Guest guest Posted August 12, 2001 Report Share Posted August 12, 2001 As I was perusing the AMOS site--where I started the WLS journey--I was intrigued by the mortality rate difference between men and women. It seems that those getting any wls the number of women out# the men by 8:1, yet the mortality is far heavier on the male side. Why is this? Are they 1)heavier 2)unhealthier 3)older 4) do they ignore s/s of serioius complications? It was a curious thing to me, something that concerns me as well, because my hubby may be following me in this journey. All the more--do I want to research and prepare for his sake. I go back to the AMOS site infrequently, only to update my profile and seek out more DS info. It is heavily on the RNY side--and if it hadn't of been for me stumbling onto a profile of someone from my home state I would have never found out about the DS. It would behoove AMOS to provide more info on DS. It seems very limited. The discussions here border on debate sometimes--but the " critical thinking " and discerning natures are positives that benefit all who visit. As a nurse and staunch pt. advocate I push people to ask questions---and more questions---the time to ask them is before jumping headlong into something so life altering. So many worry about getting the " insurance " to foot the bill at the expense of good decision making on the choice of surgery and surgeon. Some may dislike the discussions here--but they elicit a broader view of what we think this surgery is all about--both risks and benefits. Pammi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 12, 2001 Report Share Posted August 12, 2001 As I was perusing the AMOS site--where I started the WLS journey--I was intrigued by the mortality rate difference between men and women. It seems that those getting any wls the number of women out# the men by 8:1, yet the mortality is far heavier on the male side. Why is this? Are they 1)heavier 2)unhealthier 3)older 4) do they ignore s/s of serioius complications? It was a curious thing to me, something that concerns me as well, because my hubby may be following me in this journey. All the more--do I want to research and prepare for his sake. I go back to the AMOS site infrequently, only to update my profile and seek out more DS info. It is heavily on the RNY side--and if it hadn't of been for me stumbling onto a profile of someone from my home state I would have never found out about the DS. It would behoove AMOS to provide more info on DS. It seems very limited. The discussions here border on debate sometimes--but the " critical thinking " and discerning natures are positives that benefit all who visit. As a nurse and staunch pt. advocate I push people to ask questions---and more questions---the time to ask them is before jumping headlong into something so life altering. So many worry about getting the " insurance " to foot the bill at the expense of good decision making on the choice of surgery and surgeon. Some may dislike the discussions here--but they elicit a broader view of what we think this surgery is all about--both risks and benefits. Pammi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 12, 2001 Report Share Posted August 12, 2001 Having all of the particulars, on all available treatments, is the only way to make an intelligent decision. The facts need to be applied to your own lifestyle, and habit, in order to make the best decision. Can you tolerate the lifestyle post surgery, or will it drive you to not comply, causing dangerous side effects? Without different personal opinions, there would not be horse races! Good luck! Sue . > As I was perusing the AMOS site--where I started the WLS journey--I > was intrigued by the mortality rate difference between men and women. > > It seems that those getting any wls the number of women out# the men > by 8:1, yet the mortality is far heavier on the male side. Why is > this? Are they 1)heavier 2)unhealthier 3)older 4) do they ignore s/s > of serioius complications? It was a curious thing to me, something > that concerns me as well, because my hubby may be following me in > this journey. All the more--do I want to research and prepare for his > sake. > > I go back to the AMOS site infrequently, only to update my profile > and seek out more DS info. It is heavily on the RNY side--and if it > hadn't of been for me stumbling onto a profile of someone from my > home state I would have never found out about the DS. It would > behoove AMOS to provide more info on DS. It seems very limited. > > The discussions here border on debate sometimes--but the " critical > thinking " and discerning natures are positives that benefit all who > visit. As a nurse and staunch pt. advocate I push people to ask > questions---and more questions---the time to ask them is before > jumping headlong into something so life altering. So many worry about > getting the " insurance " to foot the bill at the expense of good > decision making on the choice of surgery and surgeon. > > Some may dislike the discussions here--but they elicit a broader view > of what we think this surgery is all about--both risks and benefits. > > Pammi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 One reason for the higher mortality of males with surgery is that men are more likely to have central obesity and post op abdominal compartment syndrome. in Seattle It seems that those getting any wls the number of women out# the men > by 8:1, yet the mortality is far heavier on the male side. Why is > this? