Dr. Shaw's webinar last night

[Guest guest]

I missed this, rats, can anyone give me a short update?

cathy

[Guest guest]

It just now finished, he was talking about the affect of Tylenol during pregnancy,how it goes through the placenta and affect the liver.Also he  said how its important not to prevent fever with Tylenol.

They will be doing a study   go to www.GPL4U.com/study   and you may choose which study you want to participate.Tunde

 

I missed this, rats, can anyone give me a short update?

cathy

-- " Our lives begin to end the day we become silent about things that matter. " MLK

[Guest guest]

Yes. He's just finishing up now. It was about the role of acetaminophen in Autism. I'm not a biologist or chemist and I missed the first 3 or 4 minutes, but what he says made a whole lot of sense.In a QUICK nutshell: Pregnant ladies or sick or vaccinated baby takes acetaminophen -This produces a nasty metabolite (NAPQI) - which interferes with e-v-e-r-y-t-h-i-n-g. glutamylcesteine (sp?) will turn to pyroglutamic acid if a person is low in glycine. Becoming low in glycine usually happens when a person is sick and not eating (or dieting/fasting). We need the glutamylcesteine AND the glycine to produce glutathione....and we all know what happens when

we are deplete of glutathione. Acetaminophen is especially dangerous to people who already have issues with clostridia as it will block dopamine from turning to norepinephrin.High dopamine = ocd, repetative behaviors, and obsessive compulsion. It damages purkinji cells in the cerebellum. He explains that Too much acetaminophen will cause acidosis. It exasperates asthma. (There is a strong link between preg moms taking tylenol and young babies develop wheezing and asthma). Even at indicated doses, it inhibits the division of monocytes. Chelation may help to clean up NAPQI There was so much. It may be available for download in a month or two. In the mean time, keep away from it!They are going to launch three studies and are looking for participants. You can check it out here:www.GPL4U.com/study Poor guy just gave this great presentation and all I could manage was this lame interpretation. Sorry : ) With all the talk about high glutamate lately, it made me stop to wonder for a second if "high" glutamate is a problem or if it's actually low cysteine that makes glutemate look high? L-glutamate+cysteine=glutamylcesteine (I know I'm butchering that word!) andglutamylcesteine+glycine=glutathione.Anyone have any thoughts? To: mb12valtrex Sent: Wednesday, January 11, 2012 10:47 PM Subject: Dr. Shaw's webinar last night

I missed this, rats, can anyone give me a short update?

cathy

[Guest guest]

Oh! There will be a facebook page: acetaminophenandautism : ) -Tammy To: "mb12valtrex " <mb12valtrex > Sent: Wednesday,

January 11, 2012 11:25 PM Subject: Re: Dr. Shaw's webinar last night

Yes. He's just finishing up now. It was about the role of acetaminophen in Autism. I'm not a biologist or chemist and I missed the first 3 or 4 minutes, but what he says made a whole lot of sense.In a QUICK nutshell: Pregnant ladies or sick or vaccinated baby takes acetaminophen -This produces a nasty metabolite (NAPQI) - which interferes with e-v-e-r-y-t-h-i-n-g. glutamylcesteine (sp?) will turn to pyroglutamic acid if a person is low in glycine. Becoming low in glycine usually happens when a person is sick and not eating (or dieting/fasting). We need the glutamylcesteine AND the glycine to produce glutathione....and we all

know what happens when

we are deplete of glutathione. Acetaminophen is especially dangerous to people who already have issues with clostridia as it will block dopamine from turning to norepinephrin.High dopamine = ocd, repetative behaviors, and obsessive compulsion. It damages purkinji cells in the cerebellum. He explains that Too much acetaminophen will cause acidosis. It exasperates asthma. (There is a strong link between preg moms taking tylenol and young babies develop wheezing and asthma). Even at indicated doses, it inhibits the division of monocytes. Chelation may help to clean up NAPQI There was so much. It may be available for download in a month or two. In the mean time, keep away from it!They are going to launch three studies and are looking for participants. You can check it out here:www.GPL4U.com/study Poor guy just gave this great presentation and all I could manage was this lame interpretation. Sorry : ) With all the talk about high glutamate lately, it made me stop to wonder for a second if "high" glutamate is a problem or if it's actually low cysteine that makes glutemate look high? L-glutamate+cysteine=glutamylcesteine (I know I'm butchering that word!) andglutamylcesteine+glycine=glutathione.Anyone have any thoughts?

