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The 8th International Conference on Alzheimer's and Related Disorders is

being held in Stockholm from July 20 to 25.

You might ask " so what? "

I learned of this conference as both of Terry's docs are attending and one

is delivering a paper. I have scanned the program at

http://www.alz.org/internationalconference/programs.htm

and find it to be of broad interest in the arena of nuero-degenerative

disease processes, especially dementia. The latter, dementia, has recently

been added on a preliminary basis to Terry's Dx. We are exploring this

further with her new neurologist.

In any events, some folks may find scanning the program to be of interest.

PS As a sidelight, both of Terry's docs, one neurologist and one

neuropsychologist, are Co Directors of the Memory Disorders Clinic in the

Neurology Department at the University of Virginia. This Clinic also

includes on movement disorder specialist, who is not nearly as broad in the

neurological outlook as the Co Directors. The message I have gotten is

that you neurologists with a broad behavioral background for folks with

MSA, PSP, DLBD, etc. Note that PD is not explicitly included. Terry

original saw the movement disorder specialist at UVa and he was baffled by

her case and referred her to the Co Director of this Clinic. We are glad

for his referral.The medical support has been much better ever since the

referral.

Sennewald Charlottesville, Virginia

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,

Actually dementia is very associated with PD and LBD (and the tau disorders like

PSP and CBGD) more so than MSA. In MSA the cognitive functions are more

affected as Fisher and others have pointed out - it is not memory but the

analyzing portion of the brain which seems to have problems.

In fact as the PD patient gets older, they are very likely to have at least some

dementia. I don't remember the exact figures but by 60 something like 10% of

the PD patients had some form of dementia, by 70 y.o. up to about 30% and by

80-90 it was very high.

Am I miss-reading your email? You seem to be saying just the opposite. The

first neurologist who saw Charlotte and diagnosed her as PD was more into

Altzheimers than the movement disorder specialists we later went to, but she was

not as good at working with specific symptoms and adjusting meds. Charlotte had

no memory problems except when she had infection. However, she could not

process info (like driving) well at all and felt the road was closing in on her.

But the night before she died, she told us not to put the trash out because the

Town does not pick up trash the day after Thanksgiving. That sort of mental

capability seems to be standard for MSA as many report the same thing - slowed

analyzation, but memory is fine.

Take care, Bill Werre

=======================================

>

>

> Date: 2002/07/19 Fri AM 08:42:43 CDT

> To: shydrager

> Subject: Conference in Sweden

>

> The 8th International Conference on Alzheimer's and Related Disorders is

> being held in Stockholm from July 20 to 25.

>

> You might ask " so what? "

>

> I learned of this conference as both of Terry's docs are attending and one

> is delivering a paper. I have scanned the program at

>

> http://www.alz.org/internationalconference/programs.htm

>

> and find it to be of broad interest in the arena of nuero-degenerative

> disease processes, especially dementia. The latter, dementia, has recently

> been added on a preliminary basis to Terry's Dx. We are exploring this

> further with her new neurologist.

>

> In any events, some folks may find scanning the program to be of interest.

>

> PS As a sidelight, both of Terry's docs, one neurologist and one

> neuropsychologist, are Co Directors of the Memory Disorders Clinic in the

> Neurology Department at the University of Virginia. This Clinic also

> includes on movement disorder specialist, who is not nearly as broad in the

> neurological outlook as the Co Directors. The message I have gotten is

> that you neurologists with a broad behavioral background for folks with

> MSA, PSP, DLBD, etc. Note that PD is not explicitly included. Terry

> original saw the movement disorder specialist at UVa and he was baffled by

> her case and referred her to the Co Director of this Clinic. We are glad

> for his referral.The medical support has been much better ever since the

> referral.

>

> Sennewald Charlottesville, Virginia

>

>

>

> If you do not wish to belong to shydrager, you may

> unsubscribe by sending a blank email to

>

> shydrager-unsubscribe

>

>

>

>

>

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Thanks , I remembered reading abstracts from this group's last

conference that included lots of research on the synuclein protein

(implicated as a possible cause of MSA) so I just scanned for that among

their abstracts on the website you provided. Some of theme look especially

relevant to MSA and the other parkinson-plus disorders.

