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Re: Re: VIOXX-Liane

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This is interesting.. I don't try and keep up with all the latest info on

medications outside my specialty area. However, I recall that both Vioxx

and Celebrex are COX II inhibitors, the newest class of NSAIDs. Neither is

supposed to inhibit the COX I pathway- which is the cause of the risk to the

stomach lining by other NSAIDs.

In all my professional drug info programs Vioxx and Celebrex have the same

warnings, " Due to rofecoxib's (or celecoxib's) specificity for the COX-2

cyclooxygenase pathway, it has the potential to cause less gastropathy and

risk of GI bleeding; however, severe gastrointestinal adverse events have

occurred in patients receiving rofecoxib (or celecoxib).

If there is new research out there or solid clinical experience indicating

that Celebrex/celexoxib is easier on the stomach than Vioxx/rofecoxib, I'd

really like to know that and take another look at which my patients are

having prescribed by other providers. Sounds like you have info I need,

Liane.

in Seattle

Re: VIOXX

> >

> :::::::::::waving arms:::::::::::::::

> I know!

> Celebrex predisposes your stomach to ulcers and is not recommended for

> those w/a history of bleeding ulcers, GI bleeds, Stomach surgery, etc.

>

> Hugs,

> Liane

>

> >

> > Before surgery I had been taking Celebrex or Vioxx and Dr. told

> me

> > to take Vioxx. I believe it easier on the stomach (can't remember

> the

> > reason right now).

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Thanks, Liane. I'm on a psychopharmacology mail list and find that clinical

experience often precedes journal articles and double blind studies by

years. So the anecdotal reports of today may well be the research supported

findings in time. I'll look tomorrow and see if both are on our hospital

formulary. And will pay attention to which is being rx'd to my patients by

their PCP. Appreciate your info... you wave your hand well

thanks,

in Seattle

----- Original Message -----

> --- In duodenalswitch@y..., > -

> Ummm... No, not really. I know only that I worked in geriatrics for

> a while, and all the docs switched from celebrex to Vioxx. The one

> patient that was left on Celebrex was also on Coumadin for a history

> of A Fib.. and he had a serious GI bleed after being on Celebrex only

> about a month. The Doctors all decided it was due to a combination of

> things: the celebrex, the coumadin, and the fact that this man ate

> very little (and DS-ers would have 2 out of 3 of those..). I asked

> about the switch when it happened, & the answer I was given was that

> Vioxx seemed to have fewer problems with GI bleeding as compared to

> Celebrex (in the elderly). There's no definitive proof yet, just

> anecdotal information from personal experiences. I got the same answer

> from 4 Doctors & one NP, so I decided that it was what they were

> seeing personally. Sorry to have gotten your hopes up.

> Hugs,

> Liane

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Thanks, Liane. I'm on a psychopharmacology mail list and find that clinical

experience often precedes journal articles and double blind studies by

years. So the anecdotal reports of today may well be the research supported

findings in time. I'll look tomorrow and see if both are on our hospital

formulary. And will pay attention to which is being rx'd to my patients by

their PCP. Appreciate your info... you wave your hand well

thanks,

in Seattle

----- Original Message -----

> --- In duodenalswitch@y..., > -

> Ummm... No, not really. I know only that I worked in geriatrics for

> a while, and all the docs switched from celebrex to Vioxx. The one

> patient that was left on Celebrex was also on Coumadin for a history

> of A Fib.. and he had a serious GI bleed after being on Celebrex only

> about a month. The Doctors all decided it was due to a combination of

> things: the celebrex, the coumadin, and the fact that this man ate

> very little (and DS-ers would have 2 out of 3 of those..). I asked

> about the switch when it happened, & the answer I was given was that

> Vioxx seemed to have fewer problems with GI bleeding as compared to

> Celebrex (in the elderly). There's no definitive proof yet, just

> anecdotal information from personal experiences. I got the same answer

> from 4 Doctors & one NP, so I decided that it was what they were

> seeing personally. Sorry to have gotten your hopes up.

