ALA - direct inhibitor of retroviral replication

[Guest guest]

Why would anyone not want to get rid of the metals? Just because there is no

longer an autism diagnosis does not mean there won & #39;t be health problems in

teen years or adulthood such as alzheimers.

Kulp wrote:

>  

> OK I had never heard of anyone who still has metals,but has lost their

autism diagnosis.

>  

>

                                        \

             

> From: Christel King < christelking1@ myfairpoint. net > Subject: Re:

Re: ALA - direct inhibitor of retroviral replication To:

mb12 valtrex@ yahoogroups. com Date: Friday, November 20, 2009, 8:59 AM

>  

> then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate!   not ALL autistic kids have

mercury issues!  my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n  we had tested 2 weeks prior

developmental testing and he was 6 months advanced.  after 7 hours after his

MMR he became autistic, lost lang, no eye contact, started seizering, ect.  we

tested him 2 weeks after this because I had no idea what had happened to my son,

he then had the develeopment of a 2-4 month old.  we LITERIALLY lost 14-16

months development from the MMR.....NOT EVERYTHING IS METALS~!  if it was he

would have become autistic when the thermitor exploded at 6 months old in our

dishwasher and he didn't then!!!!  it would also mean that my son would STILL

be autistic since we haven't chelated with

> your therory and he is not!

>  

> Re: ALA - direct inhibitor of retroviral replication

> “Could it be that the chelators work as antiretrovirals by removing

mercury? “ , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached btw some of

these studies are in vitro, no mercury in sight, so the answer is NO they do not

work as antivirals through chelating mercury, at least not exclusively (meaning

to say that who knows maybe heavy metals are also directly utilised by some

viruses) And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would

> not be unfolding in the same way when chelators added. Having said that there

are studies on herpes/CMV and others that indicate same things going on.   The

influence of divalent cations on the stability of human rotavirus. The influence

of divalent cations on the stability of human rotavirus was investigated using

the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate

infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions

in solution but not by magnesium ions. Rotavirus isolates were found to be much

less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of

virus morphology at intervals during the course of the experiment and treatment

with the chelating agents EDTA and EGTA suggests that loss of infectivity

coincides with the removal of the outer capsid layer and that calcium may be

required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA,

> Beards GM, Thouless ME, Flewett TH Biochemical studies of primate

retroviruses. In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and

> type D primate retroviruses in persistently infected human cells. From

experiments demonstrating a disintegrating activity of chelating agents (EDTA,

EGTA) and certain psychoactive drugs (including trifluoperazine) on various

primate and nonprimate retroviruses it is concluded that divalent cations,

probably Ca2+ ions, and possibly also cation-binding proteins are associated

with retroviral membranes and that complexing of these components results in

loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch

Geschwulstforsch. 1983;53(3):267- 78.

>

[Guest guest]

There are kids who are in a 'maintained recovery'. They need supplements and

diet on an ongoing basis. If you chelate long enough, you shouldn't need the

supps or diet anymore.

>

>

> From: Christel King <christelking1@ myfairpoint. net>

> Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> To: mb12 valtrex@ yahoogroups. com

> Date: Friday, November 20, 2009, 8:59 AM

>

>

>  

>

>

> then I guess we don't have mercury in the brain because our son isn't autistic

anymore....and we didn't chelate!   not ALL autistic kids have mercury

issues!  my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n  we had tested 2 weeks prior

developmental testing and he was 6 months advanced.  after 7 hours after his

MMR he became autistic, lost lang, no eye contact, started seizering, ect.  we

tested him 2 weeks after this because I had no idea what had happened to my son,

he then had the develeopment of a 2-4 month old.  we LITERIALLY lost 14-16

months development from the MMR.....NOT EVERYTHING IS METALS~!  if it was he

would have become autistic when the thermitor exploded at 6 months old in our

dishwasher and he didn't then!!!!  it would also mean that my son would STILL

be autistic since we haven't chelated with your therory and he is not!

>  

>

> Re: ALA - direct inhibitor of retroviral replication

>

> “Could it be that the chelators work as antiretrovirals by removing mercury?

“

>

> , that is not the case, that is not how chelators work here. They work

by removing ions from cation-binding viral proteins, causing unfolding/loss of

protein functionality. For example by opening zinc-finger proteins and similar.

There are other mechanisms observed, such as inhibiting viral DNA synthesis

through RNA reductase deactivation, see attached

>

> btw some of these studies are in vitro, no mercury in sight, so the answer is

NO they do not work as antivirals through chelating mercury, at least not

exclusively (meaning to say that who knows maybe heavy metals are also directly

utilised by some viruses)

>

> And whatever is observed to happen in HIV only is only because HIV is studied

lot more than others. There are no studies that show other viral proteins would

not be unfolding in the same way when chelators added. Having said that there

are studies on herpes/CMV and others that indicate same things going on.

>

>

>  

> The influence of divalent cations on the stability of human rotavirus.

> The influence of divalent cations on the stability of human rotavirus was

investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

>

>

> Biochemical studies of primate retroviruses.

> In the present paper, recent biochemical studies of retroviruses carried out

in our laboratory are summarized. Protein compositions, peptide maps of internal

structural proteins, neighborhoods of major structural proteins, and serological

properties of reverse transcriptases of type D virus isolates from human cells

(including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2

cells) were compared with those of type D viruses from Old World (Mason-Pfizer

virus of rhesus monkeys, langur virus) and New World (squirrel monkey

retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a

> disintegrating activity of chelating agents (EDTA, EGTA) and certain

psychoactive drugs (including trifluoperazine) on various primate and nonprimate

retroviruses it is concluded that divalent cations, probably Ca2+ ions, and

possibly also cation-binding proteins are associated with retroviral membranes

and that complexing of these components results in loss of viral infectivity.

Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

>

[Guest guest]

Yep and seizures later on too.

> > From: Christel King < christelking1@ myfairpoint. net > Subject: Re:

Re: ALA - direct inhibitor of retroviral replication To:

mb12 valtrex@ yahoogroups. com Date: Friday, November 20, 2009, 8:59 AM

> >  

> > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate!   not ALL autistic kids have

mercury issues!  my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n  we had tested 2 weeks prior

developmental testing and he was 6 months advanced.  after 7 hours after his

MMR he became autistic, lost lang, no eye contact, started seizering, ect.  we

tested him 2 weeks after this because I had no idea what had happened to my son,

he then had the develeopment of a 2-4 month old.  we LITERIALLY lost 14-16

months development from the MMR.....NOT EVERYTHING IS METALS~!  if it was he

would have become autistic when the thermitor exploded at 6 months old in our

dishwasher and he didn't then!!!!  it would also mean that my son would STILL

be autistic since we haven't chelated with

> > your therory and he is not!

