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In a message dated 3/8/2004 10:21:07 AM Central Standard Time,

bcweston3@... writes:

1717-1G->A

my girls carry 1717-1G-7A si this the same mutation? And are you saying that

the medication will work on this mutation? Deb A

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One thing that confuses me. Stop mutations are often directly

treated as synomous with Class I function mutations. My unborn

daughter has the 1717-1G->A mutation which is a splice junction

defect but still falls into the Class I grouping. For some reason,

they just lump Class I as a group when mentioning using Gentamycin to

create a " pass through " effect for stop mutations. Does anyone know,

micro-biologically speaking, a reason why Gentamycin would not work

for other Class I mutations like frameshift and splice junction?

A good site for more technical explanations.

http://www.hosppract.com/genetics/9706gen.htm

> > Could anyone explain to me what these are?

> >

> > Joe

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In a message dated 3/8/2004 2:29:05 PM Central Standard Time,

bcweston3@... write

Well we carry that mutation and the common mutation the Delta F508 and it

wasn't our CF clinic I was fighting with about the Ceftin it was our

Pediatrician

doctor we were fighting with and yes they do SUCK!! I have had many battles

with them but thank god I don't deal with them anymore then we have too. You

are the first person I have heard of too that carry the same gene as my girls.

Where do you live again?? Hey maybe we are related LOL!! Deb A

Woo Hoo! Finally found someone with our mutations. First off, I have

read some of your messages and I think your doctor SUCKS! I

recommend trying to find another CF center.

About our mutations, I think they are the same, the 1G->A is the

splice mutation component of the description and 1717 is the location

of the mutation.

I do not think this testing applies, would like to know for sure,

because I have seen other articles about using Gentamycin as a

passthrough mechanism for other diseases and they treated splice

mutations and stop mutations as two different entities.

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In a message dated 3/9/2004 10:10:35 AM Central Standard Time,

bcweston3@... writes:

Brad, Blythe, Birgess (no CF), and Baby on the way

We just found out that I carry the 1717-1G-7A and my husband carries the

Delta F508. I got it from my mother who got it from her mother. I found out that

my Grandmothers sister had a daughter that had a son with CF that died at 23

from complication from a lung transplant. We still have not found CF on my

husband side. I am Irish and German and my husband is mostly German I think he

might have some Irish in him to but not sure I will have to ask him about that.

I

do know that Attig is very German. those name are not familiar on my side but

I will have to go through my hubbies family history book and see if there are

any of those names in his history. My grandmothers last name was Yeggy and my

Great Grandparents name was Wadle.

Our daughters are doing pretty well they are 11 w/cf, 10 wo/cf, and 6 w/cf.

My oldest fights with polyps a lot and has pseudomonas in her nose and we can't

seem to get rid of it so we are constantly doing treatment to prevent

pseudomonas from getting into her lungs. My youngest hasn't been on treatment

since

the being of the fall. We have been trying the glutathione so I don't know if

that is why she has had such a good winter. But it is the only thing we have

done differently. My oldest just went into clinic yesterday to see if the

pseudomonas is still there so I will let you know how that goes when we fight

out.

How far along is your wife? And how did you find out your new baby has CF all

ready? My oldest was 6 when she was diagnosed and my youngest was 1. Deb A

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Thanks for the clarification. That is what I thought. If you look

at the Hopkins mutation database you can quickly pick up the

stop mutations usually have 0 pancreatic sufficiency whereas, other

class 1 mutations like 551D and 1717-1G->A have similar pancreatic

sufficiency to F508D.

http://www.hopkinsmedicine.org/cfgenotyping/database.htm

> > In a message dated 3/8/2004 10:21:07 AM Central Standard Time,

> > bcweston3@y... writes:

> > 1717-1G->A

> > my girls carry 1717-1G-7A si this the same mutation? And are

you

> saying that

> > the medication will work on this mutation? Deb A

> >

> >

> >

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Same here. My wife has the Delta F508 and I have that 1717

mutation. We live in Dallas. I don't know whether I got this

mutation from my mother or father but both families have been in

America for a LONG time (Scotch-Irish and English decent came over in

1600s). Maybe these last names are familiar to you, Hallock, Pollock,

Weston, Crawford, or Boddy.

Like I said before, we are waiting on our unborn daughter who has

CF. So we really don't fully know what to expect. How old are your

daughters and how are they doing?

Thanks,

Brad, Blythe, Birgess (no CF), and Baby on the way

> In a message dated 3/8/2004 2:29:05 PM Central Standard Time,

> bcweston3@y... write

>

> Well we carry that mutation and the common mutation the Delta F508

and it

> wasn't our CF clinic I was fighting with about the Ceftin it was

our Pediatrician

> doctor we were fighting with and yes they do SUCK!! I have had many

battles

> with them but thank god I don't deal with them anymore then we have

too. You

> are the first person I have heard of too that carry the same gene

as my girls.

> Where do you live again?? Hey maybe we are related LOL!! Deb A

>

>

>

>

> Woo Hoo! Finally found someone with our mutations. First off, I

have

> read some of your messages and I think your doctor SUCKS! I

> recommend trying to find another CF center.

>

> About our mutations, I think they are the same, the 1G->A is the

> splice mutation component of the description and 1717 is the

location

> of the mutation.

>

> I do not think this testing applies, would like to know for sure,

> because I have seen other articles about using Gentamycin as a

> passthrough mechanism for other diseases and they treated splice

> mutations and stop mutations as two different entities.

>

>

>

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