New, strange pain - imagine that....

[Guest guest]

Lynn ~

Hey sweetie, so sorry you are having so many

weird pains. From what I have learned, that is

one symptom of neuro-toxins......even Dad is

getting them now, from the chemo.

I have had them for years. I have never detoxed

tho, so, I cant wait to be able to do so.

With my heart attacks, I had no sweating, nor

usual heart attack symptoms, so I cant advise

there. Peace of mind is always good. I wonder

if you took mycoplex if that would help you get

rid of some of the pains you have been having.

IMHO, lupus, MS, and fibro just may have mycoplasma

and mycotoxin components to them.

The mycoplasma and mycotoxins are also neuro-toxins ! !

I pray you have peace, I KNOW you will have

healing !

Love Dede

http://www.rain-tree.com/myco.htm talks about mycoplasmas and rheumatoid diseases.......

http://www.rain-tree.com/mycoresearch.htm more here ck it out ! !

http://www.immunesupport.com/library/showarticle.cfm?ID=3066

– The Missing Link in Fatiguing Illnesses

by Guthrie, R.Ph.ImmuneSupport.com07-18-2001

Mysterious parasitesImmune DisruptionHow Mycoplasmas Operate

Detecting MycoplasmasCurrent Mycoplasma ResearchResources

ALL RIGHTS RESERVED. Reproduced with the kind permission of Alternative Medicine magazine, September, 2001 (#43). For subscription information call 800-333-HEAL (4325). Website: www.alternativemedicine.com Guthrie R.Ph, is a clinical pharmacist with hospital, business and residential experience, who researches scientifically validated integrative medical approaches. These mysterious microorganisms can play a major role in a wide range of diseases including rheumatoid arthritis, chronic fatigue and fibromyalgia syndromes, multiple sclerosis, Gulf War illness, Crohn’s disease and other inflammatory bowel diseases, diabetes and even aggressive cancers. Without proper diagnosis and treatment of mycoplasma infections, curing these conditions can be difficult or impossible. Staff Sergeant Sharron Nicolson, Crew Chief of an Army Blackhawk helicopter, was happy to see everyone return safely from their last deep mission into Iraqi territory. She and her unit would soon join the thousands of U.S. military personnel headed home from the Gulf War, and Sharron was looking forward to finishing her pilot training. But shortly after returning to the U.S. in 1991, Sharron began experiencing constant fatigue, muscle and joint pain and other debilitating symptoms similar to those associated with Chronic Fatigue Syndrome (CFS). She found it impossible to meet the demands of flight school and sadly realized her dream of a flying career was over. When routine medical tests revealed no answers, Sharron started looking for more help. At the time, she was unaware that over 50,000 soldiers had returned from the Gulf War with similar symptoms. (this number has now grown to over 100,000.) Fortunately, Sharron had the advantage of being the daughter of two top researchers in molecular and cellular biology: Drs Garth L. and L. Nicolson. At the time of Sharron’s return home in the early 1990s, Garth Nicolson, Ph.D., was an esteemed researcher and academic, holding the Bruton, Jr. Chair in Cancer Research at the University of Texas M.D. Center. Nicolson, Ph.D., a former instructor in the Department of Immunology and Microbiology at Baylor College of Medicine, was also a world-renowned molecular biologist. The Nicolsons were compelled into action on behalf of their daughter and other veterans whose disabling symptoms were being misdiagnosed as post-traumatic stress disorder and/or other conditions. The Nicolsons realized that Sharron was experiencing similar symptoms to what had experienced years earlier. The cause of ’s pain and fatigue had finally been diagnosed as an infection of invasive mycoplasmas. The Nicolsons knew these little-known microscopic masters of hide-and-seek were generally responsive to certain antibiotics. They put Sharron on a course of doxycycline antibiotic therapy and she dramatically improved. Word spread and members of other Airborne and Special Force Units who had similar symptoms began asking for assistance. The Nicolsons, anxious to help, began researching what became known as Gulf War Illness (GWI). It did not take long for them to realize that there was a significant overlap in the symptoms of GWI, CFS and FMS and other conditions that fall under the umbrella term of ‘fatiguing illnesses.’ Mysterious parasites Mycoplasmas are the smallest self-replicating organisms known to science. Viruses are even smaller, but they lack the genetic machinery to self-replicate. There are hundreds of types of mycoplasmas that can be found in plants, insects and animals, but only a few can be found in the blood and tissues throughout the human body. Not all mycoplasmas found in humans are pathogenic (disease-causing). Mycoplasmas have some of the simplest genomes among bacteria. The best known pathogenic mycoplasma, M. pneumoniae, the cause of ‘walking pneumonia,’ contains only 677 protein-coding sequences (by comparison, E. coli contains about 4,000). Mycopasmas do not contain the genes needed for amino and fatty acid or vitamin synthesis; thus, they need to steal certain amino acids, fats, vitamins and other nutrients from host cells in order to survive. Simply put, they are parasitic bacteria. Garth Nicolson explains, “Once in the cell, they steal lipids (fats) like cholesterol from the mitochondria, the components of a cell that produces energy. This makes the mitochondria ‘leaky,’ and they lose electrons. This is similar to a battery running down when the insulation around the battery is removed. This may be why patients with intracellular pathogenic mycoplasmas are almost always fatigued. They have run their cellular batteries down, so that less high energy molecules are available, and they are exhausted at the cellular level.†Immune Disruption Mycoplasmas can also disrupt the normal orchestration and organization of the host’s immune system. They can cause lymphocytes (white blood cells that bear the major responsibility of the immune system) to secrete inflammatory cytokines (proteins that facilitate cell-to-cell communication), which leads to swelling, inflammation and either stimulation or suppression of the immune system. Because pathogenic mycoplasmas leaving a cell they have infected can incorporate much of the host’s cell surface material into their own surface structure, they can instigate an autoimmune response in which the immune system starts attacking the host’s own cells, a process that can result in severe tissue damage and pain. Meanwhile the mycoplasmas evade the immune system by hiding inside host cells or fusing with the cellular membrane of the host cells. Certain pathogenic mycoplasmas can also invade lymphocytes and disrupt their functioning without provoking an immune response. Using a trick known as ‘molecular mimicry,†mycoplasmas can even closely resemble host structures to fool the immune system into thinking that they are normal host cells. After invading host cells, mycoplasmas can trigger the release of “reactive oxygen†free radicals that modify the RNA and DNA of the cells, an event that can eventually lead to malignant transformation. This phenomenon has been observed in a laboratory study in which benign (non-cancerous) cells infected by mycoplasmas became irreversibly malignant (cancerous) after 18 cell divisions. Dr. Nicolson has been working with two colleagues, Drs Darryl See and Ferre Akbarpour, of the Immune Institute in Huntington Beach. Their research has found that nearly 90% of certain late stage cancer patients show infection with pathogenic mycoplasmas. These mycoplasmas appear to drive the progression of cancer cells, making them more malignant and metastatic (capable of spreading throughout the body). Mycoplasmas can also invade the lining of blood vessels, where they appear to facilitate the release of biochemicals that can cause vasculitis (inflammation of blood vessels due to infection) and the formation of plaque inside blood vessel wall surfaces. How Mycoplasmas Operate Mycoplasmas are well equipped to play biological sleight-of-hand, appearing then disappearing, changing shape, shuffling their surface components, ducking into cells, then parading as normal citizens of the human flora dressed in clothes stolen from the cells they invaded. They’re elusive because they are pleomorphic (structurally changing). They do not have rigid cell walls like most bacteria; instead they possess fluid lipid (water insoluble fate) outer surfaces, and like tiny jellyfish, they can squeeze, bend and move into tight spaces. They can also slide right through laboratory and hospital filters used to produce or maintain sterility – making them one of the most common contaminants in diagnostic laboratories and vaccine manufacturing. In one recent study of vaccines, mycoplasmas were found to contaminate about six percent of commercial vaccines. These microorganisms have been quite