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1. BBC 13 March 2008 Vitamin D 'cuts risk of diabetes'

Exposure to sunlight allows the body to manufacture vitamin D

Giving young children vitamin D supplements may reduce their risk of

developing type 1 diabetes later in life, research suggests. Children who took

supplements were around 30% less likely to develop the condition than those who

did not. T1 results from the immune system destruction of pancreatic cells which

produce the hormone insulin.

The study appears in Archivesof Disease in Childhood. T1 is most common among

people of European descent, with around 2million Europeans and North Americans

affected. It is becoming increasingly common, and it is estimated that the

number of new cases

will rise by 40% between 2000 and 2010. The team pooled data from 5 studies

examining the effect of vitamin D supplementation. Not only did the use of

supplements appear to reduce the risk, the effect was dose dependent - the

higher and more regular the dose, the lower the likelihood of developing the

disease. Previous research has found that people newly diagnosed with T1have

lower concentrations of vitamin D than those without the condition [and ] that

T1 is more common in countries where exposure to sunlight - which enables the

body to manufacture vitamin D - is lower. For instance, a child in Finland was

400 times more likely to develop the disease than a child in Venezuela. Separate

research has linked low levels of vitamin D and sunlight to other autoimmune

disorders, including multiple sclerosis and rheumatoid arthritis. Further

evidence of vitamin D's role comes from the fact that pancreatic beta cells and

immune cells carry receptors or docking bays for the active forms of the

vitamin.

It is thought that vitamin D helps to keep the immune system healthy, and may

protect cells from damage caused by chemicals which control inflammation. Dr V

King, of Diabetes UK, said: " Much more research, in particular controlled trials

which compares the results when one group of people are given vitamin D

supplements and one group is not, are needed before we can confirm a concrete

association between vitamin D and T1. " Governnment [uK] experts recommend

vitamin D supplementation for at least the first two years of a child's life.

2.%% 13 Mar 2008 2, 17, 4629 s Hopkins Health Alerts:Diabetes - Stopping

Insulin Resistance Before It Leads to Diabetes In this Health Alert, s

Hopkins endocrinologists discuss intriguing research that affects all who have

T2DM. Researchers have identified a fat cell protein that may predict insulin

resistance and help doctors to prescribe treatments to prevent or slow the

development of T2DM.

In a healthy person the pancreas makes enough insulin to keep the supply and

use of glucose in balance. In DM, the glucose balancing system is disrupted,

either because too little insulin is produced or because the body's cells do not

respond to insulin normally -- a condition called insulin resistance. The result

is an unhealthy

rise in blood glucose levels. If DM is left untreated, the two principal

dangers are the immediate results of high blood glucose levels (which include

excessive urination, dehydration, intense thirst, and fatigue) and long-term

complications that can affect your eyes, nerves, kidneys, and large blood

vessels.

A screening test to identify people with insulin resistance could allow

doctors to prescribe treatments or lifestyle measures to stop or slow the

development of T2. Researchers have taken a step toward such a screening test,

according to a new study reported in The New England Journal of Medicine (Volume

354, page 2552 ). The team identified increased blood levels of a protein

produced and secreted by fat cells in people who become insulin resistant. The

protein's name: retinol-binding protein 4 (RBP4). Scientists already know that

people with insulin resistance have increased levels of RBP4. The new study

found that levels of RBP4 rose in parallel with the severity of insulin

resistance in people who were obese or had prediabetes or T2 and in healthy

people with a family history of the disease. Researchers are trying to determine

whether lowering RBP4 with medication makes cells more sensitive to insulin.

However, the study also found that about two thirds of the participants were

able to decrease their RBP4 levels and reduce insulin resistance with a

tried-and-true lifestyle measure: exercise.

3.%% Eye (2008) 22, Can we predict which patients are at risk of having an

ungradeable digital image for screening for diabetic retinopathy? Conclusion -

Inability to visualise the fovea is the commonest cause of an ungradeable image

from digital retinal photography. Age and posterior subcapsular cataract were

best predictors of ungradeable status of nonmydriatic fundus photographs.C 2008

The Royal College of Ophthalmologists

4.%% MW -Diabetes Linked to Risk of Endometrial Cancer

(Reuters Health) Mar 12 - T2DM is associated with endometrial cancer,

regardless of the presence of most other risk factors, according to new

findings. " A positive association has been observed in nearly all studies of

T2DM in relation to the incidence of endometrial cancer, " Researchers write.

" Given the adverse effect of obesity on the incidence of both DM and endometrial

cancer, investigators have adjusted for obesity in a number of these studies, "

they note. . .This team examined whether the risk of endometrial cancer among T2

women differs with respect to other endometrial cancer risk factors, [1303

cases;1779 controls.] An association was observed between T2 and endometrial

cancer (odds ratio = 1.7). The association was stronger among women with a DM

diagnosis

within 5 years (OR = 2.6) than those with a more distant diagnosis. " Among

women with no history of hypertension, those with DM had virtually no elevation

in risk compared with those without DM, whereas among women with hypertension,

the corresponding odds ratio was 2.1. " The risk of endometrial cancer associated

with DM appeared not to vary greatly with respect to menopausal hormone use,

cigarette smoking, age, race, education, or parity. " They conclude that T2DM is

associated with endometrial cancer irrespective of the presence of other risk

factors, " except possibly hypertension and extreme obesity. " Am J Epidemiol

2008;167

5.%% MW - Metabolic Syndrome Linked to Silent Ischemic Brain Lesions March 14,

2008 - Metabolic syndrome (MetS) is associated with the prevalence of silent

brain lesions independent of other risk factors for ischemic stroke, according

to the results of new a study. " Metabolic syndrome (MetS) is a recognized risk

factor for stroke, but it is unclear whether MetS is also related to subclinical

ischemic lesions, " write the researchers " We examined the association

of MetS with the prevalence of silent brain infarction, periventricular

hyperintensity, and subcortical white matter lesions in healthy adults. " [(MRI)

