Re: RECOVERYMAZE OXALATES CALCIUM SUPP

[Guest guest]

Dear Rob's mom,

If you have problems with high oxalate foods,then you might want to eat yogurt

or take calcium supplements with meals . (However,the yogurt is so much better.)

However,please be aware that there are many reasons as to why a food

may be problematic. Nuts,beans,cashews and peanut butter are all high

oxalate foods but they are also hard to digest. Another reason a child

would not tolerate these foods might be an allergy to nuts and

peanuts. If you are not getting great results with SCD,then try to

eliminate the nuts,beans,cashews and peanut butter first before

eliminating all the other high oxalate foods. Many high oxalate fruits

and vegetables are very nutricious and your child may need them to

heal.

Most ASD children have an oxalate problem because of a damaged gut.

Your first priority should be to heal the gut with SCD. The SCD yogurt

is your best ally to heal the gut,don't give up on SCD without trying

the yogurt!

Clarification for newcomers to our list:

Most children with autism get good results with dairy free SCD. Doing

dairy free SCD is worth a million dollars but SCD with goat yogurt is

worth 2 million dollars. LOL

I hope to soon post about the safety of goat yogurt for children with autism.

Mimi

> Dear recoverymaze

> So could you spell it out again for me. Do we want to take calcium

> supplement swith high or low oxalate foods?

> Thank you Rob's mom

>

> WEBSITE LINKS CORRECTION/Re: Oxylates, Phenol,

> Salicylate and Food Intolerances and SCDiet

>

>

> I just corrected the website links.

> http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed & cmd=Retrieve & dop

> t=Abstract & list_uids=9092314 & query_hl=5 & itool=pubmed_docsum

>

> factors affecting the risk for kidney stones in women

>

> 1: Ann Intern Med. 1997 Apr 1;126(7):497-504.

>

> Comparison of dietary calcium with supplemental calcium and other

> nutrients as factors affecting the risk for kidney stones in women.

>

> Curhan GC, Willett WC, Speizer FE, Spiegelman D, Stampfer MJ.

>

> Department of Nutrition, Harvard School of Public Health, Boston, MA

> 02115, USA.

>

> BACKGROUND: Calcium intake is believed to play an important role in

> the formation of kidney stones, but data on the risk factors for stone

> formation in women are limited. OBJECTIVE: To examine the association

> between intake of dietary and supplemental calcium and the risk for

> kidney stones in women. DESIGN: Prospective cohort study with 12-year

> follow-up. SETTING: Several U.S. states. PARTICIPANTS: 91,731 women

> participating in the Nurses' Health Study I who were 34 to 59 years of

> age in 1980 and had no history of kidney stones. MEASUREMENTS:

> Self-administered food-frequency questionnaires were used to assess

> diet in 1980, 1984, 1986, and 1990. The main outcome measure was

> incident symptomatic kidney stones. RESULTS: During 903,849

> person-years of follow-up, 864 cases of kidney stones were documented.

> After adjustment for potential risk factors, intake of dietary calcium

> was inversely associated with risk for kidney stones and intake of

> supplemental calcium was positively associated with risk. The relative

> risk for stone formation in women in the highest quintile of dietary

> calcium intake compared with women in the lowest quintile was 0.65

> (95% CI, 0.50 to 0.83). The relative risk in women who took

> supplemental calcium compared with women who did not was 1.20 (CI,

> 1.02 to 1.41). In 67% of women who took supplemental calcium, the

> calcium either was not consumed with a meal or was consumed with meals

> whose oxalate content was probably low. Other dietary factors showed

> the following relative risks among women in the highest quintile of

> intake compared with those in the lowest quintile: sucrose, 1.52 (CI,

> 1.18 to 1.96); sodium, 1.30 (CI, 1.05 to 1.62); fluid, 0.61 (CI, 0.48

> to 0.78); and potassium, 0.65 (CI, 0.51 to 0.84). CONCLUSIONS: High

> intake of dietary calcium appears to decrease risk for symptomatic

> kidney stones, whereas intake of supplemental calcium may increase

> risk. Because dietary calcium reduces the absorption of oxalate, the

> apparently different effects caused by the type of calcium may be

> associated with the timing of calcium ingestion relative to the amount

> of oxalate consumed. However, other factors present in dairy products

> (the major source of dietary calcium) could be responsible for the

> decreased risk seen with dietary calcium.