successful in adapting to many environments, infecting everything from insects to elephants, plants to people. Generally, they are species-specific, but there appear to be many exceptions. Garth Nicolson relates more than one case in which the pets of GWI or CFS patients were exhibiting similar symptoms as their owners, and then tested positive for the same mycoplasmas. No one knows for sure how contagious mycoplasmas are, but it appears transmission may occur among infected people in close proximity for extended periods of time. Not everyone who is exposed becomes sick. For example, when Nicolson studied Gulf War veterans’ families who became sick with symptoms similar to GWI, he found that not every member of the family became sick, but those that did become ill had the same infection as found in the sick veteran. Detecting Mycoplasmas When the Nicolsons began to explore the connection between GWI and mycoplasma, they first had to figure out how to screen people with GWI signs and symptoms for the presence of these pathogens. This was easier said than done. Since mycoplasmas are extremely small, change shape and lack rigid and distinctive cell walls, they’re impossible to find using conventional microbiology techniques. They won’t grow in a standard culture medium, and they are not usually revealed by standard tests that look for antibodies (proteins made by a white blood cell as a primary defense against foreign substances). Some people do show antibody responses to certain mycoplasmas, but antibody tests are still not specific enough to make a diagnosis. Using a technique called nucleoprotein gene tracking developed by the Nicolsons, they were able to identify mycoplasma genetic elements in white blood cells of GWI patients. However, conventional Polymerase Chain Reaction (PCR0 tests performed by Army pathologists did not confirm the presence of mycoplasma DNA. Eventually, the Nicolsons developed a new PCR test based on techniques used by forensic pathologists to test for DNA from crime scenes. This test revealed that over 40% of the GWI patients were positive for “invasive†mycoplasma (not mycoplasma in superficial sites such as nose, throat and genitourinary tract). The Nicolsons found mycoplasmas, especially M. fermentans, inside tissues and in certain white blood cells – the very cells that are normally involved in the destruction of pathogenic invaders. “Mycoplasmas are not found systemically in most normal subjects – only a few percent of asymptomatic subjects have evidence of mycoplasma in their blood. I don’t consider oral mycoplasma, or mycoplasma at other superficial sites to be evidence of an infection. It is more likely simple bacterial colonization, and unless these mycoplasma invade the epithelial cell layer (a thin layer of tissue that covers a surface or lines a cavity), they are probably benign nonpathogenic residents,’ explains Nicolson. The researchers’ results were significant and published in several journals. Other investigators, especially those working with Gulf War Vets, were able to duplicate the results, but the Nicolson’s work was largely dismissed or ignored by the Department of Defense. However, in February, 2000, psychiatrist Lt. Col. Engel, M.D., director of the Gulf War Illness Center at Walter Army Medical Center, presented pivotal information to a CFS coordinating board at the National Institutes of Health. A study conducted independently for the U.S. Departments of Defense and Veterans Affairs demonstrated that approximately 40% of more than 1,600 GWI patients were positive for mycoplasma infections, and 80% of those were positive for M. fermentans. Lt. Col. Engel also stated that he felt that these infections might also be an important cause of CFS. The study findings nearly duplicated the figures that the Nicolsons had reported earlier: 45% positive for mycoplasma; 80% with M. fermentans. Currently, other prominent researchers are corroborating the role of mycoplasma in disease. The number of known conditions in which mycoplasmas play a role is growing, thanks to advances in detection. Mycoplasmas are now said to be contributors, or at least cofactors, in a number of conditions, including CFS/CFIDS, fibromyalgia syndrome (FMS), lupus, multiple sclerosis (MS), psoriasis, scleroderma, Chrohn’s diseases, solid cancers, leukemia, lymphoma, Amyotrophic Lateral Sclerosis (ALS), pelvic inflammatory disease (PID), asthma, atypical pneumonia, Sjogren’s syndrome, interstitial cystitis, Alzheimer’s and cardiovascular diseases. Mycoplasmas have also been associated with a variety if autoimmune diseases that can cause definite changes in nerve conduction, demyelation (a degenerative process that erodes away the myelin sheath that normally protects nerve fibers) and sensitivity. Dr. Nicolson says that the role of mycoplasmas in various illnesses and diseases is now gradually being accepted, especially in those once long-suspected as “psychological.†Acceptance is due to the recognition that symptoms cannot be explained solely by psychological criteria, and because discrete clinical markers have been discovered. For example, the vascultitis (inflammation of blood vessels) found in mycoplasma-positive patients correlates with evidence of mycoplasma-induced abnormalities in blood cells and proteins related to blood clotting. Current Mycoplasma Research Recently, Dr. Nicolson has focused on various autoimmune neurological diseases such as ALS, MS, Lyme disease and others. For example, approximately 85% of patients with ALS (“Lou Gehrig’s diseaseâ€) tested positive for systemic mycoplasma infections, and most of those infections involve M. fermentans and/or M. hominis. Dr. Nicolson is working closely with Drs. See and Akbarpour on ALS, a condition in which patients lose control of their motor and skeletal muscles over a period of two to five years. Their research revealed that almost all ALS patients have co-infections with a virus from the enterovirus family (a virus related to the polio virus that replicated mostly in the gastrointestinal tract) – and mycoplasmas. The three doctors have been conducting a clinical treatment study of ALS utilizing antibiotics, antivirals and nerve growth factors. They are seeing positive results so far, as measured by increases in muscle strength. Other illnesses often have multiple strains of mycoplasmas, or mycoplasmas combined with co-infections of other bacteria or viruses. “In recent published studies from our laboratory, most CFS and FMS patients had multiple mycoplasmal infections. The number of different mycoplasmal species in these patients increased with the number of years the patients were sick and with the severity of their illness,†says Dr. Nicolson. “We have found that when the few asymptomatic subjects have blood mycoplasmal infections, they have only one species, versus when we examine patients who are sick with various chronic illnesses, they usually have multiple species of mycoplasmas and other infections such as the cytomegalovirus. In Lyme disease, we often find mycoplasmal co-infections, most frequently, M. fermentans, along with the Borrelia that causes it. This makes sense when you consider that insects, such as the ticks that carry the Borrelia, also can carry mycoplasmas. Dr. Eli Mordechai of Medical Diagnostics Lab of New Jersey has exactly the same findings in Lyme disease patients.†All the researchers above agree that long-term antibiotics must be initiated to treat mycoplasmal infections. Additional strategies must be applied to protect and strengthen the immune system and provide essential nutrients and vitamins. “We always try to use the least toxic approaches in working with pathologies, so we use a lot of natural products,†Dr. See says. For example, probiotics and undenatured whey protein isolates are used to support the GI tract – a combination that helps prevent overgrowth of undesirable microorganisms. “However,†adds Dr. See, “in our experience, and in the literature, we have found non other way to deal with mycoplasmas than fairly long-term treatment with certain antibiotics.†Fortunately, the Nicolsons and their colleagues have succeeded in helping many veterans and others infected with mycoplasmas, but controversies surrounding their work and these mysterious microorganisms still persist. Says Dr. Nicolson, “Future efforts to explain and treat a variety of illnesses that currently have unknown etiologies (causes) will undoubtedly focus more on chronic infections as underlying causes or as opportunistic infections in immune-impaired patients. We have found that chronic infections caused by mycoplasmas, viruses and other microorganisms cannot be ignored, because these patients will remain ill and not recover from their illnesses if these infections remain untreated. Resources Additional information on mycoplasma treatment, yeast overgrowth, nutrition, and treating multiple infections associated with mycoplasmas can be found on the Institute for Molecular Medicine’s Web site: www.immed.org

