Guest guest Posted March 11, 2011 Report Share Posted March 11, 2011 Well yes and no best you read the following it all starts with Chol. =============================================== The Big T How your lifestyle influences your Testosterone levels — Part 1 by M. Berardi Like it or not (and I'm sure T-mag readers really like it), Testosterone is the hormone of the decade. The granddaddy of the male hormones has gotten more media attention over the last few years than any other hormone around. Heck, I even heard a rumor that some crazy bodybuilding media guys were thinking of naming a magazine after it. Can you imagine that? While Testosterone (the hormone, of course) has been the target of much bad press, I think that if you asked this big dog of hormones what he thought of all of this, he would bark out something to the effect of " What of it? I must be doing something right if they keep talking about me! Now can't you see I'm trying to work this shaved little poodle over here? " Although the popular media has made Testosterone out to be a destructive bad guy, researchers have been slowly but surely embracing its use. Clinical trials have been conducted in diverse groups of individuals from HIV wasting patients and burn victims to people with compromised immunity, along with older men whose " Testosterone " hasn't been up in years. There have even been a number of recent trials investigating the use of Testosterone in healthy weight trained men. So where do I sign up? The results of these investigations have shown that Testosterone is not the demon the medical community once thought it to be and that it actually can be of great benefit to certain individuals and, in certain patients, possesses very few risks. I'm pretty positive though, that the use of Testosterone will never be condoned for use in healthy weight trained males. To this end, us law abiding citizens have to do the best we can with what we've got to work with. So let's talk about how our own body provides us with the big T and what we can do, both naturally and with dietary supplements, to maximize our T levels. When most people think of steroids, they tend only to think of Testosterone. This, my friends, is yet another fact which tends to make me believe that T is the hormone of the decade. Testosterone, however, is only one member of the steroid family. Some of the other steroids in this family include cholesterol, progesterone, the estrogens, cortisol, and aldosterone. Although these molecules are part of the same family and have strikingly similar structures, their functions differ like night and day. This is important to recognize because although the steroids tend to act very differently, they are subject to similar rules with respect to biochemistry and metabolism. For a simplified view of steroid metabolism in the body, you can assume that all steroid hormones begin with cholesterol. From cholesterol, steroid metabolites are formed in various tissues of the body. For example, enzymes in the adrenal glands are responsible for converting cholesterol into cortisol, while enzymes in the gonads are responsible for converting cholesterol to Testosterone. With this simplified view, it's easy to make the mistake of thinking that by simply providing the body with more cholesterol (make that two large fries, please), we can make more Testosterone. This is a mistake because the body has regulatory mechanisms that control hormone production. These regulatory mechanisms, not your bedtime prayers to the iron gods, are what determine which steroid metabolites will ultimately be formed. So the next important questions are, what magic does it take to make Testosterone out of cholesterol (now don't get too excited, you can't do this in your bath tub), what regulates this conversion, and ultimately, what regulates Testosterone production? In order to get the gonads to produce T, the body has a chain of command that must be dealt with just like any smooth running business. In business, the action plan comes down from the CEO to upper management, the plan is solidified and delegated to the production team, and the production team gets the job done. Well, in the body, a portion of the brain called the hypothalamus is the CEO, the pituitary gland is the upper management, and the testes are the production team members. As in business, the buck stops with the CEO/hypothalamus, which is known as a " pulse generator, " because during the day it sends out pulses of hormones that are designed to stimulate other organs. With respect to T, the hypothalamus sends out numerous daily pulses of GNRH (gonadotropin