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 One reason for the higher mortality of males with surgery is that men are more likely to have central obesity and post op abdominal compartment syndrome. in Seattle It seems that those getting any wls the number of women out# the men > by 8:1, yet the mortality is far heavier on the male side. Why is > this? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 , Wonderful !!!! I am an 'apple', too ... ugh ... What on earth is 'abdominal compartment syndrome' ??? Doesn't sound good :-) Bye, Donna marym@... To: duodenalswitch 08/13/2001 cc: 09:36 AM Subject: Re: WLS Mortality Please respond to duodenalswitch One reason for the higher mortality of males with surgery is that men are more likely to have central obesity and post op abdominal compartment syndrome. in Seattle It seems that those getting any wls the number of women out# the men > by 8:1, yet the mortality is far heavier on the male side. Why is > this? ---------------------------------------------------------------------- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 , Wonderful !!!! I am an 'apple', too ... ugh ... What on earth is 'abdominal compartment syndrome' ??? Doesn't sound good :-) Bye, Donna marym@... To: duodenalswitch 08/13/2001 cc: 09:36 AM Subject: Re: WLS Mortality Please respond to duodenalswitch One reason for the higher mortality of males with surgery is that men are more likely to have central obesity and post op abdominal compartment syndrome. in Seattle It seems that those getting any wls the number of women out# the men > by 8:1, yet the mortality is far heavier on the male side. Why is > this? ---------------------------------------------------------------------- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 The way it was explained to me is that men tend to have most of their fat concentrated in their abdomen making it more difficult to conduct this surgery. Joe Frost spoke of the role that " Abdominal Compartment Syndrome " played in his surgical complications. He did not say whether it was more prevalent in men, but given the above information, I could see the relationship... from NE Philly, age 39 Open DS 6/27/01 - Dr. Pomp/Mt. Sinai/NYC - BMI 63 6 1/2 weeks out - 30+lbs down Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 The way it was explained to me is that men tend to have most of their fat concentrated in their abdomen making it more difficult to conduct this surgery. Joe Frost spoke of the role that " Abdominal Compartment Syndrome " played in his surgical complications. He did not say whether it was more prevalent in men, but given the above information, I could see the relationship... from NE Philly, age 39 Open DS 6/27/01 - Dr. Pomp/Mt. Sinai/NYC - BMI 63 6 1/2 weeks out - 30+lbs down Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 At 9:55 AM -0400 8/13/01, Donna Joostema wrote: >, > >Wonderful !!!! I am an 'apple', too ... ugh ... >What on earth is 'abdominal compartment syndrome' ??? Doesn't sound good >:-) > Rao Ivatury, MD, of the Medical College of Virginia reviewed the pathophysiology and diagnosis of intra-abdominal hypertension and intra-abdominal compartment syndrome which, he pointed out, are not synonymous terms.[2] Elevated intra-abdominal pressure with systemic signs and symptoms of that pressure results in ACS. IAH is the measurement of the pressure. The elevated pressure measurement, along with the associated clinical changes, constitutes ACS. Because these patients are repeatedly resuscitated they enter a cycle of ischemia/reperfusion, which is known to be deadly to organs; systemic inflammatory response syndrome appears initially, progressing to multiple organ dysfunction syndrome and finally death. Abdominal compartment syndrome was first described in the 1890s. Only over the last several years, however, have surgeons begun to clearly define the constellation of disorders associated with an excess of pressure in the abdominal cavity. ACS is likely to develop after any event that leads to an acute increase in the volume of abdominal contents sufficient to cause pressure-related