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Hey,

Need advice on whether to try Armour or go for straight T3??

Just got some more results back from my doctor. I have been on 30mg of HC for a

month because my saliva labs came back pretty low. However, i haven't felt any

better at all from taking it. Today i got back my urine results and the cortisol

level was optimal along with Aldosterone. Also, my blood cortisol levels have

always been right at the top of the range/borderline high. So my doctor has told

me to stop the HC.

He has prescribes me Armour and DHEA.

My thyroid labs are as follows:

Free t3 5.3 (3.5-6.5)

Free t4 18.1 (10-19)

TSH 2.44

I have poor circulation, can't sweat, brain fog - heaps of hypoT symptoms. My

Urine test confirmed reasonably low t3 along with very low Growth hormone.. i am

only 21.

Any thoughts on if i should try the Armour or avoid this? I have just sent away

for Iron and RT3 so that won't be back for a week or two i would imagine.

Todd

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First you cannto get accurate cortisol testing while ON HC. Your doctor

is dangerous tellign you to stop HC for this reason. Stable temps when

done wiht proper proceedure means you are stable ON THAT DOSE OF HC it

does nto mean you are ready to wean off HC and HC should ALWAYS be

weaned not just stopped. Stopping it from 25 mg a dya could kill you.

Next, your thyroid labs show one of two things, HYPERTHYROID or thyrodi

that is pooling due to low cortisol. The high T4 would indicate to me

that you DO have a reverse T3 problem but with your T4 that high and

still a high TSH, I am suspicious of a pituitary problem an think a pit

scan shoudl be done to see if there is a producing tumor. This may be

overkill , but high TSH is NOT normal wiht high RT3 or wiht high FT3 and

FT4 and yours are near top of range. Takign more thyroid of any kind is

not likely to help this until you find out what ELSE is going on. Either

severely low cortiosl STILL meaning you may need more HC thn the 25mg

you are on, or a pit tumor producingf excess TSH.

--

http://nthadrenalsweb.org/

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

http://faqhelp.webs.com/

http://health.groups.yahoo.com/group/RT3_T3/

http://www.thyroid-rt3.com/

http://groups.yahoo.com/group/HypoPets/

http://www.stopthethyroidmadness.com/

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Sorry i pasted the wrong one, these are some highlighted sections.

With Type 2 Hypothyroidism, the thyroid gland produces " normal " amounts of

hormone, but the cells are unable to utilize the hormone properly. Some experts

call this thyroid hormone resistance (which may be regarded as similar to

insulin resistance).

drawn heavily from Mark Starr's book, Hypothyroidism Type 2: The

Epidemic.

Faulty thyroid receptors on the cell membranes as well as mitochondrial

mutations can cause a hypothyroid condition. " Defective thyroid receptors, "

Starr writes, " may prevent a sufficient supply of hormones that are circulating

in our blood from reaching the mitochondria and other crucial sites such as the

nucleus of the cell. The nucleus is where the thyroid hormones activate genes

and stimulate protein synthesis, among a host of other tasks. " [emphasis added]

The beneficial symbiotic relationship between mitochondria and thyroid hormone

works both ways. Adequate levels of thyroid hormone not only increase

mitochondria number and function, but as Starr points out, " mitochondrial

mutations appear to be largely responsible for the metabolic defects at the

cellular level, which result in a hypothyroid-like condition. . . . Defects in

mitochondria, as well as synthetic toxins, impair thyroid hormone metabolism at

the cellular level. " 2 Not surprisingly,

Inadequate thyroid hormone at the cellular level also negatively impacts other

glands. " Without the crucial influence of thyroid hormones, " Starr emphasizes,

" proper maturation and function of the other hormone glands is not possible. " 4

To compensate for the weakness and low metabolism caused by inadequate thyroid

hormone, other parts of the body overwork, including the adrenals and the

sympathetic nervous system. This may cause the subject to temporarily experience

a rapid heartbeat, and/or feel hyperactive, jittery and restless—until

exhaustion sets in from the unnatural attempts to compensate for low thyroid

hormone levels. More often, though, the majority of sufferers simply feel

fatigued and weak most of the time.

