(No subject)

[Guest guest]

Hi Group;

Before sending anyone an email I guess there are a couple of things I should

do...

Turn on a light, and read what I've written before I push send. I'm very

embarrased about my poor grammer, syntax, punctuation and spelling, but;

what the hell!

'

[Guest guest]

re:

what is toxic thyroidism? thank you for your advice i am just scared to go

to the endocrinoligist and they say it is all in my head then where do i turn?

allisonrose

[Guest guest]

hey guys it me allisonrose

i need to find an endocrinologist in sherman oaks area

thank you allisonrose

[Guest guest]

Tell them to check your thyroid. Do either a thyroid panel or a antithyroid

antibodies test. Hyperthyroidism is frequently misdiagnosed as anxiety or

something else. Get them to do the blood work.

Good luck.

Lynn

(no subject)

HI, I AM A 24 YEAR OLD WHO DOESN'T EVEN KNOW WHICH WAY IS UP ANYMORE...I

A MARRIED WITH A 3 YR OLD GIRL.....I HAVE ALWAYS BEEN HYPER/ANXIOUS (IN A

SENSE) EXCITED AND A FAST TALKER...PEOPLE ALWAYS THOUGHT I WAS ON DRUGS

YIKES!

I RECENTLY HAVE HAD THE WORST EXPERIENCE OF MY LIFE IN THE MONTH OF

THANKSGIVING NOVEMBER 99 I TOOK THE EMERGENCY BIRTH CONTROL...AFTER THAT MY

LIFE HASN'T BEEN THE SAME...I HAVE NEVER BEEN ABLE TO TAKE BIRTHH CONTROL

THOUGH..after i have my period i notice my heart totally races i notice it

when i go to bed and when i wake up and throughout the day,....i feel

nauseas

and have loose stools i have this feeling of a wave come over me and this

wierd internal like tremble happen i notice my right hand gets shakey and my

muscles feel slightly shakey my palms sweat and i get hot flashes/night

sweats. i eat and eat and will eat a huge meal and dessert and be hungery

an

hour later but i am only 100 pounds...when i first got sick though i dropped

to 94 pounds i have loose stools and i get slitley dizzy...tonight i notice

i

have a heavy pressure or lump feeling in my throat and a heavy feeling under

my chin...i have been to the heart dr/internist/everyone says anxiety? help

help

------------------------------------------------------------------------

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[Guest guest]

Hello ALLISONROSE;

It seems that you may have found the right place to

talk about your difficulties. My advice would be to get to endocrinologist

(an MD specializing in the the glands) ASAP. Your symptoms certainly

parallel my own before I was diagnosed with toxic hyperthroidism, in fact,

they are identical. As it turned out, being scared and so stressed only made

it worse. " Regular " doctors misdiagnosed me too, I spent over a week in

intensive care before an endo was called and correctly diagnosed me. I even

submitted to a cardiac catheterization because my doctor was sure I'd had a

heart attact after he dismissed his first diagnosis of pulmonary embolism

with its hundreds of diaagnostic lung x-rays. My husband suspected me of

alcohol or drug use because of the shaking tremors of my entire body. He was

getting angry at me since my doctor dismissed the shaking as " nerves " . I can

really idenetify with your distress. Please get yourself to the specialist

in endocrinology and let us know how things turn out. My doctor put me on

medication and suggested I have radioactive iodine to " Kill " the thyroid. I

opted to try medication alone and I warn you it takes several weeks to kick

in, but it does the job. Try not to let yourself get rushed into an

irreversible treatment before you've had a chance to view all your options.

Stay glued to this board and ask lots of questions as there are many very

knowledgable people here who will take the time to respond. Sorry to go on

so.

Peace and Light,

'

[Guest guest]

Rose,

If no one has a suggestion, which doubt, check yellow pages OR go to

switchboard and check there. Good luck!