> >  

> > Re: ALA - direct inhibitor of retroviral replication

> > “Could it be that the chelators work as antiretrovirals by removing

mercury? “ , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached btw some of

these studies are in vitro, no mercury in sight, so the answer is NO they do not

work as antivirals through chelating mercury, at least not exclusively (meaning

to say that who knows maybe heavy metals are also directly utilised by some

viruses) And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would

> > not be unfolding in the same way when chelators added. Having said that

there are studies on herpes/CMV and others that indicate same things going on.

  The influence of divalent cations on the stability of human rotavirus. The

influence of divalent cations on the stability of human rotavirus was

investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA,

> > Beards GM, Thouless ME, Flewett TH Biochemical studies of primate

retroviruses. In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and

> > type D primate retroviruses in persistently infected human cells. From

experiments demonstrating a disintegrating activity of chelating agents (EDTA,

EGTA) and certain psychoactive drugs (including trifluoperazine) on various

primate and nonprimate retroviruses it is concluded that divalent cations,

probably Ca2+ ions, and possibly also cation-binding proteins are associated

with retroviral membranes and that complexing of these components results in

loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch

Geschwulstforsch. 1983;53(3):267- 78.

> >

>

[Guest guest]

Yep and seizures later on too.

> > From: Christel King < christelking1@ myfairpoint. net > Subject: Re:

Re: ALA - direct inhibitor of retroviral replication To:

mb12 valtrex@ yahoogroups. com Date: Friday, November 20, 2009, 8:59 AM

> >  

> > then I guess we don't have mercury in the brain because our son isn't

autistic anymore....and we didn't chelate!   not ALL autistic kids have

mercury issues!  my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n  we had tested 2 weeks prior

developmental testing and he was 6 months advanced.  after 7 hours after his

MMR he became autistic, lost lang, no eye contact, started seizering, ect.  we

tested him 2 weeks after this because I had no idea what had happened to my son,

he then had the develeopment of a 2-4 month old.  we LITERIALLY lost 14-16

months development from the MMR.....NOT EVERYTHING IS METALS~!  if it was he

would have become autistic when the thermitor exploded at 6 months old in our

dishwasher and he didn't then!!!!  it would also mean that my son would STILL

be autistic since we haven't chelated with

> > your therory and he is not!

> >  

> > Re: ALA - direct inhibitor of retroviral replication

> > “Could it be that the chelators work as antiretrovirals by removing

mercury? “ , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached btw some of

these studies are in vitro, no mercury in sight, so the answer is NO they do not

work as antivirals through chelating mercury, at least not exclusively (meaning

to say that who knows maybe heavy metals are also directly utilised by some

viruses) And whatever is observed to happen in HIV only is only because HIV is

studied lot more than others. There are no studies that show other viral

proteins would

> > not be unfolding in the same way when chelators added. Having said that

there are studies on herpes/CMV and others that indicate same things going on.

  The influence of divalent cations on the stability of human rotavirus. The

influence of divalent cations on the stability of human rotavirus was

investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA,

> > Beards GM, Thouless ME, Flewett TH Biochemical studies of primate

retroviruses. In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and

> > type D primate retroviruses in persistently infected human cells. From

experiments demonstrating a disintegrating activity of chelating agents (EDTA,

EGTA) and certain psychoactive drugs (including trifluoperazine) on various

primate and nonprimate retroviruses it is concluded that divalent cations,

probably Ca2+ ions, and possibly also cation-binding proteins are associated

with retroviral membranes and that complexing of these components results in

loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch

Geschwulstforsch. 1983;53(3):267- 78.

> >

>

[Guest guest]

I am begining to understand now,why all these vaccine damaged children recover.There is a world of difference between getting a vaccine or two with thimerosol,after a child has had a healthy time in the womb,and a child who had spent nine months taking in the mercury from a mother's mouthful of fillings,and then being born with a methylation defect.

Subject: Re: ALA - direct inhibitor of retroviral replicationTo: mb12 valtrex Date: Tuesday, November 24, 2009, 6:19 AM

Christal,I also have an MMR kid. It makes me feel very hopeful that your son is recovered. My son is really close, but not quite there. He avoids conversing with people and when he is comfortable enough to converse with someone, he sounds kind of like the Sheldon character in "the big bang theory" show. He can get angry when people try to make him participate in a conversation that he is not interested in because it forces him out of the obsessions he is working in his head. Also he gets upset when his short answers come off as rude when he didn't intend them to be that way. He is like a foreigner that speaks the language but doesn't get the nuances, abstractions, or customs that go with itI'd love to hear anything you have to say about how you "kicked the MMR." My son tested high for aluminum many years back, but never came up high for mercury, even after DMSA and ALA. Can anyone tell us how a high mercury burden would present

differently in symptoms than say other metals, yeast, strep, clostridia, viruses? We keep talking about how our kids are so different yet so alike. What were the symptoms of "Mad Hatter" again?Jen > > > > From: Christel King <christelking1@ >> > Subject: Re: Re: ALA - direct inhibitor of retroviral replication> > To: mb12 valtrex@ yahoogroups. com> > Date: Friday, November 20, 2009, 8:59 AM> > > > > > > > > > > > > > > > Â > > > > > > > > > > > > > > > > > > > > > > > >

