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anyone with enlarged lymph nodes w/ saline implants?cance...

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Hello a ~

Welcome to the group ! I am so glad you found

us, as we have loads of information that can be helpful for you as well as a large group of women that are so

kind, and loving and supportive.

If you look in the archives, you will see a photo section, and if you look in those photo albums, you will see pictures of breasts with rashes on them. This is very common in people with breast implants.

Swollen lymph nodes are very common too. All implants are made out of silicone, and there are many toxic chemicals that they use to make the silicone, I will put a list at the end of this note to you. The insides of the implants can be different, there can be cohesive silicone or gel silicone or saline filled implants. I think at times they have tried other things in them too.

Anyway, your body has natural defenses. One of them is macrophages, and the macrophages will go try to protect you by fighting the foreign material and degrade it, and it will fleck off and go through your blood stream, and all your filtering systems, like lymph nodes, and others.

Here is the list of ingredients used in silicone, they are all known neuro-toxins ! :

1)Methyl Ethyl Ketone

2)Cyclobexanone

3)Isopropyl Alcohol

4)Denatured Alcohol

5)Acetone

6)Urethane

7)Poly vinyl Chloride

8)Lacquer Thinner

9)Ethyl Acetate

10)Epoxy Resin

11)Epoxy Hardener

12)Amine

13)Printing Ink

14)Toluene

15)Freon

16)Silica

17)Flux

18)Solder

]19)Chlorplatinic Acid

20)Metal Cleaning Acid

21)Formaldehyde

22)Talcum Powder

23)Color Pigmentation (Printers Ink)

24)Oakite

25)Cyanoacyrylates

26)Ethylene Oxide

27)Carob Black

28)Xylene

29)Hexone

30)Benzene

31)Hexanone 2

32)Thixon-OSN-2

33)Rubber

34)Acid Stearic

35)Zinc Oxide

36)Naptha

37)Phenol

38)Methylene Chloride

39)Platinum Salt

Your body is tired of dealing with all those toxins.

The debris that comes off the silicone shell and goes around in your body can form cystic masses in there and even get stuck in your lymph nodes and become cystic. This is very common, here are a couple medical articles discussing the issues I just described to you.

You might want to print this out and take with you to appointments

http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=117237

(To read the entire study)

The initial body's reaction to the implanted material is the inflammatory response that induces recruitment and activation of different cells [7]. The magnitude of any inflammatory response can be related to the level of activation of macrophages. This activation occurs both in inflammatory and in adaptive immune responses, and involves phenotypic and functional changes [8]. Criteria widely used for activation are the ability to inhibit intracellular proliferation of microorganisms, the increased production of reactive oxygen intermediates and the enhanced expression of MHC and co-stimulatory molecules [9,10]. Recently, Naim et al. showed that silicone elastomer preadsorbed with plasma proteins activated human monocytes in vitro to secrete pro-inflammatory cytokines [11]. Besides, silicone gels and oils activated macrophages in female A.SW mice: increased production of IL-6 and IL-1â was obtained from macrophages collected from silicone fluid- and silicone oil-treated mice when cultured with increasing amounts of lipopolysaccharide [12].

Activated macrophages exhibited an increased expression of adhesion (CD54 and CD44) and co-stimulatory (CD86) molecules and an enhanced production of oxidant metabolites and NO.

Look at the SMALL AMOUNT used in the study, compared to the surface and content of breast implants...YIKES! No wonder our immune systems become overwhelmed.

Figure 1 Silicone induces differential recruitment of leukocytes. Rat peritoneal cells (n = 4/group) were obtained after 45 days, 48 h and 24 h of the i.p. injection of 1 ml of silicone. Differential cell counting was assessed by microscopic observation of cytospin(more ...)

In silicone breast implants, chronic inflammation seems to be the most relevant process with accumulations of lymphocytes and monocytes [16]. Even if a silicone gel filled breast implant does not rupture, small amounts of low molecular weight fluid DMPS may permeate (bleed or sweat) out of the implant into the surrounding tissue [17]. Hydrophobic materials such as silicone do not migrate well and are coated instead with host proteins [18], and within one hour elastomers are at least 70 % covered with host proteins [19]. Apparently, recruited inflammatory cells do not respond to DMPS itself but to adsorbed, partially denatured plasma proteins such as IgG, albumin, fibronectin and complement components [20]. It has been suggested that liposome-like structures can be formed within the body of an implant, involving water-soluble and hydrophobic constituents [5]. When administered in vivo liposomes interact almost exclusively with the mononuclear phagocytic system [21] and in an i.p. injection resident macrophages take up liposomes in large quantities and monocytes can be recruited from the general circulation [22,23]. This could account for the persistent stimulation that we found in our study, after several days of DMPS in the peritoneal cavity. Perhaps the association of vesicular and lipoidal structures with host proteins could facilitate this strong stimulatory capacity. However the activity of "naked" DMPS should also be considered. Previous work support the stimulatory capacity of silicone in vitro without the influence of plasma proteins: when peritoneal macrophages are cultured on silicone-coated plates, their cytotoxic activity on cancer cells is markedly augmented and the activity of antigen presenting cell is enhanced [24].

The activation of macrophages is an important event involved in inflammation, T cell activation and adjuvanticity. Adhesion and co-stimulatory molecules are up-regulated with key implications on antigen presentation and T cell priming [8]. In rats, strong stimulatory capacity for primary immune response is associated with the expression of the co-stimulatory ligands CD80 and CD86 [25]. After DMPS injection in peritoneal macrophages the MFI for CD54, CD44 and CD86 increased significantly in all injected rats (24 h, 48 h and 45 days after injection) providing evidences of cellular activation status.

After activation, cytokines, reactive oxygen intermediates and NO that belong to the molecular repertoire of activated macrophages are up-regulated [8]. The cytokine stimulating capacity of silicone has been already demonstrated [26]. Macrophages layered on DMPS and silicone rubber with or without protein adsorption produce variable levels of IL-1â, IL-6 and TNF-á depending on the polymer and adsorbed protein [12]. Moreover, chronic loading of macrophages with silicone particles derived of dialysis tubing results in augmented release of IL-1 [26].

I would say the best thing would be to get them out. It is very important

to have them removed properly, which is: enbloc removal where they cut around the scar capsule and remove the implant with the scar capsule still around the implant so that any implant debris will still be contained inside there. then use of drain tubes to keep the swelling down, and carry off debris that may be there as well. You want them to try their best not to break the implant just in case they are full of bacteria and mold and fungus, you can also see photos of saline implants that are red brown and black and speckeld and full of mold and bacteria.

The good thing is you can get better ! with proper removal and detoxing and eating healthy, you can get your health back. It takes patients and time, and then more patients, but it can be done.

WE are all here for you ! Please read in the archives and also look at the photos, This will help you some and get you more familiar.

Hugs and Prayers ~

Dede

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