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Good morning all,

As most here on the list know, Dr. Levesque’s of Cedar Sinai in Los Angles

California, is in the process of his second stage research of stem cell

study for Parkinson’s in January 2003 and also he is doing one patient with

MSA to see if it works as it did for the one patient of PD who is now

completely well and living a normal life.

If you don’t remember completely, he actually use’s your own stem cells to

generate many more and than reinserts them back into your own brain. In

doing so, there is no rejection because they are yours.

We just hope he is successful for if so, it will benefit so many. Also if

the single trial on the one patient who has MSA works, than the door will

perhaps be open. Of course this all takes time but he is a lot closer to

the cure than anyone else, so lets pray he has total success.

Listed below is an article that relates to fetal stem cell, that is so

controversy unlike Dr. Levesque’s procedure.

E-MOVE reports from the Seventh International Congress

of Parkinson's

Disease

and Movement Disorders, November 10-14 in Miami,

Florida. Poster (P) and

page

numbers are from Movement Disorders 2002;17(suppl 5).

Transplantation of fetal tissue does not improve

parkinsonian

disability, and

can cause off-medication dyskinesias, according to

results from a new

double-blind study presented in a platform session.

The lack of

symptomatic

benefit occurred despite significant improvements seen

with PET imaging.

Thirty-four patients were randomized to receive

bilateral grafting of 4

fetal

tissues per side, 1 tissue per side, or sham surgery

(partial burr hole

without penetration of the dura), similar to the

previous double-blind

surgical trial by Freed et al. Unlike that trial,

tissues were held for

less

than 48 hours before transplantation, and all patients

received

immunosuppression for six months after surgery. Other

differences

included

the number of tissues used (4 or 1 vs. 2), the target

site (posterior

putamen

vs. caudate and putamen), trial duration (24 vs. 12

months), and primary

outcome variable (UPDRS motor score vs. quality of

life). Fluorodopa

uptake

was assessed via PET imaging in a subset of patients.

Thirty-one patients completed the trial. Two patients

died during the

trial,

and 3 afterward, for causes unrelated to the

procedure. Post-mortem

examination was performed on all patients. While

placebo patients showed

virtually no tyrosine hydroxylase staining in the

striatum, transplanted

patients did, indicating striatal innervation. In

patients with 4

tissues

" the surrounding striatum was very well innervated, "

according to Dr.

Warren

Olanow, who presented the results.

PET results indicated a significant dose-dependent

increase versus

baseline

in fluorodopa uptake, with no change in placebo

patients and an

approximate

one-third increase in patients receiving 4 tissues.

Despite these histochemical and imaging improvements,

no significant

differences were seen in clinical measures. Increase

(worsening) from

baseline in the UPDRS motor score while off medication

was 9.4 for

placebo,

3.5 for 1 tissue, and -0.72 for 4 tissues (p=0.096 for

4 vs placebo).

Dr.

Olanow noted results for treated patients improved for

approximately 9

months, then worsened, possibly suggesting a delayed

immune response. No

differences were seen for on time without dyskinesias,

total off time,

ADL

scores, or levodopa dose required. Patients with

initially lower UPDRS

scores

did respond significantly better to treatment than to

placebo, while

those

with higher scores did not.

No placebo patients, but 13 of 23 treated patients,

developed

off-medication

dyskinesias, similar in kind to those seen in the

Freed trial. Three

patients

required surgical treatment to control them.

" Despite the hope and promise of open label trials,

fetal translation in

our

study failed to meet its primary or secondary

endpoints, " Dr. Olanow

concluded.

Best Regards,

" tenacity's man "

See our personal website and photo site:

http://www.pdhangout.com

http://community.webshots.com/user/tenacitywins

_________________________________________________________________

MSN 8 with e-mail virus protection service: 2 months FREE*

http://join.msn.com/?page=features/virus

Link to comment
Share on other sites

Good morning all,

As most here on the list know, Dr. Levesque’s of Cedar Sinai in Los Angles

California, is in the process of his second stage research of stem cell

study for Parkinson’s in January 2003 and also he is doing one patient with

MSA to see if it works as it did for the one patient of PD who is now

completely well and living a normal life.