brain scans of 1151 healthy Japanese subjects] MetS was significantly associated

with silent brain infarction, periventricular hyperintensity (PVH), and

subcortical white matter lesions (SWML) after adjustment for age and other

clinical factors. Among the components of MetS, high blood pressure was

associated

with all 3 types of lesions, whereas dyslipidemia was associated with SWML and

elevated fasting blood glucose levels were associated with PVH. There were

positive trends between the number of MetS components and the prevalence of

silent lesions. The positive trend between MetS components and silent lesions

could be used as a diagnostic tool to predict and prevent future stroke. "

Stroke. Published online March 6, 2008.

6.%% MW - More Support for Statins in Diabetics January 11, 2008 - A new

meta-analysis, combining data on more than 18000 diabetic

patients across 14 trials, helps support the current wisdom that all diabetic

patients at " sufficiently high risk " for vascular events can benefit from

statins The study is the latest analysis from the

Cholesterol Treatment Trialists (CTT) Collaborators and indicates that statins

reduce deaths from vascular causes in diabetics, without

increasing the risk from non-vascular causes, to a degree similar to that seen

in nondiabetics. According to one of the CTT collaborators, the findings should

help quell debate spurred by the recent

4D and ASPEN trials, which found no clear benefit of statins in diabetics. In

the current meta-analysis, the effects of statins were not only similar in

people with and without DM, they were also similar in diabetics with and without

vascular disease, an important finding since many of the diabetics in the study

did not have vascular disease at baseline. " It also did not matter whether they

were young or old, men or women, obese or not obese, had mild kidney disease or

not, " " Most important of all, it didn't matter what their starting cholesterol

was, so even people with below-average LDL [low-density lipoprotein]

cholesterol: if they were diabetic and at increased risk, they benefited from

the statin. "

7.%% MW - Insulin Glargine Mix Improves Glucose Control in Diabetic Children

(Reuters Health) Mar 18 - Insulin glargine mixed with rapid-acting insulin

analogs is more effective than standard treatment with neutral protamine

Hagedorn and rapid-acting insulin analogs in children with newly diagnosed T1DM

researchers report. " we tested the hypothesis that insulin glargine mixed with

rapid-acting insulin analogs is superior to the standard therapy of neutral

protamine Hagedorn twice a day with rapid-acting insulin analogs. " . . in an

earlier study they established that both could be combined in the same syringe,

reducing daily injections from four to two. In the current study, the team

studied 42 children with new-onset T1. . the team found that A1C in the

combination group had fallen from 6.8% at baseline to 6.7%. " The fact that we

were able to mix glargine with rapid-acting insulin analogs in the same

syringe, " she concluded, " decreased the number of injections, which will further

help adherence to the regimen. " Pediatrics 2008;121

8.%% MW - Type 2 Dabetes in People of South Asian Origin: Potential Strategies

for Prevention 3/11/2008

Abstract - South Asians have a one in three lifetime risk for the development

of diabetes, developing the condition 10 years earlier than Europids. The high

social and economic burden of DM in South Asians is augmented by greater rates

of attrition from renal and cardiovascular disease. Conclusions - Prevention (or

at least delay) in the development of DM is feasible with rigorous lifestyle

intervention or drug therapy in people at high risk of DM. Trials that have

shown particular benefit include a significant lifestyle component - exercise

levels of 30 minutes per day, plus at least two-monthly dietary advice or

education sessions for the first year. If the early data from the IDPP study is

borne out, serious consideration of the early use of metformin in high-risk

South Asians should be undertaken. Early treatment of hypercholesterolaemia with

statins and hypertension with ACE-inhibitors may also contribute to the

prevention of DM in South Asian people. The greatest challenge lies in the

development of culturally appropriate interventions using a whole community

approach. It is important that interventions to prevent diabetes

in South Asians start early, and involve families and communities.

9.%% MW - Metabolic Syndrome a Possible Risk Factor for Depression (Reuters

Health) Mar 11 - Metabolic syndrome may be a " predisposing factor for the

development of depression, " Finnish researchers report. [the team followed a

large group of middle-aged

7 years. At baseline they assessed depressive symptoms using the Beck

Depression Inventory. They also assessed the presence of metabolic syndrome in

the subjects, using the modified National Cholesterol Education Program Adult

Treatment Panel III criteria.

Non-depressed subjects at baseline with metabolic syndrome were twice as

likely as subjects without metabolic syndrome to have symptoms of depression at

follow-up 7 years later, with an odds ratio of 2.2 for both sexes. Effective

prevention and treatment

of the metabolic syndrome may, in turn, reduce the incidence of depression.

" In the future, new long-term studies are needed to elucidate the long-term

course and prognosis, proper treatment and specific symptom picture of

depression related to metabolic syndrome, " they J Clin Psychiatry 2008;69.

