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HI Edith

This is an interesting theory and development in understanding this condition

(OA). It could certainly explain, in part, my left hip OA. It does seem to

tally with my post-op rehabilitation (physiotherapy challenges) which revealed

massive atrophy of tissues (muscles, tendons etc.) all the way from my foot up

to and including the small of my back (psoas is a good example), pelvic bone

non-alignment, contraction of certain sets (hamstring etc.), leg shortness (the

part which is not attributable to the loss of cartilage and femur cysts and

femur/acetabulum deformation due to bone-on-bone pressure), lack of flexion

(pelvic and lumbar areas, lack of rotation (internal at knee and hip joints),

etc.

However, as I said, this theory could explain my condition in part only as I

have OA in other joints as well which are unlikely to have been caused by

posture/gait and/or use and development of muscle and tendon sets (both elbows,

both shoulders, left knee, most fingers’ joints etc. – in fact, you can see

the sclerotic bone growth [osteophytes] on my fingers’ joints which will

eventually lead to bent fingers, I’m sure). I think that in my case this is

partly genetic – I can see very similar patterns in my mother’s bones/joints

(BTW, she refused a THR despite being in agony for a few years now, but being 83

now she is entitled to doubt the benefits v. risks of such major op/trauma) –

and, probably, partly bio/socio-specific (I think that some people with OA have

particular [in]ability in metabolising calcium-related substances [maybe in

dairy products?] that lead to the sclerotic deposits of calcium forming bone

growths near joints and destroying cartilage there in the process; that’s why,

I think, this condition could be equally attributed to lack of calcium in some

cases and to excess of calcium in others). However, these are my “theoriesâ€

and I am a complete layman and amateur in these matters, but I do recall that

many a doctor had said that OA-type conditions appear to be influenced, if not

caused, by dietary habits.

Nevertheless, the theory espoused by your physio friend is certainly very

interesting and, I’d say, in some cases probably plausible. Thanks for that

info.

Regards

Dan

* +44 (0)7974 981-407

* +44 (0)20 8501-2573

@ dan.milosevic@...

_____

From: ecrow

Sent: 25 April 2004 09:13

To: surfacehippy

Subject: Some thoughts from a physio

Hi,

I happened upon a physio at my pool trip today and got talking about why

hips get OA and children have problems doing splits etc.......

She said that at a conference last year the physio's were told that at last

they had, via MRI's proof, that a large % of OA in hip joints could be

attributed to muscle problems........ one and/or some being tight etc......

'Why' comes from a variety of reasons and often to do with how they were

developed as young ones.......whether faults were corrected etc and the

types of exercises we got to do etc........... Some being genetic given that

gymninists tend to belong to gym parents who have far looser moving muscles

etc than the general population and often problem type muscles tend to run

in families. Maybe natural selection would have weeded us out long ago if we

still depended on fleeing predators or hunter/gathering all day to get

something to eat........... smile

She said that the MRI's indicated that rarely did actual bone structure have

much to do with OA......... Apparently the use of MRI;s has increased their

knowledge of how all this works greatly........and they can do tests etc

with people standing etc to see just what a muscle set is actually

doing.........

She seemed somewhat apologetic that it has taken so long to get this

proved..........though I think it is somewhat typical human that we need to

have evidence via a MRI etc before we accept the obvious at

times.............and again I think I will be nagging some of my relatives

to check out their teens etc to try heading off hassles later...........

Anyway thought I would share that in light of our discussions the last few

days.......

Edith LBHR Dr. L Walter Syd Aust 8/02

_____

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Guest guest

> HI Edith

>

> This is an interesting theory and development in understanding this

condition (OA). It could certainly explain, in part, my left hip

OA. It does seem to tally with my post-op rehabilitation

(physiotherapy challenges) which revealed massive atrophy of tissues

(muscles, tendons etc.) all the way from my foot up to and including

the small of my back (psoas is a good example), pelvic bone non-

alignment, contraction of certain sets (hamstring etc.), leg

shortness (the part which is not attributable to the loss of

cartilage and femur cysts and femur/acetabulum deformation due to

bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

lack of rotation (internal at knee and hip joints), etc.

>

> However, as I said, this theory could explain my condition in part

only as I have OA in other joints as well which are unlikely to have

been caused by posture/gait and/or use and development of muscle and

tendon sets (both elbows, both shoulders, left knee, most fingers’

joints etc. †" in fact, you can see the sclerotic bone growth

[osteophytes] on my fingers’ joints which will eventually lead to

bent fingers, I’m sure). I think that in my case this is partly

genetic †" I can see very similar patterns in my mother’s

bones/joints (BTW, she refused a THR despite being in agony for a few

years now, but being 83 now she is entitled to doubt the benefits v.

risks of such major op/trauma) †" and, probably, partly bio/socio-

specific (I think that some people with OA have particular [in]

ability in metabolising calcium-related substances [maybe in dairy

products?] that lead to the sclerotic deposits of calcium forming

bone growths near joints and destroying cartilage there in the

process; that’s why, I think, this condition could be equally

attributed to lack of calcium in some cases and to excess of calcium

in others). However, these are my “theories†and I am a complete

layman and amateur in these matters, but I do recall that many a

doctor had said that OA-type conditions appear to be influenced, if

not caused, by dietary habits.

>

> Nevertheless, the theory espoused by your physio friend is

certainly very interesting and, I’d say, in some cases probably

plausible. Thanks for that info.

>

> Regards

> Dan

>

> * +44 (0)7974 981-407

> * +44 (0)20 8501-2573

> @ dan.milosevic@i...

> _____

>

> From: ecrow [mailto:ecrow@c...]

> Sent: 25 April 2004 09:13

> To: surfacehippy

> Subject: Some thoughts from a physio

>

> Hi,

>

> I happened upon a physio at my pool trip today and got talking

about why

> hips get OA and children have problems doing splits etc.......

>

> She said that at a conference last year the physio's were told that

at last

> they had, via MRI's proof, that a large % of OA in hip joints could

be

> attributed to muscle problems........ one and/or some being tight

etc......

> 'Why' comes from a variety of reasons and often to do with how they

were

> developed as young ones.......whether faults were corrected etc and

the

> types of exercises we got to do etc........... Some being genetic

given that

> gymninists tend to belong to gym parents who have far looser moving

muscles

> etc than the general population and often problem type muscles tend

to run

> in families. Maybe natural selection would have weeded us out long

ago if we

> still depended on fleeing predators or hunter/gathering all day to

get

> something to eat........... smile

>

> She said that the MRI's indicated that rarely did actual bone

structure have

> much to do with OA......... Apparently the use of MRI;s has

increased their

> knowledge of how all this works greatly........and they can do

tests etc

> with people standing etc to see just what a muscle set is actually

> doing.........

>

> She seemed somewhat apologetic that it has taken so long to get this

> proved..........though I think it is somewhat typical human that we

need to

> have evidence via a MRI etc before we accept the obvious at

> times.............and again I think I will be nagging some of my

relatives

> to check out their teens etc to try heading off hassles

later...........

>

> Anyway thought I would share that in light of our discussions the

last few

> days.......

>

> Edith LBHR Dr. L Walter Syd Aust 8/02

>

>

>

>

> _____

>

>

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Share on other sites

Guest guest

> HI Edith

>

> This is an interesting theory and development in understanding this

condition (OA). It could certainly explain, in part, my left hip

OA. It does seem to tally with my post-op rehabilitation

(physiotherapy challenges) which revealed massive atrophy of tissues

(muscles, tendons etc.) all the way from my foot up to and including

the small of my back (psoas is a good example), pelvic bone non-

alignment, contraction of certain sets (hamstring etc.), leg

shortness (the part which is not attributable to the loss of

cartilage and femur cysts and femur/acetabulum deformation due to

bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

lack of rotation (internal at knee and hip joints), etc.

>

> However, as I said, this theory could explain my condition in part

only as I have OA in other joints as well which are unlikely to have

been caused by posture/gait and/or use and development of muscle and

tendon sets (both elbows, both shoulders, left knee, most fingers’

joints etc. †" in fact, you can see the sclerotic bone growth

[osteophytes] on my fingers’ joints which will eventually lead to

bent fingers, I’m sure). I think that in my case this is partly

genetic †" I can see very similar patterns in my mother’s

bones/joints (BTW, she refused a THR despite being in agony for a few

years now, but being 83 now she is entitled to doubt the benefits v.

risks of such major op/trauma) †" and, probably, partly bio/socio-

specific (I think that some people with OA have particular [in]

ability in metabolising calcium-related substances [maybe in dairy

products?] that lead to the sclerotic deposits of calcium forming

bone growths near joints and destroying cartilage there in the

process; that’s why, I think, this condition could be equally

attributed to lack of calcium in some cases and to excess of calcium

in others). However, these are my “theories†and I am a complete

layman and amateur in these matters, but I do recall that many a

doctor had said that OA-type conditions appear to be influenced, if

not caused, by dietary habits.

>

> Nevertheless, the theory espoused by your physio friend is

certainly very interesting and, I’d say, in some cases probably

plausible. Thanks for that info.

>

> Regards

> Dan

>

> * +44 (0)7974 981-407

> * +44 (0)20 8501-2573

> @ dan.milosevic@i...

> _____

>

> From: ecrow [mailto:ecrow@c...]

> Sent: 25 April 2004 09:13

> To: surfacehippy

> Subject: Some thoughts from a physio

>

> Hi,

>

> I happened upon a physio at my pool trip today and got talking

about why

> hips get OA and children have problems doing splits etc.......

>

> She said that at a conference last year the physio's were told that

at last

> they had, via MRI's proof, that a large % of OA in hip joints could

be

> attributed to muscle problems........ one and/or some being tight

etc......

> 'Why' comes from a variety of reasons and often to do with how they

were

> developed as young ones.......whether faults were corrected etc and

the

> types of exercises we got to do etc........... Some being genetic

given that

> gymninists tend to belong to gym parents who have far looser moving

muscles

> etc than the general population and often problem type muscles tend

to run

> in families. Maybe natural selection would have weeded us out long

ago if we

> still depended on fleeing predators or hunter/gathering all day to

get

> something to eat........... smile

>

> She said that the MRI's indicated that rarely did actual bone

structure have

> much to do with OA......... Apparently the use of MRI;s has

increased their

> knowledge of how all this works greatly........and they can do

tests etc

> with people standing etc to see just what a muscle set is actually

> doing.........

>

> She seemed somewhat apologetic that it has taken so long to get this

> proved..........though I think it is somewhat typical human that we

need to

> have evidence via a MRI etc before we accept the obvious at

> times.............and again I think I will be nagging some of my

relatives

> to check out their teens etc to try heading off hassles

later...........

>

> Anyway thought I would share that in light of our discussions the

last few

> days.......

>

> Edith LBHR Dr. L Walter Syd Aust 8/02

>

>

>

>

> _____

>

>

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Guest guest

Hi Dan,

Sounds like you have quite a deal of recovery to do there with getting back

reasonable hip performance........... I know what zilch muscle etc has meant

recovery wise thus have a fair idea of the picture.......... and sorry about

your mum - I have a friend whose dad is similiar age and condition and it

isn't pretty to watch the suffering.

You then raise another interesting question as I too have OA in other joints

in very similiar places i.e. both knees, shoulder, fingers, wrist, spine

etc. Now at the tender age of 18 when my hip was fused it did already have

signs of arthritis but that was given to be from the impact of osteomylitis

in the hip area before and at that point the arthritis had gone septic along

with an infection sitting right beside the joint.........so there was almost

a panic to fuse the hip........

When I finally found a surgeon willing to help me all these years later he

could tell me where I had arthritis problems now without me telling him or

even seeing me ( I live 1500 miles from him) due he said, to the result of

the stress on the body of dealing with a fused hip all those years. It was

one reason I was so trusting of him from day one as I normally have had to

explain it all to other doctors I had consulted for they didn't seem to have

a clue, but here was one who knew about what was happening and could

actually tell me............. Needless to say he is one of the best OS in my

country..............

So it seems that automatically one stands a high chance of developing OA

into quite a few places in the body simply because one is stressing

everything to carry a joint as significant as the hip - and with that much

atrophication you certainly would have been carrying the joint. Which is

another reason it is often said here that waiting years for a hip

replacement isn't just about the hip joint itself...... everything else gets

to wear the results.

I don't discount what you say about calcium etc but even that field is still

being sorted out........ i.e. for years doctors told women just eating

calcium would help osteoporosis where now it is becoming more and more

obvious it is more to do with hormones and without the right ones you can

eat all the calcium you like with little affect. I also do know that some

research is throwing up information that certain people can lack particular

enzeme creation via the liver which also seems to play a role in RA and

other arthritis conditions. Now whether that is heriditary or dietary will

no doubt come to light too............ and maybe just that during life one

has damaged the liver through things such as massive doses of say antibotics

when young .............. which is one of my favourites for blame with

myself.......smile.

The one thing I think we can all know is much of this is

individualistic...........there will be patterns that people basically

follow but few people follow the same history, lived and walked the same

roads, ate the same food, were subjected to the same stresses etc.........

so it is hard to lump us all in one category and say this is what happened.

Meanwhile best of luck in getting the hip muscles going again and dealing

with the rest of the OA joints....... It is a challenge I am pretty familiar

with...............