If you don’t remember completely, he actually use’s your own stem cells to

generate many more and than reinserts them back into your own brain. In

doing so, there is no rejection because they are yours.

We just hope he is successful for if so, it will benefit so many. Also if

the single trial on the one patient who has MSA works, than the door will

perhaps be open. Of course this all takes time but he is a lot closer to

the cure than anyone else, so lets pray he has total success.

Listed below is an article that relates to fetal stem cell, that is so

controversy unlike Dr. Levesque’s procedure.

E-MOVE reports from the Seventh International Congress

of Parkinson's

Disease

and Movement Disorders, November 10-14 in Miami,

Florida. Poster (P) and

page

numbers are from Movement Disorders 2002;17(suppl 5).

Transplantation of fetal tissue does not improve

parkinsonian

disability, and

can cause off-medication dyskinesias, according to

results from a new

double-blind study presented in a platform session.

The lack of

symptomatic

benefit occurred despite significant improvements seen

with PET imaging.

Thirty-four patients were randomized to receive

bilateral grafting of 4

fetal

tissues per side, 1 tissue per side, or sham surgery

(partial burr hole

without penetration of the dura), similar to the

previous double-blind

surgical trial by Freed et al. Unlike that trial,

tissues were held for

less

than 48 hours before transplantation, and all patients

received

immunosuppression for six months after surgery. Other

differences

included

the number of tissues used (4 or 1 vs. 2), the target

site (posterior

putamen

vs. caudate and putamen), trial duration (24 vs. 12

months), and primary

outcome variable (UPDRS motor score vs. quality of

life). Fluorodopa

uptake

was assessed via PET imaging in a subset of patients.

Thirty-one patients completed the trial. Two patients

died during the

trial,

and 3 afterward, for causes unrelated to the

procedure. Post-mortem

examination was performed on all patients. While

placebo patients showed

virtually no tyrosine hydroxylase staining in the

striatum, transplanted

patients did, indicating striatal innervation. In

patients with 4

tissues

" the surrounding striatum was very well innervated, "

according to Dr.

Warren

Olanow, who presented the results.

PET results indicated a significant dose-dependent

increase versus

baseline

in fluorodopa uptake, with no change in placebo

patients and an

approximate

one-third increase in patients receiving 4 tissues.

Despite these histochemical and imaging improvements,

no significant

differences were seen in clinical measures. Increase

(worsening) from

baseline in the UPDRS motor score while off medication

was 9.4 for

placebo,

3.5 for 1 tissue, and -0.72 for 4 tissues (p=0.096 for

4 vs placebo).

Dr.

Olanow noted results for treated patients improved for

approximately 9

months, then worsened, possibly suggesting a delayed

immune response. No

differences were seen for on time without dyskinesias,

total off time,

ADL

scores, or levodopa dose required. Patients with

initially lower UPDRS

scores

did respond significantly better to treatment than to

placebo, while

those

with higher scores did not.

No placebo patients, but 13 of 23 treated patients,

developed

off-medication

dyskinesias, similar in kind to those seen in the

Freed trial. Three

patients

required surgical treatment to control them.

" Despite the hope and promise of open label trials,

fetal translation in

our

study failed to meet its primary or secondary

endpoints, " Dr. Olanow

concluded.

Best Regards,

" tenacity's man "

See our personal website and photo site:

http://www.pdhangout.com

http://community.webshots.com/user/tenacitywins

_________________________________________________________________

MSN 8 with e-mail virus protection service: 2 months FREE*

http://join.msn.com/?page=features/virus

Link to comment
Share on other sites

,

I have still not seen a clear description of HOW Dr. Levesque grows

(clones) your own stem cells or HOW he harvests live neurons (or stem

cells) from your brain. Do you have an article or scientific paper

describing this process? The article I read before, implied a SCNT form

of growth which does not use " fetal tissue " - HOWEVER it (SCNT) does use

stem cells from blastocysts or at least eggs. It was not clear, so I do

not know how it worked and I am interested.

In any case - it does use cell " cloning " so under laws proposed in

Congress at this time by Brownback (R-KA) it would be illegal in the USA

if that law passes. We must all look carefully at proposed laws, as they

CAN hurt us.