10.%% MW - Gender Impacts Anorexigenic, Memory Effects of Intranasal Insulin

Reuters Health Information 2008. C 2008 Reuters Ltd. Mar 07 - Insulin

administered intranasally acutely decreases

food intake in men but not in women; on the other hand, it acutely improves

memory function in women but not in men, investigators observed in a small

study. " Our findings indicate that gender is a critical factor in brain insulin

signaling that affects both food intake and cognitive functions, " study

investigator Dr. C.Benedict said. The findings further suggest that intranasal

insulin " may be helpful in the treatment of cognitive and metabolic disorders

like Alzheimer's disease and obesity that are assumed to derive at least in part

from malfunctions of central nervous insulin signaling, " Central nervous insulin

is critically involved in the regulation of body weight and memory formation,

the investigators point out .The team assessed the effects of a single

intranasal dose (160 IU) of regular human insulin or vehicle on food intake and

memory processing in 32 healthy, normal-weight men and women. " Because

intranasal insulin reaches the brain but bypasses the circulation, it's possible

to assess insulin effects on the central nervous system without affecting blood

glucose levels, " " Importantly, gender differences will have to be considered in

the possible future development of intranasal insulin therapeutics. " J Clin

Endocrin Metab 2008.

11.%% MW -Breast Cancer Risk May Be Reduced After Gestational Diabetes Mar 07

- Women who develop gestational diabetes [GDM] appear to have a lower risk of

breast cancer later in life, according to a new report. [1526 white women;798

Hispanic women with breast cancer; matched with a similar number of controls.]

DM overall and T2 were not associated with breast cancer, they report, but there

was a statistically significant inverse association between gestational diabetes

and breast cancer (odds ratio 0.70). The effect was stronger

when gestational diabetes was diagnosed before age 35 years

" Should the inverse association between GDM and breast cancer be

observed in future studies, " they conclude, " potential biologic explanations

should be further investigated. " Am J Epidemiol 2008;167

12.%% Medical News Today - Least Likely To Monitor Their Blood Glucose Are

Minority, Low-Income Diabetics 17 Mar 2008

Black and Hispanic adults with insulin-treated DM are less likely than whites

to monitor their blood glucose, according to a new study presented at the

American Heart Association's 48th Annual Conference on Cardiovascular Disease

Epidemiology and Prevention.

The disparities were greatest for low-income Hispanics, said the lead author

of the study. " Minority and financially vulnerable adults with insulin-treated

diabetes appear to have lower reported rates of self-monitoring of blood glucose

(SMBG) - a vital disease management component. " Efforts to improve diabetes

control, including the collection and use of SMBG data in Hispanic and black

populations with diabetes (particularly those on insulin), are warranted given

that Hispanics and blacks have a higher frequency of diabetes-related

complications compared to whites. We need to better understand income's role in

racial and ethnic disparities in SMBG to offer effective programs and policies

to improve SMBG by minorities. " [16,630 adults 19 years + with insulin-treated

DM]

Source: Astle American Heart Association

13.%% Medical News Today - Mechanisms Suggest New Way To Treat Heart Disease

Among Diabetics 14 Mar 2008 Researchers have discovered how diabetes, by driving

inflammation and slowing blood

flow, dramatically accelerates atherosclerosis, according to research to be

published in the March 14 edition of the journal Circulation Research. Experts

once believed that atherosclerosis, or hardening of the arteries, developed when

too much cholesterol clogged arteries with fatty deposits called plaques. When

blood vessels became completely blocked, heart attacks and strokes occurred.

Today

most agree that the reaction of the body's immune system to fatty build-up,

more than the build-up itself, creates heart attack risk. Immune cells traveling

with the blood mistake fatty deposits for intruders, akin to bacteria, home in

on them, and attack. This causes inflammation that makes plaques more likely to

swell, rupture

and cut off blood flow. Making matters worse, nearly 21 million Americans have

diabetes, a disease where patients' cells cannot efficiently take in dietary

sugar, causing it to build up in the blood. In part because DM increases

atherosclerosis-related inflammation,

DM patients are twice as likely to have a heart attack or stroke.

Past work has shown that high blood sugar has two effects on cells lining

blood vessels as part of atheroslerosis. First, it increases the production of

free radicals, highly reactive molecules that tear about sensitive cell

components like DNA, causing premature cell death (apoptosis). This process also

reduces the availability of nitric oxide

(NO), which would otherwise enable blood vessels to relax and blood flow to

increase. In contrast to diabetes, exercise and good diet bring about faster

blood flow through blood vessels. The force created by fast, steady blood flow

as it drags along blood vessel walls has been shown by recent studies to protect

arteries from atherosclerosis.

Physical force has emerged recently as a key player in bodily function,

capable of kicking off biochemical processes (e.g. weightlifting thickens bone).

" Inflammation is blood vessels is one of the main drivers of atherosclerosis,

and DM makes it much worse, " said a study author. " Our study argues that a

pathway surrounding a key signaling enzyme both protects the heart in normal

cases, and is sabotaged by the chemicals produced in diabetes. We believe we

have found a new therapeutic target for the treatment of DM-related damage to

blood vessels. "

How Diabetes Does It- In people without DM, fast blood flow triggers an enzyme

called extracellular signal-regulated kinase 5 (ERK-5). ERK5 in turn signals

endothelial nitric oxide synthase (eNOS) to produce more nitric oxide and dilate

blood vessels. It also activates

Kruppel-like factor 2 (KLF2) and peroxisome proliferator-activated receptor-g

(PPARg), both of which block the ability of pro-inflammatory immune cells to

home in on and adhere to diseased portions of blood vessels.

The current results provides the first mechanistic description of how diabetes

takes away the ability of fast blood flow force to protect blood vessels,

arguing that it does so by interfering with ERK5

and its signaling partners. The team showed that molecules called advanced

glycation end products (AGEs), produced in greater levels by patients with

diabetes, interfere with ERK5 cardioprotection.

