Guest guest Posted June 15, 2008 Report Share Posted June 15, 2008 I have searched for the answer to this question using onibasu but have found conflicting answers. I want to increase stomach acid to help with absorption but if I take TMG is this enough on it's own or should I add some Betaine Hcl as well? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 15, 2008 Report Share Posted June 15, 2008 TMG will not do anything for stomach acid, it's different than HCI. I got them confused also, . Does TMG increase stomach acid? I have searched for the answer to this question using onibasu but have found conflicting answers. I want to increase stomach acid to help with absorption but if I take TMG is this enough on it's own or should I add some Betaine Hcl as well? Quote Link to comment Share on other sites More sharing options...
Guest guest Posted June 22, 2008 Report Share Posted June 22, 2008 >, what are acetylcholine precursors and acetylcholinesterase >inhibitors? I'm a little confused about the relationship with TMG? > There is no relationship (that I'm aware of) between TMG and ACh (acetylcholine) precursors/acetylcholinesterase (AChE) inhibitors regarding effects on stomach acidity – I mentioned acetylcholine elevating agents, because they enable some people to boost their cognitive functioning (a nice bonus) as well as their stomach acidity. I believe I may have realized why you're experiencing some confusion regarding TMG and stomach acid. As you know, when people are suffering from hypochlordhydria (stomach acid deficiency), a reliable therapy involves supplementing (approx. mid-way through meals) with betaine (or glutamic acid) HCl. HCl (as a dietary supplement is always chelated to another molecule (e.g., betaine or glutamic acid). Importantly, the betaine (or glutamic acid) component has no effect on stomach ph – it just goes for the ride. This is relevant, because betaine (anhydrous) is also known as TMG. So, if you had someone who understood that TMG and betaine are one in the same, but didn't understand that the reason betaine HCl supplementation is an effective means of increasing stomach acidity is because of the HCl moiety (and not the betaine), then they could erroneously conclude that TMG (betaine anhydrous) can be used to decrease stomach ph. An ACh (acetylcholine) precursor is, simply, something that converts into acetylcholine within the body. Some examples include 1) choline (as bitartrate, chloride, or citrate), 2) DMAE/centrophenoxine, 3) phosphatidylcholine (a constituent of lecithin), 4) CDP-choline, and 5) alpha-GPC. The first one does not cross the BBB (blood brain barrier) very well and is also the most likely to produce GI issues. Four & five are less selective than the others (i.e., they increase brain dopamine (DA) and norepinephrine (NE) concentrations as well as ACh). Some people benefit from DA/NE increases, and others do not. So, I can offer a few options to aid in selection: 1) take the Braverman Nature Assessment (which provides insight into potential neurotransmitter deficiencies/excesses), 2) read " In Search of a Designer Brain (part 1 & 2) " by Fowkes to increase your understanding of which neurotransmitters affect which mood/behavioral states – this would allow you to speculate about which neurotransmitter(s) you may want to increase, or 3) try the different precursors, and see which one(s) best agree(s) with your system. It is worth mentioning that some research begs the question, " Is DMAE truly a legitimate ACh precursor? " Well, even if one were to overlook the excessive muscle tension that results from DMAE overdoses (a sign of cholinergic excess), any respectable biochemist knows that Dimethylaminoethanol (DMAE) becomes trimethylaminoethanol (choline) when it combines with an available methyl group (after crossing the BBB). I would use an ACh precursor over an AChE inhibitor unless Alzheimer's dementia (or a related condition) rendered one incapable of synthesizing ACh adequately (due to acetyltransferase deficiency). Of course, if it weren't for un-/improperly treated chronic mercury toxicity, Alzheimer's dementia may not exist. If anyone knows of an Alzheimer's victim with orderly mineral transport, please let me know. Sincerely, Quote Link to comment Share on other sites More sharing options...
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