organ dysfunction. As such, ACS can result from either blunt or penetrating trauma and surgery, as well as numerous medical conditions. It normally develops 12 to 24 hours after the first operation, especially after damage control. Intra-abdominal hypertension can develop from increased amounts of blood, other fluids, packing, and edematous bowel and other organs in the free abdominal cavity. Consequences of this increasing pressure appear gradually. Increased pressure leads to reduced hepatic arterial blood flow, decreases cardiac output, and can cause mucosal ischemia and acidosis, and eventually mesenteric thrombosis. Furthermore, studies have demonstrated that abdominal pressures of 15 to 20 mm Hg can produce oliguria, and in some models pressures beyond 20 mm Hg may lead to anuria. Dr. Ivatury cited a study by Lawrence Diebel, MD, from Wayne State University in Detroit, Mich, that demonstrated that a small elevation in intra-abdominal pressure results in decreased mesenteric artery flow and mucosal blood flow in the small intestine, despite maintenance of cardiac output.[3] Significant hemodynamic complications and compromises of intra-abdominal hypertension can translate to complications such as sepsis, multiorgan failure, and death. Once the pressure is brought down by opening the abdomen, these physiologic changes can be reversed. Diagnosis Classic signs of ACS are decreased PO2, very highly elevated PCO2, high peak inspiratory pressure, lack of urinary output, and a massively distended abdomen. A better way of diagnosing this condition, however, is through continuous intra-abdominal pressure monitoring in the intensive care unit (ICU) in all critically ill patients at high risk for these complications. Those at high risk for IAH and ACS include: Burn patients Patients with extensive abdominal trauma Patients with extensive intra-abdominal bleeding Patients with coagulopathies Patients with bowel ischemia Patients with packing Patients with massive colloidal and crystalloid resuscitation Patients with pancreatic abscesses -- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 At 9:55 AM -0400 8/13/01, Donna Joostema wrote: >, > >Wonderful !!!! I am an 'apple', too ... ugh ... >What on earth is 'abdominal compartment syndrome' ??? Doesn't sound good >:-) > Rao Ivatury, MD, of the Medical College of Virginia reviewed the pathophysiology and diagnosis of intra-abdominal hypertension and intra-abdominal compartment syndrome which, he pointed out, are not synonymous terms.[2] Elevated intra-abdominal pressure with systemic signs and symptoms of that pressure results in ACS. IAH is the measurement of the pressure. The elevated pressure measurement, along with the associated clinical changes, constitutes ACS. Because these patients are repeatedly resuscitated they enter a cycle of ischemia/reperfusion, which is known to be deadly to organs; systemic inflammatory response syndrome appears initially, progressing to multiple organ dysfunction syndrome and finally death. Abdominal compartment syndrome was first described in the 1890s. Only over the last several years, however, have surgeons begun to clearly define the constellation of disorders associated with an excess of pressure in the abdominal cavity. ACS is likely to develop after any event that leads to an acute increase in the volume of abdominal contents sufficient to cause pressure-related organ dysfunction. As such, ACS can result from either blunt or penetrating trauma and surgery, as well as numerous medical conditions. It normally develops 12 to 24 hours after the first operation, especially after damage control. Intra-abdominal hypertension can develop from increased amounts of blood, other fluids, packing, and edematous bowel and other organs in the free abdominal cavity. Consequences of this increasing pressure appear gradually. Increased pressure leads to reduced hepatic arterial blood flow, decreases cardiac output, and can cause mucosal ischemia and acidosis, and eventually mesenteric thrombosis. Furthermore, studies have demonstrated that abdominal pressures of 15 to 20 mm Hg can produce oliguria, and in some models pressures beyond 20 mm Hg may lead to anuria. Dr. Ivatury cited a study by Lawrence Diebel, MD, from Wayne State University in Detroit, Mich, that demonstrated that a small elevation in intra-abdominal pressure results in decreased mesenteric artery flow and mucosal blood flow in the small intestine, despite maintenance of cardiac output.