Fw: H.B.O.T. FOR SILICONE RELATED DISORDERS & CFS

[Guest guest]

----- Original Message -----

From: " Kathi " <pureheart@...>

Sent: Sunday, October 06, 2002 12:51 PM

Subject: " H.B.O.T. FOR SILICONE RELATED DISORDERS " & CFS

> " H.B.O.T. FOR SILICONE RELATED DISORDERS "

>

> Contact Dr M. Middleton at San Diego University for M.R.I. of Silicone

> Implanted disorders and Rupture; (619) 543-6414

>

> About 2 million women in the USA have had silicone breast implants.

>

> Dimethylpolysiloxy has been used either in implants or by injection for

> a

> variety of purposes including mammoplasty, filling of the tissue voids

> in

> reconstruction surgery and prostheses for cosmetic and other purposes,

> and

> of course silicone was used for some years for brain " Shunts "

>

> Injected silicone material was found to pose severe health problems,

> including cancer, arthritis and chronic fatigue syndrome as well as

> other

> immunobased and allergic reactions.

>

> Some Patients with breast implants, and exhibiting such symptoms have

> been

> shown by Enzyme linked Sero Assay () test to posses elevated IgG,

> IgA,

> IgM, and IgE silicone antibodies. In addition to the silicone reactions

> from

> silicone implants the polyurethane implants shells themselves are now

> known

> to break down under the metabolic activity and produce 2,4,

> toluenediamine

> and 2,6 toluenediamine, both of which are carcinogenic and able to react

>

> with the neurominic acids to produce phthalocyanine dichloride which in

> turn

> reacts with silicone to produce a precursor to silicone " stacked rings "

>

> phthalocyanines.

>

> This form of cyanide toxin induced metabolic impairment in the brain

> inhibits production of adenosine triphosphate, adequate supplies of

> which

> are required to avert lipofuscin accumulations, to promote phagocytosis

> and

> for natural detoxification. A abnormal sodium and potassium ion

> interchanges. In the course of this abnormal chemistry in the brain,

> polyunsaturated fatty acids become destabilized as the double bonds

> within

> membranes allow the easy extraction of hydrogen atoms. This starts a

> reaction in which peroxy radical combine with hydrogen to lipid

> hyperaldehydes. This series of reactions conjugated as diones can

> initiate

> chain reactions leading to numerous toxic and allergic responses in the

> patient as membranes cross link damaging their integrity and their

> essential

> proteins. The glutamatergic neuronal process can lose energy

> dramatically

> due to hypoxia or hyperglycemia and from further neurotoxins.

>

> SYMPTOMS

>

> Dopamines can be released, and the patients may suffer symptoms of

> confusion

> or intermittent panic. In the immunsuppressed environment, many strains

> of

> fungi will be able to flourish, and allergic reactions to these and

> other

> invasory micro-organisms are common as are developing allergies to

> chemicals

> ( especially hydrocarbons), the symptoms of silicone allergy are

> therefore

> associated with cyanide blockage of part of the hemoglobin oxygen

> transportation mechanism, with neurotoxin responses, and with lipofuscin

>

> accumulation.

>

> HYPERBARIC OXYGEN THERAPY ( HBOT)

>

> HBOT has been shown to disperse lipofuscins, burn out cyanide poisoning

> ( a

> category one use for hbot) and in many cases destroy a wide range of

> neuro-toxins. HBOT also has a well earned reputation as an immune system

>

> enhancer and is also able in many cases to reactivate idling neurons.

> Experience with anoxic encephalopathy has shown that neurons are able to

> be

> " idling " for up to 15 years, and still be recovered to normal activity

> as

> soon as good oxidation is present.

>

> Over 90% of all the oxygen used by humans is consumed in the

> mitochondria

> and this makes hypoxic conditions in that area singularly threatening,

> as

> the mitochondria are not very efficient in any case at controlling the

> electrical components of the mechanism of breathing. It is notable that

> restoration of good oxidation of the mitochondria usually results in the

>

> breathing difficulties of silicone reaction patients ( usually described

> as

> " choking sensations " ) resolving very quickly.

>

> SINGLE PHOTON EMISSION COMPUTER TOMOGRAPHY (BRAIN SPECT SCAN)

>

> Patients with sequel to silicone implant leakage almost always exhibit

> the

> same abnormalities on Spect Brain Scan, and seem to recover from their

> symptoms when SPECT scans become Normal. Abnormalities usually include

> bilateral cortical lack of perfusion, and this together with a history

> of

> silicone implants or past silicone injections and the symptoms described

>

> would seem to be a good rationale for HBOT treatment. Some patients have

> in

> the past been miss diagnosed as cases of Multiple Sclerosis ( MS), and

> whilst they exhibited all of the symptoms of silicone reactions they did

> not

> show the usual " platelets " distributed in the brain scans by Magnetic

> Resonance imaging (MRI)

>

> Patients have also been diagnosed as having ALS, Lupus, and sclerodrema;

> and

> have later been found to have silicone reactions instead. However, these

>

> disease process does indeed mimic the above stated. It must also be

> stated

> that Hyperbaric oxygen therapy has been used in Europe for treatment of

> MS.