Lynn

Re: (no subject)

hey guys it me allisonrose

i need to find an endocrinologist in sherman oaks area

thank you allisonrose

[Guest guest]

Why would someone reply to a distraught-sounding woman asking for help with

a suggestion to " check out " an on-line discussion of legalization of

marijiuana? Please help correct my ignorance, is this what is commonly

referred to as SPAM? Thanks a lot. Oh by the way, I noticed this person was

reluctant to even use a first name.

[Guest guest]

Check this out go to discussion/legalization of medical marijuana, after all

it's an herb that offers relief for some people.

End the unjust seizure laws.

your opinion matters.

Peace http://www.wnem5.com

(no subject)

> HI, I AM A 24 YEAR OLD WHO DOESN'T EVEN KNOW WHICH WAY IS UP

ANYMORE...I

>A MARRIED WITH A 3 YR OLD GIRL.....I HAVE ALWAYS BEEN HYPER/ANXIOUS (IN A

>SENSE) EXCITED AND A FAST TALKER...PEOPLE ALWAYS THOUGHT I WAS ON DRUGS

YIKES!

>

>I RECENTLY HAVE HAD THE WORST EXPERIENCE OF MY LIFE IN THE MONTH OF

>THANKSGIVING NOVEMBER 99 I TOOK THE EMERGENCY BIRTH CONTROL...AFTER THAT MY

>LIFE HASN'T BEEN THE SAME...I HAVE NEVER BEEN ABLE TO TAKE BIRTHH CONTROL

>THOUGH..after i have my period i notice my heart totally races i notice it

>when i go to bed and when i wake up and throughout the day,....i feel

nauseas

>and have loose stools i have this feeling of a wave come over me and this

>wierd internal like tremble happen i notice my right hand gets shakey and

my

>muscles feel slightly shakey my palms sweat and i get hot flashes/night

>sweats. i eat and eat and will eat a huge meal and dessert and be hungery

an

>hour later but i am only 100 pounds...when i first got sick though i

dropped

>to 94 pounds i have loose stools and i get slitley dizzy...tonight i notice

i

>have a heavy pressure or lump feeling in my throat and a heavy feeling

under

>my chin...i have been to the heart dr/internist/everyone says anxiety? help

>help

>

>------------------------------------------------------------------------

>LOW RATE, NO WAIT!

>Get a NextCard Visa, in 30 seconds! Get rates

>as low as 2.9% Intro or 9.9% Fixed APR and no hidden fees.

>Learn more at:

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>------------------------------------------------------------------------

>

>

[Guest guest]

Alison,

Please get the blood work done. I have been called " flaky " and dismissed by

doctors too. If you can't get to an Endo yet get to an Osteopath (DO instead of

MD). They will at least listen to all your symptoms before passing judgement.

They take one's total symptoms into consideration instead of just zeroing in on

just one.

You can demand blood tests for your thyroid, hormones. After all the doctor

and your family may consider you " flaky " etc -- that is until you get the proper

diagnosis and you start feeling better with the right treatment.

MY husband actually said that he was glad to get his wife back, after so many

years of " the way I was out of it, constantly tired etc.. " That made me kind of

mad; like we would purposely want to feel or act this way.

Also, consider starting 's supplements.

--kathleen

[Guest guest]

....I also noticed the omission of a name - interesting. Exactly how

is marijiuana related to thyroid disease? Sounds like someone has their own

agenda....

Re: (no subject)

Why would someone reply to a distraught-sounding woman asking for help with

a suggestion to " check out " an on-line discussion of legalization of

marijiuana? Please help correct my ignorance, is this what is commonly

referred to as SPAM? Thanks a lot. Oh by the way, I noticed this person was

reluctant to even use a first name.

------------------------------------------------------------------------

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Intro or 9.9% Fixed APR and no hidden fees. Apply NOW!