releasing hormone) through the blood stream. These pulses are designed to stimulate the pituitary gland to get to work. The pituitary gland then senses the pulses of GNRH and sends out a work order of its own, consisting of LH (leutinizing hormone) pulses. The LH message travels down to the leydig cells of the testis to stimulate the enzymatic conversion of cholesterol to Testosterone. Cholesterol conversion to T is no easy process and I'm not going to go into all of the details (partly because no one really knows them all). One fact that you should understand, though, is that there's a high level of complexity to this pathway and that there are many enzymes and intermediates that cholesterol has to encounter before forming T. Some of these intermediates include pregnenolone, DHEA, androstenedione, and other well-known androgens. So, although the hypothalamus might be functioning well, the pituitary might be doing the right thing, and the testis are getting the " ball " in motion, ultimately the enzymes in the leydig cells determine whether you're pumping out loads of muscle building T or simply forming other intermediates at the expense of the top dog. As a result of the process I mentioned above, T levels fluctuate wildly. If you were to measure your Testosterone levels throughout the day, you'd likely be amazed. One minute you have the hormonal profile of a hyper-muscular bull ready to " fertilize " an entire herd of cattle? and the next minute your blood profile is that of a fully menstruating Martha intent on color coordinating your powder room. These odd fluctuations occur as a result of the pulsatile nature of hormone secretion. Again, this begins with the hypothalamic pulse generator's release of GNRH. Incidentally, researchers now believe that it is this physiologic pulsatility of Testosterone that makes it anabolic. So if you can mimic this pattern of hormone release, you can stimulate muscle growth. With this hormonal cascade in mind, it's important to realize that each step in the pathway has a regulation point designed to either stimulate or inhibit pulse release. In this respect, the body is a bit of a control freak as it tends to like many control points rather than just one. In this particular case there are three main control points; the hypothalamus, the pituitary, and the testis. With this type of control, the body can maintain the Testosterone homeostasis (a sort of hormonal status quo) and prevent us from any abnormal changes in muscle development and strength. For example, if our Testosterone levels go way up, the body senses this and the hypothalamus and the pituitary produce less GNRH and LH in order to slow down T production. This, of course, is the famous negative feedback. Damn that homeostasis! Now that I'm certain you're all experts in Testosterone production (and there will be a test at the end — I'm serious!), I'd like to address one more important issue that will come up later in the article with regard to Testosterone in the body. When Testosterone is converted from cholesterol in the leydig cells of the testis, it's released into the blood stream where it embarks on an anabolic adventure. However, when in the blood, 60% of the big T released from the boys down below is bound up by a protein known as SHBG, or sex-hormone binding globulin. SHBG is produced and released by the liver. The important point is that the Testosterone bound to SHBG is biologically inactive and this is why there's an important distinction between total T and bioavailable T. Total T represents all the Testosterone in the blood, while bioavailable T represents the non SHBG bound Testosterone. There are other proteins in the blood that bind Testosterone, too, but their binding is rather weak, so this T is bioavailable and these proteins can still enter the cells to produce and effect all the things we're interested in. As I said, bioavailable T represents the Testosterone that is not SHBG bound, while free T represents the Testosterone that's not bound to any blood proteins at all. It's tricky, I know, but I hope that it's now evident that although only about 2% of the T in blood is technically considered free T, there is a larger percentage of T (about 40% or so) that is bioavailable because it's only weakly bound to non SHBG blood proteins. I'm taking you through this complex path for good reason. When trying to increase T levels in the body, one must attempt to not only increase total T. More importantly, one must attempt to increase bioavailable T. If you increase total T, but you increase SHBG to a larger extent, they you will actually have