Estradiol Issues and Continuing Treatment

[Guest guest]

Hello Everyone,

Allow me to bring you up to date with my treatment. By now, the endocrinologist

has before him two samples, both measuring testosterone and estradiol level.

One sample shows the peak concentration of E and T, taken two days after my last

injection. The other registers their nadir, the low point, a full week after

injection. Once I know the exact values, I'll post them here.

My next endocrinology appointment is at the end of this coming week. I will

start the Femara as soon as Friday. Phil believes that this particular

medication is too strong. I have no reason to doubt him. However, assuming I

do have a very elevated level of Estradiol, which lab results are likely to

confirm, is it possible that something this high octane is what I need?

I have a few questions to ask. I'd like to know what I can expect from now

going forward.

What do I need to look for in order to be able to judge Femara's effects on me?

How will I be able to know if that medication is working, or, if it isn't, that

it is not what I need? How long does it usually take for an Aromatase inhibitor

to work, roughly speaking?

The decision to try Femara was based exclusively on out of pocket cost. Since I

had an issue with my insurance not covering AndroGel, I've been seeking to find

cost-effective options that insurance is unlikely to reject. Arimidex, which

was recommended to me here, is considerably more expensive than Femara. Cost

per dose is about five times as much, but this is because Arimidex has to be

taken every day as prescribed for me, whereas Femara is only taken once a week.

As I reflect back on it, I probably have had elevated levels of Estradiol for a

long time, likely years. Something that has been puzzling me recently is why

the insulin resistance issues, which resembling reactive hypoglycemia are so

much worse after I finish working out. My personal theory is that exercise

raises testosterone levels, but the extra testosterone then gets immediately

converted to estradiol, since the body considers it excess. Anyone with

thoughts about this theory is welcome to contribute. I may be entirely off the

mark here, but when you don't have answers, you tend to come up with them on

your own.

Thanks again for all your help.

.