The fetal tissue transplant listed in your note sounds like the work

(1993-1995) which lead to the discovery of stem cells. The people in

that study were for the most part helped for 6-24 months - over 60% were

helped somewhat and only about 5% had more dyskinesia. Note that Sinemet

gives many people dyskinesia, but they accept dyskinesia in order to

keep movement. I know of a dozen people who want the movement allowed by

Sinemet and accept dyskinesia in order to keep some movement - as did

Charlotte. I also know of two people involved in the fetal tissue

transplant study and ASKED for the real cells after hearing of the risks

involved, so it was NOT a total failure. Note too, that PET exams showed

some increase in dopamine production in the people who actually got the

fetal tissue wheras the placebo group did not have the increase, showing

it did some good.

Take care, Bill Werre

==================================================================

Setzer wrote:

>Good morning all,

>

>As most here on the list know, Dr. Levesque's of Cedar Sinai in Los Angles

>California, is in the process of his second stage research of stem cell

>study for Parkinson's in January 2003 and also he is doing one patient with

>MSA to see if it works as it did for the one patient of PD who is now

>completely well and living a normal life.

>

>If you don't remember completely, he actually use's your own stem cells to

>generate many more and than reinserts them back into your own brain. In

>doing so, there is no rejection because they are yours.

>

>We just hope he is successful for if so, it will benefit so many. Also if

>the single trial on the one patient who has MSA works, than the door will

>perhaps be open. Of course this all takes time but he is a lot closer to

>the cure than anyone else, so lets pray he has total success.

>

>Listed below is an article that relates to fetal stem cell, that is so

>controversy unlike Dr. Levesque's procedure.

>

>E-MOVE reports from the Seventh International Congress

>of Parkinson's

>Disease

>and Movement Disorders, November 10-14 in Miami,

>Florida. Poster (P) and

>page

>numbers are from Movement Disorders 2002;17(suppl 5).

>

>

>Transplantation of fetal tissue does not improve

>parkinsonian

>disability, and

>can cause off-medication dyskinesias, according to

>results from a new

>double-blind study presented in a platform session.

>The lack of

>symptomatic

>benefit occurred despite significant improvements seen

>with PET imaging.

>

>Thirty-four patients were randomized to receive

>bilateral grafting of 4

>fetal

>tissues per side, 1 tissue per side, or sham surgery

>(partial burr hole

>without penetration of the dura), similar to the

>previous double-blind

>surgical trial by Freed et al. Unlike that trial,

>tissues were held for

>less

>than 48 hours before transplantation, and all patients

>received

>immunosuppression for six months after surgery. Other

>differences

>included

>the number of tissues used (4 or 1 vs. 2), the target

>site (posterior

>putamen

>vs. caudate and putamen), trial duration (24 vs. 12

>months), and primary

>outcome variable (UPDRS motor score vs. quality of

>life). Fluorodopa

>uptake

>was assessed via PET imaging in a subset of patients.

>

>Thirty-one patients completed the trial. Two patients

>died during the

>trial,

>and 3 afterward, for causes unrelated to the

>procedure. Post-mortem

>examination was performed on all patients. While

>placebo patients showed

>virtually no tyrosine hydroxylase staining in the

>striatum, transplanted

>patients did, indicating striatal innervation. In

>patients with 4

>tissues

> " the surrounding striatum was very well innervated, "

>according to Dr.

>Warren

>Olanow, who presented the results.

>

>PET results indicated a significant dose-dependent

>increase versus

>baseline

>in fluorodopa uptake, with no change in placebo

>patients and an

>approximate

>one-third increase in patients receiving 4 tissues.

>

>Despite these histochemical and imaging improvements,

>no significant

>differences were seen in clinical measures. Increase

>(worsening) from

>baseline in the UPDRS motor score while off medication

>was 9.4 for

>placebo,

>3.5 for 1 tissue, and -0.72 for 4 tissues (p=0.096 for

>4 vs placebo).

>Dr.

