OT/morning sickness/Meleah

[Guest guest]

I got terribly sick with all of my pregnancies. I discovered with #3 the

involvement of h.pylori and debilitating morning sickness. I used natural

substances to fight that bacteria and was able to stop vomitting. More

recently, I read about the connection between high copper levels in the mom and

morning sickness. High copper also impacts the body's ability to keep bacteria

and yeast under control, so I think it all goes hand in hand. Women tend to

accumulate more copper with each pregnancy. Even though you might find red meat

repulsive right now, eating helps with the copper issue, so that is a simple

thing you might try to do. There are other more complicated things, but I just

wanted to let you know of a very simple one!!!

HTH,

robin wrote:

Great.

Meleah

Re: Re: glutamine and Glutathione

A natural way to increase glutathione levels is to eliminate the

pathogenic bacteria in our guts. LPS, the poison found in pathogenic

bacteria decreases glutathione levels.

SCD is a great way to eliminate the poisons from gut pathogens and

restore the glutathione levels. There is a famous doctor who is an

expert in detoxification and he now uses SCD for detoxification.

Below is a research article that demonstrates that bacterial poisons

decrease the level of glutathione.

Mimi

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra\

ctPlus&list_uids=17291629&query_hl=2&itool=pubmed_docsum

1: Neurochem Int. 2007 Mar;50(4):671-80. Epub 2007 Jan 13.

Altered glutathione homeostasis in animals prenatally exposed to

lipopolysaccharide.

* Zhu Y,

* Carvey PM,

* Ling Z.

Department of Pharmacology, Rush University Medical Center, 1735

West on Street, Chicago, IL 60612, USA.

We previously reported that injection of bacterial

lipopolysaccharide (LPS) into gravid female rats at embryonic day 10.5

resulted in a birth of offspring with fewer than normal dopamine (DA)

neurons along with innate immunity dysfunction and many

characteristics seen in Parkinson's disease (PD) patients. The

LPS-exposed animals were also more susceptible to secondary toxin

exposure as indicated by an accelerated DA neuron loss. Glutathione

(GSH) is an important antioxidant in the brain. A disturbance in

glutathione homeostasis has been proposed for the pathogenesis of PD.

In this study, animals prenatally exposed to LPS were studied along

with an acute intranigral LPS injection model for the status of

glutathione homeostasis, lipid peroxidation, and related enzyme

activities. Both prenatal LPS exposure and acute LPS injection

produced a significant GSH reduction and increase in oxidized GSH

(GSSG) and lipid peroxide (LPO) production. Activity of

gamma-glutamylcysteine synthetase (GCS), the rate-limiting enzyme in

de novo GSH synthesis, was up-regulated in acute supranigral LPS model

but was reduced in the prenatal LPS model. The GCS light subunit

protein expression was also down-regulated in prenatal LPS model. GSH

redox recycling enzyme activities (glutathione peroxidase, GPx and

glutathione reducdase, GR) and glutathione-S-transferase (GST),

gamma-glutamyl transpeptidase (gamma-GT) activities were all increased

in prenatal LPS model. Prenatal LPS exposure and aging synergized in

GSH level and GSH-related enzyme activities except for those (GR, GST,

and gamma-GT) with significant regional variations. Additionally,

prenatal LPS exposure produced a reduction of DA neuron count in the

substantia nigra (SN). These results suggest that prenatal LPS

exposure may cause glutathione homeostasis disturbance in offspring

brain and render DA neurons susceptible to the secondary neurotoxin

insult.

PMID: 17291629 [PubMed - in process]

>

> > >

> > > They are different. L-glutamine is an amino acid. It only causes

> > neurological damage if it is in excess and converts to glutimate.

> > This is one of those controversial issues. It does help the gut and

> > therefore has also helped many with neurological conditions.

> > >

> > > Glutathione is an anti-oxidant. It is often better to create

> > more glutathione in the body by giving it's precursers, rather than

> > taking glutathione itself.

> > >

> > > mom to broderick, asd, scd 19 months

>

> I am unable to explain as I am inexperienced with supplements.

> I got the definitions by doing an Internet search.

>

> Carol F.