Edith LBHR Dr. L Walter Syd Aust 8/02

This is an interesting theory and development in understanding this

condition (OA). It could certainly explain, in part, my left hip OA. It

does seem to tally with my post-op rehabilitation (physiotherapy challenges)

which revealed massive atrophy of tissues (muscles, tendons etc.) all the

way from my foot up to and including the small of my back (psoas is a good

example), pelvic bone non-alignment, contraction of certain sets (hamstring

etc.), leg shortness (the part which is not attributable to the loss of

cartilage and femur cysts and femur/acetabulum deformation due to

bone-on-bone pressure), lack of flexion (pelvic and lumbar areas, lack of

rotation (internal at knee and hip joints), etc.

However, as I said, this theory could explain my condition in part only as I

have OA in other joints as well which are unlikely to have been caused by

posture/gait and/or use and development of muscle and tendon sets (both

elbows, both shoulders, left knee, most fingers’ joints etc. – in fact, you

can see the sclerotic bone growth [osteophytes] on my fingers’ joints which

will eventually lead to bent fingers, I’m sure). I think that in my case

this is partly genetic – I can see very similar patterns in my mother’s

bones/joints (BTW, she refused a THR despite being in agony for a few years

now, but being 83 now she is entitled to doubt the benefits v. risks of such

major op/trauma) – and, probably, partly bio/socio-specific (I think that

some people with OA have particular [in]ability in metabolising

calcium-related substances [maybe in dairy products?] that lead to the

sclerotic deposits of calcium forming bone growths near joints and

destroying cartilage there in the process; that’s why, I think, this

condition could be equally attributed to lack of calcium in some cases and

to excess of calcium in others). However, these are my “theories†and I am

a complete layman and amateur in these matters, but I do recall that many a

doctor had said that OA-type conditions appear to be influenced, if not

caused, by dietary habits.

Nevertheless, the theory espoused by your physio friend is certainly very

interesting and, I’d say, in some cases probably plausible. Thanks for that

info.

Regards

Dan

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Share on other sites

Guest guest

Hi Dan,

Sounds like you have quite a deal of recovery to do there with getting back

reasonable hip performance........... I know what zilch muscle etc has meant

recovery wise thus have a fair idea of the picture.......... and sorry about

your mum - I have a friend whose dad is similiar age and condition and it

isn't pretty to watch the suffering.

You then raise another interesting question as I too have OA in other joints

in very similiar places i.e. both knees, shoulder, fingers, wrist, spine

etc. Now at the tender age of 18 when my hip was fused it did already have

signs of arthritis but that was given to be from the impact of osteomylitis

in the hip area before and at that point the arthritis had gone septic along

with an infection sitting right beside the joint.........so there was almost

a panic to fuse the hip........

When I finally found a surgeon willing to help me all these years later he

could tell me where I had arthritis problems now without me telling him or

even seeing me ( I live 1500 miles from him) due he said, to the result of

the stress on the body of dealing with a fused hip all those years. It was

one reason I was so trusting of him from day one as I normally have had to

explain it all to other doctors I had consulted for they didn't seem to have

a clue, but here was one who knew about what was happening and could

actually tell me............. Needless to say he is one of the best OS in my

country..............

So it seems that automatically one stands a high chance of developing OA

into quite a few places in the body simply because one is stressing

everything to carry a joint as significant as the hip - and with that much

atrophication you certainly would have been carrying the joint. Which is

another reason it is often said here that waiting years for a hip

replacement isn't just about the hip joint itself...... everything else gets

to wear the results.

I don't discount what you say about calcium etc but even that field is still

being sorted out........ i.e. for years doctors told women just eating

calcium would help osteoporosis where now it is becoming more and more

obvious it is more to do with hormones and without the right ones you can

eat all the calcium you like with little affect. I also do know that some

research is throwing up information that certain people can lack particular

enzeme creation via the liver which also seems to play a role in RA and

other arthritis conditions. Now whether that is heriditary or dietary will

no doubt come to light too............ and maybe just that during life one

has damaged the liver through things such as massive doses of say antibotics

when young .............. which is one of my favourites for blame with

myself.......smile.

The one thing I think we can all know is much of this is

individualistic...........there will be patterns that people basically

follow but few people follow the same history, lived and walked the same

roads, ate the same food, were subjected to the same stresses etc.........

so it is hard to lump us all in one category and say this is what happened.

Meanwhile best of luck in getting the hip muscles going again and dealing

with the rest of the OA joints....... It is a challenge I am pretty familiar

with...............

Edith LBHR Dr. L Walter Syd Aust 8/02

This is an interesting theory and development in understanding this

condition (OA). It could certainly explain, in part, my left hip OA. It

does seem to tally with my post-op rehabilitation (physiotherapy challenges)

which revealed massive atrophy of tissues (muscles, tendons etc.) all the

way from my foot up to and including the small of my back (psoas is a good

example), pelvic bone non-alignment, contraction of certain sets (hamstring

etc.), leg shortness (the part which is not attributable to the loss of

cartilage and femur cysts and femur/acetabulum deformation due to

bone-on-bone pressure), lack of flexion (pelvic and lumbar areas, lack of

rotation (internal at knee and hip joints), etc.

However, as I said, this theory could explain my condition in part only as I

have OA in other joints as well which are unlikely to have been caused by

posture/gait and/or use and development of muscle and tendon sets (both

elbows, both shoulders, left knee, most fingers’ joints etc. – in fact, you

can see the sclerotic bone growth [osteophytes] on my fingers’ joints which

will eventually lead to bent fingers, I’m sure). I think that in my case

this is partly genetic – I can see very similar patterns in my mother’s

bones/joints (BTW, she refused a THR despite being in agony for a few years

now, but being 83 now she is entitled to doubt the benefits v. risks of such

major op/trauma) – and, probably, partly bio/socio-specific (I think that

some people with OA have particular [in]ability in metabolising

calcium-related substances [maybe in dairy products?] that lead to the

sclerotic deposits of calcium forming bone growths near joints and

destroying cartilage there in the process; that’s why, I think, this

condition could be equally attributed to lack of calcium in some cases and

to excess of calcium in others). However, these are my “theories†and I am

a complete layman and amateur in these matters, but I do recall that many a

doctor had said that OA-type conditions appear to be influenced, if not

caused, by dietary habits.

Nevertheless, the theory espoused by your physio friend is certainly very

interesting and, I’d say, in some cases probably plausible. Thanks for that

info.

Regards

Dan

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Guest guest

Hi Dan,

Sounds like you have quite a deal of recovery to do there with getting back

reasonable hip performance........... I know what zilch muscle etc has meant

recovery wise thus have a fair idea of the picture.......... and sorry about

your mum - I have a friend whose dad is similiar age and condition and it

isn't pretty to watch the suffering.

You then raise another interesting question as I too have OA in other joints

in very similiar places i.e. both knees, shoulder, fingers, wrist, spine

etc. Now at the tender age of 18 when my hip was fused it did already have

signs of arthritis but that was given to be from the impact of osteomylitis

in the hip area before and at that point the arthritis had gone septic along

with an infection sitting right beside the joint.........so there was almost

a panic to fuse the hip........

When I finally found a surgeon willing to help me all these years later he

could tell me where I had arthritis problems now without me telling him or

even seeing me ( I live 1500 miles from him) due he said, to the result of

the stress on the body of dealing with a fused hip all those years. It was

one reason I was so trusting of him from day one as I normally have had to

explain it all to other doctors I had consulted for they didn't seem to have

a clue, but here was one who knew about what was happening and could

actually tell me............. Needless to say he is one of the best OS in my

country..............

So it seems that automatically one stands a high chance of developing OA

into quite a few places in the body simply because one is stressing

everything to carry a joint as significant as the hip - and with that much

atrophication you certainly would have been carrying the joint. Which is

another reason it is often said here that waiting years for a hip

replacement isn't just about the hip joint itself...... everything else gets

to wear the results.

I don't discount what you say about calcium etc but even that field is still

being sorted out........ i.e. for years doctors told women just eating

calcium would help osteoporosis where now it is becoming more and more

obvious it is more to do with hormones and without the right ones you can

eat all the calcium you like with little affect. I also do know that some

research is throwing up information that certain people can lack particular

enzeme creation via the liver which also seems to play a role in RA and

other arthritis conditions. Now whether that is heriditary or dietary will

no doubt come to light too............ and maybe just that during life one

has damaged the liver through things such as massive doses of say antibotics

when young .............. which is one of my favourites for blame with

myself.......smile.

The one thing I think we can all know is much of this is

individualistic...........there will be patterns that people basically

follow but few people follow the same history, lived and walked the same

roads, ate the same food, were subjected to the same stresses etc.........

so it is hard to lump us all in one category and say this is what happened.

Meanwhile best of luck in getting the hip muscles going again and dealing

with the rest of the OA joints....... It is a challenge I am pretty familiar

with...............

Edith LBHR Dr. L Walter Syd Aust 8/02

This is an interesting theory and development in understanding this

condition (OA). It could certainly explain, in part, my left hip OA. It

does seem to tally with my post-op rehabilitation (physiotherapy challenges)

which revealed massive atrophy of tissues (muscles, tendons etc.) all the

way from my foot up to and including the small of my back (psoas is a good

example), pelvic bone non-alignment, contraction of certain sets (hamstring

etc.), leg shortness (the part which is not attributable to the loss of

cartilage and femur cysts and femur/acetabulum deformation due to

bone-on-bone pressure), lack of flexion (pelvic and lumbar areas, lack of

rotation (internal at knee and hip joints), etc.

However, as I said, this theory could explain my condition in part only as I

have OA in other joints as well which are unlikely to have been caused by

posture/gait and/or use and development of muscle and tendon sets (both

elbows, both shoulders, left knee, most fingers’ joints etc. – in fact, you

can see the sclerotic bone growth [osteophytes] on my fingers’ joints which

will eventually lead to bent fingers, I’m sure). I think that in my case

this is partly genetic – I can see very similar patterns in my mother’s

bones/joints (BTW, she refused a THR despite being in agony for a few years

now, but being 83 now she is entitled to doubt the benefits v. risks of such

major op/trauma) – and, probably, partly bio/socio-specific (I think that

some people with OA have particular [in]ability in metabolising

calcium-related substances [maybe in dairy products?] that lead to the

sclerotic deposits of calcium forming bone growths near joints and

destroying cartilage there in the process; that’s why, I think, this

condition could be equally attributed to lack of calcium in some cases and

to excess of calcium in others). However, these are my “theories†and I am

a complete layman and amateur in these matters, but I do recall that many a

doctor had said that OA-type conditions appear to be influenced, if not

caused, by dietary habits.

Nevertheless, the theory espoused by your physio friend is certainly very

interesting and, I’d say, in some cases probably plausible. Thanks for that

info.

Regards

Dan

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Hi Susie,

I think we may be talking about 2 different things here............ It is

really what you were having the MRI for and what the OS was looking

for............. the MRI you had would still tell him some of the story

about

the state of your muscles regardless of what it said about the

bones.........and that was what I was referring too........... According to

the physio the MRI has been quite a useful tool for researches to be able to

see what muscles were doing over time etc............ and I know in my own

case the OS referred to what he saw as the state of the muscles,( extremely

poor) from the MRI he had me have............ he was also interested in bone

state given my situation etc but he wasn't looking for anything like your

hassle - the hip was long passed that being fused. And yes MRI's are not

totally perfect in what they show........ like xrays etc nothing beats going

in for a look see..........

And then sadly by the time most people hit the doctor with hip pain, the OS

is at the point of looking for bone damage and bone malfunction around the

hip joint.......... From what I know of human nature very few 'see' the

doctor until it is quite painful and therefore so much catch 22 stuff would

be happening..........i.e. muscles also are in trouble because the bones are

rubbing as well as anything that may have been in trouble before

that.............. This is partly because we are scared of the outcome,

don't have time, don't want to bother others with our problems, don't have a

clear idea of why these things happen etc. And how life is.........

I am taking it that the conference report the physio is referring too is

where researchers are going back to finding individuals before big damage

(without big OA damaged joints etc) and looking at what happens over

time............ In these cases they would be using MRI's to watch the

development of the whole picture and specifically the role played by

muscles. I totally agree those individuals would fall into a very different

category to someone like yourself with some genetic irregularity in the

formation of the hip joint..........

Edith LBHR Dr. L Walter Syd Aust 8/02

Edith, I must tell you about my MRI experience. Upon having my MRI,

the tech placed a pillow under my knees and declared that that was

the proper positioning for a hip MRI...I questioned this placement

but was firmly told that the pillow was necessary...well, to make a

long story short, this altered the alignment of my hip joint making

the protrusio element (which is my big problem) less defined. For me,

the MRI did not tell the tale. It did, however show that I do not

have AVN, for which I am grateful, but my simple x-ray proved much

more valuable. Also, there are a multitude of problems that cause

irregular joint geometry and it's the irregular joint geometry that

after time, I suspect, causes the osteoartritis. My OS told me that

often the MRI is not particularly useful. Susie

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Hi Susie,

I think we may be talking about 2 different things here............ It is

really what you were having the MRI for and what the OS was looking

for............. the MRI you had would still tell him some of the story

about

the state of your muscles regardless of what it said about the

bones.........and that was what I was referring too........... According to

the physio the MRI has been quite a useful tool for researches to be able to

see what muscles were doing over time etc............ and I know in my own

case the OS referred to what he saw as the state of the muscles,( extremely

poor) from the MRI he had me have............ he was also interested in bone

state given my situation etc but he wasn't looking for anything like your

hassle - the hip was long passed that being fused. And yes MRI's are not

totally perfect in what they show........ like xrays etc nothing beats going

in for a look see..........