To: mb12valtrex Sent: Wednesday, January 11, 2012 10:47 PM Subject: Dr. Shaw's webinar last night

I missed this, rats, can anyone give me a short update?

cathy

[Guest guest]

He's doing a replay of the presentation on Friday, January 20th. It was good...I'll listen again. -Tammy To: "mb12valtrex " <mb12valtrex > Sent: Wednesday, January 11, 2012 11:25 PM Subject: Re: Dr. Shaw's webinar last night

Yes. He's just finishing up now. It was about the role of acetaminophen in Autism. I'm not a biologist or chemist and I missed the first 3 or 4 minutes, but what he says made a whole lot of sense.In a QUICK nutshell: Pregnant ladies or sick or vaccinated baby takes acetaminophen -This produces a nasty metabolite (NAPQI) - which interferes with e-v-e-r-y-t-h-i-n-g. glutamylcesteine (sp?) will turn to pyroglutamic acid if a person is low in glycine. Becoming low in glycine usually happens when a person is sick and not eating (or dieting/fasting). We need the glutamylcesteine AND the glycine to produce glutathione....and we all

know what happens when

we are deplete of glutathione. Acetaminophen is especially dangerous to people who already have issues with clostridia as it will block dopamine from turning to norepinephrin.High dopamine = ocd, repetative behaviors, and obsessive compulsion. It damages purkinji cells in the cerebellum. He explains that Too much acetaminophen will cause acidosis. It exasperates asthma. (There is a strong link between preg moms taking tylenol and young babies develop wheezing and asthma). Even at indicated doses, it inhibits the division of monocytes. Chelation may help to clean up NAPQI There was so much. It may be available for download in a month or two. In the mean time, keep away from it!They are going to launch three studies and are looking for participants. You can check it out here:www.GPL4U.com/study Poor guy just gave this great presentation and all I could manage was this lame interpretation. Sorry : ) With all the talk about high glutamate lately, it made me stop to wonder for a second if "high" glutamate is a problem or if it's actually low cysteine that makes glutemate look high? L-glutamate+cysteine=glutamylcesteine (I know I'm butchering that word!) andglutamylcesteine+glycine=glutathione.Anyone have any thoughts?

To: mb12valtrex Sent: Wednesday, January 11, 2012 10:47 PM Subject: Dr. Shaw's webinar last night

I missed this, rats, can anyone give me a short update?

cathy

[Guest guest]

The webinar focussed on the use of acetaminophen and its effect on the reduction of glutathione and other processes within the body. He talked about the detoxification pathways and how some are unable to excrete the acetaminophen through normal pathways which ultimately leads to liver damage/or the creation of an enzyme which interferes with detoxification. Use of acetaminophen while you were pregnant or in conjunction with vaccines before the age of 3 also appears to be an issue. He also discussed how the byproducts of acetaminophen (when not properly excreted) can lead to malfunction in synthesis of many proteins and creation of neurotransmitters as well as affecting binding sites of neurotransmitters and cannabinoid receptors. There was also a connection drawn to significantly less dendritic branching in the brain and areas of the brain like the corpus callosum (and two others which I can't recall the names of) that appeared to be affected by an adverse reaction to the acetaminophen.There is also some question about the strength and purity of many children's pain reliever's that were manufactured by and in some specific plants. Problems have been documented back as far as 2003, but may reach further back in time.Wish I had taken notes. I missed this, rats, can anyone give me a short update? cathy

[Guest guest]

roflmaaaaaaaoooooooooooooooooooo! ! Did you read your summary in comparison to mine????? Wow. I totally feel like a 3rd grader.Sad but true: I took notes. Maybe we should just leave this job to you from now on : ) -Tammy From:

To: mb12valtrex Sent: Wednesday, January 11, 2012 11:41 PM Subject: Re: Dr. Shaw's webinar last night