>

>

[Guest guest]

Actually, I eat red meat very well during my pregnancies for some reason...as

long as I am not the one preparing it. What natural substances did you use for

the bacteria. I have no doubt that I'm one big toxic mess.

Meleah

Re: Re: glutamine and Glutathione

A natural way to increase glutathione levels is to eliminate the

pathogenic bacteria in our guts. LPS, the poison found in pathogenic

bacteria decreases glutathione levels.

SCD is a great way to eliminate the poisons from gut pathogens and

restore the glutathione levels. There is a famous doctor who is an

expert in detoxification and he now uses SCD for detoxification.

Below is a research article that demonstrates that bacterial poisons

decrease the level of glutathione.

Mimi

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra\

ctPlus&list_uids=17291629&query_hl=2&itool=pubmed_docsum

1: Neurochem Int. 2007 Mar;50(4):671-80. Epub 2007 Jan 13.

Altered glutathione homeostasis in animals prenatally exposed to

lipopolysaccharide.

* Zhu Y,

* Carvey PM,

* Ling Z.

Department of Pharmacology, Rush University Medical Center, 1735

West on Street, Chicago, IL 60612, USA.

We previously reported that injection of bacterial

lipopolysaccharide (LPS) into gravid female rats at embryonic day 10.5

resulted in a birth of offspring with fewer than normal dopamine (DA)

neurons along with innate immunity dysfunction and many

characteristics seen in Parkinson's disease (PD) patients. The

LPS-exposed animals were also more susceptible to secondary toxin

exposure as indicated by an accelerated DA neuron loss. Glutathione

(GSH) is an important antioxidant in the brain. A disturbance in

glutathione homeostasis has been proposed for the pathogenesis of PD.

In this study, animals prenatally exposed to LPS were studied along

with an acute intranigral LPS injection model for the status of

glutathione homeostasis, lipid peroxidation, and related enzyme

activities. Both prenatal LPS exposure and acute LPS injection

produced a significant GSH reduction and increase in oxidized GSH

(GSSG) and lipid peroxide (LPO) production. Activity of

gamma-glutamylcysteine synthetase (GCS), the rate-limiting enzyme in

de novo GSH synthesis, was up-regulated in acute supranigral LPS model

but was reduced in the prenatal LPS model. The GCS light subunit

protein expression was also down-regulated in prenatal LPS model. GSH

redox recycling enzyme activities (glutathione peroxidase, GPx and

glutathione reducdase, GR) and glutathione-S-transferase (GST),

gamma-glutamyl transpeptidase (gamma-GT) activities were all increased

in prenatal LPS model. Prenatal LPS exposure and aging synergized in

GSH level and GSH-related enzyme activities except for those (GR, GST,

and gamma-GT) with significant regional variations. Additionally,

prenatal LPS exposure produced a reduction of DA neuron count in the

substantia nigra (SN). These results suggest that prenatal LPS

exposure may cause glutathione homeostasis disturbance in offspring

brain and render DA neurons susceptible to the secondary neurotoxin

insult.

PMID: 17291629 [PubMed - in process]

>

> > >

> > > They are different. L-glutamine is an amino acid. It only causes

> > neurological damage if it is in excess and converts to glutimate.

> > This is one of those controversial issues. It does help the gut and

> > therefore has also helped many with neurological conditions.

> > >

> > > Glutathione is an anti-oxidant. It is often better to create

> > more glutathione in the body by giving it's precursers, rather than

> > taking glutathione itself.

> > >

> > > mom to broderick, asd, scd 19 months

>

> I am unable to explain as I am inexperienced with supplements.

> I got the definitions by doing an Internet search.

>

> Carol F.

>

>

[Guest guest]

Initially GSE, which I now feel is too much of a broad spectrum antimicrobial,

but I was desperate! The GSE did stop the vomiting though, so I knew I " killed "

something in there that was making me sick! After a week or so on GSE, I

switched to L-glutamine, which is the natural cure for ulcers/h. pylori -- but

it is suddenly not recommended during pregnancy for fear that in excess it will

convert to glutamate. I had a naturopath supervise me taking it & it really

helped. I also took DGL, a safe form of licorice (safe because it won't raise

your blood pressure). That really helped with the burning sensation in my

stomach. That's good that you can still eat meat, that should help.