http://www.immed.org/signsympt.htm this is very interesting ! ! ! ! ! ! ! !

http://www.immed.org/index.htm

For further Information on diseases and syndromes that could have an

important infectious component, such as:

Fatiguing Illnesses

Autoimmune Diseases

Neurodegenerative Diseases

Chronic Fatigue Syndrome

Myalgic Encephalomyelitis

Fibromyalgia Syndrome

Gulf War Illnesses

Other Illnesses:

Chronic Asthma

Chronic Bronchitis

Chronic Pneumonia

Vascular inflammation

Urogenital diseases

HIV

Heart Diseases

Rheumatoid Arthritis

Scleroderma

Sarcoidosis

Ankylosing Spondylitis

Graves’ Disease

Hashimoto’s Disease

Inflammatory Bowel Diseases (IBD)

Lupus (SLE)

Crohn’s Disease

Reiter’s Disease

Amyotrophic Lateral Sclerosis (ALS)Multiple Sclerosis (MS)

Alzheimer’s Disease (AD)

Parkinson’s Disease (PD)

Peripheral Neuropathies

Other Neurological Disorders:

Autistic Spectrum Disorders

Autism

ADHD, ADD

Asperger Syndrome

StrokeStart the year off right. Easy ways to stay in shape in the new year.

[Guest guest]

Lynn ~

Hey sweetie, so sorry you are having so many

weird pains. From what I have learned, that is

one symptom of neuro-toxins......even Dad is

getting them now, from the chemo.

I have had them for years. I have never detoxed

tho, so, I cant wait to be able to do so.

With my heart attacks, I had no sweating, nor

usual heart attack symptoms, so I cant advise

there. Peace of mind is always good. I wonder

if you took mycoplex if that would help you get

rid of some of the pains you have been having.

IMHO, lupus, MS, and fibro just may have mycoplasma

and mycotoxin components to them.

The mycoplasma and mycotoxins are also neuro-toxins ! !

I pray you have peace, I KNOW you will have

healing !

Love Dede

http://www.rain-tree.com/myco.htm talks about mycoplasmas and rheumatoid diseases.......

http://www.rain-tree.com/mycoresearch.htm more here ck it out ! !