And then sadly by the time most people hit the doctor with hip pain, the OS

is at the point of looking for bone damage and bone malfunction around the

hip joint.......... From what I know of human nature very few 'see' the

doctor until it is quite painful and therefore so much catch 22 stuff would

be happening..........i.e. muscles also are in trouble because the bones are

rubbing as well as anything that may have been in trouble before

that.............. This is partly because we are scared of the outcome,

don't have time, don't want to bother others with our problems, don't have a

clear idea of why these things happen etc. And how life is.........

I am taking it that the conference report the physio is referring too is

where researchers are going back to finding individuals before big damage

(without big OA damaged joints etc) and looking at what happens over

time............ In these cases they would be using MRI's to watch the

development of the whole picture and specifically the role played by

muscles. I totally agree those individuals would fall into a very different

category to someone like yourself with some genetic irregularity in the

formation of the hip joint..........

Edith LBHR Dr. L Walter Syd Aust 8/02

Edith, I must tell you about my MRI experience. Upon having my MRI,

the tech placed a pillow under my knees and declared that that was

the proper positioning for a hip MRI...I questioned this placement

but was firmly told that the pillow was necessary...well, to make a

long story short, this altered the alignment of my hip joint making

the protrusio element (which is my big problem) less defined. For me,

the MRI did not tell the tale. It did, however show that I do not

have AVN, for which I am grateful, but my simple x-ray proved much

more valuable. Also, there are a multitude of problems that cause

irregular joint geometry and it's the irregular joint geometry that

after time, I suspect, causes the osteoartritis. My OS told me that

often the MRI is not particularly useful. Susie

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Share on other sites

Guest guest

Hi Edith

I certainly plan to discuss with Mr Krikler (the OS who resurfaced my left hip)

this theory when I see him on Thursday for my first post-op check-up (he

operated on me on 16th February). By the way, it seems that the weather will

conspire against me riding my bike the 100 miles from my home in London to the

Rugby hospital where I had my operation and where he holds the post-op clinic.

Shame, as I was planning to show off and was looking forward to the 200 mile

round trip.

I will also check the liver theory. Having had a particularly severe form of

hepatitis B when I was a kid (I was warned then that my liver was permanently

damaged, but you could have fooled me as I have led a super normal and active

life since: I was extremely into sports, some up to professional level and some

others to instructor level; I ate all types of food in four out of five

continents and drank, moderately, all types of alcohol; travelled the world and

stayed in a number of different countries and cultures) and having had excessive

doses of antibiotics (it was fashionable in the 50’s, 60’s and 70’s to

treat any and all conditions with antibiotics which led to the need for stronger

types and higher doses when a real need arose; I even developed an allergy to

tetracycline due to heavy dosages; mind you, I am following the debate about

preventive antibiotic application prior to dental treatment with interest now as

I have not taken a dose of antibiotics for the last 25 years and have had a

tooth filling replaced and my teeth cleaned only a couple of weeks ago; neither

has my OS suggested any such preventive course nor has my dentist thought it

necessary and even if either did I would have refused preventive use of

antibiotics) I would like to find out how much of a factor this might have been

in inducing OA.

Thanks for your thoughts and ideas. I find this site tremendously helpful in

getting a more accurate and thorough understanding of the mysteries of our

bodies through the various experiences that people post about.

Regards

Dan

* +44 (0)7974 981-407

* +44 (0)20 8501-2573

@ dan.milosevic@...

_____

From: ecrow

Sent: 26 April 2004 08:59

To: surfacehippy

Subject: Re: Some thoughts from a physio

Hi Dan,

Sounds like you have quite a deal of recovery to do there with getting back

reasonable hip performance........... I know what zilch muscle etc has meant

recovery wise thus have a fair idea of the picture.......... and sorry about

your mum - I have a friend whose dad is similiar age and condition and it

isn't pretty to watch the suffering.

You then raise another interesting question as I too have OA in other joints

in very similiar places i.e. both knees, shoulder, fingers, wrist, spine

etc. Now at the tender age of 18 when my hip was fused it did already have

signs of arthritis but that was given to be from the impact of osteomylitis

in the hip area before and at that point the arthritis had gone septic along

with an infection sitting right beside the joint.........so there was almost

a panic to fuse the hip........

When I finally found a surgeon willing to help me all these years later he

could tell me where I had arthritis problems now without me telling him or

even seeing me ( I live 1500 miles from him) due he said, to the result of

the stress on the body of dealing with a fused hip all those years. It was

one reason I was so trusting of him from day one as I normally have had to

explain it all to other doctors I had consulted for they didn't seem to have

a clue, but here was one who knew about what was happening and could

actually tell me............. Needless to say he is one of the best OS in my

country..............

So it seems that automatically one stands a high chance of developing OA

into quite a few places in the body simply because one is stressing

everything to carry a joint as significant as the hip - and with that much

atrophication you certainly would have been carrying the joint. Which is

another reason it is often said here that waiting years for a hip

replacement isn't just about the hip joint itself...... everything else gets

to wear the results.

I don't discount what you say about calcium etc but even that field is still

being sorted out........ i.e. for years doctors told women just eating

calcium would help osteoporosis where now it is becoming more and more

obvious it is more to do with hormones and without the right ones you can

eat all the calcium you like with little affect. I also do know that some

research is throwing up information that certain people can lack particular

enzeme creation via the liver which also seems to play a role in RA and

other arthritis conditions. Now whether that is heriditary or dietary will

no doubt come to light too............ and maybe just that during life one

has damaged the liver through things such as massive doses of say antibotics

when young .............. which is one of my favourites for blame with

myself.......smile.

The one thing I think we can all know is much of this is

individualistic...........there will be patterns that people basically

follow but few people follow the same history, lived and walked the same

roads, ate the same food, were subjected to the same stresses etc.........

so it is hard to lump us all in one category and say this is what happened.

Meanwhile best of luck in getting the hip muscles going again and dealing

with the rest of the OA joints....... It is a challenge I am pretty familiar

with...............

Edith LBHR Dr. L Walter Syd Aust 8/02

This is an interesting theory and development in understanding this

condition (OA). It could certainly explain, in part, my left hip OA. It

does seem to tally with my post-op rehabilitation (physiotherapy challenges)

which revealed massive atrophy of tissues (muscles, tendons etc.) all the

way from my foot up to and including the small of my back (psoas is a good

example), pelvic bone non-alignment, contraction of certain sets (hamstring

etc.), leg shortness (the part which is not attributable to the loss of

cartilage and femur cysts and femur/acetabulum deformation due to

bone-on-bone pressure), lack of flexion (pelvic and lumbar areas, lack of

rotation (internal at knee and hip joints), etc.

However, as I said, this theory could explain my condition in part only as I

have OA in other joints as well which are unlikely to have been caused by

posture/gait and/or use and development of muscle and tendon sets (both

elbows, both shoulders, left knee, most fingers’ joints etc. – in fact, you

can see the sclerotic bone growth [osteophytes] on my fingers’ joints which

will eventually lead to bent fingers, I’m sure). I think that in my case

this is partly genetic – I can see very similar patterns in my mother’s

bones/joints (BTW, she refused a THR despite being in agony for a few years

now, but being 83 now she is entitled to doubt the benefits v. risks of such

major op/trauma) – and, probably, partly bio/socio-specific (I think that

some people with OA have particular [in]ability in metabolising

calcium-related substances [maybe in dairy products?] that lead to the

sclerotic deposits of calcium forming bone growths near joints and

destroying cartilage there in the process; that’s why, I think, this

condition could be equally attributed to lack of calcium in some cases and

to excess of calcium in others). However, these are my “theories†and I am

a complete layman and amateur in these matters, but I do recall that many a

doctor had said that OA-type conditions appear to be influenced, if not

caused, by dietary habits.

Nevertheless, the theory espoused by your physio friend is certainly very

interesting and, I’d say, in some cases probably plausible. Thanks for that

info.

Regards

Dan

_____

Link to comment
Share on other sites

Guest guest

Hi Edith

I certainly plan to discuss with Mr Krikler (the OS who resurfaced my left hip)

this theory when I see him on Thursday for my first post-op check-up (he

operated on me on 16th February). By the way, it seems that the weather will

conspire against me riding my bike the 100 miles from my home in London to the

Rugby hospital where I had my operation and where he holds the post-op clinic.

Shame, as I was planning to show off and was looking forward to the 200 mile

round trip.

I will also check the liver theory. Having had a particularly severe form of

hepatitis B when I was a kid (I was warned then that my liver was permanently

damaged, but you could have fooled me as I have led a super normal and active

life since: I was extremely into sports, some up to professional level and some

others to instructor level; I ate all types of food in four out of five

continents and drank, moderately, all types of alcohol; travelled the world and

stayed in a number of different countries and cultures) and having had excessive

doses of antibiotics (it was fashionable in the 50’s, 60’s and 70’s to

treat any and all conditions with antibiotics which led to the need for stronger

types and higher doses when a real need arose; I even developed an allergy to

tetracycline due to heavy dosages; mind you, I am following the debate about

preventive antibiotic application prior to dental treatment with interest now as

I have not taken a dose of antibiotics for the last 25 years and have had a

tooth filling replaced and my teeth cleaned only a couple of weeks ago; neither

has my OS suggested any such preventive course nor has my dentist thought it

necessary and even if either did I would have refused preventive use of

antibiotics) I would like to find out how much of a factor this might have been

in inducing OA.

Thanks for your thoughts and ideas. I find this site tremendously helpful in

getting a more accurate and thorough understanding of the mysteries of our

bodies through the various experiences that people post about.

Regards

Dan

* +44 (0)7974 981-407

* +44 (0)20 8501-2573

@ dan.milosevic@...

_____

From: ecrow

Sent: 26 April 2004 08:59

To: surfacehippy

Subject: Re: Some thoughts from a physio

Hi Dan,

Sounds like you have quite a deal of recovery to do there with getting back

reasonable hip performance........... I know what zilch muscle etc has meant

recovery wise thus have a fair idea of the picture.......... and sorry about

your mum - I have a friend whose dad is similiar age and condition and it

isn't pretty to watch the suffering.

You then raise another interesting question as I too have OA in other joints

in very similiar places i.e. both knees, shoulder, fingers, wrist, spine

etc. Now at the tender age of 18 when my hip was fused it did already have

signs of arthritis but that was given to be from the impact of osteomylitis

in the hip area before and at that point the arthritis had gone septic along

with an infection sitting right beside the joint.........so there was almost

a panic to fuse the hip........

When I finally found a surgeon willing to help me all these years later he

could tell me where I had arthritis problems now without me telling him or

even seeing me ( I live 1500 miles from him) due he said, to the result of

the stress on the body of dealing with a fused hip all those years. It was

one reason I was so trusting of him from day one as I normally have had to

explain it all to other doctors I had consulted for they didn't seem to have

a clue, but here was one who knew about what was happening and could

actually tell me............. Needless to say he is one of the best OS in my

country..............

So it seems that automatically one stands a high chance of developing OA

into quite a few places in the body simply because one is stressing

everything to carry a joint as significant as the hip - and with that much

atrophication you certainly would have been carrying the joint. Which is

another reason it is often said here that waiting years for a hip

replacement isn't just about the hip joint itself...... everything else gets

to wear the results.

I don't discount what you say about calcium etc but even that field is still

being sorted out........ i.e. for years doctors told women just eating

calcium would help osteoporosis where now it is becoming more and more

obvious it is more to do with hormones and without the right ones you can

eat all the calcium you like with little affect. I also do know that some

research is throwing up information that certain people can lack particular

enzeme creation via the liver which also seems to play a role in RA and

other arthritis conditions. Now whether that is heriditary or dietary will

no doubt come to light too............ and maybe just that during life one

has damaged the liver through things such as massive doses of say antibotics

when young .............. which is one of my favourites for blame with

myself.......smile.

The one thing I think we can all know is much of this is

individualistic...........there will be patterns that people basically

follow but few people follow the same history, lived and walked the same

roads, ate the same food, were subjected to the same stresses etc.........

so it is hard to lump us all in one category and say this is what happened.

Meanwhile best of luck in getting the hip muscles going again and dealing

with the rest of the OA joints....... It is a challenge I am pretty familiar

with...............

Edith LBHR Dr. L Walter Syd Aust 8/02

This is an interesting theory and development in understanding this

condition (OA). It could certainly explain, in part, my left hip OA. It

does seem to tally with my post-op rehabilitation (physiotherapy challenges)

which revealed massive atrophy of tissues (muscles, tendons etc.) all the

way from my foot up to and including the small of my back (psoas is a good

example), pelvic bone non-alignment, contraction of certain sets (hamstring

etc.), leg shortness (the part which is not attributable to the loss of

cartilage and femur cysts and femur/acetabulum deformation due to

bone-on-bone pressure), lack of flexion (pelvic and lumbar areas, lack of

rotation (internal at knee and hip joints), etc.