The problem with this scenario is that most of the time, the mitochondria in

toxic and defective cells are unable to convey to the brain their need for

thyroid hormone, even if it's urgently required. In fact, according to numerous

studies, people whose mitochondria tested abnormal nonetheless had normal

thyroid hormone levels in their blood. Modern thyroid blood tests, Starr reminds

us, do not detect Type 2 hypothyroidism " because thyroid hormone levels [in the

bloodstream] may be normal, but they are not high enough to stimulate the . . .

defective mitochondria into normal activity. " [emphasis added] 7 Nor are the

blood thyroid hormone levels high enough to induce the resistant receptor sites

on the cells to start accepting hormone. Any part of the cell can be involved in

the failure to process and utilize thyroid hormone.

Treatment for Type 2 Hypothyroidism

1. Replacement Hormone

Whether the person's thyroid gland is not producing enough hormone, or the cells

are unable (for whatever reason) to process what the gland is producing, the

treatment is the same: replacement hormone.

From the perspective of conventional medical training, flooding the system with

thyroid hormone, in amounts greater than what laboratory blood tests might

indicate are useful or prudent, may seem questionable. But consider the highly

dysfunctional state of the mitochondria and/or cell receptors. If you saturate

the tissues with enough hormone, and for a long enough period, even

malfunctioning mitochondria and stubborn receptor sites will start processing

and utilizing the hormone. Once the body begins to function correctly, it has

the potential to self-correct. Then, conceivably, the hormone dosage can be

reduced. This points to the need for careful monitoring of people with Type 2

hypothyroidism.

http://www.nenahsylver.com/files/nenah_sylver_phd_-__hypothyroidism_type_2.pdf

>

> That article points to a site abotu DHEA, nto thyroid resistance, I

> haven't time ot go through it all can you copy and paste any relevant

> material?

>

> --

>

> http://nthadrenalsweb.org/

>

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

> http://faqhelp.webs.com/

>

> http://health.groups.yahoo.com/group/RT3_T3/

> http://www.thyroid-rt3.com/

>

> http://groups.yahoo.com/group/HypoPets/

> http://www.stopthethyroidmadness.com/

>

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Sorry i pasted the wrong one, these are some highlighted sections.

With Type 2 Hypothyroidism, the thyroid gland produces " normal " amounts of

hormone, but the cells are unable to utilize the hormone properly. Some experts

call this thyroid hormone resistance (which may be regarded as similar to

insulin resistance).

drawn heavily from Mark Starr's book, Hypothyroidism Type 2: The

Epidemic.

Faulty thyroid receptors on the cell membranes as well as mitochondrial

mutations can cause a hypothyroid condition. " Defective thyroid receptors, "

Starr writes, " may prevent a sufficient supply of hormones that are circulating

in our blood from reaching the mitochondria and other crucial sites such as the

nucleus of the cell. The nucleus is where the thyroid hormones activate genes

and stimulate protein synthesis, among a host of other tasks. " [emphasis added]

The beneficial symbiotic relationship between mitochondria and thyroid hormone

works both ways. Adequate levels of thyroid hormone not only increase

mitochondria number and function, but as Starr points out, " mitochondrial

mutations appear to be largely responsible for the metabolic defects at the

cellular level, which result in a hypothyroid-like condition. . . . Defects in

mitochondria, as well as synthetic toxins, impair thyroid hormone metabolism at

the cellular level. " 2 Not surprisingly,

Inadequate thyroid hormone at the cellular level also negatively impacts other

glands. " Without the crucial influence of thyroid hormones, " Starr emphasizes,

" proper maturation and function of the other hormone glands is not possible. " 4

To compensate for the weakness and low metabolism caused by inadequate thyroid

hormone, other parts of the body overwork, including the adrenals and the

sympathetic nervous system. This may cause the subject to temporarily experience

a rapid heartbeat, and/or feel hyperactive, jittery and restless—until

exhaustion sets in from the unnatural attempts to compensate for low thyroid

hormone levels. More often, though, the majority of sufferers simply feel

fatigued and weak most of the time.