> And Als, and well as lupus . HBOT is considered Investigational for the

> above stated disorders in the USA

>

> D.

>

> Reference:

> Occasional Review

> Use of Hyperbaric Oxygen in Rheumatic Diseases:And Critical Analysis

> D.J. Wallace 1,2 S. Silverman.2* J.Goldstein 3,and D. 4

> 1Department of Medicine Cedar-Sinai Medical Center 2UCLA School of

> Medicine,

> Los Angeles, CA. 3 The Chronic Fatigue Syndrome Institute. Anaheim CA.

> And 4

> The Hyperbaric Oxygen Institute, San Bernardino, CA USA

>

> Printed with Permission

>

> ******************************

>

>

> Chronic Fatigue Syndrome, FM, and Lyme

>

> (Mycoplasma associated disorders)

>

> The etiology of Chronic Fatigue Syndrome (CFS) and Fibromyalgia (FM) is

> still not entirely clear, and has been variously suggested as an

> infection

> in its own right, or as the sequel to other infections or neuro-toxic

> insults--notably Epstein Barr Virus (EBV) and allergic reaction to a

> number

> of substances including, but not limited to, silicone, chemical crop

> sprays,

> and petroleum products.

>

>

> In any event, the mechanism in physiology which causes the symptoms of

> chronic fatigue is insufficiently understood but can be explained by the

>

> following hypothesis or postulate which is put forward on the basis that

> it

> explains both the mechanism and the therapy.

>

>

> Muscular and other cellular activity requires energy, and this in turn

> requires glycolysis, which relies upon the burning of blood carried

> sugars

> with blood carried oxygen. The release of energy by this chemical

> process

> produces waste chemicals, notably lactates, and these in turn impose the

>

> symptoms of fatigue until they are fixed and removed by the same blood

> chemistry. This is the Krebs cycle normal to all life.

>

>

> Under ordinary circumstances, oxygen is transported to the body cells by

>

> this chemical cycle in which it combines with hemoglobin in the

> erythrocytes

> of the blood stream, and the same chemical (HGB) removes and fixes the

> waste

> lactates for disposal as carbon dioxide into the pulmonary system.

>

>

> In order to flow through the venous system, erythrocytes (RBCs) must be

> deformed, since they are larger in diameter than the veins in which they

>

> must travel. This is to ensure that a maximum surface contact area is

> available.

>

>

> In cases of Chronic Fatigue Syndrome, either the internal pressure in a

> percentage of the RBCs is elevated, or the permeability of their cell

> walls

> is lowered, or both. The result is that these RBCs are not able to

> deform

> and travel in the microcirculatory system. This deprives the cells,

> which

> the RBCs should serve of oxygen, and allows accumulated lactates to

> produce

> the symptoms of Chronic Fatigue Syndrome.

>

>

> CONCLUSIONS:

>

>

> The hypothesis can be supported in that if you exhaust a fit subject,

> his or

> her RBCs will behave as above roughly in the same percentages as the

> subject

> 's proportionate fatigue, but will recover to normal as the subject

> recovers. The Chronic Fatigue patient's blood, on the other hand, will

> remain abnormal in this aspect, and the patient will remain fatigued.

>

>

> TREATMENT:

>

>

> Hyperbaric Oxygen Therapy at 2 ATA - 2.4 ATA produces an increased

> elasticity in the RBCs and at the same time seems to reduce the Delta P

> between their contents and the surrounding medium. There is also a

> probability that the dissolved oxygen in the plasma may oxidize whatever

>

> substance is responsible for the decrease in RBC cell wall permeability.

>

> Certainly, the most immediate effect of Hyperbaric Oxygen Therapy is to

> relieve the cellular hypoxia, which is a feature of Chronic Fatigue

> Syndrome. Sixty minutes of treatment twice daily for 30 -40 treatments,

> followed by weekly treatments prn seems to resolve CFS symptoms in the

> majority of patients, and eventually to resolve them completely.

>

>

> We also highly recommend Antibiotic therapy (IV) along with IV mega

> Vitamin

> therapy.

>

>

> Underwood, D.O., M.D., J.D.

> Medical Director

> Rapid Recovery Hyperbarics

> Health Enhancement Centers

>

>

> Printed with Permission

>

>

[Guest guest]

I had those choking sensations when I got sick. It was weird. I don't get them anymore.

I had 22 sessions of HBOT. I hope it helped--there was no dramatic recovery for me, but I am sure I got the benefit of the extra oxygen in my system. I was supposed to have about 40 sessions, but could only afford to stay long enough for the 22 sessions. I wonder if I had done all the recommended sessions and continued to have boosters once in awhile if I would have gotten better sooner?

This is expensive therapy, by the way...