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------------------------------------------------------------------------

[Guest guest]

Carolann, I use to feel like that when I was not on Tapazole. Fine, I

thought, one day then, in bed the next. I thought it was CFS! Sheri Lynn

[Guest guest]

Hi Group,

I was just wondering if it is normal with Graves to be full of energy one

day and then really tired and worn out the next. I hate feeling like this

and hope the tapazol helps to bring my levels down so I can start feeling

like a human once again.

Thanks....Carolann

[Guest guest]

Sheri,,,,I wonder what causes that? It is soooooo annoying and makes me want

to cry. I guess I'll get over it....lol

Thanks for the reply

Carolann

[Guest guest]

I feel the same way, Carolann.

Deb

[Guest guest]

Deb....I sympathize with you. I know how badly I feel and realize that there

is not much we can do about either. It's so hard to fight depression and

convince yourself that this too shall pass. It's such a wonderful gift to be

able to communicate with others who have the same condition. I am going to

purchase this new book that came out called " The Emerging Mind " , I saw the

author on Larry King last week and the book sounds like it going to be a

very valuable read. It talks about the connection and proof (due to MRI

imaging) between mind and body and how it affects our health and so many

other interesting concepts. I'll let everyone know how it turns out.

Thanks Carolann

[Guest guest]

In a message dated 4/11/00 4:54:45 PM Pacific Daylight Time,

Gillies55@... writes:

<< Dear ,

Your site is great!!! I want to thank you and the group for such wonderful

support. When my sister was diagnosed with 's Disease 8 years ago

(ironically 's D is an over production of copper!) she and my mother

(who actually was caring for her at the peak of W.D.) had no place like

this

to turn to. No one, except doctors that didn't know a thing about 's

and prescribed 3x too much medication and it crippled her. Sad fact.

Kat >>

Hi Kat,

Lately I've been studying 's Disease and belong to a WD group.

's is interesting to be because it is another variation of broken

copper metabolism. In 's copper isn't getting into the cells properly

so it builds up in the liver and causes damage. WD patients take zinc to

prevent copper absorption and also use molybdenum to cause copper excretion.

I think it's possible to manage 's nutritionally also and there may be

things to do for 's that aren't being done.

Today on the WD group site we're discussing the use of lithium (which

seems to facilitate copper metabolism in schizophrenics). There is a doctor

who has 's and he is pretty interesting (very intelligent but showing

signs of schizophrenia). Here is the website if you're interested:

WILSONS-LIST@...

[Guest guest]

we do not give folic acid any more for that reason and she does fine

>From: akally@...

>Reply- egroups

> egroups

>Subject: (no subject)

>Date: Wed, 24 May 2000 12:30:50 EDT

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>

>Hello,

> When you read the interactions...some things tend to jump out at you.

>One

>is the idea that folic acid prevents the side effects of methotrexate. It

>doesn't, it competes with methotrexate for entry into cells so I suppose

>(I'm

>not a doctor, only a mom) giving methotrexate at the same time that you

>give

>your child folic acid isn't the thing to do. I might have read this wrong

>or

>interpreted this the wrong way...if I have, right back.

> The next thing is that NSAIDs should not be administered before, prior

>or

>directly after methotrexate because NSAID therapy can decrease the

>clearance

>of methotrexate, resulting in elevated and prolonged serum methotrexate

>levels.

> The other thing is that caution should be exercised when salicylates

>are

>given in combination with methotrexate. Since both are weak acids,

>salicylates can impair the renal secretion of methotrexate and increase the

>risk of methotrexate toxicity. Salicylates can also displace methotrexate

>from protein-binding sites. Salicylates are ingredients found in aspirin.

> The next thing is methotrexate and steroids. Several corticosteroids

>have been reported to interact with methotrexate. Hydrocortisone has been

>shown to reduce thymidylate synthesis by methotrexate and decreases the

>cytotoxicity of methotrexate in vitro. Other in vitro studies have shown

>that

>hydrocortisone decreases the uptake of methotrexate. Prednisone has been

>shown to block methotrexate-induced inhibition of DNA synthesis.

