Guest guest Posted August 28, 2002 Report Share Posted August 28, 2002 (Hey guys, I just thought it might be a good idea to post my experiences with getting too little salt. If you tend to " veg out " like I do, as well as drink lots of fluids, you may develop some interesting side effects...........) Increasing salt completely fixed my pedal edema. I went to the doctor about it some time back, and we both approached this situation from a " protein synthesis deficiency " standpoint, but everything came back negative on that. What never crossed our radar was the fact that it could be sodium-intake related, as that is pretty absurd to consider when compared to the standard American diet. But sodium is what indeed fixed it. I initially thought my caloric intake itself might have been too low, because the edema would subside when I stopped CR for brief periods, but that was apparently due to the increased sodium intake during a return to ad lib and not from the caloric intake. Additionally, I have some abdominal distension (simliar to what Micky Snir describes) that comes and go, and sodium intake seems to have improved that situation as well. My veggie intake is so high though I am not expecting these issues to completely disappear, because my total water intake is going to be high regardless. However, the most important observation I have made is an observation that Micky has also contributed, in that I am significantly less hungry upon increasing salt. Whereas previously I could go through 4- 7 500 gram cans of sweet peas no salt added without feeling full (!), I can now eat 1-2 500 gram cans of salt added green beans and feel stuffed. I believe that I was manifesting an actual " hunger for salt " , as Micky has described. This probably is not a universal phenomenon, as some of you guys are self-avowed " salt avoiders " , but in my case it has made a world of difference in terms of satiety. I was at some points concerned about inducing a stomach rupture eating more and more no salt food! But now I just " gave up " trying to completely restrict salt, and have watched everything improved, while my blood pressure remains in the 90/60 " basement " . I am glad that Dean has entertained the idea of evaluating salt's impact on his implementation of CR. If there is anyone with the " scientific rigor " to do this issue justice, it is Dean, with his relentlessly investigative approach to things! An important issue to understand is that if the " salt reservoir " in the tissues is depleted, then the body will encounter difficulty in controlling water flux throughout various body compartments. Additionally, the renal medulla's capacity to concentrate urine is based upon its salinity, so if you flush out your kidneys too thoroughly, it may become more difficult for your kidneys to produce concentrated urine, which would translate into a " nonstop urination- sensation " marathon. In relation to the paleo man, another important thing to consider is that back in the old days, the only water people drank was what they could carry away from water sources, or water they actually drank straight from a water source. [Ever seen that PBS special Frontier House? Not paleo, but still old-school nonetheless ] I think in today's environments, with plumbing and Seven-Elevens, we are guzzling liquids at rates that the paleo man would consider unimaginable. Couple this with low-cal veggie intakes that paleo man might also consider unimaginable, and you are probably going to get some interesting phenomena in certain predisposed individuals. And the link between sodium intake and cardiovascular health is not as cut and dried as people would think. Calories are obviously going to trump whatever effect sodium is exerting. As for the dangers of the opposite end of things, here is an MRI study below where they studied hyponatremic patients suffering from primary polydipsia, a mostly psychiatric condition where you literally drink water constantly for no discernable reason. What they found was that the intermittant yet constantly recurring hyponatremic state increased the size of the " holes in their heads " , the ventricles. Increased ventricle size is associated with all sorts of long-term problems, as it understandably represents " less gray matter reserve " to work with to fight senesence-induced cognitive deterioration. Bigger holes in my head doesn't sound like a good thing to me! I'll risk the salt intake regardless of what some high-falutin' cardiologists publish about salt when they are obsessing about some of their more recalcitrant congestive heart failure patients! Now where is my salt shaker............ crdude35768@... Am J Psychiatry 1999 Jun;156(6):958-60 Related Articles, Books, LinkOut MRI changes during water loading in patients with polydipsia and intermittent hyponatremia. Leadbetter RA, Shutty MS Jr, Elkashef AM, Kirch DG, Spraggins T, Cail WS, Wu H, Bilder RM, Lieberman JA, Wyatt RJ. Clinical Studies Unit, Western State Hospital, Staunton, Va., USA. robert.leadbetter@... OBJECTIVE: Patients with polydipsia and intermittent hyponatremia have greater ventricle-brain ratios (VBRs) than matched patients without polydipsia and intermittent hyponatremia and normal subjects. Unlike previous studies, this study controlled for the impact of water loading when examining the volume of intracranial structures. METHOD: Under controlled conditions, eight male schizophrenic patients with polydipsia and intermittent hyponatremia were first assigned to either normal fluid intake or oral water loading and then the alternative condition the following day. Magnetic resonance imaging (MRI) volumetric measurements were made with the use of a standardized protocol. RESULTS: During water loading, total VBR and lateral ventricle volume significantly decreased by 13.1% and 12.6%, respectively. A strong association between change in serum sodium concentration and change in VBR was noted across conditions. CONCLUSIONS: These findings indicate that 1) water loading does not account for the diminished brain volume observed in patients with polydipsia and intermittent hyponatremia in previous studies, and 2) hyponatremia can significantly alter brain morphology on MRI. PMID: 10360142 [PubMed - indexed for MEDLINE] crddude35768@... Quote Link to comment Share on other sites More sharing options...
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