Glycation reactions cause the release of oxidizing side products like hydrogen

peroxide (H202) that drive free radical production, inflammation and cell damage

in many diseases. Researchers found that AGEs and H202 sabotage ERK5 by

encouraging the attachment

to it of a small ubiquitin-related modifier (SUMO), a protein tag used by

cells to fine-tune their control over proteins. In normal function, a cell may

extend a protein's lifespan, or send it from one part of the cell to another, by

attaching a SUMO tag. In the current study, researchers found that AGEs and H202

induced ERK5-SUMOylation

as part of disease. In addition, the team found that ERK5-SUMOylation was

increased in the aortas of diabetic mice.

This work is supported by grants from the America Heart Association and the

National Institutes of Health.

" Our experiments found that taking away the " SUMO tag " from ERK protects blood

vessels against diabetes, We believe that the SUMOylation of ERK turns off

'good' genes that are important in countering atherosclerosis. In the next

phase, we will be looking for drug candidates that can turn on ERK5 as diabetes

attempts to shut

it down. " Source: Greg University of Rochester Medical Center Press

release.

14.%% Medical News Today - FDA Approves Abbott's FreeStyle Navigator®

Continuous Glucose Monitoring System 14 Mar 2008

Click to Print The (FDA) has approved the FreeStyle Navigator® Continuous

Glucose Monitoring System in the United States for people with diabetes.

Designed to discretely and continuously measure glucose levels through a sensor

in the back of the upper arm or abdomen, [this] system provides minute-by-minute

information about which way and how quickly blood sugar levels are changing.

This information can lead to proactive adjustments that can result in tighter

glucose ranges. Before adjusting therapy for DM management based on the results

and alarms from the FreeStyle Navigator system, traditional blood glucose tests

must be performed.

" Understanding glucose trends, with the goal of minimizing fluctuations, is an

important part of improving the management of diabetes, " said endocrinologist

and Director of the International Diabetes Center " We are always looking for new

tools like these to enable people with diabetes to continuously monitor their

glucose levels, putting them on the offense, not defense, so they can take

action before a high or low glucose level occurs. " For people with DM, less time

spent with low (hypoglycemia) or high (hyperglycemia)

blood sugar has been correlated with less risk for a number of serious short-

and long-term diabetes-related complications. By measuring glucose levels

continuously, the FreeStyle Navigator system is designed to provide more and

better information than traditional fingerstick glucose measurements, which can

lead to improved diabetes management. The system monitors glucose levels by

measuring and transmitting glucose information once per

minute to the pager-sized receiver, which can be clipped to a belt or carried

in a pocket or purse. It also provides audible or vibrating alarms before

glucose levels become too high or too low, displays five directional trend

arrows to help people understand if glucose is rising or falling, and stores

historical data and glucose trend information for up to 60 days. Additionally,

the sensor and transmitter are designed to accommodate showering, swimming and a

range of normal physical activities. The accuracy, safety and efficacy of the

FreeStyle Navigator system have been demonstrated in two separate l clinical

trials, including a five-day, in-clinic study and a study of people with T1 and

T2 at home. Five-Day In-Clinic Study:[58 subjects 18-64years old] Home Use

Study: [123 people with T1 & T2 used it in their homes for 40 days, wearing the

sensor on the back of their upper arm or abdomen. A e Error Grid (CEG)

analysis demonstrated that 96.8 percent of the values were in the most accurate

zones The FreeStyle Navigator system is composed of three parts: a sensor, a

transmitter and a receiver. The sensor, worn for up to five days and then

replaced, is placed just under the skin and is attached to a plastic sensor

mount with adhesive to adhere to the skin, like a patch. The transmitter snaps

into the sensor mount and sends glucose information wirelessly to the pager-

sized receiver. The system discreetly measures glucose levels once per minute;

provides high/low glucose alarms based on customizable, physician- and

patient-determined levels; and delivers early-warning alarms that indicate if

glucose levels are likely to be too high or too low 10, 20

or 30 minutes in advance. The system also stores up to 60 days worth of

glucose information that can be analyzed by the user or a health care

professional. Indicated for people ages 18 and older, the FreeStyle Navigator

system is designed to continually record interstitial fluid glucose levels for

the purpose of improving DM

management. Navigator system are not intended to replace traditional blood

glucose monitoring.

15.%% MW -Metabolic Syndrome may Increase Operative Mortality in Patients

Undergoing Coronary Artery Bypass Graft Surgery

Future Lipidol. 2008;3(1):27-29. C2008 Future Medicine Ltd.

Abstract : Metabolic syndrome increases operative mortality in patients

undergoing coronary artery bypass grafting surgery. . Metabolic syndrome (MS) is

associated with increased risk of coronary heart disease, stroke and

cardiovascular mortality. However, little data exist on the relationship between

MS and operative mortality following coronary revascularization. This

retrospective study found that MS in patients undergoing coronary artery bypass

graft (CABG) surgery is a significant independent predictor of an increased risk

of post-operative mortality. Thus, MS may be an important and novel patient risk

factor for CABG surgery.

Abbreviations: DM - diabetes Mellitus;T1DM - type 1 diabetes mellitus T2DM -

type 2; ADA - Amer Diabetes Asso; AFB - Amer Foundation for the Blind BP - blood

pressure;HTN - hypertension; MW Medscape Web MD; FDA Federal Drug

Administration; NIH - National Institutes of Health; VA - Veterans

Administration. MNTD- Medical News Today Definitions - Dorlands 31st Ed and

Google. Disclaimer, I am a BSN RN but not a diabetic or diabetic educator.

Reports are excerpted unless otherwise noted. This project is done as a courtesy

to the blind/visually impaired and diabetic communities. Dawn Wilcox Coordinator

The Health Library at Vista Center contact above e-mail or thl@...

