Guest guest Posted September 21, 2000 Report Share Posted September 21, 2000 Barb, Not studies, but firsthand personal stories (anecdotes/testimonies) about recovery/improvement among individuals with MS and other diagnoses appears in the newly revised book put out by DAMS. My story appears in it so I received a copy of the new version recently. I don't remember the exact name or price. Iowa DAMS compiled it. On Thu, 31 Aug 2000 08:19:00 -0700, egroups wrote: > Bernie, > I really need some solid studies and research I can show my Mom re MS > recovery from mercury poisoning. Yes, she has the study about spinal fluid > returning to normal. What else can you give me? > Barb > [ ] re: Hg FAC > > > > > >(from FAQ) > >Chelation should be done in cycles of at 3 to 7 days on the chelator, > >followed by at least as many days off as a rest period. Many parents > >find it convenient to chelate on weekends, starting when the child gets > >home from school and stopping on Monday morning, with weekdays as rest > >days. With the schedule, sleep is only interrupted on > >weekends and parents don't have to depend on school employees to give > >the medicine. > > > > Do the authors and others believe in Huggins TDR > >philosophy which is probably the best known protocol overall?? It is > >about mercury from amalgam mostly > >but has parts relevant to other exposures: > > > >4. Huggins Total Dental Revision Protocol(35): > >(a) history questionnaire and panel of tests. > >( replace amalgam fillings starting with filling with highest negative > >current or highest negative quadrant, with supportive > >vitamin/mineral supplements. > >© extract all root canaled teeth using proper finish protocol. > >(d) test and treat cavitations and amalgam tattoos where relevant > >(e) supportive supplementation, periodic monitoring tests, evaluate need > >for further treatment(not usually needed). > > (f) avoid acute exposures/challanges to the immune system on a weekly > >7/14/21 day pattern. > > > > Note (f) regarding the above suggested DMSA protocol. If > >you do 3 days on and 4 days off, this might constitute a challange every > >7 days in violation of (f). Or if you do 5 days on and 9 days off, > >likewise or any pattern that begins on the same day > >1 or 2 weeks later. He gives medical study references plus his > >clinical experience to support his rules, and claims to have cured or > >significantly improved over 1000 cases of MS, ALS, AD, diabetes, FM, > >Lupus, etc. with about a 80 % rate on most of these. > >If thats accurate, its at least as good as anyone else is doing and I > >don't think anyone has treated as many cases as him(other than maybe > >Daunderer) and has as many dentists and doctors using his protocol. > >He says before he used this protocol, he wasn't nearly as successful. > >He also has suggested list of tests for mercury toxicity he uses. > >Bernie > > > > > > Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 6, 2004 Report Share Posted September 6, 2004 they also autopsied a 12yr. old that died during a ms attack found no trace of immune responce compromised blood brain barrier sounds like investigating ----- Original Message ----- From: Bayuk A Home away from Home a MS Support Board ; MS-Christians ; MS_Community ; MSersLife ; mscured ; low dose naltrexone ; MSfriends ; MSViews_Multiple_Sclerosis ; jjworld Sent: Monday, September 06, 2004 8:11 AM Subject: [low dose naltrexone] ms More MS news articles for March 2004 Immune system cause of multiple sclerosis questioned Study shows evidence that myelin-producing cells die before there is any immune system activity http://www.mult-sclerosis.org March 18, 2004 All About Multiple Sclerosis Prevailing medical wisdom says that multiple sclerosis is an autoimmune disease - a disease in which the body's immune system turns in on itself. Specifically, it attacks the myelin sheaths that insulate the nerve cells in the brain and spinal cord. This immune system activity produces inflammation similar to what happens in the skin when we get a pimple. Crucially, the inflammation also kills the cells responsible for producing and maintaining the myelin. These cells are called oligodendrocytes and they have long been known to die in large numbers during attacks of MS. The majority of existing treatments for the disease and a fair proportion of new treatments currently in research focus on reducing the inflammation or disabling the immune system cells responsible for it. However, a dramatic piece of new research published in The ls of Neurology threatens to turn this understanding of multiple sclerosis on its head. The study examined twelve brains of people with multiple sclerosis, concentrating on newly forming areas of disease activity called lesions. It found that the oligodendrocytes in these lesions were dying before there were any signs of inflammation. This implies that it is not the inflammation that causes the death of the oligodendrocytes in multiple sclerosis but the other way around. The inflammation occurs in response to the oligodendrocyte cell death. The authors, Barnett