>

> PMID: 9092314 [PubMed - indexed for MEDLINE]

>

> [2]

>

>

>

> http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed & cmd=Retrieve & dop

> t=Abstract & list_uids=9761503 & query_hl=8 & itool=pubmed_docsum

>

> 1: Nephrol Dial Transplant. 1998 Sep;13(9):2241-7

>

> . High-calcium intake abolishes hyperoxaluria and reduces urinary

> crystallization during a 20-fold normal oxalate load in humans.

>

> Hess B, Jost C, Zipperle L, Takkinen R, Jaeger P.

>

> Department of Medicine, University Hospital, Berne, Switzerland.

>

> BACKGROUND: The aim of the study was to test whether increasing

> dietary calcium intake lowers intestinal oxalate absorption and

> thereby prevents hyperoxaluria and urinary crystallization during a

> 20-fold normal oxalate load in healthy subjects. METHODS: Fourteen

> healthy male volunteers (age 23-44 years, BMI 21.5-27.7 kg/m2)

> collected 24-h urines while on free-choice diet as well as on two

> standardized diets. The latter contained 2545 kcal, 2500 ml of mineral

> water, 102 g of protein, 13.6 g of sodium chloride and 2220 mg of

> oxalate (approximately 20-fold content of an average diet). Subjects

> were studied twice while on the standardized diet, once while eating a

> normal amount of calcium (1211 mg/day, oxalate-rich diet), and once

> while eating 3858 mg of calcium/day (calcium and oxalate-rich diet).

> RESULTS: Compared with the free-choice diet (322+/-36 micromol/d), UOx

> x V increased to 780+/-72 micromol/d on the oxalate-rich diet

> (P=0.001) and fell again to 326+/-31 micromol/d on calcium and

> oxalate-rich diet (P=0.001 vs oxalate-rich diet). Urinary glycolate (a

> metabolic precursor of Ox) always remained below the upper limit of

> the normal range and did not change between different diets,

> indicating that changes in UOX x V reflect respective variations in

> intestinal absorption of Ox. Uca x V was 4.60+/-0.45 mmol/d on the

> free-choice diet and 3.20+/-0.32 mmol/d on the oxalate-rich diet

> (P=0.011 vs free-choice diet); it increased to 7.28+/-0.74 mmol/d on

> the calcium- and oxalate-rich diet (P=0.001 vs free-choice and

> oxalate-rich diets). As indicated by the AP (CaOx) index (Tiselius),

> urinary supersaturation did not vary significantly between the three

> diets. In freshly voided morning urines (studied in 8/14 subjects) on

> the oxalate-rich diet, CaOx crystals or crystal aggregates of up to 80

> microm diameter were found in 5/8 urines, whereas this never occurred

> on the free-choice diet and only t once on the calcium- and

> oxalate-rich diet. CONCLUSION: Increasing calcium intake while eating

> Ox-rich food prevents dietary hyperoxaluria and reduces CaOx

> crystallization in healthy subjects. This further illustrates that

> dietary counseling to idiopathic calcium-stone formers should ensure

> sufficient calcium intake, especially during oxalate-rich meals.

>

> Publication Types:

>

> * Clinical Trial

>

> * Randomized Controlled Trial

>

> PMID: 9761503 [PubMed - indexed for MEDLINE]

>

>

>

> For information on the Specific Carbohydrate Diet, please read the book

> _Breaking the Vicious Cycle_ by Elaine Gottschall and read the

> following websites:

> http://www.breakingtheviciouscycle.info

> and

> http://www.pecanbread.com

>

>