>Olanow noted results for treated patients improved for

>approximately 9

>months, then worsened, possibly suggesting a delayed

>immune response. No

>differences were seen for on time without dyskinesias,

>total off time,

>ADL

>scores, or levodopa dose required. Patients with

>initially lower UPDRS

>scores

>did respond significantly better to treatment than to

>placebo, while

>those

>with higher scores did not.

>

>No placebo patients, but 13 of 23 treated patients,

>developed

>off-medication

>dyskinesias, similar in kind to those seen in the

>Freed trial. Three

>patients

>required surgical treatment to control them.

>

> " Despite the hope and promise of open label trials,

>fetal translation in

>our

>study failed to meet its primary or secondary

>endpoints, " Dr. Olanow

>concluded.

>

>

>Best Regards,

> " tenacity's man "

>

>See our personal website and photo site:

>http://www.pdhangout.com

>http://community.webshots.com/user/tenacitywins

>

>

>

>

>_________________________________________________________________

>MSN 8 with e-mail virus protection service: 2 months FREE*

>http://join.msn.com/?page=features/virus

>

>

>If you do not wish to belong to shydrager, you may

>unsubscribe by sending a blank email to

>

>shydrager-unsubscribe

>

>

>

>

>

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Share on other sites

,

I have still not seen a clear description of HOW Dr. Levesque grows

(clones) your own stem cells or HOW he harvests live neurons (or stem

cells) from your brain. Do you have an article or scientific paper

describing this process? The article I read before, implied a SCNT form

of growth which does not use " fetal tissue " - HOWEVER it (SCNT) does use

stem cells from blastocysts or at least eggs. It was not clear, so I do

not know how it worked and I am interested.

In any case - it does use cell " cloning " so under laws proposed in

Congress at this time by Brownback (R-KA) it would be illegal in the USA

if that law passes. We must all look carefully at proposed laws, as they

CAN hurt us.

The fetal tissue transplant listed in your note sounds like the work

(1993-1995) which lead to the discovery of stem cells. The people in

that study were for the most part helped for 6-24 months - over 60% were

helped somewhat and only about 5% had more dyskinesia. Note that Sinemet

gives many people dyskinesia, but they accept dyskinesia in order to

keep movement. I know of a dozen people who want the movement allowed by

Sinemet and accept dyskinesia in order to keep some movement - as did

Charlotte. I also know of two people involved in the fetal tissue

transplant study and ASKED for the real cells after hearing of the risks

involved, so it was NOT a total failure. Note too, that PET exams showed

some increase in dopamine production in the people who actually got the

fetal tissue wheras the placebo group did not have the increase, showing

it did some good.

Take care, Bill Werre

==================================================================

Setzer wrote:

>Good morning all,

>

>As most here on the list know, Dr. Levesque's of Cedar Sinai in Los Angles

>California, is in the process of his second stage research of stem cell

>study for Parkinson's in January 2003 and also he is doing one patient with

>MSA to see if it works as it did for the one patient of PD who is now

>completely well and living a normal life.

>

>If you don't remember completely, he actually use's your own stem cells to

>generate many more and than reinserts them back into your own brain. In

>doing so, there is no rejection because they are yours.

>

>We just hope he is successful for if so, it will benefit so many. Also if

>the single trial on the one patient who has MSA works, than the door will

>perhaps be open. Of course this all takes time but he is a lot closer to

>the cure than anyone else, so lets pray he has total success.

>

>Listed below is an article that relates to fetal stem cell, that is so

>controversy unlike Dr. Levesque's procedure.

>

>E-MOVE reports from the Seventh International Congress

>of Parkinson's

>Disease

>and Movement Disorders, November 10-14 in Miami,

>Florida. Poster (P) and

>page

>numbers are from Movement Disorders 2002;17(suppl 5).

>

>

>Transplantation of fetal tissue does not improve

>parkinsonian

>disability, and

>can cause off-medication dyskinesias, according to

>results from a new

>double-blind study presented in a platform session.

>The lack of

>symptomatic

>benefit occurred despite significant improvements seen

>with PET imaging.