Honestly, I could write a book of everything I tried to alleviate my morning

sickness. I even did hypnosis! The 3 things above worked best, because I was

concentrating on the h. pylori -- but you may find better ways to address it (an

alternative to GSE for instance). IMO, anything that helps get rid of that

particular bacteria will help! At some later time, you may want to address the

copper issue that is likely behind it all. A lot of high copper moms have ASD

kids.

-

robin wrote:

Actually, I eat red meat very well during my pregnancies for some

reason...as long as I am not the one preparing it. What natural substances did

you use for the bacteria. I have no doubt that I'm one big toxic mess.

Meleah

Re: Re: glutamine and Glutathione

A natural way to increase glutathione levels is to eliminate the

pathogenic bacteria in our guts. LPS, the poison found in pathogenic

bacteria decreases glutathione levels.

SCD is a great way to eliminate the poisons from gut pathogens and

restore the glutathione levels. There is a famous doctor who is an

expert in detoxification and he now uses SCD for detoxification.

Below is a research article that demonstrates that bacterial poisons

decrease the level of glutathione.

Mimi

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra\

ctPlus&list_uids=17291629&query_hl=2&itool=pubmed_docsum

1: Neurochem Int. 2007 Mar;50(4):671-80. Epub 2007 Jan 13.

Altered glutathione homeostasis in animals prenatally exposed to

lipopolysaccharide.

* Zhu Y,

* Carvey PM,

* Ling Z.

Department of Pharmacology, Rush University Medical Center, 1735

West on Street, Chicago, IL 60612, USA.

We previously reported that injection of bacterial

lipopolysaccharide (LPS) into gravid female rats at embryonic day 10.5

resulted in a birth of offspring with fewer than normal dopamine (DA)

neurons along with innate immunity dysfunction and many

characteristics seen in Parkinson's disease (PD) patients. The

LPS-exposed animals were also more susceptible to secondary toxin

exposure as indicated by an accelerated DA neuron loss. Glutathione

(GSH) is an important antioxidant in the brain. A disturbance in

glutathione homeostasis has been proposed for the pathogenesis of PD.

In this study, animals prenatally exposed to LPS were studied along

with an acute intranigral LPS injection model for the status of

glutathione homeostasis, lipid peroxidation, and related enzyme

activities. Both prenatal LPS exposure and acute LPS injection

produced a significant GSH reduction and increase in oxidized GSH

(GSSG) and lipid peroxide (LPO) production. Activity of

gamma-glutamylcysteine synthetase (GCS), the rate-limiting enzyme in

de novo GSH synthesis, was up-regulated in acute supranigral LPS model

but was reduced in the prenatal LPS model. The GCS light subunit

protein expression was also down-regulated in prenatal LPS model. GSH

redox recycling enzyme activities (glutathione peroxidase, GPx and

glutathione reducdase, GR) and glutathione-S-transferase (GST),

gamma-glutamyl transpeptidase (gamma-GT) activities were all increased

in prenatal LPS model. Prenatal LPS exposure and aging synergized in

GSH level and GSH-related enzyme activities except for those (GR, GST,

and gamma-GT) with significant regional variations. Additionally,

prenatal LPS exposure produced a reduction of DA neuron count in the

substantia nigra (SN). These results suggest that prenatal LPS

exposure may cause glutathione homeostasis disturbance in offspring

brain and render DA neurons susceptible to the secondary neurotoxin

insult.

PMID: 17291629 [PubMed - in process]

>

> > >

> > > They are different. L-glutamine is an amino acid. It only causes

> > neurological damage if it is in excess and converts to glutimate.

> > This is one of those controversial issues. It does help the gut and

> > therefore has also helped many with neurological conditions.

> > >

> > > Glutathione is an anti-oxidant. It is often better to create

> > more glutathione in the body by giving it's precursers, rather than

> > taking glutathione itself.

> > >

> > > mom to broderick, asd, scd 19 months

>

> I am unable to explain as I am inexperienced with supplements.

> I got the definitions by doing an Internet search.