http://www.immunesupport.com/library/showarticle.cfm?ID=3066

– The Missing Link in Fatiguing Illnesses

by Guthrie, R.Ph.ImmuneSupport.com07-18-2001

Mysterious parasitesImmune DisruptionHow Mycoplasmas Operate

Detecting MycoplasmasCurrent Mycoplasma ResearchResources

ALL RIGHTS RESERVED. Reproduced with the kind permission of Alternative Medicine magazine, September, 2001 (#43). For subscription information call 800-333-HEAL (4325). Website: www.alternativemedicine.com Guthrie R.Ph, is a clinical pharmacist with hospital, business and residential experience, who researches scientifically validated integrative medical approaches. These mysterious microorganisms can play a major role in a wide range of diseases including rheumatoid arthritis, chronic fatigue and fibromyalgia syndromes, multiple sclerosis, Gulf War illness, Crohn’s disease and other inflammatory bowel diseases, diabetes and even aggressive cancers. Without proper diagnosis and treatment of mycoplasma infections, curing these conditions can be difficult or impossible. Staff Sergeant Sharron Nicolson, Crew Chief of an Army Blackhawk helicopter, was happy to see everyone return safely from their last deep mission into Iraqi territory. She and her unit would soon join the thousands of U.S. military personnel headed home from the Gulf War, and Sharron was looking forward to finishing her pilot training. But shortly after returning to the U.S. in 1991, Sharron began experiencing constant fatigue, muscle and joint pain and other debilitating symptoms similar to those associated with Chronic Fatigue Syndrome (CFS). She found it impossible to meet the demands of flight school and sadly realized her dream of a flying career was over. When routine medical tests revealed no answers, Sharron started looking for more help. At the time, she was unaware that over 50,000 soldiers had returned from the Gulf War with similar symptoms. (this number has now grown to over 100,000.) Fortunately, Sharron had the advantage of being the daughter of two top researchers in molecular and cellular biology: Drs Garth L. and L. Nicolson. At the time of Sharron’s return home in the early 1990s, Garth Nicolson, Ph.D., was an esteemed researcher and academic, holding the Bruton, Jr. Chair in Cancer Research at the University of Texas M.D. Center. Nicolson, Ph.D., a former instructor in the Department of Immunology and Microbiology at Baylor College of Medicine, was also a world-renowned molecular biologist. The Nicolsons were compelled into action on behalf of their daughter and other veterans whose disabling symptoms were being misdiagnosed as post-traumatic stress disorder and/or other conditions. The Nicolsons realized that Sharron was experiencing similar symptoms to what had experienced years earlier. The cause of ’s pain and fatigue had finally been diagnosed as an infection of invasive mycoplasmas. The Nicolsons knew these little-known microscopic masters of hide-and-seek were generally responsive to certain antibiotics. They put Sharron on a course of doxycycline antibiotic therapy and she dramatically improved. Word spread and members of other Airborne and Special Force Units who had similar symptoms began asking for assistance. The Nicolsons, anxious to help, began researching what became known as Gulf War Illness (GWI). It did not take long for them to realize that there was a significant overlap in the symptoms of GWI, CFS and FMS and other conditions that fall under the umbrella term of ‘fatiguing illnesses.’ Mysterious parasites Mycoplasmas are the smallest self-replicating organisms known to science. Viruses are even smaller, but they lack the genetic machinery to self-replicate. There are hundreds of types of mycoplasmas that can be found in plants, insects and animals, but only a few can be found in the blood and tissues throughout the human body. Not all mycoplasmas found in humans are pathogenic (disease-causing). Mycoplasmas have some of the simplest genomes among bacteria. The best known pathogenic mycoplasma, M. pneumoniae, the cause of ‘walking pneumonia,’ contains only 677 protein-coding sequences (by comparison, E. coli contains about 4,000). Mycopasmas do not contain the genes needed for amino and fatty acid or vitamin synthesis; thus, they need to steal certain amino acids, fats, vitamins and other nutrients from host cells in order to survive. Simply put, they are parasitic bacteria. Garth Nicolson explains, “Once in the cell, they steal lipids (fats) like cholesterol from the mitochondria, the components of a cell that produces energy. This makes the mitochondria ‘leaky,’ and they lose electrons. This is similar to a battery running down when the insulation around the battery is removed. This may be why patients with intracellular pathogenic mycoplasmas are almost always fatigued. They have run their cellular batteries down, so that less high energy molecules are available, and they are exhausted at the cellular level.†Immune Disruption Mycoplasmas can also disrupt the normal orchestration and organization of the host’s immune system. They can cause lymphocytes (white blood cells that bear the major responsibility of the immune system) to secrete inflammatory cytokines (proteins that facilitate cell-to-cell communication), which leads to swelling, inflammation and either stimulation or suppression of the immune system. Because pathogenic mycoplasmas leaving a cell they have infected can incorporate much of the host’s cell surface material into their own surface structure, they can instigate an autoimmune response in which the immune system starts attacking the host’s own cells, a process that can result in severe tissue damage and pain. Meanwhile the mycoplasmas evade the immune system by hiding inside host cells or fusing with the cellular membrane of the host cells. Certain pathogenic mycoplasmas can also invade lymphocytes and disrupt their functioning without provoking an immune response. Using a trick known as ‘molecular mimicry,†mycoplasmas can even closely resemble host structures to fool the immune system into thinking that they are normal host cells. After invading host cells, mycoplasmas can trigger the release of “reactive oxygen†free radicals that modify the RNA and DNA of the cells, an event that can eventually lead to malignant transformation. This phenomenon has been observed in a laboratory study in which benign (non-cancerous) cells infected by mycoplasmas became irreversibly malignant (cancerous) after 18 cell divisions. Dr. Nicolson has been working with two colleagues, Drs Darryl See and Ferre Akbarpour, of the Immune Institute in Huntington Beach. Their research has found that nearly 90% of certain late stage cancer patients show infection with pathogenic mycoplasmas. These mycoplasmas appear to drive the progression of cancer cells, making them more malignant and metastatic (capable of spreading throughout the body). Mycoplasmas can also invade the lining of blood vessels, where they appear to facilitate the release of biochemicals that can cause vasculitis (inflammation of blood vessels due to infection) and the formation of plaque inside blood vessel wall surfaces. How Mycoplasmas Operate Mycoplasmas are well equipped to play biological sleight-of-hand, appearing then disappearing, changing shape, shuffling their surface components, ducking into cells, then parading as normal citizens of the human flora dressed in clothes stolen from the cells they invaded. They’re elusive because they are pleomorphic (structurally changing). They do not have rigid cell walls like most bacteria; instead they possess fluid lipid (water insoluble fate) outer surfaces, and like tiny jellyfish, they can squeeze, bend and move into tight spaces. They can also slide right through laboratory and hospital filters used to produce or maintain sterility – making them one of the most common contaminants in diagnostic laboratories and vaccine manufacturing. In one recent study of vaccines, mycoplasmas were found to contaminate about six percent of commercial vaccines. These microorganisms have been quite successful in adapting to many environments, infecting everything from insects to elephants, plants to people. Generally, they are species-specific, but there appear to be many exceptions. Garth Nicolson relates more than one case in which the pets of GWI or CFS patients were exhibiting similar symptoms as their owners, and then tested positive for the same mycoplasmas. No one knows for sure how contagious mycoplasmas are, but it appears transmission may occur among infected people in close proximity for extended periods of time. Not everyone who is exposed becomes sick. For example, when Nicolson studied Gulf War veterans’ families who became sick with symptoms similar to GWI, he found that not every member of the family became sick, but those that did become ill had the same infection as found in the sick veteran. Detecting Mycoplasmas When the Nicolsons began to explore the connection between GWI and mycoplasma, they first had to figure out how to screen people with GWI signs and symptoms for the presence of these pathogens. This was easier said than done. Since mycoplasmas are extremely small, change shape and lack rigid and distinctive cell walls, they’re impossible to find using conventional microbiology techniques. They won’t grow in a standard culture medium, and they are not usually revealed by standard tests that look for antibodies (proteins made by a white blood cell as a primary defense against foreign substances). Some people do show antibody responses to certain mycoplasmas, but antibody tests are still not specific enough to make a diagnosis. Using a technique called nucleoprotein gene tracking developed by the Nicolsons, they were able to identify mycoplasma genetic elements in white blood cells of GWI patients. However, conventional Polymerase Chain Reaction (PCR0 tests performed by Army pathologists did not confirm the presence of mycoplasma DNA. Eventually, the Nicolsons developed a new PCR test based on techniques used by forensic pathologists to test for DNA from crime scenes. This test revealed that over 40% of the GWI patients were positive for “invasive†mycoplasma (not mycoplasma in superficial sites such as nose, throat and genitourinary tract). The Nicolsons found mycoplasmas, especially M. fermentans, inside tissues and in certain white blood cells – the very cells that are normally involved in the destruction of pathogenic invaders. “Mycoplasmas are not found systemically in most normal subjects – only a few percent of asymptomatic subjects have evidence of mycoplasma in their blood. I don’t consider oral mycoplasma, or mycoplasma at other superficial sites to be evidence of an infection. It is more likely simple bacterial colonization, and unless these mycoplasma invade the epithelial cell layer (a thin layer of tissue that covers a surface or lines a cavity), they are probably benign nonpathogenic residents,’ explains Nicolson. The researchers’ results were significant and published in several journals. Other investigators, especially those working with Gulf War Vets, were able to duplicate the results, but the Nicolson’s work was largely dismissed or ignored by the Department of Defense. However, in February, 2000, psychiatrist Lt. Col. Engel, M.D., director of the Gulf War Illness Center at Walter Army Medical Center, presented pivotal information to a CFS coordinating board at the National Institutes of Health. A study conducted independently for the U.S. Departments of Defense and Veterans Affairs demonstrated that approximately 40% of more than 1,600 GWI patients were positive for mycoplasma infections, and 80% of those were positive for M. fermentans. Lt. Col. Engel also stated that he felt that these infections might also be an important cause of CFS. The study findings nearly duplicated the figures that the Nicolsons had reported earlier: 45% positive for mycoplasma; 80% with M. fermentans. Currently, other prominent researchers are corroborating the role of mycoplasma in disease. The number of known conditions in which mycoplasmas play a role is growing, thanks to advances in detection. Mycoplasmas are now said to be contributors, or at least cofactors, in a number of conditions, including CFS/CFIDS, fibromyalgia syndrome (FMS), lupus, multiple sclerosis (MS), psoriasis, scleroderma, Chrohn’s diseases, solid cancers, leukemia, lymphoma, Amyotrophic Lateral Sclerosis (ALS), pelvic inflammatory disease (PID), asthma, atypical pneumonia, Sjogren’s syndrome, interstitial cystitis, Alzheimer’s and cardiovascular diseases. Mycoplasmas have also been associated with a variety if autoimmune diseases that can cause definite changes in nerve conduction, demyelation (a degenerative process that erodes away the myelin sheath that normally protects nerve fibers) and sensitivity. Dr. Nicolson says that the role of mycoplasmas in various illnesses and diseases is now gradually being accepted, especially in those once long-suspected as “psychological.†Acceptance is due to the recognition that symptoms cannot be explained solely by psychological criteria, and because discrete clinical markers have been discovered. For example, the vascultitis (inflammation of blood vessels) found in mycoplasma-positive patients correlates with evidence of mycoplasma-induced abnormalities in blood cells and proteins related to blood clotting. Current Mycoplasma Research Recently, Dr. Nicolson has focused on various autoimmune neurological diseases such as ALS, MS, Lyme disease and others. For example, approximately 85% of patients with ALS (“Lou Gehrig’s diseaseâ€) tested positive for systemic mycoplasma infections, and most of those infections involve M. fermentans and/or M. hominis. Dr. Nicolson is working closely with Drs. See and Akbarpour on ALS, a condition in which patients lose control of their motor and skeletal muscles over a period of two to five years. Their research revealed that almost all ALS patients have co-infections with a virus from the enterovirus family (a virus related to the polio virus that replicated mostly in the gastrointestinal tract) – and mycoplasmas. The three doctors have been conducting a clinical treatment study of ALS utilizing antibiotics, antivirals and nerve growth factors. They are seeing positive results so far, as measured by increases in muscle strength. Other illnesses often have multiple strains of mycoplasmas, or mycoplasmas combined with co-infections of other bacteria or viruses. “In recent published studies from our laboratory, most CFS and FMS patients had multiple mycoplasmal infections. The number of different mycoplasmal species in these patients increased with the number of years the patients were sick and with the severity of their illness,†says Dr. Nicolson. “We have found that when the few asymptomatic subjects have blood mycoplasmal infections, they have only one species, versus when we examine patients who are sick with various chronic illnesses, they usually have multiple species of mycoplasmas and other infections such as the cytomegalovirus. In Lyme disease, we often find mycoplasmal co-infections, most frequently, M. fermentans, along with the Borrelia that causes it. This makes sense when you consider that insects, such as the ticks that carry the Borrelia, also can carry mycoplasmas. Dr. Eli Mordechai of Medical Diagnostics Lab of New Jersey has exactly the same findings in Lyme disease patients.†All the researchers above agree that long-term antibiotics must be initiated to treat mycoplasmal infections. Additional strategies must be applied to protect and strengthen the immune system and provide essential nutrients and vitamins. “We always try to use the least toxic approaches in working with pathologies, so we use a lot of natural products,†Dr. See says. For example, probiotics and undenatured whey protein isolates are used to support the GI tract – a combination that helps prevent overgrowth of undesirable microorganisms. “However,†adds Dr. See, “in our experience, and in the literature, we have found non other way to deal with mycoplasmas than fairly long-term treatment with certain antibiotics.†Fortunately, the Nicolsons and their colleagues have succeeded in helping many veterans and others infected with mycoplasmas, but controversies surrounding their work and these mysterious microorganisms still persist. Says Dr. Nicolson, “Future efforts to explain and treat a variety of illnesses that currently have unknown etiologies (causes) will undoubtedly focus more on chronic infections as underlying causes or as opportunistic infections in immune-impaired patients. We have found that chronic infections caused by mycoplasmas, viruses and other microorganisms cannot be ignored, because these patients will remain ill and not recover from their illnesses if these infections remain untreated. Resources Additional information on mycoplasma treatment, yeast overgrowth, nutrition, and treating multiple infections associated with mycoplasmas can be found on the Institute for Molecular Medicine’s Web site: www.immed.org