However, as I said, this theory could explain my condition in part only as I

have OA in other joints as well which are unlikely to have been caused by

posture/gait and/or use and development of muscle and tendon sets (both

elbows, both shoulders, left knee, most fingers’ joints etc. – in fact, you

can see the sclerotic bone growth [osteophytes] on my fingers’ joints which

will eventually lead to bent fingers, I’m sure). I think that in my case

this is partly genetic – I can see very similar patterns in my mother’s

bones/joints (BTW, she refused a THR despite being in agony for a few years

now, but being 83 now she is entitled to doubt the benefits v. risks of such

major op/trauma) – and, probably, partly bio/socio-specific (I think that

some people with OA have particular [in]ability in metabolising

calcium-related substances [maybe in dairy products?] that lead to the

sclerotic deposits of calcium forming bone growths near joints and

destroying cartilage there in the process; that’s why, I think, this

condition could be equally attributed to lack of calcium in some cases and

to excess of calcium in others). However, these are my “theories†and I am

a complete layman and amateur in these matters, but I do recall that many a

doctor had said that OA-type conditions appear to be influenced, if not

caused, by dietary habits.

Nevertheless, the theory espoused by your physio friend is certainly very

interesting and, I’d say, in some cases probably plausible. Thanks for that

info.

Regards

Dan

_____

Link to comment
Share on other sites

Guest guest

> Hi Edith

>

> I certainly plan to discuss with Mr Krikler (the OS who resurfaced

my left hip) this theory when I see him on Thursday for my first post-

op check-up (he operated on me on 16th February). By the way, it

seems that the weather will conspire against me riding my bike the

100 miles from my home in London to the Rugby hospital where I had my

operation and where he holds the post-op clinic. Shame, as I was

planning to show off and was looking forward to the 200 mile round

trip.

>

> I will also check the liver theory. Having had a particularly

severe form of hepatitis B when I was a kid (I was warned then that

my liver was permanently damaged, but you could have fooled me as I

have led a super normal and active life since: I was extremely into

sports, some up to professional level and some others to instructor

level; I ate all types of food in four out of five continents and

drank, moderately, all types of alcohol; travelled the world and

stayed in a number of different countries and cultures) and having

had excessive doses of antibiotics (it was fashionable in the 50’s,

60’s and 70’s to treat any and all conditions with antibiotics

which led to the need for stronger types and higher doses when a real

need arose; I even developed an allergy to tetracycline due to heavy

dosages; mind you, I am following the debate about preventive

antibiotic application prior to dental treatment with interest now as

I have not taken a dose of antibiotics for the last 25 years and have

had a tooth filling replaced and my teeth cleaned only a couple of

weeks ago; neither has my OS suggested any such preventive course nor

has my dentist thought it necessary and even if either did I would

have refused preventive use of antibiotics) I would like to find out

how much of a factor this might have been in inducing OA.

>

> Thanks for your thoughts and ideas. I find this site tremendously

helpful in getting a more accurate and thorough understanding of the

mysteries of our bodies through the various experiences that people

post about.

>

> Regards

> Dan

>

> * +44 (0)7974 981-407

> * +44 (0)20 8501-2573

> @ dan.milosevic@i...

> _____

>

> From: ecrow [mailto:ecrow@c...]

> Sent: 26 April 2004 08:59

> To: surfacehippy

> Subject: Re: Some thoughts from a physio

>

>

> Hi Dan,

>

> Sounds like you have quite a deal of recovery to do there with

getting back

> reasonable hip performance........... I know what zilch muscle etc

has meant

> recovery wise thus have a fair idea of the picture.......... and

sorry about

> your mum - I have a friend whose dad is similiar age and condition

and it

> isn't pretty to watch the suffering.

>

> You then raise another interesting question as I too have OA in

other joints

> in very similiar places i.e. both knees, shoulder, fingers, wrist,

spine

> etc. Now at the tender age of 18 when my hip was fused it did

already have

> signs of arthritis but that was given to be from the impact of

osteomylitis

> in the hip area before and at that point the arthritis had gone

septic along

> with an infection sitting right beside the joint.........so there

was almost

> a panic to fuse the hip........

>

> When I finally found a surgeon willing to help me all these years

later he

> could tell me where I had arthritis problems now without me telling

him or

> even seeing me ( I live 1500 miles from him) due he said, to the

result of

> the stress on the body of dealing with a fused hip all those years.

It was

> one reason I was so trusting of him from day one as I normally have

had to

> explain it all to other doctors I had consulted for they didn't

seem to have

> a clue, but here was one who knew about what was happening and could

> actually tell me............. Needless to say he is one of the best

OS in my

> country..............

>

> So it seems that automatically one stands a high chance of

developing OA

> into quite a few places in the body simply because one is stressing

> everything to carry a joint as significant as the hip - and with

that much

> atrophication you certainly would have been carrying the joint.

Which is

> another reason it is often said here that waiting years for a hip

> replacement isn't just about the hip joint itself...... everything

else gets

> to wear the results.

>

> I don't discount what you say about calcium etc but even that field

is still

> being sorted out........ i.e. for years doctors told women just

eating

> calcium would help osteoporosis where now it is becoming more and

more

> obvious it is more to do with hormones and without the right ones

you can

> eat all the calcium you like with little affect. I also do know

that some

> research is throwing up information that certain people can lack

particular

> enzeme creation via the liver which also seems to play a role in RA

and

> other arthritis conditions. Now whether that is heriditary or

dietary will

> no doubt come to light too............ and maybe just that during

life one

> has damaged the liver through things such as massive doses of say

antibotics

> when young .............. which is one of my favourites for blame

with

> myself.......smile.

>

> The one thing I think we can all know is much of this is

> individualistic...........there will be patterns that people

basically

> follow but few people follow the same history, lived and walked the

same

> roads, ate the same food, were subjected to the same stresses

etc.........

> so it is hard to lump us all in one category and say this is what

happened.

>

> Meanwhile best of luck in getting the hip muscles going again and

dealing

> with the rest of the OA joints....... It is a challenge I am pretty

familiar

> with...............

>

> Edith LBHR Dr. L Walter Syd Aust 8/02

>

>

>

>

>

>

> This is an interesting theory and development in understanding this

> condition (OA). It could certainly explain, in part, my left hip

OA. It

> does seem to tally with my post-op rehabilitation (physiotherapy

challenges)

> which revealed massive atrophy of tissues (muscles, tendons etc.)

all the

> way from my foot up to and including the small of my back (psoas is

a good

> example), pelvic bone non-alignment, contraction of certain sets

(hamstring

> etc.), leg shortness (the part which is not attributable to the

loss of

> cartilage and femur cysts and femur/acetabulum deformation due to

> bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

lack of

> rotation (internal at knee and hip joints), etc.

>

> However, as I said, this theory could explain my condition in part

only as I

> have OA in other joints as well which are unlikely to have been

caused by

> posture/gait and/or use and development of muscle and tendon sets

(both

> elbows, both shoulders, left knee, most fingers’ joints etc. †"

in fact, you

> can see the sclerotic bone growth [osteophytes] on my fingers’

joints which

> will eventually lead to bent fingers, I’m sure). I think that in

my case

> this is partly genetic †" I can see very similar patterns in my

mother’s

> bones/joints (BTW, she refused a THR despite being in agony for a

few years

> now, but being 83 now she is entitled to doubt the benefits v.

risks of such

> major op/trauma) †" and, probably, partly bio/socio-specific (I

think that

> some people with OA have particular [in]ability in metabolising

> calcium-related substances [maybe in dairy products?] that lead to

the

> sclerotic deposits of calcium forming bone growths near joints and

> destroying cartilage there in the process; that’s why, I think,

this

> condition could be equally attributed to lack of calcium in some

cases and

> to excess of calcium in others). However, these are my

“theories†and I am

> a complete layman and amateur in these matters, but I do recall

that many a

> doctor had said that OA-type conditions appear to be influenced, if

not

> caused, by dietary habits.

>

> Nevertheless, the theory espoused by your physio friend is

certainly very

> interesting and, I’d say, in some cases probably plausible.

Thanks for that

> info.

>

> Regards

> Dan

> Are you or are you not kidding about riding your bike 200 miles for

your follow up appointment? Seriously though Dan, I'm also following

the antibiotic idea. Fortunately, I don't feel that I have used

antibiotics excessively. Interesting theory as is the dietary ..it

also grabs my attention and surely plays a role in many

disorders.....and then there's the hormone theory, long twitch/slow

twitch muscle theory, and the lax joint theory, the genetic theory,

etc, etc, etc. Who did did your resurfacing? How old are you? Susie

>

> _____

>

>

Link to comment
Share on other sites

Guest guest

> Hi Edith

>

> I certainly plan to discuss with Mr Krikler (the OS who resurfaced

my left hip) this theory when I see him on Thursday for my first post-

op check-up (he operated on me on 16th February). By the way, it

seems that the weather will conspire against me riding my bike the

100 miles from my home in London to the Rugby hospital where I had my

operation and where he holds the post-op clinic. Shame, as I was

planning to show off and was looking forward to the 200 mile round

trip.

>

> I will also check the liver theory. Having had a particularly

severe form of hepatitis B when I was a kid (I was warned then that

my liver was permanently damaged, but you could have fooled me as I

have led a super normal and active life since: I was extremely into

sports, some up to professional level and some others to instructor

level; I ate all types of food in four out of five continents and

drank, moderately, all types of alcohol; travelled the world and

stayed in a number of different countries and cultures) and having

had excessive doses of antibiotics (it was fashionable in the 50’s,

60’s and 70’s to treat any and all conditions with antibiotics

which led to the need for stronger types and higher doses when a real

need arose; I even developed an allergy to tetracycline due to heavy

dosages; mind you, I am following the debate about preventive

antibiotic application prior to dental treatment with interest now as

I have not taken a dose of antibiotics for the last 25 years and have

had a tooth filling replaced and my teeth cleaned only a couple of

weeks ago; neither has my OS suggested any such preventive course nor

has my dentist thought it necessary and even if either did I would

have refused preventive use of antibiotics) I would like to find out

how much of a factor this might have been in inducing OA.

>

> Thanks for your thoughts and ideas. I find this site tremendously

helpful in getting a more accurate and thorough understanding of the

mysteries of our bodies through the various experiences that people

post about.

>

> Regards

> Dan

>

> * +44 (0)7974 981-407

> * +44 (0)20 8501-2573

> @ dan.milosevic@i...

> _____

>

> From: ecrow [mailto:ecrow@c...]

> Sent: 26 April 2004 08:59

> To: surfacehippy

> Subject: Re: Some thoughts from a physio

>

>

> Hi Dan,

>

> Sounds like you have quite a deal of recovery to do there with

getting back

> reasonable hip performance........... I know what zilch muscle etc

has meant

> recovery wise thus have a fair idea of the picture.......... and

sorry about

> your mum - I have a friend whose dad is similiar age and condition

and it

> isn't pretty to watch the suffering.

>

> You then raise another interesting question as I too have OA in

other joints

> in very similiar places i.e. both knees, shoulder, fingers, wrist,

spine

> etc. Now at the tender age of 18 when my hip was fused it did

already have

> signs of arthritis but that was given to be from the impact of

osteomylitis

> in the hip area before and at that point the arthritis had gone

septic along

> with an infection sitting right beside the joint.........so there

was almost

> a panic to fuse the hip........

>

> When I finally found a surgeon willing to help me all these years

later he

> could tell me where I had arthritis problems now without me telling

him or

> even seeing me ( I live 1500 miles from him) due he said, to the

result of

> the stress on the body of dealing with a fused hip all those years.

It was

> one reason I was so trusting of him from day one as I normally have

had to

> explain it all to other doctors I had consulted for they didn't

seem to have

> a clue, but here was one who knew about what was happening and could

> actually tell me............. Needless to say he is one of the best

OS in my

> country..............

>

> So it seems that automatically one stands a high chance of

developing OA

> into quite a few places in the body simply because one is stressing

> everything to carry a joint as significant as the hip - and with

that much

> atrophication you certainly would have been carrying the joint.

Which is

> another reason it is often said here that waiting years for a hip

> replacement isn't just about the hip joint itself...... everything

else gets

> to wear the results.

>

> I don't discount what you say about calcium etc but even that field

is still

> being sorted out........ i.e. for years doctors told women just

eating

> calcium would help osteoporosis where now it is becoming more and

more

> obvious it is more to do with hormones and without the right ones

you can

> eat all the calcium you like with little affect. I also do know

that some

> research is throwing up information that certain people can lack

particular

> enzeme creation via the liver which also seems to play a role in RA

and

> other arthritis conditions. Now whether that is heriditary or

dietary will

> no doubt come to light too............ and maybe just that during

life one

> has damaged the liver through things such as massive doses of say

antibotics

> when young .............. which is one of my favourites for blame

with

> myself.......smile.

>

> The one thing I think we can all know is much of this is

> individualistic...........there will be patterns that people

basically

> follow but few people follow the same history, lived and walked the

same

> roads, ate the same food, were subjected to the same stresses

etc.........

> so it is hard to lump us all in one category and say this is what

happened.

>

> Meanwhile best of luck in getting the hip muscles going again and

dealing

> with the rest of the OA joints....... It is a challenge I am pretty

familiar

> with...............