>Dexamethasone and prednisolone do not interfere with the cytotoxicity of

>methotrexate.

> Once again, I'm only a lay person so to combat these possible adverse

>reactions.... I do the following. I give Tally his methotrexate 4 hours

>later than I do all of his other medications. I normally give it to him

>at

>midnight on Saturday.

> I hope this helps you in your search.

> kathy

>Interactions

>

>

>------------------------------------------------------------------------------

>--

>

>Leucovorin is a reduced folate that, when given after methotrexate

>administration, bypasses methotrexate-induced enzyme blockade

>intracellularly

>and allows DNA synthesis to proceed. Leucovorin is used clinically as a

>rescue agent to reduce methotrexate toxicity in normal tissues. Monitoring

>of

>methotrexate serum concentrations will determine the appropriate dose and

>duration of leucovorin therapy. Excessive methotrexate-induced hematologic

>and gastrointestinal toxicity may occur if the dosage and duration of

>leucovorin therapy is inadequate. Leucovorin appears unlikely to interfere

>with the tumor cell response to methotrexate; tumor cells have higher

>methotrexate polyglutamate formation intracellularly and are thus more

>resistant to leucovorin rescue.

>

>Folic acid, vitamin B9, is not effective for methotrexate rescue therapy

>since folic acid requires dihydrofolate reductase for bioactivation and

>methotrexate inhibits this enzyme. Therefore folic acid should not be used

>to

>prevent methotrexate toxicity. Do not refer to leucovorin as " folinic acid "

>as this could be confused with folic acid and result in serious systemic

>methotrexate toxicity should the wrong rescue treatment be prescribed.

>Folic

>acid may compete with methotrexate for entry into cells.

>

>Nonsteroidal antiinflammatory drugs (NSAIDs) should not be administered

>prior

>to, concomitantly, or following intermediate or high doses of methotrexate.

>Concomitant administration of NSAIDs with high dose methotrexate therapy

>has

>been reported to elevate and prolong serum concentrations of methotrexate

>resulting in deaths from severe hematologic and gastrointestinal toxicity.

>Caution should be used when NSAIDs are administered concurrently with lower

>doses of methotrexate. In patients with rheumatoid arthritis, methotrexate

>has been given concurrently with NSAIDs without apparent problems. It

>should

>be noted that the doses of methotrexate used in rheumatoid arthritis are

>lower than those used in psoriasis or malignant disease; these higher doses

>may lead to unexpected toxicity in combination with NSAIDs. In general,

>NSAID

>therapy can decrease the clearance of methotrexate, resulting in elevated

>and

>prolonged serum methotrexate levels. The potential for an interaction

>between

>methotrexate and celecoxib or rofecoxib has not been evaluated in large

>trials; patients should be monitored closely for signs of methotrexate

>toxicity while receiving these combinations. In a small pharmacokinetic

>study, methotrexate pharmacokinetics were not affected by celecoxib in

>arthritis patients receiving maintenance doses of methotrexate. Rofecoxib

>has

>been reported to decrease methotrexate clearance in one small study.

>Concurrent use of NSAIDs may lead to an increased risk of GI bleeding in

>patients with methotrexate-induced thrombocytopenia.

>

>Caution should be exercised when salicylates are given in combination with

>methotrexate. Since both are weak acids, salicylates can impair the renal

>secretion of methotrexate and increase the risk of methotrexate toxicity.

>Salicylates can also displace methotrexate from protein-binding sites.

>Although the risk for drug interactions with methotrexate is greatest

>during

>HD-MTX therapy, it has been recommended that any of these drugs be used

>cautiously with methotrexate even when methotrexate is used in low doses

>for

>the treatment of rheumatoid arthritis. A significantly higher incidence of

>leukopenia has been reported in patients taking aspirin during methotrexate

>therapy. Large doses of salicylates (>= 6 g/day) can cause

>hypoprothrombinemia, an additional risk factor for bleeding in patients

>with

>thrombocytopenia due to methotrexate.