> Hugs,

> Liane

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Guest guest

Thanks, Liane. I'm on a psychopharmacology mail list and find that clinical

experience often precedes journal articles and double blind studies by

years. So the anecdotal reports of today may well be the research supported

findings in time. I'll look tomorrow and see if both are on our hospital

formulary. And will pay attention to which is being rx'd to my patients by

their PCP. Appreciate your info... you wave your hand well

thanks,

in Seattle

----- Original Message -----

> --- In duodenalswitch@y..., > -

> Ummm... No, not really. I know only that I worked in geriatrics for

> a while, and all the docs switched from celebrex to Vioxx. The one

> patient that was left on Celebrex was also on Coumadin for a history

> of A Fib.. and he had a serious GI bleed after being on Celebrex only

> about a month. The Doctors all decided it was due to a combination of

> things: the celebrex, the coumadin, and the fact that this man ate

> very little (and DS-ers would have 2 out of 3 of those..). I asked

> about the switch when it happened, & the answer I was given was that

> Vioxx seemed to have fewer problems with GI bleeding as compared to

> Celebrex (in the elderly). There's no definitive proof yet, just

> anecdotal information from personal experiences. I got the same answer

> from 4 Doctors & one NP, so I decided that it was what they were

> seeing personally. Sorry to have gotten your hopes up.

> Hugs,

> Liane

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I had written:

....recent articles indicate that both of

them (the cox2 inhibitors) may give short-term relief, but may

also cause more long-term damage to cartilage.

Some of you asked for the reference(s). I could not find

the original article(s) that i had seen, but today located another

article. Excerpts are included below. it would appear that

there still is no free lunch!

--Steve

=================================================

http://www.lef.org/magazine/mag2001/jan2001_report_cox2_1.html

ARTHRITIS UPDATE

Drugs That Inhibit COX-2 May Cause Tissue Damage

by: Faloon

[part of] Page 1 of 3

The Scales Tilt on the Side of Nature

Drugs that inhibit the cyclooxyenase-2 (COX-2) enzyme have shown

efficacy in alleviating inflammation and pain caused by arthritis.

Celebrex and Vioxx are two popular COX-2 inhibitors that are being

aggressively marketed to arthritis patients by drug companies.

A new study published in the Journal of Immunology(1) acknowledges

the temporary benefits of COX-2 inhibitors, but identifies a

potential long-term problem that could lead to cartilage and other

tissue degeneration if these drugs are taken over an extended time

period.

The authors of this study found that COX-2

inhibitors cause metabolic

imbalances that can result in the over production of two toxic

cytokines, tumor necrosis factor alpha (TNF-a) and interleukin one

beta (IL1B). Both TNF-a and IL-1B have been shown to play a role

in

the cartilage destruction and the

inflammation process.(2-4) TNF-a

and IL-1B have been found to be elevated in the synovial fluid

and

the cartilage of osteoarthritis patients.(2,5) Thus, the short term

beneficial effects of these agents on arthritic pain and

inflammation

may be achieved at the cost of an increased propensity to long

term

tissue damage caused by TNF-a and

IL1B.

Too much TNF-a results in a host of aging-related disorders

including

autoimmune disease, congestive heart failure, insulin resistance

and

catabolic wasting.(6-10) When TNF-a attacks the linings of the

joints, the result is inflammation, pain and eventual

immobility.(11-13)

Over expression of the destructive cytokines TNF-a and IL-1B are

not

the only problem that COX-2 inhibitors may induce. While COX-2

inhibitors suppress an inflammatory fatty-acid called

prostaglandin

E2, they fail to block the formation of a joint-destroying

cytokine

called leukotriene B4.(14) This all

helps explain why COX-2

inhibitors do not always provide complete relief from arthritis

symptoms.

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Guest guest

I had written:

....recent articles indicate that both of

them (the cox2 inhibitors) may give short-term relief, but may

also cause more long-term damage to cartilage.

Some of you asked for the reference(s). I could not find

the original article(s) that i had seen, but today located another

article. Excerpts are included below. it would appear that

there still is no free lunch!

--Steve

=================================================

http://www.lef.org/magazine/mag2001/jan2001_report_cox2_1.html

ARTHRITIS UPDATE

Drugs That Inhibit COX-2 May Cause Tissue Damage

by: Faloon

[part of] Page 1 of 3

The Scales Tilt on the Side of Nature

Drugs that inhibit the cyclooxyenase-2 (COX-2) enzyme have shown

efficacy in alleviating inflammation and pain caused by arthritis.

Celebrex and Vioxx are two popular COX-2 inhibitors that are being

aggressively marketed to arthritis patients by drug companies.

