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Fwd: [HED] Mechanisms of thermogenesis induced by low protein diets.

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I thought I would forward this from another board and look forward to your

insight and replies.

Yours Truly,

Dan Holt

Begin forwarded message:

From: Holt <danthemanholt@...>

Date: April 13, 2009 2:46:17 PM PDT

" High-Everything-Diet "

<High-Everything-Diet >

Subject: Re: [HED] Mechanisms of thermogenesis induced by low protein diets.

Reply-High-Everything-Diet

It seems that the three most important PUFAs to take in from

The diet are AA, DTA, and DHA.

I would imagine that most people have a DHA deficiency as most fats that contain

DHA also contain EPA in higher quantities which gets in the way of DHA

absorption.

It would be ideal to eat foods the have a higher ratio of DHA over EPA, and eat

foods that contain AA and DTA.

I have not been able to find a list that tells you what foods contain DTA. I

know that eggs contain AA, and the free range eggs would also carry the DHA.

The best PUFA containing fat is from an animal brain such as a cow. The fatty

profile is a 2-1 ratio of DHA to EPA. It is also high in AA. Based on the

profile I looked at DTA is not specified.

AA (Arachidonic acid) has 20 carbons and 4 double bonds.

DHA (Docosahexaenioc acid) is 22 carbons and 6 double bonds.

DTA (Docosatetraenoic acid) is 22 carbons and 4 double bonds

Yours Truly,

Dan Holt

On Apr 13, 2009, at 1:45 PM, " Will " <wlorkovic@...> wrote:

> > http://www.ncbi.nlm.nih.gov/pubmed/6827996

> >

> > Mechanisms of thermogenesis induced by low protein diets.

> > Rothwell NJ, Stock MJ, Tyzbir RS.

>

> Here's another study showing that a low-protein, high-fat,

> high-carb diet helps make brown fat.

>

This one talks about how w-3's are better than w-6's in generating more BAT

activity but since no one here is a huge fan of PUFA's, the point is probably

moot, LOL!

http://cat.inist.fr/?aModele=afficheN & cpsidt=1478094

High-fat diets relative to a low-fat diet increased brown adipose tissue

uncoupling protein I mRNA level. The increases were greater with fats rich in

n-3 PUFA than with n-6 PUFA. A high-safflower-oil diet, compared to a low-fat

diet, doubled the leptin mRNA level in white adipose tissue. However, high-fat

diets rich in n-3 PUFA failed to increase it. Compared to a low-fat diet,

high-fat diets down-regulated the glucose transporter 4 mRNA level in white

adipose tissue. However, the decreases were attenuated with high-fat diets rich

in n-3 PUFA. It is suggested that the alterations in gene expression in adipose

tissue contribute to the physiological activities of n-3 PUFA in preventing body

fat accumulation and in regulating glucose metabolism in rats.

This is only part of the abstract. No access to the full article.

Will

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