>

>Thirty-four patients were randomized to receive

>bilateral grafting of 4

>fetal

>tissues per side, 1 tissue per side, or sham surgery

>(partial burr hole

>without penetration of the dura), similar to the

>previous double-blind

>surgical trial by Freed et al. Unlike that trial,

>tissues were held for

>less

>than 48 hours before transplantation, and all patients

>received

>immunosuppression for six months after surgery. Other

>differences

>included

>the number of tissues used (4 or 1 vs. 2), the target

>site (posterior

>putamen

>vs. caudate and putamen), trial duration (24 vs. 12

>months), and primary

>outcome variable (UPDRS motor score vs. quality of

>life). Fluorodopa

>uptake

>was assessed via PET imaging in a subset of patients.

>

>Thirty-one patients completed the trial. Two patients

>died during the

>trial,

>and 3 afterward, for causes unrelated to the

>procedure. Post-mortem

>examination was performed on all patients. While

>placebo patients showed

>virtually no tyrosine hydroxylase staining in the

>striatum, transplanted

>patients did, indicating striatal innervation. In

>patients with 4

>tissues

> " the surrounding striatum was very well innervated, "

>according to Dr.

>Warren

>Olanow, who presented the results.

>

>PET results indicated a significant dose-dependent

>increase versus

>baseline

>in fluorodopa uptake, with no change in placebo

>patients and an

>approximate

>one-third increase in patients receiving 4 tissues.

>

>Despite these histochemical and imaging improvements,

>no significant

>differences were seen in clinical measures. Increase

>(worsening) from

>baseline in the UPDRS motor score while off medication

>was 9.4 for

>placebo,

>3.5 for 1 tissue, and -0.72 for 4 tissues (p=0.096 for

>4 vs placebo).

>Dr.

>Olanow noted results for treated patients improved for

>approximately 9

>months, then worsened, possibly suggesting a delayed

>immune response. No

>differences were seen for on time without dyskinesias,

>total off time,

>ADL

>scores, or levodopa dose required. Patients with

>initially lower UPDRS

>scores

>did respond significantly better to treatment than to

>placebo, while

>those

>with higher scores did not.

>

>No placebo patients, but 13 of 23 treated patients,

>developed

>off-medication

>dyskinesias, similar in kind to those seen in the

>Freed trial. Three

>patients

>required surgical treatment to control them.

>

> " Despite the hope and promise of open label trials,

>fetal translation in

>our

>study failed to meet its primary or secondary

>endpoints, " Dr. Olanow

>concluded.

>

>

>Best Regards,

> " tenacity's man "

>

>See our personal website and photo site:

>http://www.pdhangout.com

>http://community.webshots.com/user/tenacitywins

>

>

>

>

>_________________________________________________________________

>MSN 8 with e-mail virus protection service: 2 months FREE*

>http://join.msn.com/?page=features/virus

>

>

>If you do not wish to belong to shydrager, you may

>unsubscribe by sending a blank email to

>

>shydrager-unsubscribe

>

>

>

>

>

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Share on other sites

Bill,

Here is information for you and all to read. Some are Scientific and others

are News Paper and Journal articles. I hope all of you will take the time to

actually read everything that I have found. It is cutting edge w/o religeous

or moral problems/issues involved.

“tenacity’s man”

======================================================================

Since stem cells divide endlessly, a single sample started from a human

fetus could provide all that's needed. But the recipient's immune system

might attack these as foreign. Perhaps the patient's own body is a better

source of stem cells. At Cedars-Sinai, scientists isolate stem cells from

tissue saved during brain operations on Parkinson's patients. In the lab,

these stem cells produce new brain cells. These in turn mature into dopamine

makers, the specific kind of brain cells that people with Parkinson's lack.

Finally, they are put back into the patients' brains. Even if this works,

however, the approach has an obvious shortcoming. The only source of these

brain stem cells is the patient's own brain, not a particularly accessible

reservoir.

http://www.cryonics.org/May99.html

======================================================================

Washington, May 24, 2002

Neural Adult Stem Cells Alleviate Symptoms in Parkinson's Disease

Tremors and other Parkinson's disease symptoms have largely disappeared from

a California man since he underwent a procedure wherein doctors obtained

stem cells from his brain, grew them in the laboratory, and injected them

back into his brain in an attempt to treat his Parkinson's disease.