less bioavailable T for muscle building purposes! A great example of this is the use of both thyroid drugs and tamoxifen (nolvadex). Both may increase total T levels in the body, but both also increase SHBG to a large extent. Although you may get a bit of a T surge with each (hurray!), the increase in SHBG may bind up any extra, and actually decrease your bioavailable T (boo!). Well, now that the class is up to speed with our physiology and endocrinology (will someone please wake up Mr. Luoma! — he's always falling asleep during my physiology lectures), we can dive, full force, into how lifestyle factors including things like diet, training, recreational drugs, over the counter medications, altitude, and how psychological mood states influence T levels. There's an abundance of Testosterone literature out there and some of it is applicable for us while some is not, but to a science geek like me who both likes facts and likes being big and lean, it's all interesting nevertheless. Oh wait, I almost forgot! Before we go on, I promised a test didn't I? Settle down! Although there are no actual grades on this test, I hope that you take away a few fundamental things from this article. If you can answer these questions, you're ready to take on next week's article in which I'll review a number of environmental and lifestyle factors that can influence your levels of free T, total T, and bioavailable T. Rest easy, next week's article ties in all that you learned this week and makes some recommendations about how to up the T levels. And next week there won't be a test! Co-Moderator Phil > From: zorroceasar <zorroceasar@...> > Subject: Now TOTALY confused ! > > Date: Friday, March 11, 2011, 1:49 PM > I READ THIS ON THE WORLDCLASS > BODYBUILDING FITNESS FORUM: > > > LH > > Luteinizing hormone is responsible for the trigger of > testosterone release in the body. Basically, LH which is > secreted in the Testes triggers the body to produce > Testosterone. Once this is done the hypothalmus picks up on > it in a negative feed back process (inhibition of GnRH). > Thus, LH " checks " itself. > > What does this mean? > > Well, LH is a fine balance. Not enough, and your body > doesn't produce Test. Too much, same effect. Now introduce > synthetic Testosterone into the loop and what do we have? A > break in the cycle. Boom...body stops producing LH. > > Now wait a minute. If we're on cycle why would we want to > tell the body produce LH when it doesn't need to? People > think your testicles shrink due to lowered Test production. > No no no. This is DIRECTLY linked to LH and FSH depletion. > Test is NOT made in the testes. LH is made in the Testes > which SIGNALS the body to make Test. Well if there's excess > Test introduced why do we need LH? Are you guys seeing the > link yet? > > Excess test = no need for LH = shutdown of LH = Testicle > shrinkage > > So what are we aiming for? Well we're aiming for just > enough LH production to keep the loop working, BUT not too > much where the body downgrades receptors because it's gone > haywire. > > I THOUGHT THE PITUARY GLAND PRODUCES LH > I THOUGHT TESTES PRODUCES TESTOSTERONE > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW LH WORKS > AM I WRONG OR WHAT IS HAPPENING > > > > > > > ------------------------------------ > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 11, 2011 Report Share Posted March 11, 2011 Here is part two. ================================================================= the Big T — Part 2 How your lifestyle influences your testosterone levels by M. Berardi Testosterone, Environment, and Lifestyle After last week's physiology and endocrine primer, I thought you might need a break, a chance to let the smoking gray matter cool down. All that science is heavy. As a result of your week off, I hope you're ready to move on to this week's topic. This week we delve into the factors that affect T and what you can do about your own T levels. A Testosterone Summer Summer time is just about gone, and in addition to there having been lots of extra daylight and vacation time, there seems to have been lots of extra Testosterone floating around. This may just be a personal observation but in the summer, men and women seem more likely to want to meet, and as my 10th grade musical hero, Tone Loc, would say? do the wild thing. Ever wonder why? Sure it may have something to do with the fact that there's less thread and more tanned flesh readily visible. But it may be something else as well. In a study examining the effects of season on total Testosterone levels, men with average T levels of 666 ng/dl (normal levels are between 300-1000 ng/dl) were studied during the months of April, July, October, January, and April again.