These presentations appear to deal with understanding the function of

alpha-synuclein and why alpha-synuclein forms aggregates in brain cells and

also look at ways to inhibit(or stop) this aggregation ie. potential future

treatments. It's really interesting to see where in the world these

researchers are working. Lots going on in Japan, China and Europe.

ARE ?-SYNUCLEIN DERIVATIVES AN ALTERNATIVE APPROACH FOR TREATMENT OF

NEURODEGENERATIVE DISORDERS

Manfred Windisch1, Eliezer Masliah2, Rockenstein2, Birgit

Hutter-Paier1

1JSW-Research GmbH, Rankengasse 28, Graz, A-8020, Styria, Austria;

2University of California at San Diego, 9500 Gilman Drive, La Jolla, CA

92093-0624, California, United States

Session: Synucleins - P3

INHIBITION AND MECHANISM OF ALPHA-SYNUCLEIN AGGREGATION

Zagorski1, Mihaela Apetri1, Vernon 2

1Case Western Reserve University, Dept of Chemistry, Cleveland, 44106-7078,

Ohio, United States; 2Case Western Reserve University, Dept of Biohemistry,

Cleveland, 44106, Ohio, United States

Session: Lewy-body related diseases - P4

THE DUAL EFFECTS OF a-SYNUCLEIN ON THE NEURONAL SURVIVAL

Ji-Heui Seo, Jong-Cheol Rah, Se Hoon Choi, Jae Kyung Shin, KyoungSik Min,

Yoo-Hun Suh

Coll. of Medicine, Seoul National University, Dept. of Pharmacology, 28

Yongon-dong, Chongno-gu, Seoul, 110-799, Republic of Korea

Session: Alzheimers disease - P3

INHIBITION OF a-SYNUCLEIN(68-78) AGGREGATION AND TOXICITY BY AN N-METHYLATED

ANALOGUE

G Brent Irvine1, M Bodles1, Brett Greer1, M A El-Agnaf2, J

S Guthrie1

1Queen's University Belfast, School of Biology & Biochemistry, Belfast, BT9

7BL, United Kingdom;