The problem with this scenario is that most of the time, the mitochondria in

toxic and defective cells are unable to convey to the brain their need for

thyroid hormone, even if it's urgently required. In fact, according to numerous

studies, people whose mitochondria tested abnormal nonetheless had normal

thyroid hormone levels in their blood. Modern thyroid blood tests, Starr reminds

us, do not detect Type 2 hypothyroidism " because thyroid hormone levels [in the

bloodstream] may be normal, but they are not high enough to stimulate the . . .

defective mitochondria into normal activity. " [emphasis added] 7 Nor are the

blood thyroid hormone levels high enough to induce the resistant receptor sites

on the cells to start accepting hormone. Any part of the cell can be involved in

the failure to process and utilize thyroid hormone.

Treatment for Type 2 Hypothyroidism

1. Replacement Hormone

Whether the person's thyroid gland is not producing enough hormone, or the cells

are unable (for whatever reason) to process what the gland is producing, the

treatment is the same: replacement hormone.

From the perspective of conventional medical training, flooding the system with

thyroid hormone, in amounts greater than what laboratory blood tests might

indicate are useful or prudent, may seem questionable. But consider the highly

dysfunctional state of the mitochondria and/or cell receptors. If you saturate

the tissues with enough hormone, and for a long enough period, even

malfunctioning mitochondria and stubborn receptor sites will start processing

and utilizing the hormone. Once the body begins to function correctly, it has

the potential to self-correct. Then, conceivably, the hormone dosage can be

reduced. This points to the need for careful monitoring of people with Type 2

hypothyroidism.

http://www.nenahsylver.com/files/nenah_sylver_phd_-__hypothyroidism_type_2.pdf

>

> That article points to a site abotu DHEA, nto thyroid resistance, I

> haven't time ot go through it all can you copy and paste any relevant

> material?

>

> --

>

> http://nthadrenalsweb.org/

>

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

> http://faqhelp.webs.com/

>

> http://health.groups.yahoo.com/group/RT3_T3/

> http://www.thyroid-rt3.com/

>

> http://groups.yahoo.com/group/HypoPets/

> http://www.stopthethyroidmadness.com/

>

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High creatinine confirms high cortisol. From what I understand it is

caused by the breakdown of protein tissues in the body which is the

catabolixc effects of high cortiosl. Low thyrodi makes this worse but

yes I would start the adaptogens first and I woudl also start LOW on

DHEA as it cna convert to estrgen in men and when cortisol is bnot righ

tit tends ot do this more often. I woudl start no higher than 10mg a day

to see how you feel on it. Periods of FEELING hot cna be form high

cortils or from adrenaline.

--

http://nthadrenalsweb.org/

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

http://faqhelp.webs.com/

http://health.groups.yahoo.com/group/RT3_T3/

http://www.thyroid-rt3.com/

http://groups.yahoo.com/group/HypoPets/

http://www.stopthethyroidmadness.com/

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Share on other sites

Guest guest

High creatinine confirms high cortisol. From what I understand it is

caused by the breakdown of protein tissues in the body which is the

catabolixc effects of high cortiosl. Low thyrodi makes this worse but

yes I would start the adaptogens first and I woudl also start LOW on

DHEA as it cna convert to estrgen in men and when cortisol is bnot righ

tit tends ot do this more often. I woudl start no higher than 10mg a day

to see how you feel on it. Periods of FEELING hot cna be form high

cortils or from adrenaline.