(21) It appears that T levels increase about 12% during July and 15% during October and return to April levels throughout the rest of the winter. So remember, summer and early fall may be the time to start a growth phase since T will be the highest. In addition, the winter might be a nice time to cycle prohormones or other Testosterone boosters in an effort to keep the T up to summer highs. One side note. No one knows why T levels fluctuate this way, but researchers speculate that T levels may be regulated by light-dark cycles. If this is true, then perhaps exposing yourself to extra sunlight throughout the winter months may keep T levels up. Who knows, a little extra winter tanning might even help to keep the T up, but that's just speculation. Give it a try. If your T doesn't go up, then at least you'll have a nice tan. Training With A Capital T There's nothing new about the fact that acute exercise increases T levels. It's been shown that in men, T levels rise both during exercise and up to about 15 minutes after exercise. But not all exercise increases T levels. Certain workloads and intensities produce no increase at all. Exercise at a certain threshold, however, leads to T increases that are proportional to the amount of muscle mass involved in the workout, the amount of total work (sets X reps), and the intensity. With respect to weight training, a powerlifting-type workout consisting of 3 sets of 5 reps with 3 minutes rest between sets and a bodybuilding-type workout consisting of 3 sets of 10 reps with 1 minute rest between sets were compared. It appears that total T levels were increased for 15 minutes following exercise and that the bodybuilding workout (67% increase) is superior to the powerlifting workout (32% increase) for increasing total T levels.(14) What about an activity like running? In a study done comparing elite distance runners with sedentary men, it was interesting to note that at rest, the sedentary men had 54% more total and free T floating around in their blood than the runners. It seems to be the case that most volume-training athletes have lower levels of T. The volume threshold seems to be at about 8 hours of exercise per week. In this study, the runners did, however, have larger free T increases in response to the running. Here's a table of the results: Running Intensity and Duration Sedentary Runners Elite Runners 80% HR max for 120 minutes 47% Increase 76% Increase 80% HR max for 20 minutes 31% Increase 62% Increase 50% HR max for 120 minutes Nothing Nothing 50% HR max for 20 minutes Nothing Nothing From these results, it appears that intense running (80% of max HR) can lead to increases in free T similar to the increases seen with resistance training.(6) If your goal is to increase your Testosterone, though, I wouldn't necessarily recommend very intense continuous running or a very high volume of exercise (greater than 8 hours per week). Doing so would probably ensure that any increased T as a result of a single exercise session would probably be canceled out by lower resting levels of T. If you still insist on volume training, supplementing with over-the-counter androgens might help normalize T levels during intense periods of training. Unfortunately and paradoxically, although T levels are increased directly after a training bout, some research has shown that they might then take a nosedive into subnormal levels for 1-3 days after the exercise session. This means that although your squat day might jack your T levels up for a little while, ultimately those T levels are destined to come back down and end up lower than you started with. They might also stay low for a day or two before coming back to normal. With that said, my advice would be to stop training altogether. Well, no, but I thought that might get your attention. Actually, inserting rest days between intense exercise bouts might be a better solution to allow T to come back to normal before hammering out another intense workout. Of course, you could always rely on OTC androgens, but since intense exercise might always promote this effect and you don't want to stay on androgens year round, perhaps program manipulation might be the best option. Mood, Sex, and T Most people think that stress and depression are psychological phenomenon that have minimal impact on the body. Wrong! Mood states and stressors have major effects on physical systems, especially the endocrine system. In a bad mood? Stress and depression tend to cause the release of a hormone called corticotropin-releasing hormone (CRH) from our friend, the hypothalamus. This hormone ultimately leads to increases in the release of cortisol, the nasty catabolic adrenal hormone that munches up hard-earned muscle tissue with extreme prejudice. Increases in CRH and cortisol as a result of both physical and psychological stress, including depression, anxiety, injury, fever, disease, etc. lead to decreased T levels.