>

> Carol F.

>

>

[Guest guest]

How would I know if I have high copper? When will I ever have a safe time to

address that? I assume I wouldn't want to do that when I'm pregnant or nursing

which is the case pretty much all the time. This is the first pregnancy that

didn't come along right on top of weening a baby. I weened in November,

and didn't get pregnant until January. I started up cycles in September.

Usually, I would start up cycles right around the time I ween, and then I'd end

up pregnant a month later. Of course, I don't contracept because I'm Catholic.

Well, not because I'm Catholic. Because it's wrong. I actually haven't usedc

it for aboutten years...when I found out what it does. Anyway, that's another

subject.

Meleah

Re: Re: glutamine and Glutathione

A natural way to increase glutathione levels is to eliminate the

pathogenic bacteria in our guts. LPS, the poison found in pathogenic

bacteria decreases glutathione levels.

SCD is a great way to eliminate the poisons from gut pathogens and

restore the glutathione levels. There is a famous doctor who is an

expert in detoxification and he now uses SCD for detoxification.

Below is a research article that demonstrates that bacterial poisons

decrease the level of glutathione.

Mimi

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra\

ctPlus&list_uids=17291629&query_hl=2&itool=pubmed_docsum

1: Neurochem Int. 2007 Mar;50(4):671-80. Epub 2007 Jan 13.

Altered glutathione homeostasis in animals prenatally exposed to

lipopolysaccharide.

* Zhu Y,

* Carvey PM,

* Ling Z.

Department of Pharmacology, Rush University Medical Center, 1735

West on Street, Chicago, IL 60612, USA.

We previously reported that injection of bacterial

lipopolysaccharide (LPS) into gravid female rats at embryonic day 10.5

resulted in a birth of offspring with fewer than normal dopamine (DA)

neurons along with innate immunity dysfunction and many

characteristics seen in Parkinson's disease (PD) patients. The

LPS-exposed animals were also more susceptible to secondary toxin

exposure as indicated by an accelerated DA neuron loss. Glutathione

(GSH) is an important antioxidant in the brain. A disturbance in

glutathione homeostasis has been proposed for the pathogenesis of PD.

In this study, animals prenatally exposed to LPS were studied along

with an acute intranigral LPS injection model for the status of

glutathione homeostasis, lipid peroxidation, and related enzyme

activities. Both prenatal LPS exposure and acute LPS injection

produced a significant GSH reduction and increase in oxidized GSH

(GSSG) and lipid peroxide (LPO) production. Activity of

gamma-glutamylcysteine synthetase (GCS), the rate-limiting enzyme in

de novo GSH synthesis, was up-regulated in acute supranigral LPS model

but was reduced in the prenatal LPS model. The GCS light subunit

protein expression was also down-regulated in prenatal LPS model. GSH

redox recycling enzyme activities (glutathione peroxidase, GPx and

glutathione reducdase, GR) and glutathione-S-transferase (GST),

gamma-glutamyl transpeptidase (gamma-GT) activities were all increased

in prenatal LPS model. Prenatal LPS exposure and aging synergized in

GSH level and GSH-related enzyme activities except for those (GR, GST,

and gamma-GT) with significant regional variations. Additionally,

prenatal LPS exposure produced a reduction of DA neuron count in the

substantia nigra (SN). These results suggest that prenatal LPS

exposure may cause glutathione homeostasis disturbance in offspring

brain and render DA neurons susceptible to the secondary neurotoxin

insult.

PMID: 17291629 [PubMed - in process]

>

> > >

> > > They are different. L-glutamine is an amino acid. It only causes

> > neurological damage if it is in excess and converts to glutimate.

> > This is one of those controversial issues. It does help the gut and

> > therefore has also helped many with neurological conditions.

> > >

> > > Glutathione is an anti-oxidant. It is often better to create

> > more glutathione in the body by giving it's precursers, rather than

> > taking glutathione itself.

> > >

> > > mom to broderick, asd, scd 19 months

>

> I am unable to explain as I am inexperienced with supplements.

> I got the definitions by doing an Internet search.

>

> Carol F.