and Prineas of the Institute of Clinical Neurosciences at the University of Sydney, Australia don't deny that the inflammation might cause some of the damage seen in multiple sclerosis but they do paint a radically new picture of of the disease. They suggest that the first stage of the development of a new multiple sclerosis lesion is mass suicide of the oligodendrocytes over a relatively small area. This process is called apoptosis or programmed cell death and is a normal response in the human body during growth and repair. If cells were allowed to grow and divide without limits, they would form a cancer. Similarily, cells infected by viruses or cells that are no longer needed by the body will often cell kill themselves. Barnett and Prineas observed oligodendrocytes in which the central nucleus was shrivelling up - a typical sign of a cell committing suicide. Other cells in the brain were also changing. Microglia, another type of maintenance cell which can swallow up dead and dying cells, were forming long extensions ready to engulf the dead and dying oligodendrocytes. Additionally, a group of proteins, called complement, which are responsible for activating the body's rubbish-collecting cells, had collected on the myelin. Crucially, the rubbish-collecting cells of the immune system, the macrophages, had not yet appeared in the lesion. Within one or two days of lesion formation, all the oligodendrocytes had disappeared. The authors suggest that they had been swallowed up by the microglia. The spaces that they had once occupied were now full of liquid forming what is known as edema. The next stage seems to be the invasion of immune system cells. Macrophages now start to appear, together with T cells, the orchestrators of the immune response. These initiate and take part in inflammation. The macrophages start to gobble up the myelin left over by the vanished oligodendrocytes. The final stage would appear to be regeneration. Oligodendrocyte precursor cells, cells that have the ability to develop into new oligodendrocytes, move in to replace the lost cells. They are fed special chemicals called trophic factors by the macrophages and the process of remyelination can begin. It is important to bear in mind that this was a study of only 12 brains and further work needs to be done to validate the studies findings. However, if this work reflects what is actually happening in multiple sclerosis, then its implications are earth shattering: Multiple sclerosis will no longer be an autoimmune disease. A lot of text books are going to have to be rewritten. Treatments that target inflammation will not not addressing the root cause of the diease. This does not mean that they are not effective to some degree but that they can never be as good as treatments that target the death of the oligodendrocytes. All the animal models of multiple sclerosis are poor representations of the disease in that they are all primarily autoimmune models. Perhaps this is why so many treatments that are so effective in mouse models prove to make no difference to multiple sclerosis in humans. For animal models to be valid, they would need to show the kind of disease process described by Barnett and Prineas. Researchers will need to change direction. Whilst work on oligodendrocyte precursor cells becomes more important than ever, work on describing the inflammation process in multiple sclerosis needs to take a back-seat. Importantly, researchers need to find out why oligodendrocytes are dying and what can be done to stop them. Quite how the world of multiple sclerosis research will react to this paper is unclear. Thus far, Barnett and Prineas's paper seems to have been met with a deafening silence which is why I decided to write this piece. Source: Relapsing and remitting multiple sclerosis: Pathology of the newly forming lesion H. Barnett, MBBS, W. Prineas, MBBS * ls of Neurology, Feb 23, 2004 http://www3.interscience.wiley.com/cgi-bin/abstract/107629227/ABSTRACT Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 7, 2004 Report Share Posted September 7, 2004 Tom, I just looked at the site you gave - www.mult-sclerosis.org. If you look at the bit where it says famous people with MS. What do you notice? Well I noticed what an incredibly talented bunch of people they were and most of them are involved in the arts. That either means that people only become famous if they are artistic, or they are artistic as a result of the MS, or the statistics for getting MS are high if you are an artist. I am myself an artist. Just Curious. Audrey -- Bayuk <tbayuk@...