Levesque and his team at Cedars-Sinai Medical Center in Los Angeles

performed the work. They removed 50 to 100 cells from the brain of Dennis

, a San Clemente, CA man with Parkinson's, grew them in the laboratory

for several months, and then in March 1999 injected about six million of the

cells into the patient's brain. Dopamine, a chemical neurotransmitter, is

lacking in Parkinson's disease patients. With this treatment, there was an

initial increase in the man's brain of dopamine, which leveled off again a

year later, to pre-surgery levels. Still, an 83 percent reduction in

symptoms has persisted. " It's not just psychological. His motor improvement

is real. And the improvement is beyond the level for placebo effects, "

Levesque said. Mr. , the patient, has great praises for the procedure:

" Two years ago I couldn't put my contact lenses in without a big problem.

Now it's no problem. And I don't have to take any anti-rejection medication

because the cells are myself. " Because the cells are derived from the

patient himself, such a procedure precludes any need for

anti-immune-rejection medication, with its strong side effects. With such a

treatment, patients could " essentially grow their own cures from a few

starter cells taken from their own brains. " This research shows that human

embryos need not be killed in order to treat the diseases of others.

Sources: Weiss, Rick, " Stem Cell Transplant Works in Calif. Case;

Parkinson's Traits Largely Disappear, " Washington Post, April 9, 2002, Page

A8;

http://www.newsnet5.com/health/1488252/detail.html

http://www.techtv.com/news/scitech/story/0,24195,3381344,00.html

http://www.theratech.com/english/products/cell/neuro.html

http://www.neurosurgery.org/health/news/detail.asp?PressID=178

Medscape Medical News

Autologous Neural Stem Cells Improve PD Symptoms

NEW YORK (MedscapeWire) Apr 10 — Autologous neural stem cells improved motor

function and reduced levodopa requirements in a single patient with

Parkinson's disease (PD), according to data presented on April 8 at the 70th

annual meeting of the American Association of Neurological Surgeons (AANS)

in Chicago, Illinois.

" One of the most significant findings of this study was the patient's

combined clinical improvement over time, " study author Michel F. Lévesque,

MD, FRSC, says in a news release. " After six months of the transplantation

culture in vitro, the number of neural stem cells exceeded several

millions. " Lévesque is the director of Cedars-Sinai Medical Center's

Neurofunctional Surgery Center.

The patient was a 57-year-old man diagnosed at age 46 years with idiopathic

PD, initially responsive to dopaminergic agents. When his severe tremor

became disabling, neural stem cells were removed during a single

stereotactic craniotomy, expanded in vitro over several months, then

transplanted in the left putamen.

At 3 months after transplantation, the patient's motor scores on his usual

medications improved by 37% on blinded neurologic examination, and

fluoro-DOPA PET studies showed a 55.6% increase in dopamine uptake. At 1

year posttransplantation, his Unified Parkinson's Disease Rating Scale

(UPDRS) improved by 81% while on medication and 83% while off medication.

Equivalent levodopa intake was reduced by 50%.

Potential advantages of autologous neuronal transplantation as a treatment

in PD include avoidance of ethical concerns surrounding embryonic stem

cells, absence of immune reactions at the site of transplantation, improved

survival of grafted cells, lower risks of transmission of infectious

disease, and no need for immunosuppressants or steroids.

" This form of treatment has the potential for making neural stem cell

therapy acceptable and available to a large number of patients, " Lévesque

says.

Reviewed by D. Vogin, MD

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Share on other sites

Bill,

Here is information for you and all to read. Some are Scientific and others

are News Paper and Journal articles. I hope all of you will take the time to

actually read everything that I have found. It is cutting edge w/o religeous

or moral problems/issues involved.

“tenacity’s man”

======================================================================

Since stem cells divide endlessly, a single sample started from a human

fetus could provide all that's needed. But the recipient's immune system

might attack these as foreign. Perhaps the patient's own body is a better

source of stem cells. At Cedars-Sinai, scientists isolate stem cells from

tissue saved during brain operations on Parkinson's patients. In the lab,

these stem cells produce new brain cells. These in turn mature into dopamine

makers, the specific kind of brain cells that people with Parkinson's lack.