>

>Methotrexate is partially bound to plasma proteins, and drugs that can

>displace methotrexate from these proteins, such as oral sulfonylureas,

>phenylbutazone, phenytoin, or sulfonamides could cause methotrexate-induced

>toxicity. Due to the potential toxicity of methotrexate, interactions with

>any of these drugs can be very serious even if methotrexate is administered

>in low doses such as in the treatment of rheumatic diseases. When given in

>combination with methotrexate, sulfamethoxazole-trimethoprim, SMX-TMP, may

>be

>associated with additive antifolate effects and increased bone marrow

>suppression due to the trimethoprim component.

>

>Oral antibiotics such as tetracyclines, chloramphenicol, and nonabsorbable

>broad-spectrum antibiotics, may decrease intestinal absorption of

>methotrexate or interfere with enterohepatic circulation by inhibiting

>bowel

>flora and suppressing metabolism of the drug by bacteria. Tetracyclines and

>chloramphenicol may displace methotrexate from protein binding sites

>leading

>to increased methotrexate levels. A case report describes a patient who

>received oral doxycycline in combination with her eleventh course of

>high-dose methotrexate. Methotrexate serum concentrations indicated a

>prolonged half-life and the patient developed severe gastrointestinal

>toxicity and myelosuppression including neutropenic fever. This resulted in

>two prolonged hospital stays and a delay in her next course of

>chemotherapy.[2730]

>

>Penicillins may reduce the renal clearance of methotrexate. Increased serum

>concentrations of methotrexate with concomitant hematologic and

>gastrointestinal toxicity have been observed with concurrent administration

>of high or low doses of methotrexate and penicillins. Patients should be

>carefully monitored while receiving this combination.

>

>Probenecid decreases the renal elimination of methotrexate. Renal tubular

>transport of methotrexate is decreased by probenecid since methotrexate is

>secreted through the same mechanism as other weak organic acids. Probenecid

>has also been associated with decreased clearance of methotrexate from the

>CSF. Patients receiving this combination should be closely monitored.

>Nephrotoxic agents such as amphotericin B, aminoglycosides, or cisplatin

>may

>interfere with the renal elimination of methotrexate. Amphotericin B has

>also

>been reported to alter cell membrane structure and permeability resulting

>in

>increased influx of methotrexate.

>

>Omeprazole has been reported to delay methotrexate elimination in one

>patient. A drug interaction was implied since previously methotrexate

>elimination was normal in the absence of omeprazole. Methotrexate undergoes

>active tubular secretion in the kidney and omeprazole may interfere with

>renal ATPase. Although the patient was not rechallenged with omeprazole,

>omeprazole should be used cautiously in patients receiving high-dose

>methotrexate. Subsequent reports have not indicated an interaction.

>

>Patients receiving other agents that cause hepatotoxicity including ethanol

>or systemic vitamin A analogs (acitretin, alitretinoin, bexarotene,

>isotretinoin, and tretinoin, ATRA) could be at increased risk and monitored

>closely while receiving combination therapy. Although no longer available,

>etretinate has been shown to increase methotrexate serum concentrations and

>cases of hepatotoxicity have also been reported in patients receiving

>etretinate and methotrexate concomitantly. Concurrent use of vitamin A

>analogs and methotrexate may result in additive photosensitivity.

>

>Methotrexate may increase the photosensitizing effects of photodynamic

>therapy with methoxsalen or aminolevulinic acid.

>

>Concurrent administration of oral methotrexate with food may delay the

>absorption of methotrexate and decrease the maximum serum concentration.

>Methotrexate should be given 1 hour before or 2 hours after a meal.

>

>Concurrent use of methotrexate with other agents that cause bone marrow or

>immune suppression such as other antineoplastic agents or

>immunosuppressives

>may result in additive effects.