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http://www.eset.com

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1. Nature Clinical Practice Endocrinology & Metabolism (2008) 4

Artificial pancreas is feasible for pediatric patients with type 1

diabetes The most promising treatment for pediatric patients with T1 is

an artificial

pancreas, which links continuous blood-glucose monitoring to

computerized control of a subcutaneous insulin pump. Such systems are

feasible in adults,

but inherent delays in subcutaneous insulin delivery along with

peripheral insulin resistance in puberty could increase the risks of

postprandial glucose-level

excursion and subsequent hypoglycemia. Weinzimer and colleagues

hypothesized that these risks could be reduced

by manual administration of a preprandial 'priming' insulin bolus.

Full text is available 1. Personal subscription 2. 7 day single article

pass for US$18 © 2008 Nature Publishing Group

2.%% Nature Clinical Practice Endocrinology & Metabolism (2008) 4

Diabetes is associated with poor outcomes after bypass surgery

Critical limb ischemia can be treated with vascular bypass surgery, but

if ischemia does not resolve, lower-limb amputation might become

necessary. In

developed countries, the majority of such amputations are performed in

patients with diabetes. Whether the outcomes of bypass surgery are less

favorable

in patients with diabetes than in patients without diabetes is, however,

unclear. Malmstedt and colleagues performed a nationwide,

population-based cohort

study to compare postoperative amputation-free survival in patients with

and without diabetes. Full text [see #1]

3.%% Nature Clinical Practice Endocrinology & Metabolism (2008) 4

Diabetic nephropathy—an evaluation of potential risk factors in young

patients with type 1 diabetes mellitus Raile et al. have performed a

prospective

epidemiological study of risk factors for diabetic

nephropathy in a large cohort of patients with T1DM. In almost 28,000

patients with childhood onset of T1 disease duration, HbA1c level, lipid

abnormalities,

and BP were identified as risk factors for microalbuminuria. In

addition, male sex was a risk factor for macroalbuminuria. Full text

[see #1]

4.%% Nature Clinical Practice Endocrinology & Metabolism (2008) 4

Treatment of impaired glucose tolerance in childhood Impaired glucose

tolerance is a prediabetic condition that is a major risk factor

for subsequent development of T2DM. As T2 has become common among obese

adolescents, options for the management of children

with impaired glucose tolerance are discussed in this Viewpoint.

Full text [see #1]

5.%% Nature Clinical Practice Endocrinology & Metabolism (2008) 4

Prevention of type 1 diabetes: the time has come Type 1 diabetes

mellitus is a chronic process of many years, during which insulin

production slowly decreases. This key feature makes the disease a

potential candidate for treatment but also for prevention against

symptoms becoming clinically

overt. This Review discusses issues of diagnosis and risk

stratification, and the development of prevention strategies. Full text

[see #1]

6.%% MW - Risk for Metabolic Syndrome Linked to Sleep Duration May 21,

2008 — Risk for metabolic syndrome was associated with sleep duration,

according

to the results of a cross-sectional community-based cohort study. " Short

and long sleep duration have been linked to various risk factors for

cardiovascular

disease, " write M. Hall, PhD, and team " In the present study, we

evaluated the

relationship between sleep duration and presence of the metabolic

syndrome, which is a cluster of physiologically interrelated risk

factors for cardiometabolic

disease. " [1214 participants aged 30- 54

were divided into 4 groups on the basis of their reported sleep

duration.] Adjusted odds for metabolic syndrome were increased 45%

in both short and long sleepers vs those sleeping 7-8 hours per night.

" Additional studies are needed to evaluate temporal relationships among

these measures,

the behavioral and physiologic mechanisms that link the two, and their

impact on subsequent cardiometabolic disease. " Sleep. 2008;31

7.%% Erectile Dysfunction in Diabetics Predictive of Coronary Artery

Disease Journal of the American College of Cardiology, May 21, 2008, as

reported

by WebMD Erectile dysfunction (ED) is a " powerful " predictor of

coronary heart disease/cardiovascular disease events, including death,

in T2DM. In the

first of 2 separate studies, Italian investigators showed that among 291

type 2 diabetic men with angiographically proven asymptomatic coronary

artery

disease, the presence of ED was associated with a doubling of risk for a

major cardiac adverse event over 4 years. In the second study,

researchers in

Hong Kong looked at 2306 men with T2 and found that ED was an

independent predictor for coronary heart events (hazard ratio, 1.58;

after adjustment for

other covariates.

8.%% MW - Oxidized LDL Associated With Metabolic Syndrome

Heartwire May 21, 2008 — A higher concentration of oxidized low-density

lipoprotein (LDL)-cholesterol was associated with increased incidence of

metabolic

syndrome overall, as well as

its components of abdominal obesity, hyperglycemia, and

hypertriglyceridemia in a new population-based study. The senior author

commented to: " This is

another piece of evidence suggesting that oxidized LDL is harmful and is

a signal of future heart disease risk even in people who are currently

young and

healthy. " The authors explain that studies in cellular and animal models

have suggested that oxidized LDL, which accounts for only a minor

fraction of

LDL (0.001%-5%), contributes to processes that lead to the incidence of

the metabolic syndrome, but this association has not been tested in

humans. [CARDIA

study -5115 participants ages 18-30; followed 20 years. After adjustment

for many different variables, oxidized LDL showed a graded relation to

incident

metabolic syndrome, with those in the highest quintile of oxidized LDL

having a 3.5-times increased risk of metabolic syndrome compared with

those in the

lowest. They conclude: " As yet, it is not possible to conclude whether

oxidized LDL is a marker related to mechanistic underlying factors on

the pathway

to the development of metabolic syndrome or whether it is by itself a

functional intermediary in this pathway. However, the strong association

of oxidized

LDL with the incidence of metabolic syndrome is consistent with a causal

role. " JAMA 2008;299.