[3] Significant hemodynamic complications and compromises of intra-abdominal hypertension can translate to complications such as sepsis, multiorgan failure, and death. Once the pressure is brought down by opening the abdomen, these physiologic changes can be reversed. Diagnosis Classic signs of ACS are decreased PO2, very highly elevated PCO2, high peak inspiratory pressure, lack of urinary output, and a massively distended abdomen. A better way of diagnosing this condition, however, is through continuous intra-abdominal pressure monitoring in the intensive care unit (ICU) in all critically ill patients at high risk for these complications. Those at high risk for IAH and ACS include: Burn patients Patients with extensive abdominal trauma Patients with extensive intra-abdominal bleeding Patients with coagulopathies Patients with bowel ischemia Patients with packing Patients with massive colloidal and crystalloid resuscitation Patients with pancreatic abscesses -- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 From what I read, or what I could understand of what I read :-), it doesn't really seem that this should be a huge concern of mine. Maybe I should borrow trouble elsewhere, but thanks for the information .... this was a new one for me !! Bye, Donna Steve Goldstein <steve-goldstein@c To: duodenalswitch ox.rr.com> cc: duodenalswitch , " Donna Joostema " 08/13/2001 11:00 Subject: Re: Re: WLS Mortality AM Please respond to duodenalswitch At 9:55 AM -0400 8/13/01, Donna Joostema wrote: >, > >Wonderful !!!! I am an 'apple', too ... ugh ... >What on earth is 'abdominal compartment syndrome' ??? Doesn't sound good >:-) > Rao Ivatury, MD, of the Medical College of Virginia reviewed the pathophysiology and diagnosis of intra-abdominal hypertension and intra-abdominal compartment syndrome which, he pointed out, are not synonymous terms.[2] Elevated intra-abdominal pressure with systemic signs and symptoms of that pressure results in ACS. IAH is the measurement of the pressure. The elevated pressure measurement, along with the associated clinical changes, constitutes ACS. Because these patients are repeatedly resuscitated they enter a cycle of ischemia/reperfusion, which is known to be deadly to organs; systemic inflammatory response syndrome appears initially, progressing to multiple organ dysfunction syndrome and finally death. Abdominal compartment syndrome was first described in the 1890s. Only over the last several years, however, have surgeons begun to clearly define the constellation of disorders associated with an excess of pressure in the abdominal cavity. ACS is likely to develop after any event that leads to an acute increase in the volume of abdominal contents sufficient to cause pressure-related organ dysfunction. As such, ACS can result from either blunt or penetrating trauma and surgery, as well as numerous medical conditions. It normally develops 12 to 24 hours after the first operation, especially after damage control. Intra-abdominal hypertension can develop from increased amounts of blood, other fluids, packing, and edematous bowel and other organs in the free abdominal cavity. Consequences of this increasing pressure appear gradually. Increased pressure leads to reduced hepatic arterial blood flow, decreases cardiac output, and can cause mucosal ischemia and acidosis, and eventually mesenteric thrombosis. Furthermore, studies have demonstrated that abdominal pressures of 15 to 20 mm Hg can produce oliguria, and in some models pressures beyond 20 mm Hg may lead to anuria. Dr. Ivatury cited a study by Lawrence Diebel, MD, from Wayne State University in Detroit, Mich, that demonstrated that a small elevation in intra-abdominal pressure results in decreased mesenteric artery flow and mucosal blood flow in the small intestine, despite maintenance of cardiac output.[3] Significant hemodynamic complications and compromises of intra-abdominal hypertension can translate to complications such as sepsis, multiorgan failure, and death. Once the pressure is brought down by opening the abdomen, these physiologic changes can be reversed. Diagnosis Classic signs of ACS are decreased PO2, very highly elevated PCO2, high peak inspiratory pressure, lack of urinary output, and a massively distended abdomen. A better way of diagnosing this condition, however, is through continuous intra-abdominal pressure monitoring in the intensive care unit (ICU) in all critically ill patients at high risk for these complications. Those at high risk for IAH and ACS include: Burn patients Patients with extensive abdominal trauma Patients with extensive intra-abdominal bleeding Patients with coagulopathies Patients with bowel ischemia Patients with packing Patients with massive colloidal and crystalloid resuscitation Patients with pancreatic abscesses -- ---------------------------------------------------------------------- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 From what I read, or what I could understand of what I read :-), it doesn't really seem that this should be a huge concern of mine. Maybe I should borrow trouble elsewhere, but thanks for the information .... this was a new one for me !! Bye, Donna Steve Goldstein <steve-goldstein@c To: duodenalswitch ox.rr.com> cc: duodenalswitch , " Donna Joostema " 08/13/2001 11:00 Subject: Re: Re: WLS Mortality AM Please respond to duodenalswitch At 9:55 AM -0400 8/13/01, Donna Joostema wrote: >, > >Wonderful !!!! I am an 'apple', too ... ugh ... >What on earth is 'abdominal compartment syndrome' ??? Doesn't sound good >:-) > Rao Ivatury, MD, of the Medical College of Virginia reviewed the pathophysiology and diagnosis of intra-abdominal hypertension and intra-abdominal compartment syndrome which, he pointed out, are not synonymous terms.