>

>In patients receiving immunosuppressant therapy with methotrexate, antibody

>development to infliximab was lower as compared to patients not receiving

>concurrent immunosuppression. In addition, the incidence of

>infusion-related

>reactions was lower in these patients. Rheumatoid arthritis patients who

>received methotrexate in combination with infliximab had higher serum

>concentrations of infliximab as compared to those who received infliximab

>alone. The impact of concurrent infliximab therapy and immunosuppression on

>the development of malignancies is unknown.

>

>The immune response of the immunocompromised patient to vaccines is

>decreased

>and higher doses or more frequent boosters may be required. Despite these

>dose increases, the immune response may still be suboptimal. Live virus

>vaccines are contraindicated during therapy with antineoplastic agents due

>to

>the potentiation of virus replication, adverse reactions to the virus, and

>the immunocompromised status of the patient. Those undergoing

>antineoplastic

>therapy should not be exposed to others who have recently received the oral

>poliovirus vaccine (OPV). Estimates for postponing vaccination vary from 3

>months to 1 year following discontinuation of treatment depending of the

>type

>of antineoplastic agent used and the disease state of the patient.

>

>Several corticosteroids have been reported to interact with methotrexate.

>Hydrocortisone has been shown to reduce thymidylate synthesis by

>methotrexate

>and decreases the cytotoxicity of methotrexate in vitro. Other in vitro

>studies have shown that hydrocortisone decreases the uptake of

>methotrexate.

>Prednisone has been shown to block methotrexate-induced inhibition of DNA

>synthesis. Dexamethasone and prednisolone do not interfere with the

>cytotoxicity of methotrexate.

>

>Drugs with similar pharmacologic activity, such as pyrimethamine, and

>trimetrexate, may lead to additive antifolate effects and bone marrow

>suppression.

>

>Methotrexate given 3—24 hours before fluorouracil, 5-FU, increases

>nucleotide

>formation and enhances cell kill and toxicity. However, when fluorouracil

>or

>floxuridine are given prior to methotrexate (0—24 hours), the

>cytotoxicity of

>methotrexate is decreased due to the inhibition of thymidylate synthetase.

>

>When methotrexate is given 3—24 hours prior to L-asparaginase,

>methotrexate

>blocks protein synthesis and decreases L-asparaginase toxicity. However,

>when

>L-asparaginase is given prior to or concurrently with methotrexate,

>cellular

>uptake of methotrexate is decreased resulting in decreased efficacy. It is

>recommended to give L-asparaginase at least 10—12 days prior to

>methotrexate

>or shortly after methotrexate administration.

>

>Methotrexate improves the cyctotoxic activity of cytarabine when given

>either

>before or after cytarabine. Methotrexate enhances activation of cytarabine

>by

>an unknown mechanism.

>

>Vincristine given 0—1 hour prior to methotrexate results in blockade of

>methotrexate efflux out of the cell. The administration methotrexate

>followed

>in 8—48 hours by vincristine results in enhanced therapeutic effects and

>increased methotrexate uptake.

>

>Concomitant use of methotrexate and 6-mercaptopurine, 6-MP, results in a

>31%

>increase in mercaptopurine levels.

>

>Methotrexate given prior to cyclophosphamide has been shown to immediately

>slow the biotransformation of cyclophosphamide and probably the activation

>of

>cyclophosphamide.

>

>Concurrent use of etoposide, VP-16, or teniposide, VM-26, with methotrexate

>results in decreased methotrexate efflux out of cells. There is a potential

>for increased methotrexate efficacy and/or toxicity with these

>combinations.

>

>In vitro studies have shown that allopurinol administered one hour prior to

>methotrexate may decrease the therapeutic effects of methotrexate.