>

> Edith LBHR Dr. L Walter Syd Aust 8/02

>

>

>

>

>

>

> This is an interesting theory and development in understanding this

> condition (OA). It could certainly explain, in part, my left hip

OA. It

> does seem to tally with my post-op rehabilitation (physiotherapy

challenges)

> which revealed massive atrophy of tissues (muscles, tendons etc.)

all the

> way from my foot up to and including the small of my back (psoas is

a good

> example), pelvic bone non-alignment, contraction of certain sets

(hamstring

> etc.), leg shortness (the part which is not attributable to the

loss of

> cartilage and femur cysts and femur/acetabulum deformation due to

> bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

lack of

> rotation (internal at knee and hip joints), etc.

>

> However, as I said, this theory could explain my condition in part

only as I

> have OA in other joints as well which are unlikely to have been

caused by

> posture/gait and/or use and development of muscle and tendon sets

(both

> elbows, both shoulders, left knee, most fingers’ joints etc. †"

in fact, you

> can see the sclerotic bone growth [osteophytes] on my fingers’

joints which

> will eventually lead to bent fingers, I’m sure). I think that in

my case

> this is partly genetic †" I can see very similar patterns in my

mother’s

> bones/joints (BTW, she refused a THR despite being in agony for a

few years

> now, but being 83 now she is entitled to doubt the benefits v.

risks of such

> major op/trauma) †" and, probably, partly bio/socio-specific (I

think that

> some people with OA have particular [in]ability in metabolising

> calcium-related substances [maybe in dairy products?] that lead to

the

> sclerotic deposits of calcium forming bone growths near joints and

> destroying cartilage there in the process; that’s why, I think,

this

> condition could be equally attributed to lack of calcium in some

cases and

> to excess of calcium in others). However, these are my

“theories†and I am

> a complete layman and amateur in these matters, but I do recall

that many a

> doctor had said that OA-type conditions appear to be influenced, if

not

> caused, by dietary habits.

>

> Nevertheless, the theory espoused by your physio friend is

certainly very

> interesting and, I’d say, in some cases probably plausible.

Thanks for that

> info.

>

> Regards

> Dan

> Are you or are you not kidding about riding your bike 200 miles for

your follow up appointment? Seriously though Dan, I'm also following

the antibiotic idea. Fortunately, I don't feel that I have used

antibiotics excessively. Interesting theory as is the dietary ..it

also grabs my attention and surely plays a role in many

disorders.....and then there's the hormone theory, long twitch/slow

twitch muscle theory, and the lax joint theory, the genetic theory,

etc, etc, etc. Who did did your resurfacing? How old are you? Susie

>

> _____

>

>

Link to comment
Share on other sites

Guest guest

Hi Susie

I’m serious about my bike ride, but will not ride there if it rains (or is

wet).

My op was done by Mr Krikler at St Cross Hospital, RUGBY, Warwickshire. I’ll

be 54 in a few months time.

In my case the lax joint theory would not apply as I am not double-jointed. I

am also unaware of collagen deficiency. I don’t know about the long switch /

slow switch muscle theory. I think that in my case it could be a combination of

the following (but I don’t know if that’s true or in which proportion these

factors may have played a part):

* Genetic

* Anomaly in calcium processing (which could be due to genetic, liver [caused by

diet and/or antibiotic abuse], dietary reasons)

* Sporting overexertion

The last one could be tied to gait posture issues.

I suspect I will never know the likely cause(s). But, the more people post

about their understanding and suspicions, to more likely we are to find out, if

by no other means than by elimination, what are the principal factors involved

in triggering OA.

Regards

Dan

PS Have you had your resurf, and if yes, when, where and by whom? Where are you

based?

* +44 (0)7974 981-407

* +44 (0)20 8501-2573

@ dan.milosevic@...

_____

From: susettejk

Sent: 26 April 2004 12:47

To: surfacehippy

Subject: Re: Some thoughts from a physio

> Hi Edith

>

> I certainly plan to discuss with Mr Krikler (the OS who resurfaced

my left hip) this theory when I see him on Thursday for my first post-

op check-up (he operated on me on 16th February). By the way, it

seems that the weather will conspire against me riding my bike the

100 miles from my home in London to the Rugby hospital where I had my

operation and where he holds the post-op clinic. Shame, as I was

planning to show off and was looking forward to the 200 mile round

trip.

>

> I will also check the liver theory. Having had a particularly

severe form of hepatitis B when I was a kid (I was warned then that

my liver was permanently damaged, but you could have fooled me as I

have led a super normal and active life since: I was extremely into

sports, some up to professional level and some others to instructor

level; I ate all types of food in four out of five continents and

drank, moderately, all types of alcohol; travelled the world and

stayed in a number of different countries and cultures) and having

had excessive doses of antibiotics (it was fashionable in the 50’s,

60’s and 70’s to treat any and all conditions with antibiotics

which led to the need for stronger types and higher doses when a real

need arose; I even developed an allergy to tetracycline due to heavy

dosages; mind you, I am following the debate about preventive

antibiotic application prior to dental treatment with interest now as

I have not taken a dose of antibiotics for the last 25 years and have

had a tooth filling replaced and my teeth cleaned only a couple of

weeks ago; neither has my OS suggested any such preventive course nor

has my dentist thought it necessary and even if either did I would

have refused preventive use of antibiotics) I would like to find out

how much of a factor this might have been in inducing OA.

>

> Thanks for your thoughts and ideas. I find this site tremendously

helpful in getting a more accurate and thorough understanding of the

mysteries of our bodies through the various experiences that people

post about.

>

> Regards

> Dan

>

> * +44 (0)7974 981-407

> * +44 (0)20 8501-2573

> @ dan.milosevic@i...

> _____

>

> From: ecrow [mailto:ecrow@c...]

> Sent: 26 April 2004 08:59

> To: surfacehippy

> Subject: Re: Some thoughts from a physio

>

>

> Hi Dan,

>

> Sounds like you have quite a deal of recovery to do there with

getting back

> reasonable hip performance........... I know what zilch muscle etc

has meant

> recovery wise thus have a fair idea of the picture.......... and

sorry about

> your mum - I have a friend whose dad is similiar age and condition

and it

> isn't pretty to watch the suffering.

>

> You then raise another interesting question as I too have OA in

other joints

> in very similiar places i.e. both knees, shoulder, fingers, wrist,

spine

> etc. Now at the tender age of 18 when my hip was fused it did

already have

> signs of arthritis but that was given to be from the impact of

osteomylitis

> in the hip area before and at that point the arthritis had gone

septic along

> with an infection sitting right beside the joint.........so there

was almost

> a panic to fuse the hip........

>

> When I finally found a surgeon willing to help me all these years

later he

> could tell me where I had arthritis problems now without me telling

him or

> even seeing me ( I live 1500 miles from him) due he said, to the

result of

> the stress on the body of dealing with a fused hip all those years.

It was

> one reason I was so trusting of him from day one as I normally have

had to

> explain it all to other doctors I had consulted for they didn't

seem to have

> a clue, but here was one who knew about what was happening and could

> actually tell me............. Needless to say he is one of the best

OS in my

> country..............

>

> So it seems that automatically one stands a high chance of

developing OA

> into quite a few places in the body simply because one is stressing

> everything to carry a joint as significant as the hip - and with

that much

> atrophication you certainly would have been carrying the joint.

Which is

> another reason it is often said here that waiting years for a hip

> replacement isn't just about the hip joint itself...... everything

else gets

> to wear the results.

>

> I don't discount what you say about calcium etc but even that field

is still

> being sorted out........ i.e. for years doctors told women just

eating

> calcium would help osteoporosis where now it is becoming more and

more

> obvious it is more to do with hormones and without the right ones

you can

> eat all the calcium you like with little affect. I also do know

that some

> research is throwing up information that certain people can lack

particular

> enzeme creation via the liver which also seems to play a role in RA

and

> other arthritis conditions. Now whether that is heriditary or

dietary will

> no doubt come to light too............ and maybe just that during

life one

> has damaged the liver through things such as massive doses of say

antibotics

> when young .............. which is one of my favourites for blame

with

> myself.......smile.

>

> The one thing I think we can all know is much of this is

> individualistic...........there will be patterns that people

basically

> follow but few people follow the same history, lived and walked the

same

> roads, ate the same food, were subjected to the same stresses

etc.........

> so it is hard to lump us all in one category and say this is what

happened.

>

> Meanwhile best of luck in getting the hip muscles going again and

dealing

> with the rest of the OA joints....... It is a challenge I am pretty

familiar

> with...............

>

> Edith LBHR Dr. L Walter Syd Aust 8/02

>

>

>

>

>

>

> This is an interesting theory and development in understanding this

> condition (OA). It could certainly explain, in part, my left hip

OA. It

> does seem to tally with my post-op rehabilitation (physiotherapy

challenges)

> which revealed massive atrophy of tissues (muscles, tendons etc.)

all the

> way from my foot up to and including the small of my back (psoas is

a good

> example), pelvic bone non-alignment, contraction of certain sets

(hamstring

> etc.), leg shortness (the part which is not attributable to the

loss of

> cartilage and femur cysts and femur/acetabulum deformation due to

> bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

lack of

> rotation (internal at knee and hip joints), etc.

>

> However, as I said, this theory could explain my condition in part

only as I

> have OA in other joints as well which are unlikely to have been

caused by

> posture/gait and/or use and development of muscle and tendon sets

(both

> elbows, both shoulders, left knee, most fingers’ joints etc. †"

in fact, you

> can see the sclerotic bone growth [osteophytes] on my fingers’

joints which

> will eventually lead to bent fingers, I’m sure). I think that in

my case

> this is partly genetic †" I can see very similar patterns in my

mother’s

> bones/joints (BTW, she refused a THR despite being in agony for a

few years

> now, but being 83 now she is entitled to doubt the benefits v.

risks of such

> major op/trauma) †" and, probably, partly bio/socio-specific (I

think that

> some people with OA have particular [in]ability in metabolising

> calcium-related substances [maybe in dairy products?] that lead to

the

> sclerotic deposits of calcium forming bone growths near joints and

> destroying cartilage there in the process; that’s why, I think,

this

> condition could be equally attributed to lack of calcium in some

cases and

> to excess of calcium in others). However, these are my

“theories†and I am

> a complete layman and amateur in these matters, but I do recall

that many a

> doctor had said that OA-type conditions appear to be influenced, if

not

> caused, by dietary habits.

>

> Nevertheless, the theory espoused by your physio friend is

certainly very

> interesting and, I’d say, in some cases probably plausible.

Thanks for that

> info.

>

> Regards

> Dan

> Are you or are you not kidding about riding your bike 200 miles for

your follow up appointment? Seriously though Dan, I'm also following

the antibiotic idea. Fortunately, I don't feel that I have used

antibiotics excessively. Interesting theory as is the dietary ..it

also grabs my attention and surely plays a role in many

disorders.....and then there's the hormone theory, long twitch/slow

twitch muscle theory, and the lax joint theory, the genetic theory,

etc, etc, etc. Who did did your resurfacing? How old are you? Susie

>

> _____

>

>

Link to comment
Share on other sites

Guest guest

Hi Susie

I’m serious about my bike ride, but will not ride there if it rains (or is

wet).

My op was done by Mr Krikler at St Cross Hospital, RUGBY, Warwickshire. I’ll

be 54 in a few months time.

In my case the lax joint theory would not apply as I am not double-jointed. I

am also unaware of collagen deficiency. I don’t know about the long switch /

slow switch muscle theory. I think that in my case it could be a combination of

the following (but I don’t know if that’s true or in which proportion these

factors may have played a part):

* Genetic

* Anomaly in calcium processing (which could be due to genetic, liver [caused by

diet and/or antibiotic abuse], dietary reasons)

* Sporting overexertion

The last one could be tied to gait posture issues.

I suspect I will never know the likely cause(s). But, the more people post

about their understanding and suspicions, to more likely we are to find out, if

by no other means than by elimination, what are the principal factors involved

in triggering OA.

Regards

Dan

PS Have you had your resurf, and if yes, when, where and by whom? Where are you

based?

* +44 (0)7974 981-407

* +44 (0)20 8501-2573

@ dan.milosevic@...

_____

From: susettejk

Sent: 26 April 2004 12:47

To: surfacehippy

Subject: Re: Some thoughts from a physio

> Hi Edith

>

> I certainly plan to discuss with Mr Krikler (the OS who resurfaced

my left hip) this theory when I see him on Thursday for my first post-

op check-up (he operated on me on 16th February). By the way, it

seems that the weather will conspire against me riding my bike the

100 miles from my home in London to the Rugby hospital where I had my

operation and where he holds the post-op clinic. Shame, as I was

planning to show off and was looking forward to the 200 mile round

trip.

>

> I will also check the liver theory. Having had a particularly

severe form of hepatitis B when I was a kid (I was warned then that

my liver was permanently damaged, but you could have fooled me as I

have led a super normal and active life since: I was extremely into

sports, some up to professional level and some others to instructor

level; I ate all types of food in four out of five continents and

drank, moderately, all types of alcohol; travelled the world and

stayed in a number of different countries and cultures) and having

had excessive doses of antibiotics (it was fashionable in the 50’s,

60’s and 70’s to treat any and all conditions with antibiotics

which led to the need for stronger types and higher doses when a real

need arose; I even developed an allergy to tetracycline due to heavy

dosages; mind you, I am following the debate about preventive

antibiotic application prior to dental treatment with interest now as

I have not taken a dose of antibiotics for the last 25 years and have

had a tooth filling replaced and my teeth cleaned only a couple of

weeks ago; neither has my OS suggested any such preventive course nor

has my dentist thought it necessary and even if either did I would

have refused preventive use of antibiotics) I would like to find out

how much of a factor this might have been in inducing OA.