> wrote: > > > More MS news articles for March 2004 > > -------------------------------------------------------------------------------- > Immune system cause of multiple sclerosis questioned > > Study shows evidence that myelin-producing cells die > before there is any immune system activity > > http://www.mult-sclerosis.org > > March 18, 2004 > > All About Multiple Sclerosis > > Prevailing medical wisdom says that multiple > sclerosis is an autoimmune disease - a disease in > which the body's immune system turns in on itself. > Specifically, it attacks the myelin sheaths that > insulate the nerve cells in the brain and spinal > cord. This immune system activity produces > inflammation similar to what happens in the skin > when we get a pimple. > > Crucially, the inflammation also kills the cells > responsible for producing and maintaining the > myelin. These cells are called oligodendrocytes and > they have long been known to die in large numbers > during attacks of MS. > > The majority of existing treatments for the disease > and a fair proportion of new treatments currently in > research focus on reducing the inflammation or > disabling the immune system cells responsible for > it. > > However, a dramatic piece of new research published > in The ls of Neurology threatens to turn this > understanding of multiple sclerosis on its head. > > The study examined twelve brains of people with > multiple sclerosis, concentrating on newly forming > areas of disease activity called lesions. It found > that the oligodendrocytes in these lesions were > dying before there were any signs of inflammation. > > This implies that it is not the inflammation that > causes the death of the oligodendrocytes in multiple > sclerosis but the other way around. The inflammation > occurs in response to the oligodendrocyte cell > death. > > The authors, Barnett and Prineas of the > Institute of Clinical Neurosciences at the > University of Sydney, Australia don't deny that the > inflammation might cause some of the damage seen in > multiple sclerosis but they do paint a radically new > picture of of the disease. > > They suggest that the first stage of the development > of a new multiple sclerosis lesion is mass suicide > of the oligodendrocytes over a relatively small > area. This process is called apoptosis or programmed > cell death and is a normal response in the human > body during growth and repair. If cells were allowed > to grow and divide without limits, they would form a > cancer. Similarily, cells infected by viruses or > cells that are no longer needed by the body will > often cell kill themselves. > > Barnett and Prineas observed oligodendrocytes in > which the central nucleus was shrivelling up - a > typical sign of a cell committing suicide. Other > cells in the brain were also changing. Microglia, > another , which are responsible for > activating the botype of maintenance cell which can swallow > up dead and dying cells, were forming long > extensions ready to engulf the dead and dying > oligodendrocytes. Additionally, a group of proteins, > called complementdy's rubbish-collecting cells, had > collected on the myelin. Crucially, the > rubbish-collecting cells of the immune system, the > macrophages, had not yet appeared in the lesion. > > Within one or two days of lesion formation, all the > oligodendrocytes had disappeared. The authors > suggest that they had been swallowed up by the > microglia. The spaces that they had once occupied > were now full of liquid forming what is known as > edema. > > The next stage seems to be the invasion of immune > system cells. Macrophages now start to appear, > together with T cells, the orchestrators of the > immune response. These initiate and take part in > inflammation. The macrophages start to gobble up the > myelin left over by the vanished oligodendrocytes. > > The final stage would appear to be regeneration. > Oligodendrocyte precursor cells, cells that have the > ability to develop into new oligodendrocytes, move > in to replace the lost cells. They are fed special > chemicals called trophic factors by the macrophages > and the process of remyelination can begin. > > It is important to bear in mind that this was a > study of only 12 brains and further work needs to be > done to validate the studies findings. However, if > this work reflects what is actually happening in > multiple sclerosis, then its implications are earth > shattering: > > a.. Multiple sclerosis will no longer be an > autoimmune disease. A lot of text books are going to > have to be rewritten. > a.. Treatments that target inflammation will not > not addressing the root cause of the diease. This > does not mean that they are not effective to some > degree but that they can never be as good as > treatments that target the death of the > oligodendrocytes. > a.. All the animal models of multiple sclerosis > are poor representations of the disease in that they > are all primarily autoimmune models. Perhaps this is > why so many treatments that are so effective in > mouse models prove to make no difference to multiple > sclerosis in humans. For animal models to be valid, > they would need to show the kind of disease process > described by Barnett and Prineas. > a.. Researchers will need to change direction. > Whilst work on oligodendrocyte precursor cells > becomes more important than ever, work on describing > the inflammation process in multiple sclerosis needs > to take a back-seat. Importantly, researchers need > to find out why oligodendrocytes are dying and what > can be done to stop them. > Quite how the