Autoimmune diseases can be separated broadly into two

> > > categories.

> > >>>>> One group is characterized by the presence of auto

antibodies

> > >>> which

> > >>>>> are broadly reactive with nuclear or cytoplasmic antigens

and

> > > do

> > >>> not

> > >>>>> demonstrate any tissue specificity. Included in this group

are

> > >>>>> diseases such as rheumatoid arthritis, SLE, mixed connective

> > >>> tissue

> > >>>>> disease, scleroderma, Sjogren's syndrome, and

> > >>>>> dermatomyositis/polymyositis. A second group of autoimmune

> > >>> diseases

> > >>>>> is characterized by autoantibodies which demonstrate tissue

> > >>>>> specificity. These diseases include thyroiditis, chronic

liver

> > >>>>> diseases (including primary biliary cirrhosis and chronic

> > > active

> > >>>>> Hepatitis), certain cases of pernicious anemia, and

myasthenia

> > >>> gravis.

> > >>>>> The autoantibodies which appear in these disease states

> > >>> demonstrate

> > >>>>> different degrees of specificity with respect to both tissue

> > > and

> > >>>>> species. Tissue-specific autoantibodies include those which

> > > react

> > >>>>> against erythrocyte stromalantigens, platelets,

antihemophilic

> > >>>>> globulin, thyroid tissue and other tissues, and g-globulin

> > >>>>> (rheumatoid factor). Autoantibodies without tissue

specificity

> > >>>>> include anti-nuclear, anti-nucleoprotien, anti-DNA, and

> > >>>>> anti-cytoplasmicantibodies. Autoantibodies directed against

> the

> > >>>>> formed elements of the blood can undoubtedly induce disease

by

> > >>>>> causing the destruction and removal of these cells from the

> > >>>>> circulation. However, it is far less certain whether other

> > > types

> > >>> of

> > >>>>> autoantibodies play pathogenic roles.

> > >>>>> Most studies of autoantibodies in both humans and animals

have

> > >>>>> concentrated on the reactivity of humoral constituents.

> > > However,

> > >>> it

> > >>>>> should be remembered that the cell-mediated immune response

is

> > > far

> > >>>>> more efficient in terms of tissue destruction. Since humoral

> > >>>>> antibodies have been shown, under appropriate

circumstances,

> to

> > >>>>> prevent cell-mediated tissue damage, it is conceivable that

> > > they

> > >>> may

> > >>>>> have a protective rather than a destructive function. There

is

> > >>>>> presently no indication whether the autoantibodies detected

in

> > >>> human

> > >>>>> disease rep-resent a primary manifestation of the disease

> > > itself

> > >>> or

> > >>>>> a secondary event stimulated by an underlying, but

unrelated,

> > >>>>> abnormality. In ether event, the mechanisms which may be

> > >>> responsible

> > >>>>> for the abrogation of natural immunologic tolerance are

worthy

> > > of

> > >>>>> consideration. Four general mechanisms have been proposed in

> > > the

> > >>>>> pathogenesis of autoantibody production:1.alterations in the

> > >>>>> structure or distribution of antigens;2.formation of cross-

> > >>> reactive

> > >>>>> antibodies following exposure to extrinsic

antigens;3.release

> > > of

> > >>>>> sequestered antigens; and4.abnormalities of immunologic

> > > responsive-

> > >>> ness.

> > >>>>> An assault on the self through molecular mimicry or

antigenic

> > >>>>> similarity between foreign antigens (virus, bacreria) and

> human

> > >>>>> tissue antigens which may end with an autoimmune disease is

> > >>>>> presented in <<http://www.immuno-sci->http://www.immuno-sci-

> > >>> lab.com/2003_cat_page27.htm>Fig.

> > >>>>> 8. This process which may strike many target tissues is

shown

> > > in

> > >>> Table 1.

> > >>>>>

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