>

> Thanks for your thoughts and ideas. I find this site tremendously

helpful in getting a more accurate and thorough understanding of the

mysteries of our bodies through the various experiences that people

post about.

>

> Regards

> Dan

>

> * +44 (0)7974 981-407

> * +44 (0)20 8501-2573

> @ dan.milosevic@i...

> _____

>

> From: ecrow [mailto:ecrow@c...]

> Sent: 26 April 2004 08:59

> To: surfacehippy

> Subject: Re: Some thoughts from a physio

>

>

> Hi Dan,

>

> Sounds like you have quite a deal of recovery to do there with

getting back

> reasonable hip performance........... I know what zilch muscle etc

has meant

> recovery wise thus have a fair idea of the picture.......... and

sorry about

> your mum - I have a friend whose dad is similiar age and condition

and it

> isn't pretty to watch the suffering.

>

> You then raise another interesting question as I too have OA in

other joints

> in very similiar places i.e. both knees, shoulder, fingers, wrist,

spine

> etc. Now at the tender age of 18 when my hip was fused it did

already have

> signs of arthritis but that was given to be from the impact of

osteomylitis

> in the hip area before and at that point the arthritis had gone

septic along

> with an infection sitting right beside the joint.........so there

was almost

> a panic to fuse the hip........

>

> When I finally found a surgeon willing to help me all these years

later he

> could tell me where I had arthritis problems now without me telling

him or

> even seeing me ( I live 1500 miles from him) due he said, to the

result of

> the stress on the body of dealing with a fused hip all those years.

It was

> one reason I was so trusting of him from day one as I normally have

had to

> explain it all to other doctors I had consulted for they didn't

seem to have

> a clue, but here was one who knew about what was happening and could

> actually tell me............. Needless to say he is one of the best

OS in my

> country..............

>

> So it seems that automatically one stands a high chance of

developing OA

> into quite a few places in the body simply because one is stressing

> everything to carry a joint as significant as the hip - and with

that much

> atrophication you certainly would have been carrying the joint.

Which is

> another reason it is often said here that waiting years for a hip

> replacement isn't just about the hip joint itself...... everything

else gets

> to wear the results.

>

> I don't discount what you say about calcium etc but even that field

is still

> being sorted out........ i.e. for years doctors told women just

eating

> calcium would help osteoporosis where now it is becoming more and

more

> obvious it is more to do with hormones and without the right ones

you can

> eat all the calcium you like with little affect. I also do know

that some

> research is throwing up information that certain people can lack

particular

> enzeme creation via the liver which also seems to play a role in RA

and

> other arthritis conditions. Now whether that is heriditary or

dietary will

> no doubt come to light too............ and maybe just that during

life one

> has damaged the liver through things such as massive doses of say

antibotics

> when young .............. which is one of my favourites for blame

with

> myself.......smile.

>

> The one thing I think we can all know is much of this is

> individualistic...........there will be patterns that people

basically

> follow but few people follow the same history, lived and walked the

same

> roads, ate the same food, were subjected to the same stresses

etc.........

> so it is hard to lump us all in one category and say this is what

happened.

>

> Meanwhile best of luck in getting the hip muscles going again and

dealing

> with the rest of the OA joints....... It is a challenge I am pretty

familiar

> with...............

>

> Edith LBHR Dr. L Walter Syd Aust 8/02

>

>

>

>

>

>

> This is an interesting theory and development in understanding this

> condition (OA). It could certainly explain, in part, my left hip

OA. It

> does seem to tally with my post-op rehabilitation (physiotherapy

challenges)

> which revealed massive atrophy of tissues (muscles, tendons etc.)

all the

> way from my foot up to and including the small of my back (psoas is

a good

> example), pelvic bone non-alignment, contraction of certain sets

(hamstring

> etc.), leg shortness (the part which is not attributable to the

loss of

> cartilage and femur cysts and femur/acetabulum deformation due to

> bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

lack of

> rotation (internal at knee and hip joints), etc.

>

> However, as I said, this theory could explain my condition in part

only as I

> have OA in other joints as well which are unlikely to have been

caused by

> posture/gait and/or use and development of muscle and tendon sets

(both

> elbows, both shoulders, left knee, most fingers’ joints etc. †"

in fact, you

> can see the sclerotic bone growth [osteophytes] on my fingers’

joints which

> will eventually lead to bent fingers, I’m sure). I think that in

my case

> this is partly genetic †" I can see very similar patterns in my

mother’s

> bones/joints (BTW, she refused a THR despite being in agony for a

few years

> now, but being 83 now she is entitled to doubt the benefits v.

risks of such

> major op/trauma) †" and, probably, partly bio/socio-specific (I

think that

> some people with OA have particular [in]ability in metabolising

> calcium-related substances [maybe in dairy products?] that lead to

the

> sclerotic deposits of calcium forming bone growths near joints and

> destroying cartilage there in the process; that’s why, I think,

this

> condition could be equally attributed to lack of calcium in some

cases and

> to excess of calcium in others). However, these are my

“theories†and I am

> a complete layman and amateur in these matters, but I do recall

that many a

> doctor had said that OA-type conditions appear to be influenced, if

not

> caused, by dietary habits.

>

> Nevertheless, the theory espoused by your physio friend is

certainly very

> interesting and, I’d say, in some cases probably plausible.

Thanks for that

> info.

>

> Regards

> Dan

> Are you or are you not kidding about riding your bike 200 miles for

your follow up appointment? Seriously though Dan, I'm also following

the antibiotic idea. Fortunately, I don't feel that I have used

antibiotics excessively. Interesting theory as is the dietary ..it

also grabs my attention and surely plays a role in many

disorders.....and then there's the hormone theory, long twitch/slow

twitch muscle theory, and the lax joint theory, the genetic theory,

etc, etc, etc. Who did did your resurfacing? How old are you? Susie

>

> _____

>

>

Link to comment
Share on other sites

Guest guest

> > Hi Edith

> >

> > I certainly plan to discuss with Mr Krikler (the OS who

resurfaced

> my left hip) this theory when I see him on Thursday for my first

post-

> op check-up (he operated on me on 16th February). By the way, it

> seems that the weather will conspire against me riding my bike the

> 100 miles from my home in London to the Rugby hospital where I had

my

> operation and where he holds the post-op clinic. Shame, as I was

> planning to show off and was looking forward to the 200 mile round

> trip.

> >

> > I will also check the liver theory. Having had a particularly

> severe form of hepatitis B when I was a kid (I was warned then that

> my liver was permanently damaged, but you could have fooled me as I

> have led a super normal and active life since: I was extremely into

> sports, some up to professional level and some others to instructor

> level; I ate all types of food in four out of five continents and

> drank, moderately, all types of alcohol; travelled the world and

> stayed in a number of different countries and cultures) and having

> had excessive doses of antibiotics (it was fashionable in the 50â

€™s,

> 60’s and 70’s to treat any and all conditions with

antibiotics

> which led to the need for stronger types and higher doses when a

real

> need arose; I even developed an allergy to tetracycline due to

heavy

> dosages; mind you, I am following the debate about preventive

> antibiotic application prior to dental treatment with interest now

as

> I have not taken a dose of antibiotics for the last 25 years and

have

> had a tooth filling replaced and my teeth cleaned only a couple of

> weeks ago; neither has my OS suggested any such preventive course

nor

> has my dentist thought it necessary and even if either did I would

> have refused preventive use of antibiotics) I would like to find

out

> how much of a factor this might have been in inducing OA.

> >

> > Thanks for your thoughts and ideas. I find this site

tremendously

> helpful in getting a more accurate and thorough understanding of

the

> mysteries of our bodies through the various experiences that people

> post about.

> >

> > Regards

> > Dan

> >

> > * +44 (0)7974 981-407

> > * +44 (0)20 8501-2573

> > @ dan.milosevic@i...

> > _____

> >

> > From: ecrow [mailto:ecrow@c...]

> > Sent: 26 April 2004 08:59

> > To: surfacehippy

> > Subject: Re: Some thoughts from a physio

> >

> >

> > Hi Dan,

> >

> > Sounds like you have quite a deal of recovery to do there with

> getting back

> > reasonable hip performance........... I know what zilch muscle

etc

> has meant

> > recovery wise thus have a fair idea of the picture.......... and

> sorry about

> > your mum - I have a friend whose dad is similiar age and

condition

> and it

> > isn't pretty to watch the suffering.

> >

> > You then raise another interesting question as I too have OA in

> other joints

> > in very similiar places i.e. both knees, shoulder, fingers,

wrist,

> spine

> > etc. Now at the tender age of 18 when my hip was fused it did

> already have

> > signs of arthritis but that was given to be from the impact of

> osteomylitis

> > in the hip area before and at that point the arthritis had gone

> septic along

> > with an infection sitting right beside the joint.........so there

> was almost

> > a panic to fuse the hip........

> >

> > When I finally found a surgeon willing to help me all these years

> later he

> > could tell me where I had arthritis problems now without me

telling

> him or

> > even seeing me ( I live 1500 miles from him) due he said, to the

> result of

> > the stress on the body of dealing with a fused hip all those

years.

> It was

> > one reason I was so trusting of him from day one as I normally

have

> had to

> > explain it all to other doctors I had consulted for they didn't

> seem to have

> > a clue, but here was one who knew about what was happening and

could

> > actually tell me............. Needless to say he is one of the

best

> OS in my

> > country..............

> >

> > So it seems that automatically one stands a high chance of

> developing OA

> > into quite a few places in the body simply because one is

stressing

> > everything to carry a joint as significant as the hip - and with

> that much

> > atrophication you certainly would have been carrying the joint.

> Which is

> > another reason it is often said here that waiting years for a hip

> > replacement isn't just about the hip joint itself......

everything

> else gets

> > to wear the results.

> >

> > I don't discount what you say about calcium etc but even that

field

> is still

> > being sorted out........ i.e. for years doctors told women just

> eating

> > calcium would help osteoporosis where now it is becoming more and

> more

> > obvious it is more to do with hormones and without the right ones

> you can

> > eat all the calcium you like with little affect. I also do know

> that some

> > research is throwing up information that certain people can lack

> particular

> > enzeme creation via the liver which also seems to play a role in

RA

> and

> > other arthritis conditions. Now whether that is heriditary or

> dietary will

> > no doubt come to light too............ and maybe just that during

> life one

> > has damaged the liver through things such as massive doses of say

> antibotics

> > when young .............. which is one of my favourites for blame

> with

> > myself.......smile.

> >

> > The one thing I think we can all know is much of this is

> > individualistic...........there will be patterns that people

> basically

> > follow but few people follow the same history, lived and walked

the

> same

> > roads, ate the same food, were subjected to the same stresses

> etc.........

> > so it is hard to lump us all in one category and say this is what

> happened.

> >

> > Meanwhile best of luck in getting the hip muscles going again and

> dealing

> > with the rest of the OA joints....... It is a challenge I am

pretty

> familiar

> > with...............

> >

> > Edith LBHR Dr. L Walter Syd Aust 8/02

> >

> >

> >

> >

> >

> >

> > This is an interesting theory and development in understanding

this

> > condition (OA). It could certainly explain, in part, my left hip

> OA. It

> > does seem to tally with my post-op rehabilitation (physiotherapy

> challenges)

> > which revealed massive atrophy of tissues (muscles, tendons etc.)

> all the

> > way from my foot up to and including the small of my back (psoas

is

> a good

> > example), pelvic bone non-alignment, contraction of certain sets

> (hamstring

> > etc.), leg shortness (the part which is not attributable to the

> loss of

> > cartilage and femur cysts and femur/acetabulum deformation due to

> > bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

> lack of

> > rotation (internal at knee and hip joints), etc.

> >

> > However, as I said, this theory could explain my condition in

part

> only as I

> > have OA in other joints as well which are unlikely to have been

> caused by

> > posture/gait and/or use and development of muscle and tendon sets

> (both

> > elbows, both shoulders, left knee, most fingers’ joints

etc. †"

> in fact, you

> > can see the sclerotic bone growth [osteophytes] on my fingersâ

€™

> joints which

> > will eventually lead to bent fingers, I’m sure). I think

that in

> my case

> > this is partly genetic †" I can see very similar patterns in

my

> mother’s

> > bones/joints (BTW, she refused a THR despite being in agony for a

> few years

> > now, but being 83 now she is entitled to doubt the benefits v.

> risks of such

> > major op/trauma) †" and, probably, partly bio/socio-specific

(I

> think that

> > some people with OA have particular [in]ability in metabolising

> > calcium-related substances [maybe in dairy products?] that lead

to

> the

> > sclerotic deposits of calcium forming bone growths near joints and

> > destroying cartilage there in the process; that’s why, I

think,

> this

> > condition could be equally attributed to lack of calcium in some

> cases and

> > to excess of calcium in others). However, these are my

>  " theories†and I am

> > a complete layman and amateur in these matters, but I do recall

> that many a

> > doctor had said that OA-type conditions appear to be influenced,

if

> not

> > caused, by dietary habits.

> >

> > Nevertheless, the theory espoused by your physio friend is

> certainly very

> > interesting and, I’d say, in some cases probably

plausible.

> Thanks for that

> > info.