http://www.immed.org/signsympt.htm this is very interesting ! ! ! ! ! ! ! !

http://www.immed.org/index.htm

For further Information on diseases and syndromes that could have an

important infectious component, such as:

Fatiguing Illnesses

Autoimmune Diseases

Neurodegenerative Diseases

Chronic Fatigue Syndrome

Myalgic Encephalomyelitis

Fibromyalgia Syndrome

Gulf War Illnesses

Other Illnesses:

Chronic Asthma

Chronic Bronchitis

Chronic Pneumonia

Vascular inflammation

Urogenital diseases

HIV

Heart Diseases

Rheumatoid Arthritis

Scleroderma

Sarcoidosis

Ankylosing Spondylitis

Graves’ Disease

Hashimoto’s Disease

Inflammatory Bowel Diseases (IBD)

Lupus (SLE)

Crohn’s Disease

Reiter’s Disease

Amyotrophic Lateral Sclerosis (ALS)Multiple Sclerosis (MS)

Alzheimer’s Disease (AD)

Parkinson’s Disease (PD)

Peripheral Neuropathies

Other Neurological Disorders:

Autistic Spectrum Disorders

Autism

ADHD, ADD

Asperger Syndrome

StrokeStart the year off right. Easy ways to stay in shape in the new year.

[Guest guest]

Lynn,

I don't know what it is . .. but it doesn't sound like Fibromyalgia . . . Fibro = muscle - Myalgia = pain.

Rogene

New, strange pain - imagine that....