> > then I guess we don't have mercury in the brain > > because our son isn't autistic anymore....and we didn't chelate!  not > > ALL autistic kids have mercury issues! my son didn't become autistic > > because he had metals, he became autistic when he got an MMR....in HOURS after > > it!n we had tested 2 weeks prior developmental testing and he was 6 months > > advanced. after 7 hours after his MMR he became autistic, lost lang, no > > eye contact, started seizering, ect. we tested him 2 weeks after this > > because I had no idea what had happened to my son, he then had the develeopment > > of a 2-4 month old. we LITERIALLY lost 14-16 months development from the > > MMR.....NOT EVERYTHING IS METALS~! if it was he would have become autistic > > when the thermitor exploded at 6 months old in our dishwasher

and he > > didn't then!!!! it would also mean that my son would STILL be > > autistic since we haven't chelated with your therory and he is not!> >  > > > > Re: ALA > > - direct inhibitor of retroviral replication> > > > â€Å"Could it be that the chelators work as > >

antiretrovirals by removing mercury? â€Å"> > > > , that is not > > the case, that is not how chelators work here. They work by removing > > ions from cation-binding viral proteins, causing unfolding/loss of > > protein functionality. For example by opening zinc-finger proteins > > and similar. There are other mechanisms observed, such as inhibiting > > viral DNA synthesis through RNA reductase deactivation, see > > attached> > > > btw some of these studies are in vitro, no mercury > > in sight, so the answer is NO they do not work as antivirals through > > chelating mercury, at least not exclusively (meaning to say that who > > knows maybe heavy metals are also directly utilised by some > > viruses)> > > > And whatever is observed to happen in HIV only is > > only

because HIV is studied lot more than others. There are no > > studies that show other viral proteins would not be unfolding in the > > same way when chelators added. Having said that there are studies on > > herpes/CMV and others that indicate same things going on. > > > > > > > > Â > > The influence of divalent cations on the > > stability of human rotavirus.> > The influence of divalent > > cations on the stability of human rotavirus was investigated using > > the indirect immunofluorescence (FA) technique in LLC-MK2 cells to > > titrate infectivity. Rotavirus infectivity was stabilized by calcium > > and strontium ions in solution but not by magnesium ions. Rotavirus > > isolates were found to be much less stable at 37 degrees C than at + > > 4 degrees C or 20 degrees. A study of virus

morphology at intervals > > during the course of the experiment and treatment with the chelating > > agents EDTA and EGTA suggests that loss of infectivity coincides > > with the removal of the outer capsid layer and that calcium may be > > required to maintain virus integrity. Arch Virol. > > 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett > > TH> > > > > > Biochemical studies of primate > > retroviruses.> > In the present paper, recent biochemical > > studies of retroviruses carried out in our laboratory are > > summarized. Protein compositions, peptide maps of internal > > structural proteins, neighborhoods of major structural proteins, and > > serological properties of reverse transcriptases of type D virus > > isolates from human cells (including Graffi's isolate termed PMFV

> > and also isolates from HeLa- and HEp-2 cells) were compared with > > those of type D viruses from Old World (Mason-Pfizer virus of rhesus > > monkeys, langur virus) and New World (squirrel monkey retrovirus) > > monkeys. The results provide various new informations on, and > > further demonstrate the diversity of primate type D viruses. Other > > studies showed that tumor promoting agents (phorbol ester TPA, > > indole alkaloid teleocidin) are able to considerably increase, in a > > transient manner, the production of type C and type D primate > > retroviruses in persistently infected human cells. From experiments > > demonstrating a disintegrating activity of chelating agents (EDTA, > > EGTA) and certain psychoactive drugs (including trifluoperazine) on > > various primate and nonprimate retroviruses it is concluded that >

> divalent cations, probably Ca2+ ions, and possibly also > > cation-binding proteins are associated with retroviral membranes and > > that complexing of these components results in loss of viral > > infectivity. Wunderlich Very, Uckert W, Sydow G Arch > > Geschwulstforsch. > > 1983;53(3):267- 78.> >>

[Guest guest]

I am begining to understand now,why all these vaccine damaged children recover.There is a world of difference between getting a vaccine or two with thimerosol,after a child has had a healthy time in the womb,and a child who had spent nine months taking in the mercury from a mother's mouthful of fillings,and then being born with a methylation defect.

Subject: Re: ALA - direct inhibitor of retroviral replicationTo: mb12 valtrex Date: Tuesday, November 24, 2009, 6:19 AM

Christal,I also have an MMR kid. It makes me feel very hopeful that your son is recovered. My son is really close, but not quite there. He avoids conversing with people and when he is comfortable enough to converse with someone, he sounds kind of like the Sheldon character in "the big bang theory" show. He can get angry when people try to make him participate in a conversation that he is not interested in because it forces him out of the obsessions he is working in his head. Also he gets upset when his short answers come off as rude when he didn't intend them to be that way. He is like a foreigner that speaks the language but doesn't get the nuances, abstractions, or customs that go with itI'd love to hear anything you have to say about how you "kicked the MMR." My son tested high for aluminum many years back, but never came up high for mercury, even after DMSA and ALA. Can anyone tell us how a high mercury burden would present

differently in symptoms than say other metals, yeast, strep, clostridia, viruses? We keep talking about how our kids are so different yet so alike. What were the symptoms of "Mad Hatter" again?Jen > > > > From: Christel King <christelking1@ >> > Subject: Re: Re: ALA - direct inhibitor of retroviral replication> > To: mb12 valtrex@ yahoogroups. com> > Date: Friday, November 20, 2009, 8:59 AM> > > > > > > > > > > > > > > > Â > > > > > > > > > > > > > > > > > > > > > > > >

> > then I guess we don't have mercury in the brain > > because our son isn't autistic anymore....and we didn't chelate!  not > > ALL autistic kids have mercury issues! my son didn't become autistic > > because he had metals, he became autistic when he got an MMR....in HOURS after > > it!n we had tested 2 weeks prior developmental testing and he was 6 months > > advanced. after 7 hours after his MMR he became autistic, lost lang, no > > eye contact, started seizering, ect. we tested him 2 weeks after this > > because I had no idea what had happened to my son, he then had the develeopment > > of a 2-4 month old. we LITERIALLY lost 14-16 months development from the > > MMR.....NOT EVERYTHING IS METALS~! if it was he would have become autistic > > when the thermitor exploded at 6 months old in our dishwasher

and he > > didn't then!!!! it would also mean that my son would STILL be > > autistic since we haven't chelated with your therory and he is not!> >  > > > > Re: ALA > > - direct inhibitor of retroviral replication> > > > â€Å"Could it be that the chelators work as > >

antiretrovirals by removing mercury? â€Å"> > > > , that is not > > the case, that is not how chelators work here. They work by removing > > ions from cation-binding viral proteins, causing unfolding/loss of > > protein functionality. For example by opening zinc-finger proteins > > and similar. There are other mechanisms observed, such as inhibiting > > viral DNA synthesis through RNA reductase deactivation, see > > attached> > > > btw some of these studies are in vitro, no mercury > > in sight, so the answer is NO they do not work as antivirals through > > chelating mercury, at least not exclusively (meaning to say that who > > knows maybe heavy metals are also directly utilised by some > > viruses)> > > > And whatever is observed to happen in HIV only is > > only