2Lancaster University, School of Biological Sciences, Lancaster, LA1 4YQ,

United Kingdom

Session: Synucleins - P3

alpha-SYNUCLEIN, ESPECIALLY THE PARKINSON'S DISEASE-ASSOCIATED MUTANTS,

FORMS PORE-LIKE ANNULAR AND TUBULAR PROTOFIBRILS

Hilal Lashuel1, Petre2, ph Wall3, Martha Simon3,

Nowak1, Walz2, Lansbury1

1Brigham and Women's Hospital/Harvard Medical School, 65 Landsdowne St,

Cambridge, 02139, Massachusetts, United States; 2Department of Cell Biology,

Harvard Medical School, 240 Longwood Ave, Boston, 02115, Massachusetts,

United States; 3Department of Biology, Brookhaven National Laboratory,

building 463, Upton, 11973, New York, United States

Session: Synucleins - P3

NEURODEGENERATION IN ALPHA-SYNUCLEIN TRANSGENIC MICE IS ASSOCIATED WITH

ACCUMULATION OF TRUNCATED ALPHA-SYNUCLEIN POLYPEPTIDES

Lee1, Wanda Stirling1, Yanqun Xu1, Neal Copeland2, Nacy 2,

Price1

1s Hopkins University, School of Medicine, Dept of Pathology, Ross 558,

Baltimore, 21205, land, United States; 2NCI, Mammalian Genetic

Laboratory, Frederick, 21702, land, United States

Session: Animal & Cellular Models Transgenic - S8

ALPHA-SYNUCLEIN (A-SYN) AS A REGULATOR OF DOPAMINE SYNTHESIS

Ruth 1, Jack Waymire2, Eva Lin1, Fengli Guo3, Zigmond1

1University of Pittsburgh School of Medicine, Department of Neurology,

Pittsburgh, 15213, Pennsylvania, United States; 2University of Texas Medical

School, Department of Neurobiology & Anatomy, Houston, 77330, Texas, United

States; 3University of Pittsburgh School of Medicine, Center for Biological

Imaging, Pittsburgh, 15213, Pennsylvania, United States

Session: Related neurodegenerative conditions - S6

TWO PARKINSON'S DISEASE-LINKED PROTEINS, a-SYNUCLEIN AND PARKIN, INTERACT IN

NORMAL HUMAN BRAIN

Schlossmacher1, Hideki Shimura1, Frosch1, Wei Ping Gai2,

Nobutaka Hattori3, Yoshikuni Mizuno3, Kosik1, Dennis Selkoe1

1Center for Neurologic Diseases, Harvard Medical School, 77 Ave Louis

Pasteur, Boston, 02115, MA, United States; 2Centre for Neuroscience,

Flinders University, Flinders University, Bedford Park, Australia;

3Department of Neurology, Juntendo University Medical School, Juntendo

University, Tokyo, Japan

Session: Lewy-body related diseases - P4

SYNUCLEIN PROFILE IN APP23 TRANSGENIC MICE

Feldmann1, Oliver Wirths1, Matthias Staufenbiel2, Gerd Multhaup3,

Konrad Beyreuther4, Bayer1

1University of Bonn, Medical Center, Sigmund-Freud-Str.25, Bonn, 53105, NRW,

Germany; 2Nervous System Research, Novartis Pharma Inc., Klybachstr.141,

Basel, 4057, Basel, Switzerland; 3ZMBH Heidelberg, Im Neuenheimer Feld 282,

Heidelberg, 69120, Germany; 4ZMBH Heidelberg, Im Neuenheimer Feld 282,

Heidelberg, 69120, Basel, Germany

Session: Synucleins - P3

MOLECULAR CLONING OF A COMPLIMENTARY DNA ENCODING A NOVEL PROTEIN THAT

INTERACTS WITH ALPHA-SYNUCLEIN

Qiulan Ma1, M Abdul Alim1, M Shahanara Hossain1, Takako Aizawa1, Kunimasa

Arima2, Piu Chan3, Mitsunobu Yoshii1, Kenji Ueda1

1Department of Neural Plasticity, Tokyo Inst Psychiatry, 2-1-8 Kamikitazawa,

Setagaya, Tokyo, 156-8585, Japan; 2Department of Lab Med, NCHMNMD, NCNP,

4-1-1 Ogawahigashi, Kodaira, 187-8551, Tokyo, Japan; 3The Sino-Japan Joint

Lab on Neurodegenerative Diseases, Beijing Inst Geriatrics, Xuanwu Hosptl,

Capital Univ Med Sciences, 45 Chang Chun Street, Xuan Wu District, Beijing,

100053, China

Session: Synucleins - P3

------------------------------------------------

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Message: 3

Date: Fri, 19 Jul 2002 9:03:38 -0500

Subject: Re: Conference in Sweden

Bill:

The addition of dementia to the Dx is new by the new neurologist and we

need to spend more time to understand his basis for this addition. Terry's

memory and general cognitive skills are not bad as long as we stick to

single tasking without a lot of other confusing inputs. When her new nuero

gets backs from Sweden, I plan to follow up with him on his perspective on

Terry. I had a lot to digest in our first appointment. He also added

" psuedo PSP to the Dx mix, so we have a lot of ground to cover. He comes

with good credentials and I am quite impressed with his approach and

knowledge in our first encounter. Another nuero in the department thinks

her Dx is FTD. Also, the new nuero had not had a chance to see the results

of the SPECT scan done last October. So we have some work to do.

,

Actually dementia is very associated with PD and LBD (and the tau disorders

like PSP and CBGD) more so than MSA. In MSA the cognitive functions are

more affected as Fisher and others have pointed out - it is not memory

but the analyzing portion of the brain which seems to have problems.

In fact as the PD patient gets older, they are very likely to have at least

some dementia. I don't remember the exact figures but by 60 something like

10% of the PD patients had some form of dementia, by 70 y.o. up to about

30% and by 80-90 it was very high.

Am I miss-reading your email? You seem to be saying just the

opposite. The first neurologist who saw Charlotte and diagnosed her as PD

was more into Altzheimers than the movement disorder specialists we later

went to, but she was not as good at working with specific symptoms and

adjusting meds. Charlotte had no memory problems except when she had

infection. However, she could not process info (like driving) well at all

and felt the road was closing in on her.