(25) To highlight the importance of this, one marker of overtraining and a catabolic state is the Testosterone-cortisol ratio. The higher the ratio, the more anabolism. The lower the ratio, well, let's not discuss that. Many studies have been conducted to examine the relationship between stress, mood, cortisol, and Testosterone. Studies in humans and animals show that both acute and chronic stress lower total Testosterone levels. Here's the evidence: > > > From: zorroceasar <zorroceasar@...> > > Subject: Now TOTALY confused ! > > > > Date: Friday, March 11, 2011, 1:49 PM > > I READ THIS ON THE WORLDCLASS > > BODYBUILDING FITNESS FORUM: > > > > > > LH > > > > Luteinizing hormone is responsible for the trigger of > > testosterone release in the body. Basically, LH which is > > secreted in the Testes triggers the body to produce > > Testosterone. Once this is done the hypothalmus picks up on > > it in a negative feed back process (inhibition of GnRH). > > Thus, LH " checks " itself. > > > > What does this mean? > > > > Well, LH is a fine balance. Not enough, and your body > > doesn't produce Test. Too much, same effect. Now introduce > > synthetic Testosterone into the loop and what do we have? A > > break in the cycle. Boom...body stops producing LH. > > > > Now wait a minute. If we're on cycle why would we want to > > tell the body produce LH when it doesn't need to? People > > think your testicles shrink due to lowered Test production. > > No no no. This is DIRECTLY linked to LH and FSH depletion. > > Test is NOT made in the testes. LH is made in the Testes > > which SIGNALS the body to make Test. Well if there's excess > > Test introduced why do we need LH? Are you guys seeing the > > link yet? > > > > Excess test = no need for LH = shutdown of LH = Testicle > > shrinkage > > > > So what are we aiming for? Well we're aiming for just > > enough LH production to keep the loop working, BUT not too > > much where the body downgrades receptors because it's gone > > haywire. > > > > I THOUGHT THE PITUARY GLAND PRODUCES LH > > I THOUGHT TESTES PRODUCES TESTOSTERONE > > > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW LH WORKS > > AM I WRONG OR WHAT IS HAPPENING > > > > > > > > > > > > > > ------------------------------------ > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 13, 2011 Report Share Posted March 13, 2011 I have read through what youve send me. I still have the same questions. This forum in bodybuilding says Lh is made in the testes and this is a well known forum and people in the forum agree with him This is his statements LH is made in the testes Testosterone is not made in the testes now everything I ve read so far in our group here over the past 5 years says otherwise I only had one response and that was from Phil saying yes and no Please clarify. > > > From: zorroceasar <zorroceasar@...> > > Subject: Now TOTALY confused ! > > > > Date: Friday, March 11, 2011, 1:49 PM > > I READ THIS ON THE WORLDCLASS > > BODYBUILDING FITNESS FORUM: > > > > > > LH > > > > Luteinizing hormone is responsible for the trigger of > > testosterone release in the body. Basically, LH which is > > secreted in the Testes triggers the body to produce > > Testosterone. Once this is done the hypothalmus picks up on > > it in a negative feed back process (inhibition of GnRH). > > Thus, LH " checks " itself. > > > > What does this mean? > > > > Well, LH is a fine balance. Not enough, and your body > > doesn't produce Test. Too much, same effect. Now introduce > > synthetic Testosterone into the loop and what do we have? A > > break in the cycle. Boom...body stops producing LH. > > > > Now wait a minute. If we're on cycle why would we want to > > tell the body produce LH when it doesn't need to? People > > think your testicles shrink due to lowered Test production. > > No no no. This is DIRECTLY linked to LH and FSH depletion. > > Test is NOT made in the testes. LH is made in the Testes > > which SIGNALS the body to make Test. Well if there's excess > > Test introduced why do we need LH? Are you guys seeing the > > link yet? > > > > Excess test = no need for LH = shutdown of LH = Testicle > > shrinkage > > > > So what are we aiming for? Well we're aiming