because HIV is studied lot more than others. There are no > > studies that show other viral proteins would not be unfolding in the > > same way when chelators added. Having said that there are studies on > > herpes/CMV and others that indicate same things going on. > > > > > > > > Â > > The influence of divalent cations on the > > stability of human rotavirus.> > The influence of divalent > > cations on the stability of human rotavirus was investigated using > > the indirect immunofluorescence (FA) technique in LLC-MK2 cells to > > titrate infectivity. Rotavirus infectivity was stabilized by calcium > > and strontium ions in solution but not by magnesium ions. Rotavirus > > isolates were found to be much less stable at 37 degrees C than at + > > 4 degrees C or 20 degrees. A study of virus

morphology at intervals > > during the course of the experiment and treatment with the chelating > > agents EDTA and EGTA suggests that loss of infectivity coincides > > with the removal of the outer capsid layer and that calcium may be > > required to maintain virus integrity. Arch Virol. > > 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett > > TH> > > > > > Biochemical studies of primate > > retroviruses.> > In the present paper, recent biochemical > > studies of retroviruses carried out in our laboratory are > > summarized. Protein compositions, peptide maps of internal > > structural proteins, neighborhoods of major structural proteins, and > > serological properties of reverse transcriptases of type D virus > > isolates from human cells (including Graffi's isolate termed PMFV

> > and also isolates from HeLa- and HEp-2 cells) were compared with > > those of type D viruses from Old World (Mason-Pfizer virus of rhesus > > monkeys, langur virus) and New World (squirrel monkey retrovirus) > > monkeys. The results provide various new informations on, and > > further demonstrate the diversity of primate type D viruses. Other > > studies showed that tumor promoting agents (phorbol ester TPA, > > indole alkaloid teleocidin) are able to considerably increase, in a > > transient manner, the production of type C and type D primate > > retroviruses in persistently infected human cells. From experiments > > demonstrating a disintegrating activity of chelating agents (EDTA, > > EGTA) and certain psychoactive drugs (including trifluoperazine) on > > various primate and nonprimate retroviruses it is concluded that >

> divalent cations, probably Ca2+ ions, and possibly also > > cation-binding proteins are associated with retroviral membranes and > > that complexing of these components results in loss of viral > > infectivity. Wunderlich Very, Uckert W, Sydow G Arch > > Geschwulstforsch. > > 1983;53(3):267- 78.> >>

[Guest guest]

Raun Kaufman, Georgia Stehlie, and Maurice's daughter recovered in an

era when chelation was not done. If metals contributed to their autism, they

were not removed and did not impede their total recoveries from autism. I had

lunch with

Raun Kaufman in 1998. He was no more autistic than Jon from the Daily

Show.

>

>

> From: Christel King <christelking1@ myfairpoint. net>

> Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> To: mb12 valtrex@ yahoogroups. com

> Date: Friday, November 20, 2009, 8:59 AM

>

>

>  

>

>

> then I guess we don't have mercury in the brain because our son isn't autistic

anymore....and we didn't chelate!   not ALL autistic kids have mercury

issues!  my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n  we had tested 2 weeks prior

developmental testing and he was 6 months advanced.  after 7 hours after his

MMR he became autistic, lost lang, no eye contact, started seizering, ect.  we

tested him 2 weeks after this because I had no idea what had happened to my son,

he then had the develeopment of a 2-4 month old.  we LITERIALLY lost 14-16

months development from the MMR.....NOT EVERYTHING IS METALS~!  if it was he

would have become autistic when the thermitor exploded at 6 months old in our

dishwasher and he didn't then!!!!  it would also mean that my son would STILL

be autistic since we haven't chelated with your therory and he is not!

>  

>

> Re: ALA - direct inhibitor of retroviral replication

>

> “Could it be that the chelators work as antiretrovirals by removing mercury?

“

>

> , that is not the case, that is not how chelators work here. They work

by removing ions from cation-binding viral proteins, causing unfolding/loss of

protein functionality. For example by opening zinc-finger proteins and similar.

There are other mechanisms observed, such as inhibiting viral DNA synthesis

through RNA reductase deactivation, see attached

>

> btw some of these studies are in vitro, no mercury in sight, so the answer is

NO they do not work as antivirals through chelating mercury, at least not

exclusively (meaning to say that who knows maybe heavy metals are also directly

utilised by some viruses)

>

> And whatever is observed to happen in HIV only is only because HIV is studied

lot more than others. There are no studies that show other viral proteins would

not be unfolding in the same way when chelators added. Having said that there

are studies on herpes/CMV and others that indicate same things going on.

>

>

>  

> The influence of divalent cations on the stability of human rotavirus.

> The influence of divalent cations on the stability of human rotavirus was

investigated using the indirect immunofluorescence (FA) technique in LLC-MK2

cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium

and strontium ions in solution but not by magnesium ions. Rotavirus isolates

were found to be much less stable at 37 degrees C than at + 4 degrees C or 20

degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

>

>

> Biochemical studies of primate retroviruses.

> In the present paper, recent biochemical studies of retroviruses carried out

in our laboratory are summarized. Protein compositions, peptide maps of internal

structural proteins, neighborhoods of major structural proteins, and serological

properties of reverse transcriptases of type D virus isolates from human cells

(including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2

cells) were compared with those of type D viruses from Old World (Mason-Pfizer

virus of rhesus monkeys, langur virus) and New World (squirrel monkey

retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a

> disintegrating activity of chelating agents (EDTA, EGTA) and certain

psychoactive drugs (including trifluoperazine) on various primate and nonprimate

retroviruses it is concluded that divalent cations, probably Ca2+ ions, and

possibly also cation-binding proteins are associated with retroviral membranes

and that complexing of these components results in loss of viral infectivity.

Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

>

[Guest guest]

God this is getting old! There is not much out there that could back up your statement and plenty of fully recovered children and adults out there who never went near a chelator. And that includes those who never did any biomed.

To go on saying in spite of all that that there is no autism without mercury and no recovery without chelation sounds a bit like religious fanaticism. If I was a new parent reasearching treatments coming across messages like this one would make me question if I was in the right place .

Lets agree to disagree but I would plead to everyone to stop making such blanket statement. They do not help anyone.