Last night I was having a shooting, sharp pain on my left side collar bone area - felt like it was on the actual bone. Could not lay on my left side. So, this morning, I still had the pain on and off most of the morning at work. Hurt like heck. Now, it has moved to my left arm bone. Again, feels like it is on the actual upper bone of my arm. Not constant, just sharp and every now and again. Any thoughts? Fibromyalgia pains? I am just scared of a heart attack but I am not sweating. I do feel a little sick to my stomach but that is about all - felt like crapola all day. Thanks,Lynn

[Guest guest]

Thanks Dede! This e-mail alone gives me piece of mind.

Love, Lynn

>

> Lynn ~

> Hey sweetie, so sorry you are having so many

> weird pains. From what I have learned, that is

> one symptom of neuro-toxins......even Dad is

> getting them now, from the chemo.

> I have had them for years. I have never detoxed

> tho, so, I cant wait to be able to do so.

> With my heart attacks, I had no sweating, nor

> usual heart attack symptoms, so I cant advise

> there. Peace of mind is always good. I wonder

> if you took mycoplex if that would help you get

> rid of some of the pains you have been having.

> IMHO, lupus, MS, and fibro just may have mycoplasma

> and mycotoxin components to them.

> The mycoplasma and mycotoxins are also neuro-toxins ! !

> I pray you have peace, I KNOW you will have

> healing !

> Love Dede

> _http://www.rain-tree.com/myco.htm_ (http://www.rain-

tree.com/myco.htm)

> talks about mycoplasmas and rheumatoid diseases.......

> _http://www.rain-tree.com/mycoresearch.htm_

> (http://www.rain-tree.com/mycoresearch.htm) more here ck it

out ! !

>

> _http://www.immunesupport.com/library/showarticle.cfm?ID=3066_

> (http://www.immunesupport.com/library/showarticle.cfm?ID=3066)

> †" The Missing Link in Fatiguing Illnesses

>

> by Guthrie, R.Ph.

> ImmuneSupport.com

>

> 07-18-2001

>

> _Mysterious parasites_

> (http://www.immunesupport.com/library/showarticle.cfm?

ID=3066#article1)

> _Immune Disruption_

> (http://www.immunesupport.com/library/showarticle.cfm?

ID=3066#article2)

> _How Mycoplasmas Operate_

> (http://www.immunesupport.com/library/showarticle.cfm?

ID=3066#article3) _Detecting Mycoplasmas_

> (http://www.immunesupport.com/library/showarticle.cfm?

ID=3066#article4)

> _Current Mycoplasma Research_

> (http://www.immunesupport.com/library/showarticle.cfm?

ID=3066#article5)

> _Resources_

> (http://www.immunesupport.com/library/showarticle.cfm?

ID=3066#article6)

> ALL RIGHTS RESERVED. Reproduced with the kind permission of

Alternative

> Medicine magazine, September, 2001 (#43). For subscription

information call

> 800-333-HEAL (4325). Website: www.alternativemedicine.com

> Guthrie R.Ph, is a clinical pharmacist with hospital,

business and

> residential experience, who researches scientifically validated

integrative

> medical approaches.

> These mysterious microorganisms can play a major role in a wide

range of

> diseases including rheumatoid arthritis, chronic fatigue and

fibromyalgia

> syndromes, multiple sclerosis, Gulf War illness, Crohn’s disease

and other

> inflammatory bowel diseases, diabetes and even aggressive cancers.

Without proper

> diagnosis and treatment of mycoplasma infections, curing these

conditions can be

> difficult or impossible.

> Staff Sergeant Sharron Nicolson, Crew Chief of an Army Blackhawk

helicopter,

> was happy to see everyone return safely from their last deep

mission into

> Iraqi territory. She and her unit would soon join the thousands of

U.S. military

> personnel headed home from the Gulf War, and Sharron was looking

forward to

> finishing her pilot training. But shortly after returning to the

U.S. in

> 1991, Sharron began experiencing constant fatigue, muscle and

joint pain and

> other debilitating symptoms similar to those associated with

Chronic Fatigue

> Syndrome (CFS). She found it impossible to meet the demands of

flight school and

> sadly realized her dream of a flying career was over.

> When routine medical tests revealed no answers, Sharron started

looking for

> more help. At the time, she was unaware that over 50,000 soldiers

had returned

> from the Gulf War with similar symptoms. (this number has now

grown to over

> 100,000.) Fortunately, Sharron had the advantage of being the

daughter of two

> top researchers in molecular and cellular biology: Drs Garth L.

and L.

> Nicolson.

> At the time of Sharron’s return home in the early 1990s, Garth

Nicolson,

> Ph.D., was an esteemed researcher and academic, holding the

Bruton, Jr.

> Chair in Cancer Research at the University of Texas M.D.

Center.

> Nicolson, Ph.D., a former instructor in the Department of

Immunology and

> Microbiology at Baylor College of Medicine, was also a world-

renowned molecular

> biologist. The Nicolsons were compelled into action on behalf of

their

> daughter and other veterans whose disabling symptoms were being

misdiagnosed as

> post-traumatic stress disorder and/or other conditions.

> The Nicolsons realized that Sharron was experiencing similar

symptoms to what

> had experienced years earlier. The cause of ’s pain

and fatigue

> had finally been diagnosed as an infection of invasive

mycoplasmas. The

> Nicolsons knew these little-known microscopic masters of hide-and-

seek were

> generally responsive to certain antibiotics. They put Sharron on a

course of

> doxycycline antibiotic therapy and she dramatically improved.

> Word spread and members of other Airborne and Special Force Units

who had

> similar symptoms began asking for assistance. The Nicolsons,

anxious to help,

> began researching what became known as Gulf War Illness (GWI). It

did not take

> long for them to realize that there was a significant overlap in

the symptoms

> of GWI, CFS and FMS and other conditions that fall under the

umbrella term

> of ‘fatiguing illnesses.’

> Mysterious parasites

> Mycoplasmas are the smallest self-replicating organisms known to

science.

> Viruses are even smaller, but they lack the genetic machinery to

self-replicate.

> There are hundreds of types of mycoplasmas that can be found in

plants,

> insects and animals, but only a few can be found in the blood and

tissues

> throughout the human body. Not all mycoplasmas found in humans are

pathogenic

> (disease-causing).

> Mycoplasmas have some of the simplest genomes among bacteria. The

best known

> pathogenic mycoplasma, M. pneumoniae, the cause of ‘walking

pneumonia,’

> contains only 677 protein-coding sequences (by comparison, E. coli

contains about

> 4,000). Mycopasmas do not contain the genes needed for amino and

fatty acid

> or vitamin synthesis; thus, they need to steal certain amino

acids, fats,

> vitamins and other nutrients from host cells in order to survive.

