Need some advice on treatment for TED

[Guest guest]

Hi,

Thyroid eye disease can occur in Hashimoto's thyroiditis, in patients with

normal thyroid levels, in Graves' disease (autoimmune hyperthyroidism), and in

people with Hashitoxicosis.

It's common for patients with Graves' disease to have an initial period of

hypothyroidism. This can last for many years, requiring replacement hormone, or

it can be very mild and only realized in retrospect after hyperthyroidism has

developed. Graves' disease occurs when you start producing TSI, which are

stimulating TSH receptor antibodies. Thyroid eye disease (Graves'

ophthalmopathy)

is caused by both blocking and stimulating TSH receptor antibodies. Blocking

antibodies contribute to hypothyroidism.

Some people with Hashimoto's thyroiditis begin producing low levels of TSI in

a condition called Hashitoxicosis. Here, the TSI cause transient or temporary

hyper symptoms although the patient is primarily hypothyroidism. Because both

stimulating and blocking antibodies are present, the risk for TED is higher.

When people have equal amounts of blocking and stimulating TSH receptor

antibodies, they have normal thyroid function tests. However, they can have some

transient symptoms of hyperT and hypoT. When these people develop TED, they have

what's called euthyroid Graves' disease.

Right now, it sounds like you could be moving into Graves' disease or have

Hashitoxicosis. Since the antibodies can cause your thryoid hormone levels to

fluctuate, it can be difficult to tell what your true thyroid status is

especially when TSH alone is measured.

With records appearing in wrong patient files, it sounds like you probably

need to take charge of things. I have a number of articles on TED on

www.suite101.com and my book on it is available through www.trafford.com, for

ealth

Press. Take care, Elaine

[Guest guest]

can they tell that you have graves if they have never tested TSI?

Re: Need some advice on treatment for TED

Hi,

Thyroid eye disease can occur in Hashimoto's thyroiditis, in patients with

normal thyroid levels, in Graves' disease (autoimmune hyperthyroidism), and in

people with Hashitoxicosis.

It's common for patients with Graves' disease to have an initial period of

hypothyroidism. This can last for many years, requiring replacement hormone,

or

it can be very mild and only realized in retrospect after hyperthyroidism has

developed. Graves' disease occurs when you start producing TSI, which are

stimulating TSH receptor antibodies. Thyroid eye disease (Graves'

ophthalmopathy)

is caused by both blocking and stimulating TSH receptor antibodies. Blocking

antibodies contribute to hypothyroidism.

Some people with Hashimoto's thyroiditis begin producing low levels of TSI in

a condition called Hashitoxicosis. Here, the TSI cause transient or temporary

hyper symptoms although the patient is primarily hypothyroidism. Because both

stimulating and blocking antibodies are present, the risk for TED is higher.

When people have equal amounts of blocking and stimulating TSH receptor

antibodies, they have normal thyroid function tests. However, they can have

some

transient symptoms of hyperT and hypoT. When these people develop TED, they

have

what's called euthyroid Graves' disease.

Right now, it sounds like you could be moving into Graves' disease or have

Hashitoxicosis. Since the antibodies can cause your thryoid hormone levels to

fluctuate, it can be difficult to tell what your true thyroid status is

especially when TSH alone is measured.

With records appearing in wrong patient files, it sounds like you probably

need to take charge of things. I have a number of articles on TED on

www.suite101.com and my book on it is available through www.trafford.com, for

ealth

Press. Take care, Elaine