A new study published in the Journal of Immunology(1) acknowledges

the temporary benefits of COX-2 inhibitors, but identifies a

potential long-term problem that could lead to cartilage and other

tissue degeneration if these drugs are taken over an extended time

period.

The authors of this study found that COX-2

inhibitors cause metabolic

imbalances that can result in the over production of two toxic

cytokines, tumor necrosis factor alpha (TNF-a) and interleukin one

beta (IL1B). Both TNF-a and IL-1B have been shown to play a role

in

the cartilage destruction and the

inflammation process.(2-4) TNF-a

and IL-1B have been found to be elevated in the synovial fluid

and

the cartilage of osteoarthritis patients.(2,5) Thus, the short term

beneficial effects of these agents on arthritic pain and

inflammation

may be achieved at the cost of an increased propensity to long

term

tissue damage caused by TNF-a and

IL1B.

Too much TNF-a results in a host of aging-related disorders

including

autoimmune disease, congestive heart failure, insulin resistance

and

catabolic wasting.(6-10) When TNF-a attacks the linings of the

joints, the result is inflammation, pain and eventual

immobility.(11-13)

Over expression of the destructive cytokines TNF-a and IL-1B are

not

the only problem that COX-2 inhibitors may induce. While COX-2

inhibitors suppress an inflammatory fatty-acid called

prostaglandin

E2, they fail to block the formation of a joint-destroying

cytokine

called leukotriene B4.(14) This all

helps explain why COX-2

inhibitors do not always provide complete relief from arthritis

symptoms.

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Guest guest

I had written:

....recent articles indicate that both of

them (the cox2 inhibitors) may give short-term relief, but may

also cause more long-term damage to cartilage.

Some of you asked for the reference(s). I could not find

the original article(s) that i had seen, but today located another

article. Excerpts are included below. it would appear that

there still is no free lunch!

--Steve

=================================================

http://www.lef.org/magazine/mag2001/jan2001_report_cox2_1.html

ARTHRITIS UPDATE

Drugs That Inhibit COX-2 May Cause Tissue Damage

by: Faloon

[part of] Page 1 of 3

The Scales Tilt on the Side of Nature

Drugs that inhibit the cyclooxyenase-2 (COX-2) enzyme have shown

efficacy in alleviating inflammation and pain caused by arthritis.

Celebrex and Vioxx are two popular COX-2 inhibitors that are being

aggressively marketed to arthritis patients by drug companies.

A new study published in the Journal of Immunology(1) acknowledges

the temporary benefits of COX-2 inhibitors, but identifies a

potential long-term problem that could lead to cartilage and other

tissue degeneration if these drugs are taken over an extended time

period.

The authors of this study found that COX-2

inhibitors cause metabolic

imbalances that can result in the over production of two toxic

cytokines, tumor necrosis factor alpha (TNF-a) and interleukin one

beta (IL1B). Both TNF-a and IL-1B have been shown to play a role

in

the cartilage destruction and the

inflammation process.(2-4) TNF-a

and IL-1B have been found to be elevated in the synovial fluid

and

the cartilage of osteoarthritis patients.(2,5) Thus, the short term

beneficial effects of these agents on arthritic pain and

inflammation

may be achieved at the cost of an increased propensity to long

term

tissue damage caused by TNF-a and

IL1B.

Too much TNF-a results in a host of aging-related disorders

including

autoimmune disease, congestive heart failure, insulin resistance

and

catabolic wasting.(6-10) When TNF-a attacks the linings of the

joints, the result is inflammation, pain and eventual

immobility.(11-13)

Over expression of the destructive cytokines TNF-a and IL-1B are

not

the only problem that COX-2 inhibitors may induce. While COX-2

inhibitors suppress an inflammatory fatty-acid called

prostaglandin

E2, they fail to block the formation of a joint-destroying

cytokine

called leukotriene B4.(14) This all

helps explain why COX-2

inhibitors do not always provide complete relief from arthritis

symptoms.

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Guest guest

For what it's worth. I took Vioxx. After 2 days, I began to feel great.

After 4 days I began to bleed. Appeared rectal. Had several tests including

a colonoscopy. It turns out I have hemrhoids(Spelling?). Anyway, I stopped

taking Vioxx.

n

Pre-Op Dr. Pomp/Mt Sinai

n Boden (maboden@...)

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