Simply put,

> they are parasitic bacteria. Garth Nicolson explains, “Once in

the cell, they

> steal lipids (fats) like cholesterol from the mitochondria, the

components of

> a cell that produces energy. This makes the mitochondria

‘leaky,’ and they

> lose electrons. This is similar to a battery running down when the

insulation

> around the battery is removed. This may be why patients with

intracellular

> pathogenic mycoplasmas are almost always fatigued. They have run

their cellular

> batteries down, so that less high energy molecules are available,

and they

> are exhausted at the cellular level.â€

> Immune Disruption

> Mycoplasmas can also disrupt the normal orchestration and

organization of the

> host’s immune system. They can cause lymphocytes (white blood

cells that

> bear the major responsibility of the immune system) to secrete

inflammatory

> cytokines (proteins that facilitate cell-to-cell communication),

which leads to

> swelling, inflammation and either stimulation or suppression of

the immune

> system.

> Because pathogenic mycoplasmas leaving a cell they have infected

can

> incorporate much of the host’s cell surface material into their

own surface

> structure, they can instigate an autoimmune response in which the

immune system

> starts attacking the host’s own cells, a process that can result

in severe tissue

> damage and pain.

> Meanwhile the mycoplasmas evade the immune system by hiding inside

host cells

> or fusing with the cellular membrane of the host cells. Certain

pathogenic

> mycoplasmas can also invade lymphocytes and disrupt their

functioning without

> provoking an immune response. Using a trick known as ‘molecular

mimicry,â€

> mycoplasmas can even closely resemble host structures to fool the

immune system

> into thinking that they are normal host cells.

> After invading host cells, mycoplasmas can trigger the release of

“reactive

> oxygen†free radicals that modify the RNA and DNA of the cells,

an event that

> can eventually lead to malignant transformation. This phenomenon

has been

> observed in a laboratory study in which benign (non-cancerous)

cells infected by

> mycoplasmas became irreversibly malignant (cancerous) after 18

cell

> divisions. Dr. Nicolson has been working with two colleagues, Drs

Darryl See and

> Ferre Akbarpour, of the Immune Institute in Huntington Beach.

Their research has

> found that nearly 90% of certain late stage cancer patients show

infection

> with pathogenic mycoplasmas. These mycoplasmas appear to drive the

progression

> of cancer cells, making them more malignant and metastatic

(capable of

> spreading throughout the body).

> Mycoplasmas can also invade the lining of blood vessels, where they

appear to

> facilitate the release of biochemicals that can cause vasculitis

> (inflammation of blood vessels due to infection) and the formation

of plaque inside

> blood vessel wall surfaces.

> How Mycoplasmas Operate

> Mycoplasmas are well equipped to play biological sleight-of-hand,

appearing

> then disappearing, changing shape, shuffling their surface

components, ducking

> into cells, then parading as normal citizens of the human flora

dressed in

> clothes stolen from the cells they invaded. They’re elusive

because they are

> pleomorphic (structurally changing). They do not have rigid cell

walls like

> most bacteria; instead they possess fluid lipid (water insoluble

fate) outer

> surfaces, and like tiny jellyfish, they can squeeze, bend and move

into tight

> spaces. They can also slide right through laboratory and hospital

filters used

> to produce or maintain sterility †" making them one of the most

common

> contaminants in diagnostic laboratories and vaccine manufacturing.

In one recent

> study of vaccines, mycoplasmas were found to contaminate about six

percent of

> commercial vaccines.

> These microorganisms have been quite successful in adapting to

many

> environments, infecting everything from insects to elephants,

plants to people.

> Generally, they are species-specific, but there appear to be many

exceptions.

> Garth Nicolson relates more than one case in which the pets of GWI

or CFS

> patients were exhibiting similar symptoms as their owners, and then

tested positive

> for the same mycoplasmas. No one knows for sure how contagious

mycoplasmas

> are, but it appears transmission may occur among infected people

in close

> proximity for extended periods of time.

> Not everyone who is exposed becomes sick. For example, when

Nicolson studied

> Gulf War veterans’ families who became sick with symptoms similar

to GWI, he

> found that not every member of the family became sick, but those

that did

> become ill had the same infection as found in the sick veteran.

> Detecting Mycoplasmas

> When the Nicolsons began to explore the connection between GWI and

> mycoplasma, they first had to figure out how to screen people with

GWI signs and

> symptoms for the presence of these pathogens. This was easier said

than done.

> Since mycoplasmas are extremely small, change shape and lack rigid

and

> distinctive cell walls, they’re impossible to find using

conventional microbiology

> techniques. They won’t grow in a standard culture medium, and

they are not

> usually revealed by standard tests that look for antibodies

(proteins made by a

> white blood cell as a primary defense against foreign substances).

Some people

> do show antibody responses to certain mycoplasmas, but antibody

tests are

> still not specific enough to make a diagnosis.

> Using a technique called nucleoprotein gene tracking developed by

the

> Nicolsons, they were able to identify mycoplasma genetic elements

in white blood

> cells of GWI patients. However, conventional Polymerase Chain

Reaction (PCR0

> tests performed by Army pathologists did not confirm the presence

of mycoplasma

> DNA.

> Eventually, the Nicolsons developed a new PCR test based on

techniques used

> by forensic pathologists to test for DNA from crime scenes. This

test revealed

> that over 40% of the GWI patients were positive for “invasiveâ€

mycoplasma

> (not mycoplasma in superficial sites such as nose, throat and

genitourinary

> tract).

> The Nicolsons found mycoplasmas, especially M. fermentans, inside

tissues and

> in certain white blood cells †" the very cells that are normally

involved in

> the destruction of pathogenic invaders. “Mycoplasmas are not

found

> systemically in most normal subjects †" only a few percent of

asymptomatic subjects have

> evidence of mycoplasma in their blood. I don’t consider oral

mycoplasma, or

> mycoplasma at other superficial sites to be evidence of an

infection. It is

> more likely simple bacterial colonization, and unless these

mycoplasma invade

> the epithelial cell layer (a thin layer of tissue that covers a

surface or

> lines a cavity), they are probably benign nonpathogenic

residents,’ explains

> Nicolson.

> The researchers’ results were significant and published in

several journals.

> Other investigators, especially those working with Gulf War Vets,

were able

> to duplicate the results, but the Nicolson’s work was largely

dismissed or

> ignored by the Department of Defense. However, in February, 2000,

psychiatrist

> Lt. Col. Engel, M.D., director of the Gulf War Illness

Center at

> Walter Army Medical Center, presented pivotal information to

a CFS

> coordinating board at the National Institutes of Health. A study

conducted

> independently for the U.S. Departments of Defense and Veterans

Affairs demonstrated that

> approximately 40% of more than 1,600 GWI patients were positive

for

> mycoplasma infections, and 80% of those were positive for M.

fermentans. Lt. Col.