Patty

----- Original Message -----

From: Heer

Sent: Monday, October 07, 2002 6:47 AM

Subject: Fw: "H.B.O.T. FOR SILICONE RELATED DISORDERS" & CFS

----- Original Message ----- From: "Kathi" <pureheart@...>Sent: Sunday, October 06, 2002 12:51 PMSubject: "H.B.O.T. FOR SILICONE RELATED DISORDERS" & CFS> "H.B.O.T. FOR SILICONE RELATED DISORDERS"> > Contact Dr M. Middleton at San Diego University for M.R.I. of Silicone> Implanted disorders and Rupture; (619) 543-6414> > About 2 million women in the USA have had silicone breast implants.> > Dimethylpolysiloxy has been used either in implants or by injection for> a> variety of purposes including mammoplasty, filling of the tissue voids> in> reconstruction surgery and prostheses for cosmetic and other purposes,> and> of course silicone was used for some years for brain "Shunts"> > Injected silicone material was found to pose severe health problems,> including cancer, arthritis and chronic fatigue syndrome as well as> other> immunobased and allergic reactions.> > Some Patients with breast implants, and exhibiting such symptoms have> been> shown by Enzyme linked Sero Assay () test to posses elevated IgG,> IgA,> IgM, and IgE silicone antibodies. In addition to the silicone reactions> from> silicone implants the polyurethane implants shells themselves are now> known> to break down under the metabolic activity and produce 2,4,> toluenediamine> and 2,6 toluenediamine, both of which are carcinogenic and able to react> > with the neurominic acids to produce phthalocyanine dichloride which in> turn> reacts with silicone to produce a precursor to silicone " stacked rings"> > phthalocyanines.> > This form of cyanide toxin induced metabolic impairment in the brain> inhibits production of adenosine triphosphate, adequate supplies of> which> are required to avert lipofuscin accumulations, to promote phagocytosis> and> for natural detoxification. A abnormal sodium and potassium ion> interchanges. In the course of this abnormal chemistry in the brain,> polyunsaturated fatty acids become destabilized as the double bonds> within> membranes allow the easy extraction of hydrogen atoms. This starts a> reaction in which peroxy radical combine with hydrogen to lipid> hyperaldehydes. This series of reactions conjugated as diones can> initiate> chain reactions leading to numerous toxic and allergic responses in the> patient as membranes cross link damaging their integrity and their> essential> proteins. The glutamatergic neuronal process can lose energy> dramatically> due to hypoxia or hyperglycemia and from further neurotoxins.> > SYMPTOMS> > Dopamines can be released, and the patients may suffer symptoms of> confusion> or intermittent panic. In the immunsuppressed environment, many strains> of> fungi will be able to flourish, and allergic reactions to these and> other> invasory micro-organisms are common as are developing allergies to> chemicals> ( especially hydrocarbons), the symptoms of silicone allergy are> therefore> associated with cyanide blockage of part of the hemoglobin oxygen> transportation mechanism, with neurotoxin responses, and with lipofuscin> > accumulation.> > HYPERBARIC OXYGEN THERAPY ( HBOT)> > HBOT has been shown to disperse lipofuscins, burn out cyanide poisoning> ( a> category one use for hbot) and in many cases destroy a wide range of> neuro-toxins. HBOT also has a well earned reputation as an immune system> > enhancer and is also able in many cases to reactivate idling neurons.> Experience with anoxic encephalopathy has shown that neurons are able to> be> "idling" for up to 15 years, and still be recovered to normal activity> as> soon as good oxidation is present.> > Over 90% of all the oxygen used by humans is consumed in the> mitochondria> and this makes hypoxic conditions in that area singularly threatening,> as> the mitochondria are not very efficient in any case at controlling the> electrical components of the mechanism of breathing. It is notable that> restoration of good oxidation of the mitochondria usually results in the> > breathing difficulties of silicone reaction patients ( usually described> as> "choking sensations") resolving very quickly.> > SINGLE PHOTON EMISSION COMPUTER TOMOGRAPHY (BRAIN SPECT SCAN)> > Patients with sequel to silicone implant leakage almost always exhibit> the> same abnormalities on Spect Brain Scan, and seem to recover from their> symptoms when SPECT scans become Normal. Abnormalities usually include> bilateral cortical lack of perfusion, and this together with a history> of> silicone implants or past silicone injections and the symptoms described> > would seem to be a good rationale for HBOT treatment. Some patients have> in> the past been miss diagnosed as cases of Multiple Sclerosis ( MS), and> whilst they exhibited all of the symptoms of silicone reactions they did> not> show the usual "platelets" distributed in the brain scans by Magnetic> Resonance imaging (MRI)> > Patients have also been diagnosed as having ALS, Lupus, and sclerodrema;> and> have later been found to have silicone reactions instead. However, these> > disease process does indeed mimic the above stated. It must also be> stated> that Hyperbaric oxygen therapy has been used in Europe for treatment of> MS.> And Als, and well as lupus . HBOT is considered Investigational for the> above stated disorders in the USA> > D. > > Reference:> Occasional Review> Use of Hyperbaric Oxygen in Rheumatic Diseases:And Critical Analysis> D.J. Wallace 1,2 S. Silverman.2* J.Goldstein 3,and D. 4> 1Department of Medicine Cedar-Sinai Medical Center 2UCLA School of> Medicine,> Los Angeles, CA. 3 The Chronic Fatigue Syndrome Institute. Anaheim CA.> And 4> The Hyperbaric Oxygen Institute, San Bernardino, CA USA> > Printed with Permission>