9.%% Adverse Childhood Experiences Linked to Obesity, Type 2 Diabetes

in Mid-adulthood 5/21/08 - Certain adverse childhood experiences

increased the risk

for obesity and T2DM in midadulthood, according to the results of a

prospective longitudinal study. " Child abuse has been associated with

poorer physical

health in adulthood. " We have examined how different stressful emotional

or neglectful childhood adversities are related to adiposity and glucose

control

in midadulthood. At 45 years of age, 9310 members of the 1958 British

birth cohort took part in a biomedical interview. Several adversities in

childhood

were associated with increased risk for obesity by 20- 50%. These

included physical, verbal, or witnessed abuse; humiliation; neglect;

strict upbringing;

physical punishment; conflict or tension; and low parental aspirations

or interest in education. Adversities with the strongest associations

with adiposity,

such as physical abuse, tended to be associated with HbA1c levels of 6

or more. In most cases, however, associations were explained by

adjusting for adiposity

or other adulthood mediators. " Some childhood adversities increase the

risk of obesity in adulthood and thereby increase the risk for T2.

..Research is

needed to understand the interrelatedness of adversities, the social

context of their occurrence, and trajectories from adversity to adult

disease. "

10.%% MNTD - High Triglycerides Common In People Who Are Obese And/Or

Diabetic, At Risk For Heart Disease 5/26/08

Diabetes researchers have identified a potential target for the

development of new therapies to treat hypertriglyceridemia, a lipid

disorder commonly seen

in people who are obese and diabetic. The scientists studied the role of

a protein known as Forkhead Box O1 (FoxO1) that mediates the metabolism

of glucose

and cholesterol. In the lab, they were able to curb the secretion of

triglycerides in animals that were obese and diabetic by inhibiting the

production

of FoxO1 in the liver. " Our latest findings suggest that we may

eventually be able to develop drug therapies that inhibit FoxO1, which

would thereby inhibit

the production of proteins that lead to elevated triglyceride levels in

people who are obese and/or who suffer from T2DM , " said the senior

author. " Hypertriglyceridemia

is a known risk factor for developing heart disease, the leading cause

of death in the US. " Their research suggests that FoxO1 is vital to the

regulation

of a protein known as microsomal triglyceride transfer protein (MTP)

which facilitates the production of very low-density lipoproteins

(VLDL), which are

produced in extreme excess in people with hyper -triglyceridemia. They

found FoxO1 mediates insulin action on the production of MTP in the

liver. The

team is now studying its function in other tissues and organs to

determine what an impact such therapies might have on children and

adults who are obese

and/or have T2. adapted by Medical News Today from original press

release.

11.%% MNTD - Benefits In Using Early Intensive Insulin Therapy To

Control Glucose 23 May 2008 In a Diabetes Special Issue of The

Lancet, researchers

have shown that patients with newly diagnosed T2DM who receive early

intensive insulin therapy may be able to improve the function of their

insulin-producing

ß-cells. The therapy regimen has also been associated with the

recovering and restoration

of blood glucose control. The researchers conducted a randomized

controlled trial in China. [382 patients;age 25 to 70 with T2] After

regular blood glucose

control (normoglycemia) was restored for a period of two weeks, patients

stopped receiving treatment and were then followed up on just diet and

exercise.

The findings of this team support the early intensive insulin therapy.

More patients achieved target blood glucose control and achieved it more

quickly

in both of the insulin groups compared to the group on oral drugs. In

addition, patients in the insulin groups significantly improved the

function of ß-cells

(pancreaticcells that produce insulin). " Although the relevant

biological mechanisms and target tissues contributing to preferential

improvement in ß-cell function remain unclear, these data suggest that

use of intensive

insulin therapy early in the course of T2 warrants further clinical

investigation. " The Lancet (2008). 371[9626]

12.%% MNTD -Anti Inflammatory Medication May Treat Type 2 Diabetes 22

May 2008 Researchers at the Joslin Diabetes Center who reported

earlier this

year that an inexpensive, non-steroidal anti-inflammatory drug called

salsalate might prevent T2DM are now reporting that the drug may also be

beneficial

in the treatment of the disease. The paper, reports on 3 studies that

demonstrate that salsalate, which has been used for decades to treat

arthritis, may

benefit patients with T2 by lowering blood sugar and reducing

inflammation. Together, these studies have led to 3 large, clinical

trials that seek to

confirm the benefit of targeting inflammation using salsalate to lower

glucose in patients with T2 or who are at risk for DM or to reduce

atherosclerosis

in patients with coronary artery disease. . It had originally been noted

nearly 150 years ago that salicylates could lower blood glucose levels,

but this

had either been forgotten or ignored. Lab studies found that the

inflammatory pathway regulated by NF-kB is activated in animals with

obesity and DM.

They went on to demonstrate that this pathway could be inhibited using

salicylates, thus showing that the effects of obesity are mediated

through inflammation.

This was not an accepted concept at the time, and is still debated in

field. The team opted to study salsalate, which is a salicylate similar

to aspirin

but that does not cause stomach upset or bleeding. " Our findings are

potentially very exciting because we show that a medication that treats

inflammation

may also treat diabetes and related medical conditions. " If we can show

in the larger clinical trials now underway that it is safe and

effective, it means

salsalate may be a new way to treat diabetes. "

Clinical and Translational Science (CTS),5:08

13.%% MNTD - Doctors Not Always Sure When To Treat BP In People With

Diabetes 20 May 2008 For people with diabetes, high blood pressure

poses a special

threat, multiplying their risk of heart attacks, strokes and kidney

problems. But a new study finds that even when people with DM show up in

their doctor's

office with a high BP reading, there's only a 50-50 chance that each of

them will get some sort of attention for it. That might mean a change to

their

medications, or a plan to follow up a few weeks later to see if the

reading is still high. What happens the other 50 percent of the time?