world of multiple sclerosis research > will react to this paper is unclear. Thus far, > Barnett and Prineas's paper seems to have been met > with a deafening silence which is why I decided to > write this piece. > Source: > > Relapsing and remitting multiple sclerosis: > Pathology of the newly forming lesion > H. Barnett, MBBS, W. Prineas, MBBS * > ls of Neurology, Feb 23, 2004 > http://www3.interscience.wiley.com/cgi-bin/abstract/107629227/ABSTRACT > ATTACHMENT part 2 image/gif name=personal.gif > ATTACHMENT part 3 image/gif name=links.gif > ATTACHMENT part 4 image/gif name=search.gif > ATTACHMENT part 5 image/gif name=about.gif ___________________________________________________________ALL-NEW Messenger - all new features - even more fun! http://uk.messenger. Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 7, 2004 Report Share Posted September 7, 2004 Along this same general line of thinking, there are many , many people who are or who have been involved in the medical industry, myself included, who now have MS. It's interesting, if nothing more. I think that you tend to notice that like people also have the disease. I'm not sure if there's any hidden meaning behind all of this but, you do tend to take notice more easily given the similarities you share with them. Would be further interesting to take a poll of what other MS'ers do or have done for a living. DesinieAudrey Henry <mumpuss@...> wrote: Tom, I just looked at the site you gave - www.mult-sclerosis.org.If you look at the bit where it says famous peoplewith MS. What do you notice?Well I noticed what an incredibly talented bunch ofpeople they were and most of them are involved in thearts.That either means that people only become famous ifthey are artistic,or they are artistic as a result of the MS, or the statistics for getting MS are high if you arean artist. I am myself an artist.Just Curious. Audrey-- Bayuk <tbayuk@...> wrote: > > > More MS news articles for March 2004> >--------------------------------------------------------------------------------> Immune system cause of multiple sclerosis questioned> > Study shows evidence that myelin-producing cells die> before there is any immune system activity > > http://www.mult-sclerosis.org > > March 18, 2004 > > All About Multiple Sclerosis > > Prevailing medical wisdom says that multiple> sclerosis is an autoimmune disease - a disease in> which the body's immune system turns in on itself.> Specifically, it attacks the myelin sheaths that> insulate the nerve cells in the brain and spinal> cord. This immune system activity produces> inflammation similar to what happens in the skin> when we get a pimple. > > Crucially, the inflammation also kills the cells> responsible for producing and maintaining the> myelin. These cells are called oligodendrocytes and> they have long been known to die in large numbers> during attacks of MS. > > The majority of existing treatments for the disease> and a fair proportion of new treatments currently in> research focus on reducing the inflammation or> disabling the immune system cells responsible for> it. > > However, a dramatic piece of new research published> in The ls of Neurology threatens to turn this> understanding of multiple sclerosis on its head. > > The study examined twelve brains of people with> multiple sclerosis, concentrating on newly forming> areas of disease activity called lesions. It found> that the oligodendrocytes in these lesions were> dying before there were any signs of inflammation. > > This implies that it is not the inflammation that> causes the death of the oligodendrocytes in multiple> sclerosis but the other way around. The inflammation> occurs in response to the oligodendrocyte cell> death. > > The authors, Barnett and Prineas of the> Institute of Clinical Neurosciences at the> University of Sydney, Australia don't deny that the> inflammation might cause some of the damage seen in> multiple sclerosis but they do paint a radically new> picture of of the disease. > > They suggest that the first stage of the development> of a new multiple sclerosis lesion is mass suicide> of the oligodendrocytes over a relatively small> area. This process is called apoptosis or programmed> cell death and is a normal response in the human> body during growth and repair. If cells were allowed> to grow and divide without limits, they would form a> cancer. Similarily, cells infected by viruses or> cells that are no longer needed by the body will> often cell kill themselves. > > Barnett and Prineas observed oligodendrocytes in> which the central nucleus was shrivelling up - a> typical sign of a cell committing suicide. Other> cells in the brain were also changing. Microglia,> another , which are responsible for> activating the botype of maintenance cell which canswallow> up dead and dying cells, were forming long> extensions ready to engulf the dead and dying> oligodendrocytes. Additionally, a group of proteins,> called complementdy's rubbish-collecting cells, had> collected on the myelin. Crucially, the> rubbish-collecting cells of the immune system, the> macrophages, had not yet appeared in the lesion. > > Within one or two days of lesion formation, all the> oligodendrocytes had disappeared. The authors> suggest that they had been swallowed up by the> microglia. The spaces that they had once occupied> were now full of liquid forming what is known as> edema. > > The next stage seems to be the invasion of immune> system cells. Macrophages now start to appear,> together with T cells, the orchestrators of the> immune response. These initiate and take part in> inflammation. The macrophages start to gobble up the> myelin left over by the vanished oligodendrocytes. > > The final stage would appear to be regeneration.