> >

> > Regards

> > Dan

> > Are you or are you not kidding about riding your bike 200 miles

for

> your follow up appointment? Seriously though Dan, I'm also

following

> the antibiotic idea. Fortunately, I don't feel that I have used

> antibiotics excessively. Interesting theory as is the dietary ..it

> also grabs my attention and surely plays a role in many

> disorders.....and then there's the hormone theory, long twitch/slow

> twitch muscle theory, and the lax joint theory, the genetic theory,

> etc, etc, etc. Who did did your resurfacing? How old are you? Susie

> >

Dan...hello again...wow! your re-surfacing must be quite a success

with you considering that bike ride! I'm impressed. I am 51 and

have not yet had surgery...I'm the person posting all the threads

about Protrusio Acetabulum...that's my three month old

diagnosis...and the reason I need the new hips. I live in Memphis,

TN where the medical community does not favor resurfacing....but I've

been doing my own investigating since the day of my diagnosis. I

know that with my particular disorder that I will lose my ROM

frighteningly fast and I can already tell that I'm on the way

there...don't know how much longer I should wait...seems so

subjective....the other two protrusio's that I've communicated with

have both gone in the direction of re-surfacing. I've also been

posting on the Hip Universe Support Group and wanted to let you know

that there's another biker guy there that would probably love to talk

with you... " smokinspokes " Thanks for your reply. Susie> > _____

> >

> >

Link to comment
Share on other sites

Guest guest

> > Hi Edith

> >

> > I certainly plan to discuss with Mr Krikler (the OS who

resurfaced

> my left hip) this theory when I see him on Thursday for my first

post-

> op check-up (he operated on me on 16th February). By the way, it

> seems that the weather will conspire against me riding my bike the

> 100 miles from my home in London to the Rugby hospital where I had

my

> operation and where he holds the post-op clinic. Shame, as I was

> planning to show off and was looking forward to the 200 mile round

> trip.

> >

> > I will also check the liver theory. Having had a particularly

> severe form of hepatitis B when I was a kid (I was warned then that

> my liver was permanently damaged, but you could have fooled me as I

> have led a super normal and active life since: I was extremely into

> sports, some up to professional level and some others to instructor

> level; I ate all types of food in four out of five continents and

> drank, moderately, all types of alcohol; travelled the world and

> stayed in a number of different countries and cultures) and having

> had excessive doses of antibiotics (it was fashionable in the 50â

€™s,

> 60’s and 70’s to treat any and all conditions with

antibiotics

> which led to the need for stronger types and higher doses when a

real

> need arose; I even developed an allergy to tetracycline due to

heavy

> dosages; mind you, I am following the debate about preventive

> antibiotic application prior to dental treatment with interest now

as

> I have not taken a dose of antibiotics for the last 25 years and

have

> had a tooth filling replaced and my teeth cleaned only a couple of

> weeks ago; neither has my OS suggested any such preventive course

nor

> has my dentist thought it necessary and even if either did I would

> have refused preventive use of antibiotics) I would like to find

out

> how much of a factor this might have been in inducing OA.

> >

> > Thanks for your thoughts and ideas. I find this site

tremendously

> helpful in getting a more accurate and thorough understanding of

the

> mysteries of our bodies through the various experiences that people

> post about.

> >

> > Regards

> > Dan

> >

> > * +44 (0)7974 981-407

> > * +44 (0)20 8501-2573

> > @ dan.milosevic@i...

> > _____

> >

> > From: ecrow [mailto:ecrow@c...]

> > Sent: 26 April 2004 08:59

> > To: surfacehippy

> > Subject: Re: Some thoughts from a physio

> >

> >

> > Hi Dan,

> >

> > Sounds like you have quite a deal of recovery to do there with

> getting back

> > reasonable hip performance........... I know what zilch muscle

etc

> has meant

> > recovery wise thus have a fair idea of the picture.......... and

> sorry about

> > your mum - I have a friend whose dad is similiar age and

condition

> and it

> > isn't pretty to watch the suffering.

> >

> > You then raise another interesting question as I too have OA in

> other joints

> > in very similiar places i.e. both knees, shoulder, fingers,

wrist,

> spine

> > etc. Now at the tender age of 18 when my hip was fused it did

> already have

> > signs of arthritis but that was given to be from the impact of

> osteomylitis

> > in the hip area before and at that point the arthritis had gone

> septic along

> > with an infection sitting right beside the joint.........so there

> was almost

> > a panic to fuse the hip........

> >

> > When I finally found a surgeon willing to help me all these years

> later he

> > could tell me where I had arthritis problems now without me

telling

> him or

> > even seeing me ( I live 1500 miles from him) due he said, to the

> result of

> > the stress on the body of dealing with a fused hip all those

years.

> It was

> > one reason I was so trusting of him from day one as I normally

have

> had to

> > explain it all to other doctors I had consulted for they didn't

> seem to have

> > a clue, but here was one who knew about what was happening and

could

> > actually tell me............. Needless to say he is one of the

best

> OS in my

> > country..............

> >

> > So it seems that automatically one stands a high chance of

> developing OA

> > into quite a few places in the body simply because one is

stressing

> > everything to carry a joint as significant as the hip - and with

> that much

> > atrophication you certainly would have been carrying the joint.

> Which is

> > another reason it is often said here that waiting years for a hip

> > replacement isn't just about the hip joint itself......

everything

> else gets

> > to wear the results.

> >

> > I don't discount what you say about calcium etc but even that

field

> is still

> > being sorted out........ i.e. for years doctors told women just

> eating

> > calcium would help osteoporosis where now it is becoming more and

> more

> > obvious it is more to do with hormones and without the right ones

> you can

> > eat all the calcium you like with little affect. I also do know

> that some

> > research is throwing up information that certain people can lack

> particular

> > enzeme creation via the liver which also seems to play a role in

RA

> and

> > other arthritis conditions. Now whether that is heriditary or

> dietary will

> > no doubt come to light too............ and maybe just that during

> life one

> > has damaged the liver through things such as massive doses of say

> antibotics

> > when young .............. which is one of my favourites for blame

> with

> > myself.......smile.

> >

> > The one thing I think we can all know is much of this is

> > individualistic...........there will be patterns that people

> basically

> > follow but few people follow the same history, lived and walked

the

> same

> > roads, ate the same food, were subjected to the same stresses

> etc.........

> > so it is hard to lump us all in one category and say this is what

> happened.

> >

> > Meanwhile best of luck in getting the hip muscles going again and

> dealing

> > with the rest of the OA joints....... It is a challenge I am

pretty

> familiar

> > with...............

> >

> > Edith LBHR Dr. L Walter Syd Aust 8/02

> >

> >

> >

> >

> >

> >

> > This is an interesting theory and development in understanding

this

> > condition (OA). It could certainly explain, in part, my left hip

> OA. It

> > does seem to tally with my post-op rehabilitation (physiotherapy

> challenges)

> > which revealed massive atrophy of tissues (muscles, tendons etc.)

> all the

> > way from my foot up to and including the small of my back (psoas

is

> a good

> > example), pelvic bone non-alignment, contraction of certain sets

> (hamstring

> > etc.), leg shortness (the part which is not attributable to the

> loss of

> > cartilage and femur cysts and femur/acetabulum deformation due to

> > bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

> lack of

> > rotation (internal at knee and hip joints), etc.

> >

> > However, as I said, this theory could explain my condition in

part

> only as I

> > have OA in other joints as well which are unlikely to have been

> caused by

> > posture/gait and/or use and development of muscle and tendon sets

> (both

> > elbows, both shoulders, left knee, most fingers’ joints

etc. †"

> in fact, you

> > can see the sclerotic bone growth [osteophytes] on my fingersâ

€™

> joints which

> > will eventually lead to bent fingers, I’m sure). I think

that in

> my case

> > this is partly genetic †" I can see very similar patterns in

my

> mother’s

> > bones/joints (BTW, she refused a THR despite being in agony for a

> few years

> > now, but being 83 now she is entitled to doubt the benefits v.

> risks of such

> > major op/trauma) †" and, probably, partly bio/socio-specific

(I

> think that

> > some people with OA have particular [in]ability in metabolising

> > calcium-related substances [maybe in dairy products?] that lead

to

> the

> > sclerotic deposits of calcium forming bone growths near joints and

> > destroying cartilage there in the process; that’s why, I

think,

> this

> > condition could be equally attributed to lack of calcium in some

> cases and

> > to excess of calcium in others). However, these are my

>  " theories†and I am

> > a complete layman and amateur in these matters, but I do recall

> that many a

> > doctor had said that OA-type conditions appear to be influenced,

if

> not

> > caused, by dietary habits.

> >

> > Nevertheless, the theory espoused by your physio friend is

> certainly very

> > interesting and, I’d say, in some cases probably

plausible.

> Thanks for that

> > info.

> >

> > Regards

> > Dan

> > Are you or are you not kidding about riding your bike 200 miles

for

> your follow up appointment? Seriously though Dan, I'm also

following

> the antibiotic idea. Fortunately, I don't feel that I have used

> antibiotics excessively. Interesting theory as is the dietary ..it

> also grabs my attention and surely plays a role in many

> disorders.....and then there's the hormone theory, long twitch/slow

> twitch muscle theory, and the lax joint theory, the genetic theory,

> etc, etc, etc. Who did did your resurfacing? How old are you? Susie

> >

Dan...hello again...wow! your re-surfacing must be quite a success

with you considering that bike ride! I'm impressed. I am 51 and

have not yet had surgery...I'm the person posting all the threads

about Protrusio Acetabulum...that's my three month old

diagnosis...and the reason I need the new hips. I live in Memphis,

TN where the medical community does not favor resurfacing....but I've

been doing my own investigating since the day of my diagnosis. I

know that with my particular disorder that I will lose my ROM

frighteningly fast and I can already tell that I'm on the way

there...don't know how much longer I should wait...seems so

subjective....the other two protrusio's that I've communicated with

have both gone in the direction of re-surfacing. I've also been

posting on the Hip Universe Support Group and wanted to let you know

that there's another biker guy there that would probably love to talk

with you... " smokinspokes " Thanks for your reply. Susie> > _____

> >

> >

Link to comment
Share on other sites

Guest guest

Hi Susie

By all means, pass my details to your biker friend.

I trust you will sort out your resurf shortly. I know next to nothing about

Protrusio Acetabulum, but from my Latin and my understanding of hip problems and

anatomy, I’d suggest that your condition must have started many, many years

ago, if not at birth (congenital?). The condition suggests to me a deformation

which should not happen suddenly. Not that it matters to you whether you were

borne with it or it erupted in the last few years.

From reading the posts on this site for nearly 15 months now, it seems that you

can’t go wrong if you get in touch with Dr DeSmet in Ghent – he seems to be

the Wizard of Hip with massive experience and a predilection for tough cases.

His success record appears to be second to none. What’s more, he seems to be

by far the cheapest of the bunch (excluding a couple of Indian OSs who trained

and operated in Birmingham, I understand, and who seem to be carrying out this

procedure extremely cheaply in India now – don’t quote me on that, but

someone might confirm this as I read it about 6-9 months ago on this site).

Let me know if I can be of any help.

Regards

Dan

* +44 (0)7974 981-407

* +44 (0)20 8501-2573

@ dan.milosevic@...

_____

From: susettejk

Sent: 26 April 2004 15:13

To: surfacehippy

Subject: Re: Some thoughts from a physio

> > Hi Edith

> >

> > I certainly plan to discuss with Mr Krikler (the OS who

resurfaced

> my left hip) this theory when I see him on Thursday for my first

post-

> op check-up (he operated on me on 16th February). By the way, it

> seems that the weather will conspire against me riding my bike the

> 100 miles from my home in London to the Rugby hospital where I had

my

> operation and where he holds the post-op clinic. Shame, as I was

> planning to show off and was looking forward to the 200 mile round

> trip.

> >

> > I will also check the liver theory. Having had a particularly

> severe form of hepatitis B when I was a kid (I was warned then that

> my liver was permanently damaged, but you could have fooled me as I

> have led a super normal and active life since: I was extremely into

> sports, some up to professional level and some others to instructor

> level; I ate all types of food in four out of five continents and

> drank, moderately, all types of alcohol; travelled the world and

> stayed in a number of different countries and cultures) and having

> had excessive doses of antibiotics (it was fashionable in the 50â

€™s,

> 60’s and 70’s to treat any and all

conditions with

antibiotics

> which led to the need for stronger types and higher doses when a

real

> need arose; I even developed an allergy to tetracycline due to

heavy

> dosages; mind you, I am following the debate about preventive

> antibiotic application prior to dental treatment with interest now

as

> I have not taken a dose of antibiotics for the last 25 years and

have

> had a tooth filling replaced and my teeth cleaned only a couple of

> weeks ago; neither has my OS suggested any such preventive course

nor

> has my dentist thought it necessary and even if either did I would

> have refused preventive use of antibiotics) I would like to find

out

> how much of a factor this might have been in inducing OA.

> >

> > Thanks for your thoughts and ideas. I find this site

tremendously

> helpful in getting a more accurate and thorough understanding of

the

> mysteries of our bodies through the various experiences that people

> post about.

> >

> > Regards

> > Dan

> >

> > * +44 (0)7974 981-407

> > * +44 (0)20 8501-2573

> > @ dan.milosevic@i...

> > _____

> >

> > From: ecrow [mailto:ecrow@c...]