--------

There are kids who are in a 'maintained recovery'. They need supplements and

diet on an ongoing basis. If you chelate long enough, you shouldn't need the

supps or diet anymore.

[Guest guest]

there wasn't a ONE thing that recovered him but a combo of addressing each of his systmptoms to get to the cause of each of them. for the MMR we did vit A protical to combate that, for the leaky gut, we addressed IGG , IGE issues, yeast, bacteria, parasites, probiotics, enzymes, glutamine, for detoxing him we used glutathione, b12, selenium, zinc, for hyperness b6 and mag, folapro for lang, LDN, DMG, b12 for pretend play EFAS, LDN, ABA, play scheduals, for low tone, carnintine, for seizers removing gluten was key, and taurine, for immune system LDN, OLE, GSE, fluconizole, zinc, vit A, vit D, amino acid combo, diet. ect

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > > > > Biochemical studies of primate retroviruses.> > > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > > >> > >> >>

[Guest guest]

Good evening, Christel,

Do you happen to have link regarding the Vitamin A protocol for MMR?

We are currently taking homeopathic remedies for his MMR. However, I was also

going to try some Amantadine.

I am very intrigued about the Vitamin A protocol.

My son was developing normally until his MMR vaccination. He did not regress

immediately meaning not in a few hours; however, after two months, we knew

something was wrong.

Now, my son has no expressive language, heavy metal issues (lead, mercury, and

arsenic), yeast issues, maybe parasites (EDS and Zyto testing show), maybe

bacteria (EDS and Zyto testing show, but OATS test do not), and MMR viral

issues.

I agree with you that you treat the symptoms, and, if you happen to get lucky

and treat one symptom that corrects other issues, that is great.

My son is getting better, but I still need to learn more.

Thank you in advance for any information.

Jerry

> > > > >

> > > > >

> > > > > From: Christel King <christelking1@>

> > > > > Subject: Re: Re: ALA - direct inhibitor of retroviral

replication

> > > > > To: mb12 valtrex

> > > > > Date: Friday, November 20, 2009, 8:59 AM

> > > > >

> > > > >

> > > > >

> > > > >

> > > > > then I guess we don't have mercury in the brain because our son

isn't autistic anymore....and we didn't chelate! not ALL autistic kids have

mercury issues! my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > > >

> > > > > Re: ALA - direct inhibitor of retroviral

replication

> > > > >

> > > > >

> > > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > > >

> > > > > , that is not the case, that is not how chelators work here.

They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > > >

> > > > > btw some of these studies are in vitro, no mercury in sight, so the

answer is NO they do not work as antivirals through chelating mercury, at least

not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > > >

> > > > > And whatever is observed to happen in HIV only is only because HIV

is studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > > >

> > > > >

> > > > >

> > > > > The influence of divalent cations on the stability of human

rotavirus.

> > > > > The influence of divalent cations on the stability of human

rotavirus was investigated using the indirect immunofluorescence (FA) technique

in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by

calcium and strontium ions in solution but not by magnesium ions. Rotavirus

isolates were found to be much less stable at 37 degrees C than at + 4 degrees C

or 20 degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > > >

> > > > >

> > > > > Biochemical studies of primate retroviruses.

> > > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > > >

> > > >

> > >

> >

>

[Guest guest]



my son has, he lost his label in 2007 of oct but still has mercury and lead issues. my daughter NT with no issues also has mercury and lead issues

Re: ALA - direct inhibitor of retroviral replication

“Could it be that the chelators work as antiretrovirals by removing mercury? “, that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attachedbtw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. The influence of divalent cations on the stability of human rotavirus.The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett THBiochemical studies of primate retroviruses.In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

[Guest guest]



not a matter of not wanting to, it's a matter of doing it safly and having over seaing by a dr and finances

Re: ALA - direct inhibitor of retroviral replication > “Could it be that the chelators work as antiretrovirals by removing mercury? “ , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses) And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would> not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. The influence of divalent cations on the stability of human rotavirus. The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA,> Beards GM, Thouless ME, Flewett TH Biochemical studies of primate retroviruses. In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and> type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78. >

[Guest guest]



some kids may never be able to go off diet. we have celiac in the family and lactose intollerence.

Re: ALA - direct inhibitor of retroviral replication> > “Could it be that the chelators work as antiretrovirals by removing mercury? “> > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > >  > The influence of divalent cations on the stability of human rotavirus.> The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > Biochemical studies of primate retroviruses.> In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a> disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.>

[Guest guest]

dana's views page would have it on it, its designed by Dr McCandless...we did the low and slow version of it of 5 drops a day

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > > > > > > > Biochemical studies of primate retroviruses.> > > > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > > > >> > > >> > >> >>

[Guest guest]

Good morning, Christel,

Thank you. How long did you do it? Did you do it until you had a Vitamin A

reaction or until test results showed the MMR Viral Titers to be low?

I want to try the Vitamin A protocol. I just don't know what I need to look for

to " check that box as complete " .

I will reference Dana's Page in a few minutes.

Happy Thanksgiving!!!!

Jerry

> > > > > >

> > > > > >

> > > > > > From: Christel King <christelking1@>

> > > > > > Subject: Re: Re: ALA - direct inhibitor of

retroviral replication

> > > > > > To: mb12 valtrex

> > > > > > Date: Friday, November 20, 2009, 8:59 AM

> > > > > >

> > > > > >

> > > > > >

> > > > > >

> > > > > > then I guess we don't have mercury in the brain because our son

isn't autistic anymore....and we didn't chelate! not ALL autistic kids have

mercury issues! my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > > > >

> > > > > > Re: ALA - direct inhibitor of retroviral

replication

> > > > > >

> > > > > >

> > > > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > > > >

> > > > > > , that is not the case, that is not how chelators work

here. They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > > > >

> > > > > > btw some of these studies are in vitro, no mercury in sight, so

the answer is NO they do not work as antivirals through chelating mercury, at

least not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > > > >

> > > > > > And whatever is observed to happen in HIV only is only because HIV

is studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > > > >

> > > > > >

> > > > > >

> > > > > > The influence of divalent cations on the stability of human

rotavirus.

> > > > > > The influence of divalent cations on the stability of human

rotavirus was investigated using the indirect immunofluorescence (FA) technique

in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by

calcium and strontium ions in solution but not by magnesium ions. Rotavirus

isolates were found to be much less stable at 37 degrees C than at + 4 degrees C

or 20 degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > > > >

> > > > > >

> > > > > > Biochemical studies of primate retroviruses.

> > > > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > > > >

> > > > >

> > > >

> > >

> >

>

[Guest guest]

for us after 2 months we broke out in measels from head to toe confirmed by 4 drs' so we pushed it through a week of break out and then dropped it and it took 2 weeks for it disappear and then lang took off

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > > > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > > > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > > > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > > > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > > > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > > > > > > > > > > Biochemical studies of primate retroviruses.> > > > > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > > > > >> > > > >> > > >> > >> >>

[Guest guest]

Did you see any seizures w this? When I tried it, the first dose produced a

seizure day! Thanks,

> > > > > > >

> > > > > > >

> > > > > > > From: Christel King <christelking1@>

> > > > > > > Subject: Re: Re: ALA - direct inhibitor of

retroviral replication

> > > > > > > To: mb12 valtrex

> > > > > > > Date: Friday, November 20, 2009, 8:59 AM

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > then I guess we don't have mercury in the brain because our son

isn't autistic anymore....and we didn't chelate! not ALL autistic kids have

mercury issues! my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > > > > >

> > > > > > > Re: ALA - direct inhibitor of retroviral

replication

> > > > > > >

> > > > > > >

> > > > > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > > > > >

> > > > > > > , that is not the case, that is not how chelators work

here. They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > > > > >

> > > > > > > btw some of these studies are in vitro, no mercury in sight, so

the answer is NO they do not work as antivirals through chelating mercury, at

least not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > > > > >

> > > > > > > And whatever is observed to happen in HIV only is only because

HIV is studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > The influence of divalent cations on the stability of human

rotavirus.

> > > > > > > The influence of divalent cations on the stability of human

rotavirus was investigated using the indirect immunofluorescence (FA) technique

in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by

calcium and strontium ions in solution but not by magnesium ions. Rotavirus

isolates were found to be much less stable at 37 degrees C than at + 4 degrees C

or 20 degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > > > > >

> > > > > > >

> > > > > > > Biochemical studies of primate retroviruses.

> > > > > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

[Guest guest]

Good evening, Christel,

Here are a few links that I found.

Just to be certain.

1. Day 1 and Day 2: High Dose of around 400,000 IU or (5,000 to 10,000 IU per

pound of child).

Do you agree with the weight adjustment ratio of IU to pound?

I read the comment in the link below from Cutler.

http://onibasu.com/archives/am/112213.html

2. Afterwards, the maintenance dose would be no more than 25,000 IU or (500 -

1,000 IU per pound of child).

Also, can you tell me what products you used?

We are looking at Klaire Labs Mycellized Vitamin A. Any recommendations for Cod

Liver Oils to use?

Thank you, again.

Jerry

> > > > > > >

> > > > > > >

> > > > > > > From: Christel King <christelking1@>

> > > > > > > Subject: Re: Re: ALA - direct inhibitor of

retroviral replication

> > > > > > > To: mb12 valtrex

> > > > > > > Date: Friday, November 20, 2009, 8:59 AM

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > then I guess we don't have mercury in the brain because our son

isn't autistic anymore....and we didn't chelate! not ALL autistic kids have

mercury issues! my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > > > > >

> > > > > > > Re: ALA - direct inhibitor of retroviral

replication

> > > > > > >

> > > > > > >

> > > > > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > > > > >

> > > > > > > , that is not the case, that is not how chelators work

here. They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > > > > >

> > > > > > > btw some of these studies are in vitro, no mercury in sight, so

the answer is NO they do not work as antivirals through chelating mercury, at

least not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > > > > >

> > > > > > > And whatever is observed to happen in HIV only is only because

HIV is studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > The influence of divalent cations on the stability of human

rotavirus.

> > > > > > > The influence of divalent cations on the stability of human

rotavirus was investigated using the indirect immunofluorescence (FA) technique

in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by

calcium and strontium ions in solution but not by magnesium ions. Rotavirus

isolates were found to be much less stable at 37 degrees C than at + 4 degrees C

or 20 degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > > > > >

> > > > > > >

> > > > > > > Biochemical studies of primate retroviruses.

> > > > > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

[Guest guest]

we used the klaire labs as well, we did 5 drops a day instead of the high dose due to the toxic if the body can't pass it fast enough, and levi at ALOT of carrots so I was to nervous to over work it, AND he took CLO from kirkmans

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > > > > > > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > > > > > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > > > > > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > > > > > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > > > > > > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > > > > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > > > > > > > > > > > > > Biochemical studies of primate retroviruses.> > > > > > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > > > > > >> > > > > >> > > > >> > > >> > >> >>

[Guest guest]

what vit A did you use? how much did you give?

we only did 5 drops a day of klaire labs drops

Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > > > > > > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > > > > > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > > > > > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > > > > > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > > > > > > > > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > > > > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > > > > > > > > > > > > > > > Biochemical studies of primate retroviruses.> > > > > > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > > > > > >> > > > > >> > > > >> > > >> > >> >>

[Guest guest]

I used Andy Cutler's suggested dosage, my son is on 10,000 IU Vit A (Solgar

brand very small gelcaps, no allergens) and 400 IU Vit D (NOW brand liquid. He

has gained speech from this combination and Vinpocetine (5mg, 3X daily)

Liz

> > > > > > > >

> > > > > > > >

> > > > > > > > From: Christel King <christelking1@>

> > > > > > > > Subject: Re: Re: ALA - direct inhibitor of

retroviral replication

> > > > > > > > To: mb12 valtrex

> > > > > > > > Date: Friday, November 20, 2009, 8:59 AM

> > > > > > > >

> > > > > > > >

> > > > > > > >

> > > > > > > >

> > > > > > > > then I guess we don't have mercury in the brain because our

son isn't autistic anymore....and we didn't chelate! not ALL autistic kids have

mercury issues! my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > > > > > >

> > > > > > > > Re: ALA - direct inhibitor of

retroviral replication

> > > > > > > >

> > > > > > > >

> > > > > > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals

by removing mercury? ââ,¬Å "

> > > > > > > >

> > > > > > > > , that is not the case, that is not how chelators work

here. They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > > > > > >

> > > > > > > > btw some of these studies are in vitro, no mercury in sight,

so the answer is NO they do not work as antivirals through chelating mercury, at

least not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > > > > > >

> > > > > > > > And whatever is observed to happen in HIV only is only because

HIV is studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > > > > > >

> > > > > > > >

> > > > > > > >

> > > > > > > > The influence of divalent cations on the stability of human

rotavirus.