> Engel also stated that he felt that these infections might also be

an important

> cause of CFS. The study findings nearly duplicated the figures

that the

> Nicolsons had reported earlier: 45% positive for mycoplasma; 80%

with M.

> fermentans.

> Currently, other prominent researchers are corroborating the role

of

> mycoplasma in disease. The number of known conditions in which

mycoplasmas play a

> role is growing, thanks to advances in detection. Mycoplasmas are

now said to

> be contributors, or at least cofactors, in a number of conditions,

including

> CFS/CFIDS, fibromyalgia syndrome (FMS), lupus, multiple sclerosis

(MS),

> psoriasis, scleroderma, Chrohn’s diseases, solid cancers,

leukemia, lymphoma,

> Amyotrophic Lateral Sclerosis (ALS), pelvic inflammatory disease

(PID), asthma,

> atypical pneumonia, Sjogren’s syndrome, interstitial cystitis,

Alzheimer’s and

> cardiovascular diseases. Mycoplasmas have also been associated with

a variety

> if autoimmune diseases that can cause definite changes in nerve

conduction,

> demyelation (a degenerative process that erodes away the myelin

sheath that

> normally protects nerve fibers) and sensitivity.

> Dr. Nicolson says that the role of mycoplasmas in various illnesses

and

> diseases is now gradually being accepted, especially in those once

long-suspected

> as “psychological.†Acceptance is due to the recognition that

symptoms

> cannot be explained solely by psychological criteria, and because

discrete

> clinical markers have been discovered. For example, the

vascultitis (inflammation

> of blood vessels) found in mycoplasma-positive patients correlates

with

> evidence of mycoplasma-induced abnormalities in blood cells and

proteins related to

> blood clotting.

> Current Mycoplasma Research

> Recently, Dr. Nicolson has focused on various autoimmune

neurological

> diseases such as ALS, MS, Lyme disease and others. For example,

approximately 85%

> of patients with ALS (“Lou Gehrig’s diseaseâ€) tested positive

for systemic

> mycoplasma infections, and most of those infections involve M.

fermentans and/or

> M. hominis.

> Dr. Nicolson is working closely with Drs. See and Akbarpour on ALS,

a

> condition in which patients lose control of their motor and

skeletal muscles over a

> period of two to five years. Their research revealed that almost

all ALS

> patients have co-infections with a virus from the enterovirus

family (a virus

> related to the polio virus that replicated mostly in the

gastrointestinal tract)

> †" and mycoplasmas. The three doctors have been conducting a

clinical

> treatment study of ALS utilizing antibiotics, antivirals and nerve

growth factors.

> They are seeing positive results so far, as measured by increases

in muscle

> strength.

> Other illnesses often have multiple strains of mycoplasmas, or

mycoplasmas

> combined with co-infections of other bacteria or viruses. “In

recent published

> studies from our laboratory, most CFS and FMS patients had multiple

> mycoplasmal infections. The number of different mycoplasmal

species in these patients

> increased with the number of years the patients were sick and with

the

> severity of their illness,†says Dr. Nicolson.

> “We have found that when the few asymptomatic subjects have blood

mycoplasmal

> infections, they have only one species, versus when we examine

patients who

> are sick with various chronic illnesses, they usually have

multiple species

> of mycoplasmas and other infections such as the cytomegalovirus.

In Lyme

> disease, we often find mycoplasmal co-infections, most frequently,

M. fermentans,

> along with the Borrelia that causes it. This makes sense when you

consider

> that insects, such as the ticks that carry the Borrelia, also can

carry

> mycoplasmas. Dr. Eli Mordechai of Medical Diagnostics Lab of New

Jersey has exactly

> the same findings in Lyme disease patients.â€

> All the researchers above agree that long-term antibiotics must be

initiated

> to treat mycoplasmal infections. Additional strategies must be

applied to

> protect and strengthen the immune system and provide essential

nutrients and

> vitamins.

> “We always try to use the least toxic approaches in working with

pathologies,

> so we use a lot of natural products,†Dr. See says. For example,

probiotics

> and undenatured whey protein isolates are used to support the GI

tract †" a

> combination that helps prevent overgrowth of undesirable

microorganisms. “

> However,†adds Dr. See, “in our experience, and in the

literature, we have found

> non other way to deal with mycoplasmas than fairly long-term

treatment with

> certain antibiotics.†Fortunately, the Nicolsons and their

colleagues have

> succeeded in helping many veterans and others infected with

mycoplasmas, but

> controversies surrounding their work and these mysterious

microorganisms still

> persist.

> Says Dr. Nicolson, “Future efforts to explain and treat a variety

of

> illnesses that currently have unknown etiologies (causes) will

undoubtedly focus

> more on chronic infections as underlying causes or as opportunistic

infections

> in immune-impaired patients. We have found that chronic infections

caused by

> mycoplasmas, viruses and other microorganisms cannot be ignored,

because these

> patients will remain ill and not recover from their illnesses if

these

> infections remain untreated.

> Resources

> Additional information on mycoplasma treatment, yeast overgrowth,

nutrition,

> and treating multiple infections associated with mycoplasmas can be

found on

> the Institute for Molecular Medicine’s Web site: www.immed.org

> _http://www.immed.org/signsympt.htm_

(http://www.immed.org/signsympt.htm)

> this is very interesting ! ! ! ! ! ! ! !

> _http://www.immed.org/index.htm_ (http://www.immed.org/index.htm)

> For further Information on diseases and syndromes that could have

an

> important infectious component, such as:

> Fatiguing Illnesses

> Autoimmune Diseases

> Neurodegenerative Diseases

> Chronic Fatigue Syndrome

> Myalgic Encephalomyelitis

> Fibromyalgia Syndrome

> Gulf War Illnesses

> Other Illnesses:

> Chronic Asthma

> Chronic Bronchitis

> Chronic Pneumonia

> Vascular inflammation

> Urogenital diseases

> HIV

> Heart Diseases

> Rheumatoid Arthritis

> Scleroderma

> Sarcoidosis

> Ankylosing Spondylitis

> Graves’ Disease

> Hashimoto’s Disease

> Inflammatory Bowel Diseases (IBD)

> Lupus (SLE)

> Crohn’s Disease

> Reiter’s Disease

> Amyotrophic Lateral Sclerosis (ALS)Multiple Sclerosis (MS)

> Alzheimer’s Disease (AD)

> Parkinson’s Disease (PD)

> Peripheral Neuropathies

> Other Neurological Disorders:

> Autistic Spectrum Disorders

> Autism

> ADHD, ADD

> Asperger Syndrome

> Stroke

>

>

>

> **************Start the year off right. Easy ways to stay in

shape.

> http://body.aol.com/fitness/winter-exercise?

NCID=aolcmp00300000002489

>