--

http://nthadrenalsweb.org/

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

http://faqhelp.webs.com/

http://health.groups.yahoo.com/group/RT3_T3/

http://www.thyroid-rt3.com/

http://groups.yahoo.com/group/HypoPets/

http://www.stopthethyroidmadness.com/

Link to comment
Share on other sites

Guest guest

High creatinine confirms high cortisol. From what I understand it is

caused by the breakdown of protein tissues in the body which is the

catabolixc effects of high cortiosl. Low thyrodi makes this worse but

yes I would start the adaptogens first and I woudl also start LOW on

DHEA as it cna convert to estrgen in men and when cortisol is bnot righ

tit tends ot do this more often. I woudl start no higher than 10mg a day

to see how you feel on it. Periods of FEELING hot cna be form high

cortils or from adrenaline.

--

http://nthadrenalsweb.org/

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

http://faqhelp.webs.com/

http://health.groups.yahoo.com/group/RT3_T3/

http://www.thyroid-rt3.com/

http://groups.yahoo.com/group/HypoPets/

http://www.stopthethyroidmadness.com/

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Share on other sites

Guest guest

Thanks heaps Val,

So definitely shouldn't try the Armour? Just get going on adaptogens and DHEA

then bring in T3 only.

I keep being told to steer away from synthetics, is Cytomel the best way to go?

>

> High creatinine confirms high cortisol. From what I understand it is

> caused by the breakdown of protein tissues in the body which is the

> catabolixc effects of high cortiosl. Low thyrodi makes this worse but

> yes I would start the adaptogens first and I woudl also start LOW on

> DHEA as it cna convert to estrgen in men and when cortisol is bnot righ

> tit tends ot do this more often. I woudl start no higher than 10mg a day

> to see how you feel on it. Periods of FEELING hot cna be form high

> cortils or from adrenaline.

>

> --

>

> http://nthadrenalsweb.org/

>

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

> http://faqhelp.webs.com/

>

> http://health.groups.yahoo.com/group/RT3_T3/

> http://www.thyroid-rt3.com/

>

> http://groups.yahoo.com/group/HypoPets/

> http://www.stopthethyroidmadness.com/

>

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Share on other sites

Guest guest

Thanks heaps Val,

So definitely shouldn't try the Armour? Just get going on adaptogens and DHEA

then bring in T3 only.

I keep being told to steer away from synthetics, is Cytomel the best way to go?

>

> High creatinine confirms high cortisol. From what I understand it is

> caused by the breakdown of protein tissues in the body which is the

> catabolixc effects of high cortiosl. Low thyrodi makes this worse but

> yes I would start the adaptogens first and I woudl also start LOW on

> DHEA as it cna convert to estrgen in men and when cortisol is bnot righ

> tit tends ot do this more often. I woudl start no higher than 10mg a day

> to see how you feel on it. Periods of FEELING hot cna be form high

> cortils or from adrenaline.

>

> --

>

> http://nthadrenalsweb.org/

>

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

> http://faqhelp.webs.com/

>

> http://health.groups.yahoo.com/group/RT3_T3/

> http://www.thyroid-rt3.com/

>

> http://groups.yahoo.com/group/HypoPets/

> http://www.stopthethyroidmadness.com/

>

Link to comment
Share on other sites

Guest guest

Thanks heaps Val,

So definitely shouldn't try the Armour? Just get going on adaptogens and DHEA

then bring in T3 only.

I keep being told to steer away from synthetics, is Cytomel the best way to go?

>

> High creatinine confirms high cortisol. From what I understand it is

> caused by the breakdown of protein tissues in the body which is the

> catabolixc effects of high cortiosl. Low thyrodi makes this worse but

> yes I would start the adaptogens first and I woudl also start LOW on

> DHEA as it cna convert to estrgen in men and when cortisol is bnot righ

> tit tends ot do this more often. I woudl start no higher than 10mg a day

> to see how you feel on it. Periods of FEELING hot cna be form high

> cortils or from adrenaline.

>

> --

>

> http://nthadrenalsweb.org/

>

http://health.groups.yahoo.com/group/NaturalThyroidHormonesADRENALS/

> http://faqhelp.webs.com/

>

> http://health.groups.yahoo.com/group/RT3_T3/

> http://www.thyroid-rt3.com/

>

> http://groups.yahoo.com/group/HypoPets/

> http://www.stopthethyroidmadness.com/

>

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