>

>Methotrexate may decrease the clearance of theophylline. Theophylline

>levels

>should be closely monitored when used concurrently with methotrexate. In a

>small number of patients with either leukemia or lymphoma and acute

>methotrexate neurotoxicity, theophylline attenuated methotrexate-induced

>neurotoxicity, a syndrome believed due to elevated adenosine CNS

>concentrations.[657]

>

>In patients with rheumatoid arthritis, disease modifying antirheumatic

>drugs,

>DMARDs (e.g., gold compounds, penicillamine, and sulfasalazine) have been

>associated with methotrexate-induced pulmonary toxicity. In non-diabetic

>patients, previous use of disease modifying drugs is one of the most

>important risk factors for methotrexate-induced pulmonary toxicity.[2148]

>Concurrent therapy of methotrexate with azathioprine or sulfasalazine may

>increase the risk of hepatotoxicity.

>

>Due to the thrombocytopenic effects of methotrexate when used as an

>antineoplastic agent, an additive risk of bleeding may be seen in patients

>receiving concomitant anticoagulants, platelet inhibitors, including

>aspirin,

>strontium-89 chloride, and thrombolytic agents. A significantly higher

>incidence of leukopenia has been seen in patients taking aspirin during

>methotrexate therapy.

>

>Because antineoplastic agents exert their toxic effects against rapidly

>growing cells, such as hematopoietic progenitor cells, sargramostim,

>GM-CSF,

>and filgrastim, G-CSF, are contraindicated for use in patients within 24

>hours of treatment with antineoplastic agents.

>

>[ Revised 2/24/00 ]

>

>

>------------------------------------------------------------------------

>Failed tests, classes skipped, forgotten locker combinations.

>Remember the good 'ol days

>1/4053/2/_/524922/_/959185867/

>------------------------------------------------------------------------

>

>For links to websites with JRA info visit:

>http://www.geocities.com/Heartland/Village/8414/Links.html

>

________________________________________________________________________

Get Your Private, Free E-mail from MSN Hotmail at http://www.hotmail.com

[Guest guest]

I asked my rheumy about this folic acid issue. She said that theer have been

a number of studies done on the effectiveness of MTX when using with Folic

Acid. And That they had found that the benfits of the folic acid in reducing

side effects was far greater than any weakenign that it may do of MTX. She

said that teh lastest study showed no signifigant evidence of a weakenign of

teh MTX when using Folic acid .

Just thought I'd throw my 2 cents in HA Kris

[Guest guest]

Hi ,

My daughter, Kelsey, is also 3 and has poly jra. She was just diagnosed in

April, and has been taking Naprosyn twice a day since then. She has also

been going to physical therapy and just started occupational therapy for her

hands, as they are her " stubborn " area right now. Kelsey has blonde hair,

blue eyes and I have never been told of scarring as a side effect. I've

never heard of it, but now I'm curious! So far, Kelsey has responded quite

well to the Naprosyn along with her therapy. There is still room for

improvement, but some people have said Naprosyn alone didn't help their

child much at all, so I feel fortunate that it has helped Kelsey as much as

it has.

About the eye exam... I was really worried about this too, but we have been

to Kelsey's first visit and it went very well. The only thing she didn't

like was the eye drops to dilate her pupils. I don't think it was painful or

anything, she just didn't like them! lol You may want to take a pair of

sunglasses for since the drops make eyes sensitive to light. Once the

dilation wears off, everything is usually back to normal.

Good luck, and I hope will only get better! )

[Guest guest]

Teri,

is so cute! She looks like a little doll!

[Guest guest]

You did it right. She's a cutie.

Diane.

[Guest guest]

WHAT a CUTIE!

As Skyler would say....

"Hubba Hubba!"

[ & Skyler]

(no subject)

Hello everybody, Here is a picture of my angel baby. is 3 yrs old . Hopefully I sent the picture right this time. I am a novice computer user. TeriFor links to websites with JRA info visit: http://www.geocities.com/Heartland/Village/8414/Links.html

[Guest guest]

Hi Teri,

is adorable! What a beautiful child. Thank you for sharing the

picture

Aloha,

Georgina

[Guest guest]

teri

thanks i checked it out and interesting

Robbin

[Guest guest]

Teri

i got this late how are yall doing?

hows your mother in law?

Robbin