> Oligodendrocyte precursor cells, cells that have the> ability to develop into new oligodendrocytes, move> in to replace the lost cells. They are fed special> chemicals called trophic factors by the macrophages> and the process of remyelination can begin. > > It is important to bear in mind that this was a> study of only 12 brains and further work needs to be> done to validate the studies findings. However, if> this work reflects what is actually happening in> multiple sclerosis, then its implications are earth> shattering: > > a.. Multiple sclerosis will no longer be an> autoimmune disease. A lot of text books are going to> have to be rewritten. > a.. Treatments that target inflammation will not> not addressing the root cause of the diease. This> does not mean that they are not effective to some> degree but that they can never be as good as> treatments that target the death of the> oligodendrocytes. > a.. All the animal models of multiple sclerosis> are poor representations of the disease in that they> are all primarily autoimmune models. Perhaps this is> why so many treatments that are so effective in> mouse models prove to make no difference to multiple> sclerosis in humans. For animal models to be valid,> they would need to show the kind of disease process> described by Barnett and Prineas. > a.. Researchers will need to change direction.> Whilst work on oligodendrocyte precursor cells> becomes more important than ever, work on describing> the inflammation process in multiple sclerosis needs> to take a back-seat. Importantly, researchers need> to find out why oligodendrocytes are dying and what> can be done to stop them. > Quite how the world of multiple sclerosis research> will react to this paper is unclear. Thus far,> Barnett and Prineas's paper seems to have been met> with a deafening silence which is why I decided to> write this piece. > Source: > > Relapsing and remitting multiple sclerosis:> Pathology of the newly forming lesion > H. Barnett, MBBS, W. Prineas, MBBS * > ls of Neurology, Feb 23, 2004 > http://www3.interscience.wiley.com/cgi-bin/abstract/107629227/ABSTRACT> ATTACHMENT part 2 image/gif name=personal.gif> ATTACHMENT part 3 image/gif name=links.gif> ATTACHMENT part 4 image/gif name=search.gif> ATTACHMENT part 5 image/gif name=about.gif ___________________________________________________________ALL-NEW Messenger - all new features - even more fun! http://uk.messenger. __________________________________________________ Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 7, 2004 Report Share Posted September 7, 2004 A poll of occupation and interests, hobbies, etc would be very interesting. ----- Original Message ----- From: Desinie low dose naltrexone Sent: Monday, September 06, 2004 8:20 PM Subject: Re: [low dose naltrexone] ms Along this same general line of thinking, there are many , many people who are or who have been involved in the medical industry, myself included, who now have MS. It's interesting, if nothing more. I think that you tend to notice that like people also have the disease. I'm not sure if there's any hidden meaning behind all of this but, you do tend to take notice more easily given the similarities you share with them. Would be further interesting to take a poll of what other MS'ers do or have done for a living. DesinieAudrey Henry <mumpuss@...> wrote: Tom, I just looked at the site you gave - www.mult-sclerosis.org.If you look at the bit where it says famous peoplewith MS. What do you notice?Well I noticed what an incredibly talented bunch ofpeople they were and most of them are involved in thearts.That either means that people only become famous ifthey are artistic,or they are artistic as a result of the MS, or the statistics for getting MS are high if you arean artist. I am myself an artist.Just Curious. Audrey-- Bayuk <tbayuk@...> wrote: > > > More MS news articles for March 2004> >--------------------------------------------------------------------------------> Immune system cause of multiple sclerosis questioned> > Study shows evidence that myelin-producing cells die> before there is any immune system activity > > http://www.mult-sclerosis.org > > March 18, 2004 > > All About Multiple Sclerosis > > Prevailing medical wisdom says that multiple> sclerosis is an autoimmune disease - a disease in> which the body's immune system turns in on itself.