Something that

others have termed " clinical inertia " takes over, say the researchers

who conducted the study. The fear is that this lack of response to high

BP readings

at clinic visits could mean that patients' pressures will keep getting

worse. The study finds little evidence supporting the idea that

providers are just

" ignoring " blood pressure problems. What really seems to have an impact

on treatment decisions is plain old uncertainty about whether the BP is

really

elevated, or providers being occupied with other medical issues.

Providers might need to spend the visit addressing more pressing

problems, some of which,

like pain, may be contributing to elevated blood pressures. Or, they

might take another reading and conclude there's no need for action. Or,

patients may

report that their pressure readings at home have been fine. More

systematic guidelines for monitoring BP in people with DM and better

guidance for when

to change treatment when pressures get too high, are needed, say the

researchers. " While there are many guidelines about treating

hypertension, there is

an amazing lack of clarity and guidance about how many blood pressures

should be taken at a clinic visit, whether those BPs should be averaged

or whether

just the lowest should be used, and how to incorporate home blood

pressure readings in decisions to intensify medications. As long as this

confusion exists,

we may not make progress in treating hypertension. " The national goal

for people with DM is less than 130/80 mm Hg. (For people without

diabetes or kidney

problems, the goal is less than 140/90, which is considered the cutoff

for Stage I hypertension.)

" Providers clearly 'trust' their own reading more than they do the

reading taken at the clinic intake point, " " But there is no evidence

that supports

that approach. In fact, the literature suggests that provider

measurements are less reliable and subject to large biases relative to

independent measures

by nurses using electronic blood pressure cuffs. " While at-home

monitoring can be important, Kerr says, the fact of the matter is that

there is no standard

for how often to monitor and how to record home pressure readings over

time. Further, patients might preferentially report only the " normal "

blood

pressures and ignore the out-of-range values. Patients should talk to

their doctors about how often to monitor and record their BP and look at

averages

over time. ls of Internal Medicine 2008, Vol. 148, 10.

14.%% Rapid Rise in Type 1 Diabetes Seen in Finnish Children

(Reuters Health) May 22 - Finland is known to have the highest incidence

of T1DM in the world and now new research indicates that cases among

children

are increasing faster than ever. Patients with T2 and those with DM from

other causes were excluded from the analysis. The average rate of T1

during the

26-year period was 42.9 cases per 100,000 per year. In 2005, the rate

per 100,000 per year was 64.2, a marked jump from the 31.4 noted in

1980. The largest

increase in cases, 4.7% annually, was noted in children under 5 years.

" A changing environment, infant and maternal diets in particular, would

seem to

be the most likely explanation for this alarming acceleration, " was the

comment in an accompanying editorial. They point out, however, that

" identification

of the probable agents is the ultimate challenge because they might be

ubiquitous, and the hardest cause of a disease to identify is that which

universally

present. " Lancet 2008;371.

15.%% MW -Modestly Elevated Glucose in Pregnancy Raises Future Diabetes

Risk (Reuters Health) May 23 - Women without gestational diabetes,[GDM]

but who

have modestly elevated glucose levels during pregnancy, face an

increased risk of subsequently developing diabetes. Gestational diabetes

is known to increase

the risk of developing T2, the authors explain, but whether less marked

elevations of blood glucose during pregnancy increase the risk of future

diabetes

remains unclear. To investigate, the team investigated whether pregnant

women who fell into oral glucose challenge test (OGCT) quartiles

[quarters] 2 through

4 have a higher risk of DM compared with women in quartile 1, and

whether pregnant women with one abnormal oral glucose tolerance test

(OGTT) value have

a greater risk compared with women with normal values. Pregnant women

with OGCT values in the 2nd to 4th quartile were 68% to 365% more likely

to develop

diabetes than were pregnant women with OGCT values in the 1st quartile,

they report. Similarly, the investigators say, pregnant women with one

abnormal

OGTT value were just over twice as likely as similar women with normal

OGTT values to develop DM. " The risk of diabetes in women with mildly

elevated OGCT

or OGTT results likely occurs because they have an intermediate form of

glucose intolerance, " the authors explain.

" Whether women who fall within this intermediate range of glucose

tolerance during pregnancy may benefit from increased diabetes

surveillance and life-style

interventions proven to reduce the risk of developing DM is unknown, "

they conclude. Diabetes Care 2008;31

16.%% Guidelines Issued About Lack of Evidence for Screening for

Gestational Diabetes May 23, 2008 — The US Preventive Services Task

Force (USPSTF) has

issued a recommendation statement and systematic review about lack of

evidence to recommend screening for

gestational diabetes mellitus (GDM) . . " Current evidence is

insufficient to assess the balance of benefits and harms of screening

for GDM, either before

or after 24 weeks' gestation, " the statement concludes. " Until there is

better evidence, clinicians should discuss screening for GDM with their

patients

and make case-by-case decisions. The discussion should include

information about the uncertain benefits and harms as well as the

frequency and uncertain

meaning of a positive screening test result. " Ann Intern Med.2008;148

17.%% Receptor for Advanced Glycation End Products (RAGEs) and

Experimental Diabetic Neuropathy Diabetes. 2008;57(4) 5/21/2008

- Several peripheral nervous system (PNS) abnormalities complicate DM..