for just > > enough LH production to keep the loop working, BUT not too > > much where the body downgrades receptors because it's gone > > haywire. > > > > I THOUGHT THE PITUARY GLAND PRODUCES LH > > I THOUGHT TESTES PRODUCES TESTOSTERONE > > > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW LH WORKS > > AM I WRONG OR WHAT IS HAPPENING > > > > > > > > > > > > > > ------------------------------------ > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 13, 2011 Report Share Posted March 13, 2011 A snippet from wikipedia that answers your question: " Luteinizing hormone (LH, also known as lutropin[1]) is a hormone produced by the anterior *pituitary gland*. " ... " In males, where LH had also been called interstitial cell-stimulating hormone (ICSH),[3] it stimulates Leydig cell production of testosterone. " The Leydig cells are in the testes. About 90% of testosterone comes from the testes, with the remaining 10% from the adrenal glands. LH (or other similar hormones like hCG) is required to tell the Leydig cells to produce testosterone. Exogenous (outside the body- i.e. pharmaceutical) testosterone tells the pituitary to release less LH. Having LH or a hormone which behaves like it is useful for a few reasons. 1) LH may have other functions besides simply triggering testosterone production. 2) Prolonged suppression of LH seems to make the testes work less efficiently when/if someone comes off of pharmaceutical testosterone. This is not simply the sluggishness the testes experience in restoring testosterone production after exogenous testosterone administration, but a permanent decrease compared to pre-TRT levels. There may be exceptions to this with specialized protocols, but generally, if one is on TRT for some time without taking hCG or LH at the same time, discontinuing TRT tends to result in diminished natural testosterone production. 3) LH ( & FSH) are necessary to adequate sperm production for fertility. ~Xian > > I READ THIS ON THE WORLDCLASS BODYBUILDING FITNESS FORUM: > > > LH > > Luteinizing hormone is responsible for the trigger of testosterone release in the body. Basically, LH which is secreted in the Testes triggers the body to produce Testosterone. Once this is done the hypothalmus picks up on it in a negative feed back process (inhibition of GnRH). Thus, LH " checks " itself. > > What does this mean? > > Well, LH is a fine balance. Not enough, and your body doesn't produce Test. Too much, same effect. Now introduce synthetic Testosterone into the loop and what do we have? A break in the cycle. Boom...body stops producing LH. > > Now wait a minute. If we're on cycle why would we want to tell the body produce LH when it doesn't need to? People think your testicles shrink due to lowered Test production. No no no. This is DIRECTLY linked to LH and FSH depletion. Test is NOT made in the testes. LH is made in the Testes which SIGNALS the body to make Test. Well if there's excess Test introduced why do we need LH? Are you guys seeing the link yet? > > Excess test = no need for LH = shutdown of LH = Testicle shrinkage > > So what are we aiming for? Well we're aiming for just enough LH production to keep the loop working, BUT not too much where the body downgrades receptors because it's gone haywire. > > I THOUGHT THE PITUARY GLAND PRODUCES LH > I THOUGHT TESTES PRODUCES TESTOSTERONE > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW LH WORKS > AM I WRONG OR WHAT IS HAPPENING > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 13, 2011 Report Share Posted March 13, 2011 Well they have it wrong read this again the LH is a message sent to the Testis from the Pituitary to tell them to make more T. ==================================================== As in business, the buck stops with the CEO/hypothalamus, which is known as a " pulse generator, " because during the day it sends out pulses of hormones that are designed to stimulate other organs. With respect to T, the hypothalamus sends out numerous daily pulses of GNRH (gonadotropin releasing hormone) through the blood stream. These pulses are designed to stimulate the pituitary gland to get to work. The pituitary gland then senses the pulses of GNRH and sends out a work order of its own, consisting of LH (leutinizing hormone) pulses. The LH message travels down to the leydig cells of the testis to stimulate the enzymatic conversion of cholesterol to Testosterone. Co-Moderator Phil > From: zorroceasar <zorroceasar@...