[2] Elevated intra-abdominal pressure with systemic signs and symptoms of that pressure results in ACS. IAH is the measurement of the pressure. The elevated pressure measurement, along with the associated clinical changes, constitutes ACS. Because these patients are repeatedly resuscitated they enter a cycle of ischemia/reperfusion, which is known to be deadly to organs; systemic inflammatory response syndrome appears initially, progressing to multiple organ dysfunction syndrome and finally death. Abdominal compartment syndrome was first described in the 1890s. Only over the last several years, however, have surgeons begun to clearly define the constellation of disorders associated with an excess of pressure in the abdominal cavity. ACS is likely to develop after any event that leads to an acute increase in the volume of abdominal contents sufficient to cause pressure-related organ dysfunction. As such, ACS can result from either blunt or penetrating trauma and surgery, as well as numerous medical conditions. It normally develops 12 to 24 hours after the first operation, especially after damage control. Intra-abdominal hypertension can develop from increased amounts of blood, other fluids, packing, and edematous bowel and other organs in the free abdominal cavity. Consequences of this increasing pressure appear gradually. Increased pressure leads to reduced hepatic arterial blood flow, decreases cardiac output, and can cause mucosal ischemia and acidosis, and eventually mesenteric thrombosis. Furthermore, studies have demonstrated that abdominal pressures of 15 to 20 mm Hg can produce oliguria, and in some models pressures beyond 20 mm Hg may lead to anuria. Dr. Ivatury cited a study by Lawrence Diebel, MD, from Wayne State University in Detroit, Mich, that demonstrated that a small elevation in intra-abdominal pressure results in decreased mesenteric artery flow and mucosal blood flow in the small intestine, despite maintenance of cardiac output.[3] Significant hemodynamic complications and compromises of intra-abdominal hypertension can translate to complications such as sepsis, multiorgan failure, and death. Once the pressure is brought down by opening the abdomen, these physiologic changes can be reversed. Diagnosis Classic signs of ACS are decreased PO2, very highly elevated PCO2, high peak inspiratory pressure, lack of urinary output, and a massively distended abdomen. A better way of diagnosing this condition, however, is through continuous intra-abdominal pressure monitoring in the intensive care unit (ICU) in all critically ill patients at high risk for these complications. Those at high risk for IAH and ACS include: Burn patients Patients with extensive abdominal trauma Patients with extensive intra-abdominal bleeding Patients with coagulopathies Patients with bowel ischemia Patients with packing Patients with massive colloidal and crystalloid resuscitation Patients with pancreatic abscesses -- ---------------------------------------------------------------------- Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 The following is a direct quote from Dr. Anthone's interview on WebMD. (http://my.webmd.com/content/article/1700.50725) Thanks, , for posting this link!! " The medical contra-indications for obesity surgery are difficult to define, but once a patient becomes so limited so they can't even walk because of their obesity, the risk for dying, either during or after surgery increases dramatically. As a matter of fact, one patient out of 200 that has weight loss or obesity surgery dies. Now, that's taking all morbidly obese patients as a group. And the highest risk patient is one that cannot walk. They already have cardiopulmonary failure. That's what that means. They're usually seen in wheelchairs, or on little motor scooters. Their ambulatory or walking status is so inhibited because of their obesity that their heart and lungs can no longer oxygenate the tissues enough to walk. So, if they can't walk before surgery, they're sure not going to walk after surgery. And, when a patient can't get up and walk after surgery all kinds of complications take place. You develop pneumonias or pressure sores or blood clots in the legs that can