The most common form of somatic nerve disease in diabetic subjects is a

diabetic

symmetric sensorimotor polyneuropathy. Chronic hyperglycemia has a

robust association with the development of complications in long-term

DM, as identified

during clinical intervention trials in both T1 & 2 . The increased

nonenzymatic glycation of proteins, leading to irreversible formation

and deposition

of reactive advanced glycation end products (AGEs), may similarly lead

to critical abnormalities within the diabetic PNS. .the receptor for

AGEs (RAGE)

has been demonstrated on hematopoietic cells and endothelial cells, as

well as spinal motor neurons and cortical neurons. RAGE has been

postulated to

contribute to the development of diabetic complications. In conclusion,

our data identify an important role for RAGE in a long-term model of

experimental

DM. Not only is heightened RAGE expression prominent and progressive at

multiple levels (and in multiple cells) of the PNS, but its expression

strictly

parallels structural and electrophysiological alterations. .Whereas

overexpression of RAGE, and the consequent aberrant signaling of neurons

and glial

cells, is not the only mechanism of neurological damage in diabetes, our

data suggest that it has an important contribution to neuropathy in this

animal

model of diabetes.

18.%% MW - Metabolic Consequences of Insulin-Related Weight Gain

5/22/2008 Question- Does the weight gain associated with insulin

therapy have metabolic consequences, such as insulin resistance?

Response from Y. Chao, MD Most of the pharmacologic

interventions, including insulin, that improve glycemic control in

patients with T2DM are

associated with weight gain. Indeed, more intensive therapies to achieve

lower glycated hemoglobin (A1C) levels often lead to more weight gain.

Because

most patients with T2 are overweight, additional weight gain may not be

desirable. For example, it may aggravate pre-existing hypertension,

exacerbate

sleep apnea, and worsen osteoarthritis. The metabolic consequences

include higher low-density lipoprotein (LDL) cholesterol and lower

high-density lipoprotein

(HDL) cholesterol. Weight gain may also increase insulin resistance,

which is present in about 90% of patients with T2. Inflammatory markers,

such as C-reactive

protein, may also increase. Although large trials have shown that

improved A1C -- even in the presence of weight gain -- leads to fewer DM

complications,

any attempt to minimize weight gain associated with insulin therapy

should further extend the benefits of better glycemic control in

patients with T2.

However, further studies will be needed to test this hypothesis.

Medscape Diabetes and Endocrinology 2008

19.%% MW - Optimizing Antidiabetic Treatment Options for Patients with

Type 2 Diabetes Mellitus and Cardiovascular Comorbidities

Pharmacotherapy. 2008;28(2): 5/20/2008 Conclusion - Patients with T2

have an inherent, elevated risk for cardiovascular disease that likely

begins

well in advance of a diagnosis of chronic hyperglycemia. As with

treatment of dyslipidemia, hypertension, and hypercoagulability, when

indicated, glycemic

control is a critical element in a multifactorial approach to management

of cardiovascular risk in patients with DM. Owing to the varied

mechanisms of

many commonly prescribed antidiabetic therapies, these agents have the

potential either to mitigate or increase the risk for cardiovascular

events in patients

with DM. In some cases, the risk imposed by the nonglycemic effects of

an agent can preclude its use, making selection of an alternative agent

more appropriate.

This is particularly true for patients with established cardiovascular

disease. Insulin is a well-studied therapy that has no apparent

potential to increase

cardiovascular risk and can be an important component of a safe and

effective regimen for glycemic control. Health care providers must

carefully consider

the potential cardiovascular risks or benefits of the numerous available

anti-diabetic agents to determine the most appropriate treatment

strategies for

their patients.

20.%% Clinical Reviews Diabetes Care -- Insulin Delivery in a Changing

World Medscape J Med. 2008;10(5): 5/20/2008

Conclusions - The long-term benefits to patients with T2DM of

maintaining optimal glycemic control within recommended limits has now

been firmly established

in key landmark studies. ..development of the new insulin analogs,

combined with innovative insulin devices and SMBG systems, makes this

goal more achievable

than ever before. The involvement and cooperation of patients is crucial

to attaining these goals, and the benefits of patient education,

together with

the use of chronic disease management tools such as log books, cannot be

underestimated. The contribution of insulin analogs to glycemic control

is further

enhanced by the range of delivery devices in which they are available.

The most widely used of these, prefilled pens and cartridge pens, help

ensure compliance

by allowing patients to administer their insulin discreetly, simply, and

accurately. Exciting developments in SMBG technology, including an

integrated

pump/CBMG system, have also increased the potential for optimal glycemic

control. Insulin therapy has evolved, and by informing and reassuring

patients

about currently available treatment options, healthcare professionals

can break down many of the barriers to achieving optimal glycemic

control.

[AACE 2008] American Association of Clinical Endocrinologists 17th

Annual Meeting and Clinical Congress

Abbreviations: DM - diabetes Mellitus;T1DM - type 1 diabetes mellitus

T2DM - type 2; BP - blood pressure; MI [myocardial infarction or heart

attack] ;HTN

- hypertension; ADA - Amer Diabetes Asso; AFB - Amer Foundation for the

Blind ; FDA Federal Drug Administration; JH - s Hopkins ; MW

Medscape Web MD;

NIH - National Institutes of Health; VA - Veterans Administration. MNTD-

Medical News Today

Definitions - Dorlands 31st Ed and Google. Disclaimer, I am a BSN RN

but not a diabetic or diabetic educator. Reports are excerpted unless

otherwise noted.

This project is done as a courtesy to the blind/visually impaired and

diabetic communities. Dawn Wilcox Coordinator The Health Library at

Vista Center

contact above e-mail or thl@...

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