The webinar focussed on the use of acetaminophen and its effect on the reduction of glutathione and other processes within the body. He talked about the detoxification pathways and how some are unable to excrete the acetaminophen through normal pathways which ultimately leads to liver damage/or the creation of an enzyme which interferes with detoxification. Use of acetaminophen while you were pregnant or in conjunction with vaccines before the age of 3 also appears to be an issue. He also discussed how the byproducts of acetaminophen (when not properly excreted) can lead to malfunction in synthesis of many proteins and creation of neurotransmitters as well as affecting binding sites of neurotransmitters and cannabinoid receptors. There was also a connection drawn to significantly less dendritic branching in the brain and areas of the brain like the corpus callosum (and two others which I can't recall the

names of) that appeared to be affected by an adverse reaction to the acetaminophen.There is also some question about the strength and purity of many children's pain reliever's that were manufactured by and in some specific plants. Problems have been documented back as far as 2003, but may reach further back in time.Wish I had taken notes. I missed this, rats, can anyone give me a short update? cathy

[Guest guest]

Got me thinking that I had a cold in the third month of my pregnancy and the

only thing that I took was tylenol may be a total of 500 mg a day for a couple

of days. So if something does happen in utero, first trimester is the perfect

time for that. Plus my son had lot of ear infections and I would give him

tylenol all the time and he got all his vaccines. Then there are parents who say

that they have been through all of this and yet their children are healthy. So

may be I had some problem with my detoxification pathways even before I got

pregnant with my son. Just some thoughts.

is

>

> > I missed this, rats, can anyone give me a short update?

> > cathy

> >

> >

>

[Guest guest]

this gets me thinking, I have a kid low in pancretic enzymes, (could be one of those chicken and egg things BUT......) this makes him look like he is low in things all the time as he can't absorb nutrients right.....however none of that changed till vaccinations. we did the tylonal thing and vacinations with our son as we were TOLD TO by the drs. and even given a hand out on doing so before hand. dose 30 min before coming in ect. makes me sick looking back. I think a lot of us moms have clostrida when we give birth and don't know it, pass it to our kids and that I bet comes into play as well. oddly I have 2 girls with extream asthma, NEITHER has ever had tylonal after all we learned through our sons issues.

From: T Lynn

Sent: Wednesday, January 11, 2012 11:25 PM

To: mb12valtrex

Subject: Re: Dr. Shaw's webinar last night

Yes. He's just finishing up now. It was about the role of acetaminophen in Autism.

I'm not a biologist or chemist and I missed the first 3 or 4 minutes, but what he says made a whole lot of sense.

In a QUICK nutshell:

Pregnant ladies or sick or vaccinated baby takes acetaminophen -

This produces a nasty metabolite (NAPQI) - which interferes with e-v-e-r-y-t-h-i-n-g. glutamylcesteine (sp?) will turn to pyroglutamic acid if a person is low in glycine. Becoming low in glycine usually happens when a person is sick and not eating (or dieting/fasting). We need the glutamylcesteine AND the glycine to produce glutathione....and we all know what happens when we are deplete of glutathione. Acetaminophen is especially dangerous to people who already have issues with clostridia as it will block dopamine from turning to norepinephrin.High dopamine = ocd, repetative behaviors, and obsessive compulsion. It damages purkinji cells in the cerebellum. He explains that Too much acetaminophen will cause acidosis. It exasperates asthma. (There is a strong link between preg moms taking tylenol and young babies develop wheezing and asthma). Even at indicated doses, it inhibits the division of monocytes. Chelation may help to clean up NAPQI

There was so much. It may be available for download in a month or two. In the mean time, keep away from it!

They are going to launch three studies and are looking for participants. You can check it out here:

www.GPL4U.com/study

Poor guy just gave this great presentation and all I could manage was this lame interpretation. Sorry : )

With all the talk about high glutamate lately, it made me stop to wonder for a second if "high" glutamate is a problem or if it's actually low cysteine that makes glutemate look high?

L-glutamate+cysteine=glutamylcesteine (I know I'm butchering that word!)

and

glutamylcesteine+glycine=glutathione.

Anyone have any thoughts?

To: mb12valtrex Sent: Wednesday, January 11, 2012 10:47 PMSubject: Dr. Shaw's webinar last night

I missed this, rats, can anyone give me a short update?cathy

[Guest guest]

I also heard him say that the accumulation of ketones from low carb diets or

fasting will interfere with detox.

>

> > I missed this, rats, can anyone give me a short update?

> > cathy

> >

> >

>

[Guest guest]

But it is a pet peeve of mine that GPL always assumes everything is clostridia

and that many parents think their kids have clostridia based on an OAT test

showing a marker for anaerobic bacteria. Just because you have an anaerobe does

not mean you have clostridia. And GPL state in their reports that it is a

marker for clostridia but fails to mention OR any other anaerobe.