> Specifically, it attacks the myelin sheaths that> insulate the nerve cells in the brain and spinal> cord. This immune system activity produces> inflammation similar to what happens in the skin> when we get a pimple. > > Crucially, the inflammation also kills the cells> responsible for producing and maintaining the> myelin. These cells are called oligodendrocytes and> they have long been known to die in large numbers> during attacks of MS. > > The majority of existing treatments for the disease> and a fair proportion of new treatments currently in> research focus on reducing the inflammation or> disabling the immune system cells responsible for> it. > > However, a dramatic piece of new research published> in The ls of Neurology threatens to turn this> understanding of multiple sclerosis on its head. > > The study examined twelve brains of people with> multiple sclerosis, concentrating on newly forming> areas of disease activity called lesions. It found> that the oligodendrocytes in these lesions were> dying before there were any signs of inflammation. > > This implies that it is not the inflammation that> causes the death of the oligodendrocytes in multiple> sclerosis but the other way around. The inflammation> occurs in response to the oligodendrocyte cell> death. > > The authors, Barnett and Prineas of the> Institute of Clinical Neurosciences at the> University of Sydney, Australia don't deny that the> inflammation might cause some of the damage seen in> multiple sclerosis but they do paint a radically new> picture of of the disease. > > They suggest that the first stage of the development> of a new multiple sclerosis lesion is mass suicide> of the oligodendrocytes over a relatively small> area. This process is called apoptosis or programmed> cell death and is a normal response in the human> body during growth and repair. If cells were allowed> to grow and divide without limits, they would form a> cancer. Similarily, cells infected by viruses or> cells that are no longer needed by the body will> often cell kill themselves. > > Barnett and Prineas observed oligodendrocytes in> which the central nucleus was shrivelling up - a> typical sign of a cell committing suicide. Other> cells in the brain were also changing. Microglia,> another , which are responsible for> activating the botype of maintenance cell which canswallow> up dead and dying cells, were forming long> extensions ready to engulf the dead and dying> oligodendrocytes. Additionally, a group of proteins,> called complementdy's rubbish-collecting cells, had> collected on the myelin. Crucially, the> rubbish-collecting cells of the immune system, the> macrophages, had not yet appeared in the lesion. > > Within one or two days of lesion formation, all the> oligodendrocytes had disappeared. The authors> suggest that they had been swallowed up by the> microglia. The spaces that they had once occupied> were now full of liquid forming what is known as> edema. > > The next stage seems to be the invasion of immune> system cells. Macrophages now start to appear,> together with T cells, the orchestrators of the> immune response. These initiate and take part in> inflammation. The macrophages start to gobble up the> myelin left over by the vanished oligodendrocytes. > > The final stage would appear to be regeneration.> Oligodendrocyte precursor cells, cells that have the> ability to develop into new oligodendrocytes, move> in to replace the lost cells. They are fed special> chemicals called trophic factors by the macrophages> and the process of remyelination can begin. > > It is important to bear in mind that this was a> study of only 12 brains and further work needs to be> done to validate the studies findings. However, if> this work reflects what is actually happening in> multiple sclerosis, then its implications are earth> shattering: > > a.. Multiple sclerosis will no longer be an> autoimmune disease. A lot of text books are going to> have to be rewritten. > a.. Treatments that target inflammation will not> not addressing the root cause of the diease. This> does not mean that they are not effective to some> degree but that they can never be as good as> treatments that target the death of the> oligodendrocytes. > a.. All the animal models of multiple sclerosis> are poor representations of the disease in that they> are all primarily autoimmune models. Perhaps this is> why so many treatments that are so effective in> mouse models prove to make no difference to multiple> sclerosis in humans. For animal models to be valid,> they would need to show the kind of disease process> described by Barnett and Prineas. > a.. Researchers will need to change direction.