> > Subject: Re: Now TOTALY confused ! > > Date: Sunday, March 13, 2011, 4:39 AM > I have read through what youve send > me. > I still have the same questions. > > This forum in bodybuilding says Lh is made in the testes > and this is a well known forum > and people in the forum agree with him > > This is his statements > LH is made in the testes > Testosterone is not made in the testes > > now everything I ve read so far in our group here > over the past 5 years says otherwise > > > I only had one response and that was from Phil saying yes > and no > Please clarify. > > > > > > > > > > > > > > From: zorroceasar <zorroceasar@...> > > > Subject: Now TOTALY confused ! > > > > > > Date: Friday, March 11, 2011, 1:49 PM > > > I READ THIS ON THE WORLDCLASS > > > BODYBUILDING FITNESS FORUM: > > > > > > > > > LH > > > > > > Luteinizing hormone is responsible for the > trigger of > > > testosterone release in the body. Basically, LH > which is > > > secreted in the Testes triggers the body to > produce > > > Testosterone. Once this is done the hypothalmus > picks up on > > > it in a negative feed back process (inhibition of > GnRH). > > > Thus, LH " checks " itself. > > > > > > What does this mean? > > > > > > Well, LH is a fine balance. Not enough, and your > body > > > doesn't produce Test. Too much, same effect. Now > introduce > > > synthetic Testosterone into the loop and what do > we have? A > > > break in the cycle. Boom...body stops producing > LH. > > > > > > Now wait a minute. If we're on cycle why would we > want to > > > tell the body produce LH when it doesn't need to? > People > > > think your testicles shrink due to lowered Test > production. > > > No no no. This is DIRECTLY linked to LH and FSH > depletion. > > > Test is NOT made in the testes. LH is made in the > Testes > > > which SIGNALS the body to make Test. Well if > there's excess > > > Test introduced why do we need LH? Are you guys > seeing the > > > link yet? > > > > > > Excess test = no need for LH = shutdown of LH = > Testicle > > > shrinkage > > > > > > So what are we aiming for? Well we're aiming for > just > > > enough LH production to keep the loop working, > BUT not too > > > much where the body downgrades receptors because > it's gone > > > haywire. > > > > > > I THOUGHT THE PITUARY GLAND PRODUCES LH > > > I THOUGHT TESTES PRODUCES TESTOSTERONE > > > > > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW > LH WORKS > > > AM I WRONG OR WHAT IS HAPPENING > > > > > > > > > > > > > > > > > > > > > ------------------------------------ > > > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 14, 2011 Report Share Posted March 14, 2011 Your getting this from a bodybuilding forum LOL Lh is made in the pituirary and stimulates the testicles to make testoserone. If you take HCG its an LH analog which stimulates testicle directly to make testosterone if you are not primary. > > > > > From: zorroceasar <zorroceasar@> > > > Subject: Now TOTALY confused ! > > > > > > Date: Friday, March 11, 2011, 1:49 PM > > > I READ THIS ON THE WORLDCLASS > > > BODYBUILDING FITNESS FORUM: > > > > > > > > > LH > > > > > > Luteinizing hormone is responsible for the trigger of > > > testosterone release in the body. Basically, LH which is > > > secreted in the Testes triggers the body to produce > > > Testosterone. Once this is done the hypothalmus picks up on > > > it in a negative feed back process (inhibition of GnRH). > > > Thus, LH " checks " itself. > > > > > > What does this mean? > > > > > > Well, LH is a fine balance. Not enough, and your body > > > doesn't produce Test. Too much, same effect. Now introduce > > > synthetic Testosterone into the loop and what do we have? A > > > break in the cycle. Boom...body stops producing LH. > > > > > > Now wait a minute. If we're on cycle why would we want to > > > tell the body produce LH when it doesn't need to? People > > > think your testicles shrink due to lowered Test production. > > > No no no. This is DIRECTLY linked to LH and FSH depletion. > > > Test is NOT made in the testes. LH is made in the Testes > > > which SIGNALS the body to make Test. Well if there's excess > > > Test introduced why do we need LH? Are you guys seeing the > > > link yet? > > > > > > Excess test = no need for LH = shutdown of LH = Testicle > > > shrinkage > > > > > > So what are we aiming for? Well we're aiming for just > > > enough LH production to keep the loop working, BUT not too > > > much where the body downgrades receptors because it's gone > > > haywire. > > > > > > I THOUGHT THE PITUARY GLAND PRODUCES LH > > > I THOUGHT TESTES PRODUCES TESTOSTERONE > > > > > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW LH WORKS > > > AM I WRONG OR WHAT IS HAPPENING > > > > > > > > > > > > > > > > > > > > > ------------------------------------ > > > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 14, 2011 Report Share Posted March 14, 2011 There are LH receptors through out the body and the brain...end of story.. > > > > > > > From: zorroceasar <zorroceasar@> > > > > Subject: Now TOTALY confused ! > > > > > > > > Date: Friday, March 11, 2011, 1:49 PM > > > > I READ THIS ON THE WORLDCLASS > > > > BODYBUILDING FITNESS FORUM: > > > > > > > > > > > > LH > > > > > > > > Luteinizing hormone is responsible for the trigger of > > > > testosterone release in the body. Basically, LH which is > > > > secreted in the Testes triggers the body to produce > > > > Testosterone. Once this is done the hypothalmus picks up on > > > > it in a negative feed back process (inhibition of GnRH). > > > > Thus, LH " checks " itself. > > > > > > > > What does this mean? > > > > > > > > Well, LH is a fine balance. Not enough, and your body > > > > doesn't produce Test. Too much, same effect. Now introduce > > > > synthetic Testosterone into the loop and what do we have? A > > > > break in the cycle. Boom...body stops producing LH. > > > > > > > > Now wait a minute. If we're on cycle why would we want to > > > > tell the body produce LH when it doesn't need to? People > > > > think your testicles shrink due to lowered Test production. > > > > No no no. This is DIRECTLY linked to LH and FSH depletion. > > > > Test is NOT made in the testes. LH is made in the Testes > > > > which SIGNALS the body to make Test. Well if there's excess > > > > Test introduced why do we need LH? Are you guys seeing the > > > > link yet? > > > > > > > > Excess test = no need for LH = shutdown of LH = Testicle > > > > shrinkage > > > > > > > > So what are we aiming for? Well we're aiming for just > > > > enough LH production to keep the loop working, BUT not too > > > > much where the body downgrades receptors because it's gone > > > > haywire. > > > > > > > > I THOUGHT THE PITUARY GLAND PRODUCES LH > > > > I THOUGHT TESTES PRODUCES TESTOSTERONE > > > > > > > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW LH WORKS > > > > AM I WRONG OR WHAT IS HAPPENING > > > > > > > > > > > > > > > > > > > > > > > > > > > > ------------------------------------ > > > > > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 14, 2011 Report Share Posted March 14, 2011 Yes here is the link , well known member and people thanking him for his wisdom. http://www.worldclassbodybuilding.com/forums/f65/hcg-hmg-and-lh-fsh-explained-71\ 345/ > > > > > > > From: zorroceasar <zorroceasar@> > > > > Subject: Now TOTALY confused ! > > > > > > > > Date: Friday, March 11, 2011, 1:49 PM > > > > I READ THIS ON THE WORLDCLASS > > > > BODYBUILDING FITNESS FORUM: > > > > > > > > > > > > LH > > > > > > > > Luteinizing hormone is responsible for the trigger of > > > > testosterone release in the body. Basically, LH which is > > > > secreted in the Testes triggers the body to produce > > > > Testosterone. Once this is done the hypothalmus picks up on > > > > it in a negative feed back process (inhibition of GnRH). > > > > Thus, LH " checks " itself. > > > > > > > > What does this mean? > > > > > > > > Well, LH is a fine balance. Not enough, and your body > > > > doesn't produce Test. Too much, same effect. Now introduce > > > > synthetic Testosterone into the loop and what do we have? A > > > > break in the cycle. Boom...body stops producing LH. > > > > > > > > Now wait a minute. If we're on cycle why would we want to > > > > tell the body produce LH when it doesn't need to? People > > > > think your testicles shrink due to lowered Test production. > > > > No no no. This is DIRECTLY linked to LH and FSH depletion. > > > > Test is NOT made in the testes. LH is made in the Testes > > > > which SIGNALS the body to make Test. Well if there's excess > > > > Test introduced why do we need LH? Are you guys seeing the > > > > link yet? > > > > > > > > Excess test = no need for LH = shutdown of LH = Testicle > > > > shrinkage > > > > > > > > So what are we aiming for? Well we're aiming for just > > > > enough LH production to keep the loop working, BUT not too > > > > much where the body downgrades receptors because it's gone > > > > haywire. > > > > > > > > I THOUGHT THE PITUARY GLAND PRODUCES LH > > > > I THOUGHT TESTES PRODUCES TESTOSTERONE > > > > > > > > THIS MAN WAS PRAISED FOR HIS EXPLANATION OF HOW LH WORKS > > > > AM I WRONG OR WHAT IS HAPPENING > > > > > > > > > > > > > > > > > > > > > > > > > > > > ------------------------------------ > > > > > > > > Quote Link to comment Share on other sites More sharing options...
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