> > Sent: 26 April 2004 08:59

> > To: surfacehippy

> > Subject: Re: Some thoughts from a physio

> >

> >

> > Hi Dan,

> >

> > Sounds like you have quite a deal of recovery to do there with

> getting back

> > reasonable hip performance........... I know what zilch muscle

etc

> has meant

> > recovery wise thus have a fair idea of the picture.......... and

> sorry about

> > your mum - I have a friend whose dad is similiar age and

condition

> and it

> > isn't pretty to watch the suffering.

> >

> > You then raise another interesting question as I too have OA in

> other joints

> > in very similiar places i.e. both knees, shoulder, fingers,

wrist,

> spine

> > etc. Now at the tender age of 18 when my hip was fused it did

> already have

> > signs of arthritis but that was given to be from the impact of

> osteomylitis

> > in the hip area before and at that point the arthritis had gone

> septic along

> > with an infection sitting right beside the joint.........so there

> was almost

> > a panic to fuse the hip........

> >

> > When I finally found a surgeon willing to help me all these years

> later he

> > could tell me where I had arthritis problems now without me

telling

> him or

> > even seeing me ( I live 1500 miles from him) due he said, to the

> result of

> > the stress on the body of dealing with a fused hip all those

years.

> It was

> > one reason I was so trusting of him from day one as I normally

have

> had to

> > explain it all to other doctors I had consulted for they didn't

> seem to have

> > a clue, but here was one who knew about what was happening and

could

> > actually tell me............. Needless to say he is one of the

best

> OS in my

> > country..............

> >

> > So it seems that automatically one stands a high chance of

> developing OA

> > into quite a few places in the body simply because one is

stressing

> > everything to carry a joint as significant as the hip - and with

> that much

> > atrophication you certainly would have been carrying the joint.

> Which is

> > another reason it is often said here that waiting years for a hip

> > replacement isn't just about the hip joint itself......

everything

> else gets

> > to wear the results.

> >

> > I don't discount what you say about calcium etc but even that

field

> is still

> > being sorted out........ i.e. for years doctors told women just

> eating

> > calcium would help osteoporosis where now it is becoming more and

> more

> > obvious it is more to do with hormones and without the right ones

> you can

> > eat all the calcium you like with little affect. I also do know

> that some

> > research is throwing up information that certain people can lack

> particular

> > enzeme creation via the liver which also seems to play a role in

RA

> and

> > other arthritis conditions. Now whether that is heriditary or

> dietary will

> > no doubt come to light too............ and maybe just that during

> life one

> > has damaged the liver through things such as massive doses of say

> antibotics

> > when young .............. which is one of my favourites for blame

> with

> > myself.......smile.

> >

> > The one thing I think we can all know is much of this is

> > individualistic...........there will be patterns that people

> basically

> > follow but few people follow the same history, lived and walked

the

> same

> > roads, ate the same food, were subjected to the same stresses

> etc.........

> > so it is hard to lump us all in one category and say this is what

> happened.

> >

> > Meanwhile best of luck in getting the hip muscles going again and

> dealing

> > with the rest of the OA joints....... It is a challenge I am

pretty

> familiar

> > with...............

> >

> > Edith LBHR Dr. L Walter Syd Aust 8/02

> >

> >

> >

> >

> >

> >

> > This is an interesting theory and development in understanding

this

> > condition (OA). It could certainly explain, in part, my left hip

> OA. It

> > does seem to tally with my post-op rehabilitation (physiotherapy

> challenges)

> > which revealed massive atrophy of tissues (muscles, tendons etc.)

> all the

> > way from my foot up to and including the small of my back (psoas

is

> a good

> > example), pelvic bone non-alignment, contraction of certain sets

> (hamstring

> > etc.), leg shortness (the part which is not attributable to the

> loss of

> > cartilage and femur cysts and femur/acetabulum deformation due to

> > bone-on-bone pressure), lack of flexion (pelvic and lumbar areas,

> lack of

> > rotation (internal at knee and hip joints), etc.

> >

> > However, as I said, this theory could explain my condition in

part

> only as I

> > have OA in other joints as well which are unlikely to have been

> caused by

> > posture/gait and/or use and development of muscle and tendon sets

> (both

> > elbows, both shoulders, left knee, most fingers’ joints

etc. †"

> in fact, you

> > can see the sclerotic bone growth [osteophytes] on my fingersâ

€™

> joints which

> > will eventually lead to bent fingers, I’m sure). I think

that in

> my case

> > this is partly genetic †" I can see very similar patterns in

my

> mother’s

> > bones/joints (BTW, she refused a THR despite being in agony for a

> few years

> > now, but being 83 now she is entitled to doubt the benefits v.

> risks of such

> > major op/trauma) †" and, probably, partly bio/socio-specific

(I

> think that

> > some people with OA have particular [in]ability in metabolising

> > calcium-related substances [maybe in dairy products?] that lead

to

> the

> > sclerotic deposits of calcium forming bone growths near joints and

> > destroying cartilage there in the process; that’s why, I

think,

> this

> > condition could be equally attributed to lack of calcium in some

> cases and

> > to excess of calcium in others). However, these are my

>  " theories†and I am

> > a complete layman and amateur in these matters, but I do recall

> that many a

> > doctor had said that OA-type conditions appear to be influenced,

if

> not

> > caused, by dietary habits.

> >

> > Nevertheless, the theory espoused by your physio friend is

> certainly very

> > interesting and, I’d say, in some cases probably

plausible.

> Thanks for that

> > info.

> >

> > Regards

> > Dan

> > Are you or are you not kidding about riding your bike 200 miles

for

> your follow up appointment? Seriously though Dan, I'm also

following

> the antibiotic idea. Fortunately, I don't feel that I have used

> antibiotics excessively. Interesting theory as is the dietary ..it

> also grabs my attention and surely plays a role in many

> disorders.....and then there's the hormone theory, long twitch/slow

> twitch muscle theory, and the lax joint theory, the genetic theory,

> etc, etc, etc. Who did did your resurfacing? How old are you? Susie

> >

Dan...hello again...wow! your re-surfacing must be quite a success

with you considering that bike ride! I'm impressed. I am 51 and

have not yet had surgery...I'm the person posting all the threads

about Protrusio Acetabulum...that's my three month old

diagnosis...and the reason I need the new hips. I live in Memphis,

TN where the medical community does not favor resurfacing....but I've

been doing my own investigating since the day of my diagnosis. I

know that with my particular disorder that I will lose my ROM

frighteningly fast and I can already tell that I'm on the way

there...don't know how much longer I should wait...seems so

subjective....the other two protrusio's that I've communicated with

have both gone in the direction of re-surfacing. I've also been

posting on the Hip Universe Support Group and wanted to let you know

that there's another biker guy there that would probably love to talk

with you... " smokinspokes " Thanks for your reply. Susie> > _____

> >

> >

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Hi Dan,

Well I certainly envy you being able to do a 100 mile bike

ride.........smile. I am thinking I am doing well with a 12 lap

swim........smile. But I do know about this urge to show off....... have it

regularly just showing people how well I can now walk.......... and

considering how many times in my life I was told by specialist etal that I

would never walk again if someone was silly enough to give me a new hip, it

feels extra special. Sadly it also proves how scatty most knowledge about a

lot of these things really is and yet most of us take it for granted that if

it is said by an OS or other health trained specialist it 'must be

true'..............

And yes one of the reasons I continue with lists like this is to gather

ideas about the why of arthritis. I don't have any history of Hepatitis of

any description, rarely drink and have always watched my diet from early

20's........ But during 6 years of my life as a teen I had extensive

antibotic treatment where everything available was thrown at the

osteomylitis I had, often with me refusing to eat for long periods or

wanting to live on meat alone for long periods, extensive blood transfusions

etc.

Then for about 20 years I had very reasonable health........ then about 10

years ago I suddenly got quite ill with tiredness and aches and

pains........I still wonder about a virus attack........which is another

avenue for body problems.......... That herald the start of a lot more pain

in a lot of joints and trips to the doctors that had them say 'well you had

osteomylitis when younger what did you expect' inferring that somehow the

problem of having high level constant infection of my bone marrow had also

left its toll. Or, yes you may have had some unnamed unisolated

virus........ Now I know that it could have been a combination of those 2

plus simply worn my body out coping with the fused hip..........

And sometimes about all I know is I can only work hard now to get my body as

healthy and fit as possible and leave the mysteries of the past back

there.............smile.

Here's hoping for a fine day for the bike ride.............

Edith LBHR Dr. L Walter Syd Aust 8/02

I certainly plan to discuss with Mr Krikler (the OS who resurfaced my left

hip) this theory when I see him on Thursday for my first post-op check-up

(he operated on me on 16th February). By the way, it seems that the weather

will conspire against me riding my bike the 100 miles from my home in London

to the Rugby hospital where I had my operation and where he holds the

post-op clinic. Shame, as I was planning to show off and was looking

forward to the 200 mile round trip.

I will also check the liver theory. Having had a particularly severe form

of hepatitis B when I was a kid (I was warned then that my liver was

permanently damaged, but you could have fooled me as I have led a super

normal and active life since: I was extremely into sports, some up to

professional level and some others to instructor level; I ate all types of

food in four out of five continents and drank, moderately, all types of

alcohol; travelled the world and stayed in a number of different countries

and cultures) and having had excessive doses of antibiotics (it was

fashionable in the 50’s, 60’s and 70’s to treat any and all conditions

with

antibiotics which led to the need for stronger types and higher doses when a

real need arose; I even developed an allergy to tetracycline due to heavy

dosages; mind you, I am following the debate about preventive antibiotic

application prior to dental treatment with interest now as I have not taken

a dose of antibiotics for the last 25 years and have had a tooth filling

replaced and my teeth cleaned only a couple of weeks ago; neither has my OS

suggested any such preventive course nor has my dentist thought it necessary

and even if either did I would have refused preventive use of antibiotics) I

would like to find out how much of a factor this might have been in inducing

OA.

Thanks for your thoughts and ideas. I find this site tremendously helpful

in getting a more accurate and thorough understanding of the mysteries of

our bodies through the various experiences that people post about.

Regards

Dan

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Hi Dan,

Well I certainly envy you being able to do a 100 mile bike

ride.........smile. I am thinking I am doing well with a 12 lap

swim........smile. But I do know about this urge to show off....... have it

regularly just showing people how well I can now walk.......... and

considering how many times in my life I was told by specialist etal that I

would never walk again if someone was silly enough to give me a new hip, it

feels extra special. Sadly it also proves how scatty most knowledge about a

lot of these things really is and yet most of us take it for granted that if

it is said by an OS or other health trained specialist it 'must be

true'..............

And yes one of the reasons I continue with lists like this is to gather

ideas about the why of arthritis. I don't have any history of Hepatitis of

any description, rarely drink and have always watched my diet from early

20's........ But during 6 years of my life as a teen I had extensive

antibotic treatment where everything available was thrown at the

osteomylitis I had, often with me refusing to eat for long periods or

wanting to live on meat alone for long periods, extensive blood transfusions

etc.

Then for about 20 years I had very reasonable health........ then about 10

years ago I suddenly got quite ill with tiredness and aches and

pains........I still wonder about a virus attack........which is another

avenue for body problems.......... That herald the start of a lot more pain

in a lot of joints and trips to the doctors that had them say 'well you had

osteomylitis when younger what did you expect' inferring that somehow the

problem of having high level constant infection of my bone marrow had also

left its toll. Or, yes you may have had some unnamed unisolated

virus........ Now I know that it could have been a combination of those 2

plus simply worn my body out coping with the fused hip..........

And sometimes about all I know is I can only work hard now to get my body as

healthy and fit as possible and leave the mysteries of the past back

there.............smile.

Here's hoping for a fine day for the bike ride.............

Edith LBHR Dr. L Walter Syd Aust 8/02

I certainly plan to discuss with Mr Krikler (the OS who resurfaced my left

hip) this theory when I see him on Thursday for my first post-op check-up

(he operated on me on 16th February). By the way, it seems that the weather

will conspire against me riding my bike the 100 miles from my home in London

to the Rugby hospital where I had my operation and where he holds the

post-op clinic. Shame, as I was planning to show off and was looking

forward to the 200 mile round trip.

I will also check the liver theory. Having had a particularly severe form

of hepatitis B when I was a kid (I was warned then that my liver was

permanently damaged, but you could have fooled me as I have led a super

normal and active life since: I was extremely into sports, some up to

professional level and some others to instructor level; I ate all types of

food in four out of five continents and drank, moderately, all types of

alcohol; travelled the world and stayed in a number of different countries

and cultures) and having had excessive doses of antibiotics (it was

fashionable in the 50’s, 60’s and 70’s to treat any and all conditions

with

antibiotics which led to the need for stronger types and higher doses when a

real need arose; I even developed an allergy to tetracycline due to heavy

dosages; mind you, I am following the debate about preventive antibiotic

application prior to dental treatment with interest now as I have not taken

a dose of antibiotics for the last 25 years and have had a tooth filling

replaced and my teeth cleaned only a couple of weeks ago; neither has my OS

suggested any such preventive course nor has my dentist thought it necessary

and even if either did I would have refused preventive use of antibiotics) I

would like to find out how much of a factor this might have been in inducing

OA.

Thanks for your thoughts and ideas. I find this site tremendously helpful

in getting a more accurate and thorough understanding of the mysteries of

our bodies through the various experiences that people post about.

Regards

Dan

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