> >

> > > I missed this, rats, can anyone give me a short update?

> > > cathy

> > >

> > >

> >

>

[Guest guest]

He also mentioned a defect in the phenol sulfur pathway reduces the ability to

produce glutathione. He also said that vit an A reduces production of vitamin a

by inhibiting P450. I believe several meds do this as well, like diflucan and

rifampin.

What I did not hear was a particular recommendation to heal this. My healing

notes includied: avoid Tylenol; give B12 to help the body make cysteine;

supplement glycine; reduce clostridia and increase healthy gut flora; support

PST; avoid accumulation of ketones.

I think he just said increase cysteine and the use b12 was my own thought to

myself. Another of my own notes was support the liver with low frequent dose

ALA. Then attempt supplementation of glutathione once everythi else is n place.

Caryn

> >

> > > I missed this, rats, can anyone give me a short update?

> > > cathy

> > >

> > >

> >

>

[Guest guest]

My sons OAT test showed his to be off the charts high.Dr.Usmans

office said this is the highest they have ever seen.They said

clostridia.So you are saying it could be something else?What else

could it be?

Thank You Caryn if you have the time,Tammy F.

But it is a pet peeve of mine that GPL always assumes

everything is clostridia and that many parents think their

kids have clostridia based on an OAT test showing a marker

for anaerobic bacteria. Just because you have an anaerobe

does not mean you have clostridia. And GPL state in their

reports that it is a marker for clostridia but fails to

mention OR any other anaerobe.

> >

> > > I missed this, rats, can anyone give me a

short update?

> > > cathy

> > >

> > >

> >

>

[Guest guest]

>

> I missed this, rats, can anyone give me a short update?

> cathy

>

Wow, some really good info here!! I will definitley not miss the Jan 20

webinar.. thanks tons everyone!!

[Guest guest]

Hi caryn,Did I understand you that vit a supplementation is not good if there is a phenol,sulfur issue? Thanks,-- Sent from my Palm Pre

He also mentioned a defect in the phenol sulfur pathway reduces the ability to produce glutathione. He also said that vit an A reduces production of vitamin a by inhibiting P450. I believe several meds do this as well, like diflucan and rifampin.

What I did not hear was a particular recommendation to heal this. My healing notes includied: avoid Tylenol; give B12 to help the body make cysteine; supplement glycine; reduce clostridia and increase healthy gut flora; support PST; avoid accumulation of ketones.

I think he just said increase cysteine and the use b12 was my own thought to myself. Another of my own notes was support the liver with low frequent dose ALA. Then attempt supplementation of glutathione once everythi else is n place.

Caryn

> >

> > > I missed this, rats, can anyone give me a short update?

> > > cathy

> > >

> > >

> >

>

[Guest guest]

actually they do, the metabolite that is let OFF by clostrida is pinpointed, its not just about A bacteria. but speicific ones. which is why there are SEVERAL markers for the possiblity of yeast and bacteria. they each mean something.

From: Tammy

Sent: Thursday, January 12, 2012 11:00 AM

To: mb12valtrex

Subject: Re: Re: Dr. Shaw's webinar last night

My sons OAT test showed his to be off the charts high.Dr.Usmans office said this is the highest they have ever seen.They said clostridia.So you are saying it could be something else?What else could it be? Thank You Caryn if you have the time,Tammy F.

But it is a pet peeve of mine that GPL always assumes everything is clostridia and that many parents think their kids have clostridia based on an OAT test showing a marker for anaerobic bacteria. Just because you have an anaerobe does not mean you have clostridia. And GPL state in their reports that it is a marker for clostridia but fails to mention OR any other anaerobe.> > > > > I missed this, rats, can anyone give me a short update?> > > cathy> > >> > >> >>

[Guest guest]

That left me very confused, too. -Tammy To: "mb12valtrex " <mb12valtrex > Sent: Thursday, January 12, 2012 12:26 PM Subject: Re: Re: Dr. Shaw's webinar last night

Hi caryn,Did I understand you that vit a supplementation is not good if there is a phenol,sulfur issue? Thanks,-- Sent from my Palm Pre

He also mentioned a defect in the phenol sulfur pathway reduces the ability to produce glutathione. He also said that vit an A reduces production of vitamin a by inhibiting P450. I believe several meds do this as well, like diflucan and rifampin.

