Guest guest Posted October 14, 2003 Report Share Posted October 14, 2003 As usual I have caused some confusion. Sorry. The action and use of ULDN is not the same as LDN. http://drgimbarzevsky.com/Naltrexone/index..html I was talking to doctor Crain about the ULDN dose range. Of course ULDN is of no value at all during the time when LDN is still active (bedtime to next morning). I take ULDN during the day as needed. I also take LDN at the Dr. Bihari dose range before bedtime, as per the instructions on LDNinfo.org. I do not take any narcotics. I rely entirely on my endogenous endorphin, maybe potentiated by ULDN. The idea is the endorphin receptors are unmasked by ULDN, or some other discomfort-causing receptors are blockaded by ULDN, or both. The worst case is I just feel a little better (about an aspirin's worth). The best case is that these other receptors are involved in MS causation and I am stopping it. Who knows? I don't. I am just trying it, to see if it helps anything. -Sullivan Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 14, 2003 Report Share Posted October 14, 2003 CHRIS, WHAT IS ULDN?? > As usual I have caused some confusion. Sorry. > > The action and use of ULDN is not the same as LDN. > > http://drgimbarzevsky.com/Naltrexone/index..html > > I was talking to doctor Crain about the ULDN dose range. > > Of course ULDN is of no value at all during the time when LDN is still > active (bedtime to next morning). > > I take ULDN during the day as needed. I also take LDN at the Dr. > Bihari dose range before bedtime, as per the instructions on > LDNinfo.org. > > I do not take any narcotics. I rely entirely on my endogenous > endorphin, maybe potentiated by ULDN. The idea is the endorphin > receptors are unmasked by ULDN, or some other discomfort-causing > receptors are blockaded by ULDN, or both. > > The worst case is I just feel a little better (about an aspirin's > worth). The best case is that these other receptors are involved in > MS causation and I am stopping it. > > Who knows? I don't. I am just trying it, to see if it helps > anything. > > -Sullivan Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 14, 2003 Report Share Posted October 14, 2003 A vanishingly small dose of naltrexone that may block a different receptor for an unknown endogenous opiate involved in opioid tolerance. It also seems to enhance or unmask the analgesic properties of other opiate painkillers. My idea is the same endogenous opiate and receptor may be involved in MS symptoms that are associated with pain and irritation. I also think residual naltrexone that is taken with the LDN therapy may be waking LDN users suddenly when it starts to block those other receptors, and stops blockading endorphin. Pure speculation on all counts. It is in a dosage range that is so low either it has zero to do with the LDN therapy or is swamped by the higher dose of LDN and can have no effect at all. Low numbers of micrograms of naltrexone are taken, either in conjunction with a normal painkilling opiate like morphine etc., or as in my case, alone, as a method of either potentiating endorphin or blockading the unknown endogenous pain-causing opiate. I believe during my high-endorphin times I walk better and faster and have a lot less dizziness so I want to givr LDN a good chance to work on me as it seems to have on others. I cannot function very well any more at low endorphin levels. I was taking ULDN during the day but not lately. I have no idea whether it works and it is a moot point for me until I can get this eye infection fixed. I am indeed on an antibiotic. -Sullivan > > As usual I have caused some confusion. Sorry. > > > > The action and use of ULDN is not the same as LDN. > > > > http://drgimbarzevsky.com/Naltrexone/index..html > > > > I was talking to doctor Crain about the ULDN dose range. > > > > Of course ULDN is of no value at all during the time when LDN is > still > > active (bedtime to next morning). > > > > I take ULDN during the day as needed. I also take LDN at the Dr. > > Bihari dose range before bedtime, as per the instructions on > > LDNinfo.org. > > > > I do not take any narcotics. I rely entirely on my endogenous > > endorphin, maybe potentiated by ULDN. The idea is the endorphin > > receptors are unmasked by ULDN, or some other discomfort-causing > > receptors are blockaded by ULDN, or both. > > > > The worst case is I just feel a little better (about an aspirin's > > worth). The best case is that these other receptors are involved > in > > MS causation and I am stopping it. > > > > Who knows? I don't. I am just trying it, to see if it helps > > anything. > > > > -Sullivan Quote Link to comment Share on other sites More sharing options...
Guest guest Posted October 15, 2003 Report Share Posted October 15, 2003 If that does something for you I would be pleased to hear it. That's exactly what I said, only I used my new word for today: potentiate -- to make more potent, I think. It either does that or blockades pain-causing opioid receptors, or both. (or not :-() Anyway your math seems fine except I've never seen a one litre dropper, and my only advice would be that it is useless to take it at night when you are already full of LDN :-) I take no blame but I am willing to consider credit... :-) -Sullivan > > > As usual I have caused some confusion. Sorry. > > > > > > The action and use of ULDN is not the same as LDN. > > > > > > http://drgimbarzevsky.com/Naltrexone/index..html > > > > > > I was talking to doctor Crain about the ULDN dose range. > > > > > > Of course ULDN is of no value at all during the time when LDN is > > still > > > active (bedtime to next morning). > > > > > > I take ULDN during the day as needed. I also take LDN at the Dr. > > > Bihari dose range before bedtime, as per the instructions on > > > LDNinfo.org. > > > > > > I do not take any narcotics. I rely entirely on my endogenous > > > endorphin, maybe potentiated by ULDN. The idea is the endorphin > > > receptors are unmasked by ULDN, or some other discomfort- causing > > > receptors are blockaded by ULDN, or both. > > > > > > The worst case is I just feel a little better (about an aspirin's > > > worth). The best case is that these other receptors are involved > > in > > > MS causation and I am stopping it. > > > > > > Who knows? I don't. I am just trying it, to see if it helps > > > anything. > > > > > > -Sullivan > > > Quote Link to comment Share on other sites More sharing options...
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