> > > > > > > > The influence of divalent cations on the stability of human

rotavirus was investigated using the indirect immunofluorescence (FA) technique

in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by

calcium and strontium ions in solution but not by magnesium ions. Rotavirus

isolates were found to be much less stable at 37 degrees C than at + 4 degrees C

or 20 degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > > > > > >

> > > > > > > >

> > > > > > > > Biochemical studies of primate retroviruses.

> > > > > > > > In the present paper, recent biochemical studies of

retroviruses carried out in our laboratory are summarized. Protein compositions,

peptide maps of internal structural proteins, neighborhoods of major structural

proteins, and serological properties of reverse transcriptases of type D virus

isolates from human cells (including Graffi's isolate termed PMFV and also

isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses

from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New

World (squirrel monkey retrovirus) monkeys. The results provide various new

informations on, and further demonstrate the diversity of primate type D

viruses. Other studies showed that tumor promoting agents (phorbol ester TPA,

indole alkaloid teleocidin) are able to considerably increase, in a transient

manner, the production of type C and type D primate retroviruses in persistently

infected human cells. From experiments demonstrating a disintegrating activity

of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > > > > > >

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

[Guest guest]

Hi Liz,

What brand did you use for the high dose of 400,000 IU- was it Solgar ? If it is then it will save me having to order from Klaire Labs ( I am in Europe and Solgar is so much easier to obtain). Also in terms of the maintanence dose, how long do you give it for ? Is it ongoing along iwth his other supplements ?Lookign forward to your answer please.

Take careJoanna

To: mb12 valtrex Sent: Fri, November 27, 2009 8:20:03 AMSubject: Re: ALA - direct inhibitor of retroviral replication

I used Andy Cutler's suggested dosage, my son is on 10,000 IU Vit A (Solgar brand very small gelcaps, no allergens) and 400 IU Vit D (NOW brand liquid. He has gained speech from this combination and Vinpocetine (5mg, 3X daily)Liz> > > > > > > > > > > > > > > > > > > > > > > > From: Christel King

<christelking1@ >> > > > > > > > Subject: Re: Re: ALA - direct inhibitor of retroviral replication> > > > > > > > To: mb12 valtrex@ yahoogroups. com> > > > > > > > Date: Friday, November 20, 2009, 8:59 AM> > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > > then I guess we don't have mercury in the brain because our son isn't autistic anymore....and we didn't chelate! not ALL autistic kids have mercury issues! my son didn't become autistic because he had metals, he became autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior developmental

testing and he was 6 months advanced. after 7 hours after his MMR he became autistic, lost lang, no eye contact, started seizering, ect. we tested him 2 weeks after this because I had no idea what had happened to my son, he then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have become autistic when the thermitor exploded at 6 months old in our dishwasher and he didn't then!!!! it would also mean that my son would STILL be autistic since we haven't chelated with your therory and he is not!> > > > > > > > > > > > > > > > Re: ALA - direct inhibitor of retroviral replication> > > > > > > > > > > > > > > > > > > > > > > > ââ,¬Å"Could it be that the chelators work as antiretrovirals by removing mercury? ââ,¬Å"> > > > > > > > > > > > > > > > , that is not the case, that is not how chelators work here. They work by removing ions from cation-binding viral proteins, causing unfolding/loss of protein functionality. For example by opening zinc-finger proteins and similar. There are other

mechanisms observed, such as inhibiting viral DNA synthesis through RNA reductase deactivation, see attached> > > > > > > > > > > > > > > > btw some of these studies are in vitro, no mercury in sight, so the answer is NO they do not work as antivirals through chelating mercury, at least not exclusively (meaning to say that who knows maybe heavy metals are also directly utilised by some viruses)> > > > > > > > > > > > > > > > And whatever is observed to happen in HIV only is only because HIV is studied lot more than others. There are no studies that show other viral proteins would not be unfolding in the same way when chelators added. Having said that there are studies on herpes/CMV and others that indicate same things going on. > > > > > > > > > > > > > > > > > > > > >

> > > > > > > > > > > The influence of divalent cations on the stability of human rotavirus.> > > > > > > > The influence of divalent cations on the stability of human rotavirus was investigated using the indirect immunofluorescence (FA) technique in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by calcium and strontium ions in solution but not by magnesium ions. Rotavirus isolates were found to be much less stable at 37 degrees C than at + 4 degrees C or 20 degrees. A study of virus morphology at intervals during the course of the experiment and treatment with the chelating agents EDTA and EGTA suggests that loss of infectivity coincides with the removal of the outer capsid layer and that calcium may be required to maintain virus integrity. Arch Virol. 1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH> > > > > > > >

> > > > > > > > > > > > > > > > Biochemical studies of primate retroviruses.> > > > > > > > In the present paper, recent biochemical studies of retroviruses carried out in our laboratory are summarized. Protein compositions, peptide maps of internal structural proteins, neighborhoods of major structural proteins, and serological properties of reverse transcriptases of type D virus isolates from human cells (including Graffi's isolate termed PMFV and also isolates from HeLa- and HEp-2 cells) were compared with those of type D viruses from Old World (Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel monkey retrovirus) monkeys. The results provide various new informations on, and further demonstrate the diversity of primate type D viruses. Other studies showed that tumor promoting agents (phorbol ester TPA, indole alkaloid teleocidin) are able to

considerably increase, in a transient manner, the production of type C and type D primate retroviruses in persistently infected human cells. From experiments demonstrating a disintegrating activity of chelating agents (EDTA, EGTA) and certain psychoactive drugs (including trifluoperazine) on various primate and nonprimate retroviruses it is concluded that divalent cations, probably Ca2+ ions, and possibly also cation-binding proteins are associated with retroviral membranes and that complexing of these components results in loss of viral infectivity. Wunderlich Very, Uckert W, Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.> > > > > > > >> > > > > > >> > > > > >> > > > >> > > >> > >> >>

[Guest guest]

OH MY GOD! Isn't that proof your child had an active measles infection that was

repressed somehow? My son's titers are sky high and he only received MMR once.

I got a waiver for the booster. I'm starting to think this could help, but I

am a little afraid to give such a high dose of vitamin A.