dislodge and travel to the heart and cause the heart not to pump blood to your lungs. Those are all real indications for a patient dying after surgery. So, we have to determine who can make it through surgery and who can't. Unfortunately, bariatric surgeons, myself in particular because I'm at a university, we get referred patients all the time who are in that situation and their obesity is already life threatening. And, I consider it almost too high a risk to perform surgery, however, we do it sometimes. " Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 The following is a direct quote from Dr. Anthone's interview on WebMD. (http://my.webmd.com/content/article/1700.50725) Thanks, , for posting this link!! " The medical contra-indications for obesity surgery are difficult to define, but once a patient becomes so limited so they can't even walk because of their obesity, the risk for dying, either during or after surgery increases dramatically. As a matter of fact, one patient out of 200 that has weight loss or obesity surgery dies. Now, that's taking all morbidly obese patients as a group. And the highest risk patient is one that cannot walk. They already have cardiopulmonary failure. That's what that means. They're usually seen in wheelchairs, or on little motor scooters. Their ambulatory or walking status is so inhibited because of their obesity that their heart and lungs can no longer oxygenate the tissues enough to walk. So, if they can't walk before surgery, they're sure not going to walk after surgery. And, when a patient can't get up and walk after surgery all kinds of complications take place. You develop pneumonias or pressure sores or blood clots in the legs that can dislodge and travel to the heart and cause the heart not to pump blood to your lungs. Those are all real indications for a patient dying after surgery. So, we have to determine who can make it through surgery and who can't. Unfortunately, bariatric surgeons, myself in particular because I'm at a university, we get referred patients all the time who are in that situation and their obesity is already life threatening. And, I consider it almost too high a risk to perform surgery, however, we do it sometimes. " Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 Wow Steve--where did you find this? Reading that against what Larry was going through the days before with the decreased urinary output-- need I say any more? Thanks so much for posting this--puts me on my toes!! Pammi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 Wow Steve--where did you find this? Reading that against what Larry was going through the days before with the decreased urinary output-- need I say any more? Thanks so much for posting this--puts me on my toes!! Pammi Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 At 8:25 PM +0000 8/13/01, psbilyeu@... wrote: >Wow Steve--where did you find this? [abdominal compartment syndrome?] http://www.medscape.com >Reading that against what Larry >was going through the days before with the decreased urinary output-- >need I say any more? Scared the heck out of me when I read it, because my belly had been greatly distended for several days post-op. Most likely in my case it was because I had discontinued my diuretic. When I went back on, the belly came down. Nopw, BTW, my PCP cut the dose of the diuretic in half (HCTZ 50 -> 25 mg), and I have not lost weight in over a week; in fact, I may have added a pound of fluid. --Steve -- Steve Goldstein, age 61 Lap BPD/DS on May 2, 2001 Dr. Elariny, INOVA Fairfax Hospital, Virginia Starting (05/02/01) BMI = 51 BMI on 08/07 = 42 Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 >abdominal >compartment syndrome. I havn't heard of this before, what exactly is Post-oip Abdominal.... Quote Link to comment Share on other sites More sharing options...
Guest guest Posted August 13, 2001 Report Share Posted August 13, 2001 Aha! I knew I didn't have to look further for that answer. Thanks again Steve! > Rao Ivatury, MD, of the Medical College of Virginia reviewed the pathophysiology and diagnosis of intra-abdominal hypertension and intra-abdominal compartment syndrome which, he pointed out, are not synonymous terms.[2] Elevated intra-abdominal pressure with systemic signs and symptoms of that pressure results in ACS. IAH is the measurement of the pressure. The elevated pressure measurement, along with the associated clinical changes, constitutes ACS. Because these patients are repeatedly resuscitated they enter a cycle of ischemia/reperfusion, which is known to be deadly to organs; systemic inflammatory response syndrome appears initially, progressing to multiple organ dysfunction syndrome and finally death. Quote Link to comment Share on other sites More sharing options...
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