>

>

[Guest guest]

It is not harmful to detox copper while pregnant and is beneficial to the baby,

per the few doctors who have studied it (primarily Dr. Eck & Dr. Larry

-- if you want to google them a bit). I would take a dietary approach

right now, which is lots of red meat and avoid dairy and some other high copper

foods (I'll look them up when I get a chance). I will send you information on

copper toxicity (testing, etc.). To a certain extent, you can dx it by symptoms

(especially in women who are most likely to have a copper issue) & getting

sicker on a later pregnancy in itself is a symptom. Today's a busy day, but

I'll get some stuff to you in the next couple of days.

-

robin wrote:

How would I know if I have high copper? When will I ever have a safe

time to address that? I assume I wouldn't want to do that when I'm pregnant or

nursing which is the case pretty much all the time. This is the first pregnancy

that didn't come along right on top of weening a baby. I weened in

November, and didn't get pregnant until January. I started up cycles in

September. Usually, I would start up cycles right around the time I ween, and

then I'd end up pregnant a month later. Of course, I don't contracept because

I'm Catholic. Well, not because I'm Catholic. Because it's wrong. I actually

haven't usedc it for aboutten years...when I found out what it does. Anyway,

that's another subject.

Meleah

Re: Re: glutamine and Glutathione

A natural way to increase glutathione levels is to eliminate the

pathogenic bacteria in our guts. LPS, the poison found in pathogenic

bacteria decreases glutathione levels.

SCD is a great way to eliminate the poisons from gut pathogens and

restore the glutathione levels. There is a famous doctor who is an

expert in detoxification and he now uses SCD for detoxification.

Below is a research article that demonstrates that bacterial poisons

decrease the level of glutathione.

Mimi

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra\

ctPlus&list_uids=17291629&query_hl=2&itool=pubmed_docsum

1: Neurochem Int. 2007 Mar;50(4):671-80. Epub 2007 Jan 13.

Altered glutathione homeostasis in animals prenatally exposed to

lipopolysaccharide.

* Zhu Y,

* Carvey PM,

* Ling Z.

Department of Pharmacology, Rush University Medical Center, 1735

West on Street, Chicago, IL 60612, USA.

We previously reported that injection of bacterial

lipopolysaccharide (LPS) into gravid female rats at embryonic day 10.5

resulted in a birth of offspring with fewer than normal dopamine (DA)

neurons along with innate immunity dysfunction and many

characteristics seen in Parkinson's disease (PD) patients. The

LPS-exposed animals were also more susceptible to secondary toxin

exposure as indicated by an accelerated DA neuron loss. Glutathione

(GSH) is an important antioxidant in the brain. A disturbance in

glutathione homeostasis has been proposed for the pathogenesis of PD.

In this study, animals prenatally exposed to LPS were studied along

with an acute intranigral LPS injection model for the status of

glutathione homeostasis, lipid peroxidation, and related enzyme

activities. Both prenatal LPS exposure and acute LPS injection

produced a significant GSH reduction and increase in oxidized GSH

(GSSG) and lipid peroxide (LPO) production. Activity of

gamma-glutamylcysteine synthetase (GCS), the rate-limiting enzyme in

de novo GSH synthesis, was up-regulated in acute supranigral LPS model

but was reduced in the prenatal LPS model. The GCS light subunit

protein expression was also down-regulated in prenatal LPS model. GSH

redox recycling enzyme activities (glutathione peroxidase, GPx and

glutathione reducdase, GR) and glutathione-S-transferase (GST),

gamma-glutamyl transpeptidase (gamma-GT) activities were all increased

in prenatal LPS model. Prenatal LPS exposure and aging synergized in

GSH level and GSH-related enzyme activities except for those (GR, GST,

and gamma-GT) with significant regional variations. Additionally,

prenatal LPS exposure produced a reduction of DA neuron count in the

substantia nigra (SN). These results suggest that prenatal LPS

exposure may cause glutathione homeostasis disturbance in offspring

brain and render DA neurons susceptible to the secondary neurotoxin

insult.

PMID: 17291629 [PubMed - in process]

>

> > >

> > > They are different. L-glutamine is an amino acid. It only causes

> > neurological damage if it is in excess and converts to glutimate.