> > > > > > >

> > > > > > >

> > > > > > > From: Christel King <christelking1@>

> > > > > > > Subject: Re: Re: ALA - direct inhibitor of

retroviral replication

> > > > > > > To: mb12 valtrex

> > > > > > > Date: Friday, November 20, 2009, 8:59 AM

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > then I guess we don't have mercury in the brain because our son

isn't autistic anymore....and we didn't chelate! not ALL autistic kids have

mercury issues! my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > > > > >

> > > > > > > Re: ALA - direct inhibitor of retroviral

replication

> > > > > > >

> > > > > > >

> > > > > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > > > > >

> > > > > > > , that is not the case, that is not how chelators work

here. They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > > > > >

> > > > > > > btw some of these studies are in vitro, no mercury in sight, so

the answer is NO they do not work as antivirals through chelating mercury, at

least not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > > > > >

> > > > > > > And whatever is observed to happen in HIV only is only because

HIV is studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > The influence of divalent cations on the stability of human

rotavirus.

> > > > > > > The influence of divalent cations on the stability of human

rotavirus was investigated using the indirect immunofluorescence (FA) technique

in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by

calcium and strontium ions in solution but not by magnesium ions. Rotavirus

isolates were found to be much less stable at 37 degrees C than at + 4 degrees C

or 20 degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > > > > >

> > > > > > >

> > > > > > > Biochemical studies of primate retroviruses.

> > > > > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

[Guest guest]

OH MY GOD! Isn't that proof your child had an active measles infection that was

repressed somehow? My son's titers are sky high and he only received MMR once.

I got a waiver for the booster. I'm starting to think this could help, but I

am a little afraid to give such a high dose of vitamin A.

> > > > > > >

> > > > > > >

> > > > > > > From: Christel King <christelking1@>

> > > > > > > Subject: Re: Re: ALA - direct inhibitor of

retroviral replication

> > > > > > > To: mb12 valtrex

> > > > > > > Date: Friday, November 20, 2009, 8:59 AM

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > then I guess we don't have mercury in the brain because our son

isn't autistic anymore....and we didn't chelate! not ALL autistic kids have

mercury issues! my son didn't become autistic because he had metals, he became

autistic when he got an MMR....in HOURS after it!n we had tested 2 weeks prior

developmental testing and he was 6 months advanced. after 7 hours after his MMR

he became autistic, lost lang, no eye contact, started seizering, ect. we tested

him 2 weeks after this because I had no idea what had happened to my son, he

then had the develeopment of a 2-4 month old. we LITERIALLY lost 14-16 months

development from the MMR.....NOT EVERYTHING IS METALS~! if it was he would have

become autistic when the thermitor exploded at 6 months old in our dishwasher

and he didn't then!!!! it would also mean that my son would STILL be autistic

since we haven't chelated with your therory and he is not!

> > > > > > >

> > > > > > > Re: ALA - direct inhibitor of retroviral

replication

> > > > > > >

> > > > > > >

> > > > > > > ââ,¬Å " Could it be that the chelators work as antiretrovirals by

removing mercury? ââ,¬Å "

> > > > > > >

> > > > > > > , that is not the case, that is not how chelators work

here. They work by removing ions from cation-binding viral proteins, causing

unfolding/loss of protein functionality. For example by opening zinc-finger

proteins and similar. There are other mechanisms observed, such as inhibiting

viral DNA synthesis through RNA reductase deactivation, see attached

> > > > > > >

> > > > > > > btw some of these studies are in vitro, no mercury in sight, so

the answer is NO they do not work as antivirals through chelating mercury, at

least not exclusively (meaning to say that who knows maybe heavy metals are also

directly utilised by some viruses)

> > > > > > >

> > > > > > > And whatever is observed to happen in HIV only is only because

HIV is studied lot more than others. There are no studies that show other viral

proteins would not be unfolding in the same way when chelators added. Having

said that there are studies on herpes/CMV and others that indicate same things

going on.

> > > > > > >

> > > > > > >

> > > > > > >

> > > > > > > The influence of divalent cations on the stability of human

rotavirus.

> > > > > > > The influence of divalent cations on the stability of human

rotavirus was investigated using the indirect immunofluorescence (FA) technique

in LLC-MK2 cells to titrate infectivity. Rotavirus infectivity was stabilized by

calcium and strontium ions in solution but not by magnesium ions. Rotavirus

isolates were found to be much less stable at 37 degrees C than at + 4 degrees C

or 20 degrees. A study of virus morphology at intervals during the course of the

experiment and treatment with the chelating agents EDTA and EGTA suggests that

loss of infectivity coincides with the removal of the outer capsid layer and

that calcium may be required to maintain virus integrity. Arch Virol.

1981;67(1):1- 9. Shirley JA, Beards GM, Thouless ME, Flewett TH

> > > > > > >

> > > > > > >

> > > > > > > Biochemical studies of primate retroviruses.

> > > > > > > In the present paper, recent biochemical studies of retroviruses

carried out in our laboratory are summarized. Protein compositions, peptide maps

of internal structural proteins, neighborhoods of major structural proteins, and

serological properties of reverse transcriptases of type D virus isolates from

human cells (including Graffi's isolate termed PMFV and also isolates from HeLa-

and HEp-2 cells) were compared with those of type D viruses from Old World

(Mason-Pfizer virus of rhesus monkeys, langur virus) and New World (squirrel

monkey retrovirus) monkeys. The results provide various new informations on, and

further demonstrate the diversity of primate type D viruses. Other studies

showed that tumor promoting agents (phorbol ester TPA, indole alkaloid

teleocidin) are able to considerably increase, in a transient manner, the

production of type C and type D primate retroviruses in persistently infected

human cells. From experiments demonstrating a disintegrating activity of

chelating agents (EDTA, EGTA) and certain psychoactive drugs (including

trifluoperazine) on various primate and nonprimate retroviruses it is concluded

that divalent cations, probably Ca2+ ions, and possibly also cation-binding

proteins are associated with retroviral membranes and that complexing of these

components results in loss of viral infectivity. Wunderlich Very, Uckert W,

Sydow G Arch Geschwulstforsch. 1983;53(3):267- 78.

> > > > > > >

> > > > > >

> > > > >

> > > >

> > >

> >

>

[Guest guest]

Christel,

Did you do a higher vitamin A dosage for 2 months to get the measles out?

That is great you got such good results on language.

> >

> > for us after 2 months we broke out in measels from head to toe confirmed by

4 drs' so we pushed it through a week of break out and then dropped it and it

took 2 weeks for it disappear and then lang took off

> >