Finally, they are put back into the patients' brains. Even if this works,

however, the approach has an obvious shortcoming. The only source of these

brain stem cells is the patient's own brain, not a particularly accessible

reservoir.

http://www.cryonics.org/May99.html

======================================================================

Washington, May 24, 2002

Neural Adult Stem Cells Alleviate Symptoms in Parkinson's Disease

Tremors and other Parkinson's disease symptoms have largely disappeared from

a California man since he underwent a procedure wherein doctors obtained

stem cells from his brain, grew them in the laboratory, and injected them

back into his brain in an attempt to treat his Parkinson's disease.

Levesque and his team at Cedars-Sinai Medical Center in Los Angeles

performed the work. They removed 50 to 100 cells from the brain of Dennis

, a San Clemente, CA man with Parkinson's, grew them in the laboratory

for several months, and then in March 1999 injected about six million of the

cells into the patient's brain. Dopamine, a chemical neurotransmitter, is

lacking in Parkinson's disease patients. With this treatment, there was an

initial increase in the man's brain of dopamine, which leveled off again a

year later, to pre-surgery levels. Still, an 83 percent reduction in

symptoms has persisted. " It's not just psychological. His motor improvement

is real. And the improvement is beyond the level for placebo effects, "

Levesque said. Mr. , the patient, has great praises for the procedure:

" Two years ago I couldn't put my contact lenses in without a big problem.

Now it's no problem. And I don't have to take any anti-rejection medication

because the cells are myself. " Because the cells are derived from the

patient himself, such a procedure precludes any need for

anti-immune-rejection medication, with its strong side effects. With such a

treatment, patients could " essentially grow their own cures from a few

starter cells taken from their own brains. " This research shows that human

embryos need not be killed in order to treat the diseases of others.

Sources: Weiss, Rick, " Stem Cell Transplant Works in Calif. Case;

Parkinson's Traits Largely Disappear, " Washington Post, April 9, 2002, Page

A8;

http://www.newsnet5.com/health/1488252/detail.html

http://www.techtv.com/news/scitech/story/0,24195,3381344,00.html

http://www.theratech.com/english/products/cell/neuro.html

http://www.neurosurgery.org/health/news/detail.asp?PressID=178

Medscape Medical News

Autologous Neural Stem Cells Improve PD Symptoms

NEW YORK (MedscapeWire) Apr 10 — Autologous neural stem cells improved motor

function and reduced levodopa requirements in a single patient with

Parkinson's disease (PD), according to data presented on April 8 at the 70th

annual meeting of the American Association of Neurological Surgeons (AANS)

in Chicago, Illinois.

" One of the most significant findings of this study was the patient's

combined clinical improvement over time, " study author Michel F. Lévesque,

MD, FRSC, says in a news release. " After six months of the transplantation

culture in vitro, the number of neural stem cells exceeded several

millions. " Lévesque is the director of Cedars-Sinai Medical Center's

Neurofunctional Surgery Center.

The patient was a 57-year-old man diagnosed at age 46 years with idiopathic

PD, initially responsive to dopaminergic agents. When his severe tremor

became disabling, neural stem cells were removed during a single

stereotactic craniotomy, expanded in vitro over several months, then

transplanted in the left putamen.

At 3 months after transplantation, the patient's motor scores on his usual

medications improved by 37% on blinded neurologic examination, and

fluoro-DOPA PET studies showed a 55.6% increase in dopamine uptake. At 1

year posttransplantation, his Unified Parkinson's Disease Rating Scale

(UPDRS) improved by 81% while on medication and 83% while off medication.

Equivalent levodopa intake was reduced by 50%.

Potential advantages of autologous neuronal transplantation as a treatment

in PD include avoidance of ethical concerns surrounding embryonic stem

cells, absence of immune reactions at the site of transplantation, improved

survival of grafted cells, lower risks of transmission of infectious

disease, and no need for immunosuppressants or steroids.

" This form of treatment has the potential for making neural stem cell

therapy acceptable and available to a large number of patients, " Lévesque

says.

Reviewed by D. Vogin, MD

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