What I did not hear was a particular recommendation to heal this. My healing notes includied: avoid Tylenol; give B12 to help the body make cysteine; supplement glycine; reduce clostridia and increase healthy gut flora; support PST; avoid accumulation of ketones.

I think he just said increase cysteine and the use b12 was my own thought to myself. Another of my own notes was support the liver with low frequent dose ALA. Then attempt supplementation of glutathione once everythi else is n place.

Caryn

> >

> > > I missed this, rats, can anyone give me a short update?

> > > cathy

> > >

> > >

> >

>

[Guest guest]

Actually I was impressed with your write up Tammy. :-) You had many of the key points with far fewer words. I tend to run on when I start talking. LOL I think we would make a good team. !!! :-)I'll be listening again on Friday too, and I think this time I will take notes. roflmaaaaaaaoooooooooooooooooooo! ! Did you read your summary in comparison to mine????? Wow. I totally feel like a 3rd grader.Sad but true: I took notes. Maybe we should just leave this job to you from now on : ) -Tammy To: mb12valtrex Sent: Wednesday, January 11, 2012 11:41 PM Subject: Re: Dr. Shaw's webinar last night The webinar focussed on the use of acetaminophen and its effect on the reduction of glutathione and other processes within the body. He talked about the detoxification pathways and how some are unable to excrete the acetaminophen through normal pathways which ultimately leads to liver damage/or the creation of an enzyme which interferes with detoxification. Use of acetaminophen while you were pregnant or in conjunction with vaccines before the age of 3 also appears to be an issue. He also discussed how the byproducts of acetaminophen (when not properly excreted) can lead to malfunction in synthesis of many proteins and creation of neurotransmitters as well as affecting binding sites of neurotransmitters and cannabinoid receptors. There was also a connection drawn to significantly less dendritic branching in the brain and areas of the brain like the corpus callosum (and two others which I can't recall the names of) that appeared to be affected by an adverse reaction to the acetaminophen.There is also some question about the strength and purity of many children's pain reliever's that were manufactured by and in some specific plants. Problems have been documented back as far as 2003, but may reach further back in time.Wish I had taken notes. I missed this, rats, can anyone give me a short update? cathy

[Guest guest]

some of that would line up with yasko research

From: T Lynn

Sent: Thursday, January 12, 2012 3:13 PM

To: mb12valtrex

Subject: Re: Re: Dr. Shaw's webinar last night

That left me very confused, too.

-Tammy

To: "mb12valtrex " <mb12valtrex > Sent: Thursday, January 12, 2012 12:26 PMSubject: Re: Re: Dr. Shaw's webinar last night

Hi caryn,Did I understand you that vit a supplementation is not good if there is a phenol,sulfur issue? Thanks,

-- Sent from my Palm Pre

He also mentioned a defect in the phenol sulfur pathway reduces the ability to produce glutathione. He also said that vit an A reduces production of vitamin a by inhibiting P450. I believe several meds do this as well, like diflucan and rifampin.What I did not hear was a particular recommendation to heal this. My healing notes includied: avoid Tylenol; give B12 to help the body make cysteine; supplement glycine; reduce clostridia and increase healthy gut flora; support PST; avoid accumulation of ketones.I think he just said increase cysteine and the use b12 was my own thought to myself. Another of my own notes was support the liver with low frequent dose ALA. Then attempt supplementation of glutathione once everythi else is n place.Caryn> > > > > I missed this, rats, can anyone give me a short update?> > > cathy> > >> > >> >>

[Guest guest]

Yes Tammy. The OAT can show anaerobic bacteria markers. The OAT does NOT

detect specifically clostridia. It is DANs and GPL that say it is clostridia.

Without a positive clostridia stool analysis or antibody test, you should not

assume it is clostridia.

> > > >

> > > > > I missed this, rats, can anyone give me a short update?

> > > > > cathy

> > > > >

> > > > >

> > > >

> > >

> >

> >

>

[Guest guest]

Unfortunately I don't remember specifically what he said about vitamin A's

effect on the detox. Maybe someone else can post after they hear the next call.

> > >

> > > > I missed this, rats, can anyone give me a short update?

> > > > cathy

> > > >

> > > >

> > >

> >

>