But the night before she died, she told us not to put the trash out because

the Town does not pick up trash the day after Thanksgiving. That sort of

mental capability seems to be standard for MSA as many report the same

thing - slowed analyzation, but memory is fine.

Take care, Bill Werre

Sennewald Charlottesville, Virginia

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,

I understand the PSP :o) BUT you state FTD - to me that is Floral Telegraph

Delivery :o) I did did give Charlotte that at times as well as Anne Pledger and

Ford :o) But I thought it was flowers and not a brain disorder :o)

Hey do you want to get together in August? It will be near the Moller's if not

at their home.

Take care, Bill Werre

> another nuero in the department thinks

> her Dx is FTD. Also, the new nuero had not had a chance to see the results

> of the SPECT scan done last October. So we have some work to do.

>

> ,

>

>

> Actually dementia is very associated with PD and LBD (and the tau disorders

> like PSP and CBGD) more so than MSA. In MSA the cognitive functions are

> more affected as Fisher and others have pointed out - it is not memory

> but the analyzing portion of the brain which seems to have problems.

>

>

> In fact as the PD patient gets older, they are very likely to have at least

> some dementia. I don't remember the exact figures but by 60 something like

> 10% of the PD patients had some form of dementia, by 70 y.o. up to about

> 30% and by 80-90 it was very high.

>

>

> Am I miss-reading your email? You seem to be saying just the

> opposocs.yahoo.com/info/terms/

>

>

>

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FTD is Frontotemporal dementia.

http://www.alzwisc.org/frontotemporaldem.html

Re: Re: Conference in Sweden

> ,

>

> I understand the PSP :o) BUT you state FTD - to me that is Floral

Telegraph Delivery :o) I did did give Charlotte that at times as well as

Anne Pledger and Ford :o) But I thought it was flowers and not a

brain disorder :o)

>

> Hey do you want to get together in August? It will be near the Moller's

if not at their home.

>

> Take care, Bill Werre

>

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Bill:

You must be pulling my leg :). FTD I believe is Frontal temporal dementia.

At the moment August looks good on any Sunday.

Message: 14

Date: Fri, 19 Jul 2002 23:29:02 -0500

Subject: Re: Re: Conference in Sweden

,

I understand the PSP :o) BUT you state FTD - to me that is Floral Telegraph

Delivery :o) I did did give Charlotte that at times as well as Anne

Pledger and Ford :o) But I thought it was flowers and not a brain

disorder :o)

Hey do you want to get together in August? It will be near the Moller's if

not at their home.

Take care, Bill Werre

> another nuero in the department thinks

> her Dx is FTD. Also, the new nuero had not had a chance to see the results

> of the SPECT scan done last October. So we have some work to do.

>

> ,

>

Sennewald Charlottesville, Virginia

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Pam:

Thanks for the clarification. Based on that page, more credence may have

to be given to the FTD Dx by Terry's original neurologist. As you may

recall from an earlier post, her new nuero - this is no. 3, no. 2 left UVa,

and no. 1 referred her to no. 2 - has a little different twist on her Dx;

he is saying in initial appointment MSA/SND with psuedo PSP[whatever that

means], and dementia. As I told Bill, I have some more work to do with no.

3 nuero when he returns from Stockholm. Maybe it is one part FTD and one

part PSP and two parts MSA/SND. That goes along with your Neapolitan ice

cream metaphor.

I am confident nuero no. 3 is quite competent based on initial meeting.

Thanks again.

Message: 3

Date: Sat, 20 Jul 2002 09:05:38 -0300

Subject: Re: Re: Conference in Sweden

FTD is Frontotemporal dementia.

http://www.alzwisc.org/frontotemporaldem.html

Re: Re: Conference in Sweden

> ,

>

> I understand the PSP :o) BUT you state FTD - to me that is Floral

Telegraph Delivery :o) I did did give Charlotte that at times as well as

Anne Pledger and Ford :o) But I thought it was flowers and not a

brain disorder :o)

>

> Hey do you want to get together in August? It will be near the Moller's

if not at their home.

>

> Take care, Bill Werre

>

Sennewald Charlottesville, Virginia

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