> Whilst work on oligodendrocyte precursor cells> becomes more important than ever, work on describing> the inflammation process in multiple sclerosis needs> to take a back-seat. Importantly, researchers need> to find out why oligodendrocytes are dying and what> can be done to stop them. > Quite how the world of multiple sclerosis research> will react to this paper is unclear. Thus far,> Barnett and Prineas's paper seems to have been met> with a deafening silence which is why I decided to> write this piece. > Source: > > Relapsing and remitting multiple sclerosis:> Pathology of the newly forming lesion > H. Barnett, MBBS, W. Prineas, MBBS * > ls of Neurology, Feb 23, 2004 > http://www3.interscience.wiley.com/cgi-bin/abstract/107629227/ABSTRACT> ATTACHMENT part 2 image/gif name=personal.gif> ATTACHMENT part 3 image/gif name=links.gif> ATTACHMENT part 4 image/gif name=search.gif> ATTACHMENT part 5 image/gif name=about.gif ___________________________________________________________ALL-NEW Messenger - all new features - even more fun! http://uk.messenger. __________________________________________________ Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 8, 2004 Report Share Posted September 8, 2004 I have read that blood testing for heavy metals only defines what metals are circulationg, not what metals reside in the body. Supposedly, the best way to determine your heavy metals is through hair analysis. Has anyone else read this opinion? Thanks. Little > This is an excerpt of a speech given to the Canadian MS Society in November of 1997. The speech was given by an ms patient that cured himself and is still cured 7 years later. > > Orthodox Medicine's idea of helping people with chronic illness is to drug them. Millions of dollars poured into MS research every year are poured right down the drain and into the bottom line of chemical companies. The ever-elusive magic drug will never be created. No drug ever cured any disease. No drug will ever cure MS. MS is not a prednisone deficiency or a deficiency of any other drug. Drugs are chemicals. They are created to suppress symptoms and to make profits for chemical companies. In the process they kill thousands of people every year and destroy the health of tens of thousands more. Modern Medicine is driven by corporate profit. Not the health of the public. > > It's no surprise that you will never hear the MS Society tell anyone to get tested for metal toxicity or mineral and DHEA deficiencies. This only involves simple blood and urine tests. Well, even simple lab testing is a threat to the MS Society. If people find that they are toxic and deficient, and they will, this would expose the Society. So the Society never utters the words " lab testing " . They have a mission - to protect themselves and their friends at the College of Physicians and Surgeons and The College of Dental Surgeons whose livelihoods depend on the MS Society helping to perpetuate the lie about the safety of dental mercury. The Dental Industry generates billions of dollars every year in revenue. It cannot afford to have the MS Society oppose mercury. The dominos would start to fall. So, it makes sure the MS Society remains solidly pro-mercury. There is too much at stake here. The Dental Industry is totally corrupt and will do anything to retain its market and its power. And so will their friends at the MS Society. > > If you want to find out the truth about dental mercury and MS you can take my example and investigate the facts I have presented to you today. My recovery was no accident. I ignored the advice of the MS Society and had my dental mercury removed, had chelation and took DHEA. My recovery was immediate and dramatic. I now help people with MS from all over the world get the information they need to start on the path to recovery. I am doing the job that the MS Society should be doing. They're too busy running for the cure. > > > > > The Ultimate Zapper > multiple sclerosis > crohn's disease > mercury toxicity > biological dentistry > root canals > parasites > DMSA chelation Quote Link to comment Share on other sites More sharing options...
Guest guest Posted September 8, 2004 Report Share Posted September 8, 2004 I did have a hair analyses done once when I was first diagnosed with M.S. back in 79 or 80 but they never found a lot of mercury just generaly high levels of zinc and other toxic chemicals as I was a welder back then. Reg. -------Original Message------- From: low dose naltrexone Date: 09/08/04 12:35:15 low dose naltrexone Subject: [low dose naltrexone] Re: ms I have read that blood testing for heavy metals only defines what metals are circulationg, not what metals reside in the body. Supposedly, the best way to determine your heavy metals is through hair analysis. Has anyone else read this opinion? Thanks. Little> This is an excerpt of a speech given to the Canadian MS Society in November of 1997. The speech was given by an ms patient that cured himself and is still cured 7 years later.> > Orthodox Medicine's idea of helping people with chronic illness is to drug them. Millions of dollars poured into MS research every year are poured right down the drain and into the bottom line of chemical companies. The ever-elusive magic drug will never be created. No drug ever cured any disease. No drug will ever cure MS. MS is not a prednisone deficiency or a deficiency of any other drug. Drugs are chemicals. They are created to suppress symptoms and to make profits for chemical companies. In the process they kill thousands of people every year and destroy the health of tens of thousands more. Modern Medicine is driven by corporate profit. Not the health of the public.