> > This is one of those controversial issues. It does help the gut and

> > therefore has also helped many with neurological conditions.

> > >

> > > Glutathione is an anti-oxidant. It is often better to create

> > more glutathione in the body by giving it's precursers, rather than

> > taking glutathione itself.

> > >

> > > mom to broderick, asd, scd 19 months

>

> I am unable to explain as I am inexperienced with supplements.

> I got the definitions by doing an Internet search.

>

> Carol F.

>

>

[Guest guest]

Thanks. I haven't been doing dairy nearly as much as I used to since we've

switched iel to SCD. I used to have lots of dairy...milk in cereal plus

at meals, cheese, ice cream...Thanks for info.

Meleah

Re: Re: glutamine and Glutathione

A natural way to increase glutathione levels is to eliminate the

pathogenic bacteria in our guts. LPS, the poison found in pathogenic

bacteria decreases glutathione levels.

SCD is a great way to eliminate the poisons from gut pathogens and

restore the glutathione levels. There is a famous doctor who is an

expert in detoxification and he now uses SCD for detoxification.

Below is a research article that demonstrates that bacterial poisons

decrease the level of glutathione.

Mimi

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstra\

ctPlus&list_uids=17291629&query_hl=2&itool=pubmed_docsum

1: Neurochem Int. 2007 Mar;50(4):671-80. Epub 2007 Jan 13.

Altered glutathione homeostasis in animals prenatally exposed to

lipopolysaccharide.

* Zhu Y,

* Carvey PM,

* Ling Z.

Department of Pharmacology, Rush University Medical Center, 1735

West on Street, Chicago, IL 60612, USA.

We previously reported that injection of bacterial

lipopolysaccharide (LPS) into gravid female rats at embryonic day 10.5

resulted in a birth of offspring with fewer than normal dopamine (DA)

neurons along with innate immunity dysfunction and many

characteristics seen in Parkinson's disease (PD) patients. The

LPS-exposed animals were also more susceptible to secondary toxin

exposure as indicated by an accelerated DA neuron loss. Glutathione

(GSH) is an important antioxidant in the brain. A disturbance in

glutathione homeostasis has been proposed for the pathogenesis of PD.

In this study, animals prenatally exposed to LPS were studied along

with an acute intranigral LPS injection model for the status of

glutathione homeostasis, lipid peroxidation, and related enzyme

activities. Both prenatal LPS exposure and acute LPS injection

produced a significant GSH reduction and increase in oxidized GSH

(GSSG) and lipid peroxide (LPO) production. Activity of

gamma-glutamylcysteine synthetase (GCS), the rate-limiting enzyme in

de novo GSH synthesis, was up-regulated in acute supranigral LPS model

but was reduced in the prenatal LPS model. The GCS light subunit

protein expression was also down-regulated in prenatal LPS model. GSH

redox recycling enzyme activities (glutathione peroxidase, GPx and

glutathione reducdase, GR) and glutathione-S-transferase (GST),

gamma-glutamyl transpeptidase (gamma-GT) activities were all increased

in prenatal LPS model. Prenatal LPS exposure and aging synergized in

GSH level and GSH-related enzyme activities except for those (GR, GST,

and gamma-GT) with significant regional variations. Additionally,

prenatal LPS exposure produced a reduction of DA neuron count in the

substantia nigra (SN). These results suggest that prenatal LPS

exposure may cause glutathione homeostasis disturbance in offspring

brain and render DA neurons susceptible to the secondary neurotoxin

insult.

PMID: 17291629 [PubMed - in process]

>

> > >

> > > They are different. L-glutamine is an amino acid. It only causes

> > neurological damage if it is in excess and converts to glutimate.

> > This is one of those controversial issues. It does help the gut and

> > therefore has also helped many with neurological conditions.

> > >

> > > Glutathione is an anti-oxidant. It is often better to create

> > more glutathione in the body by giving it's precursers, rather than

> > taking glutathione itself.

> > >

> > > mom to broderick, asd, scd 19 months

>

> I am unable to explain as I am inexperienced with supplements.

> I got the definitions by doing an Internet search.

>

> Carol F.

>

>