> > It's no surprise that you will never hear the MS Society tell anyone to get tested for metal toxicity or mineral and DHEA deficiencies. This only involves simple blood and urine tests. Well, even simple lab testing is a threat to the MS Society. If people find that they are toxic and deficient, and they will, this would expose the Society. So the Society never utters the words "lab testing". They have a mission - to protect themselves and their friends at the College of Physicians and Surgeons and The College of Dental Surgeons whose livelihoods depend on the MS Society helping to perpetuate the lie about the safety of dental mercury. The Dental Industry generates billions of dollars every year in revenue. It cannot afford to have the MS Society oppose mercury. The dominos would start to fall. So, it makes sure the MS Society remains solidly pro-mercury. There is too much at stake here. The Dental Industry is totally corrupt and will do anything to retain its market and its power. And so will their friends at the MS Society.> > If you want to find out the truth about dental mercury and MS you can take my example and investigate the facts I have presented to you today. My recovery was no accident. I ignored the advice of the MS Society and had my dental mercury removed, had chelation and took DHEA. My recovery was immediate and dramatic. I now help people with MS from all over the world get the information they need to start on the path to recovery. I am doing the job that the MS Society should be doing. They're too busy running for the cure.> > > > > The Ultimate Zapper > multiple sclerosis > crohn's disease > mercury toxicity > biological dentistry > root canals > parasites > DMSA chelation ____________________________________________________ IncrediMail - Email has finally evolved - Click Here Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 5, 2009 Report Share Posted March 5, 2009 Hi all,I remember reading an email the other day from someone who wanted to deal with MS. I wanted to share this article which might be helpful...fyi..I am a big believer in the NAET method described in the short article and would encourage you to look into it.(It also mentions severe pain for people with titanium in their bodies and how that can be cleared...)Blessings,Traci "Dove" Quote Link to comment Share on other sites More sharing options...
Guest guest Posted March 8, 2009 Report Share Posted March 8, 2009 Hi Traci, I could not access the file you sent and I do not know why as I would have loved to read it...I was diagnosed in 1987 with MS...the prognosis was that I was to become a vegetable basically. I went to 2 support groups and became totally depressed at the people in wheel chairs, depression and only speaking of the progression of this disease. For me I became frightened and angry and never returned. I knew people were healed of all kinds of illnesses but you never hear from them because they never return to doctors. I set out on a quest to find someone who was.. I was without family or friends. I did find the person and it is too long a story for here. I found an osteopathic physician who mostly did cranio-sacral work, a very spiritual man, no white coat, not affiliated with any hospital, slow on prescribing drugs but high on other things important to healing. I have been with him 20 yrs. now...I am doing well, have only had periods of time needing lots of sleep but after that enjoy a good life, even though I was never able to return to work full time...I consider this a blessing as it put me on a spiritual journey which is my path in life. I do not recommend anyone do what I did as it came from a place deep inside me and things came together after I had made a decision to heal. I no longer speak of it as nobody would believe this of me anyway but I was curious in the attachment which I was not able to download. I see you on a horse and I love horses. I have not done enough horseback riding in this lifetime.... Namaste, Morningstar >> Hi all,> > I remember reading an email the other day from someone who wanted to deal with MS. I wanted to share this article which might be helpful...fyi..I am a big believer in the NAET method described in the short article and would encourage you to look into it.> (It also mentions severe pain for people with titanium in their bodies and how that can be cleared...)> > Blessings,> Traci "Dove"> Quote Link to comment Share on other sites More sharing options...
Guest guest Posted December 19, 2009 Report Share Posted December 19, 2009 Yes Dave...to right..anything that could be a much faster and relatively easy solution to MS..would be heavily resisted by the vested interested PHARMA CARTELS...and many charities are complicitive in this...ie closed to real and safe solutions!! Maybe all MS patients should undergo chelation..to clean out all the circulatory system. AJD Quote Link to comment Share on other sites More sharing options...
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