Searching Full Texts of Amazon.com Books [Was: RE: Searching Dr. Walford's Books]

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ALL:Amazon.dom is providing an awesome! research tool. Currently, this key word search feature will search the full text of over 120,000 books.I searched for IGF-1 & Insulin separately. Here's an example below of the excerpts it found from Dr. Walford's B120YD book.BTW, an earlier post explains how to see (and save) the full text of the page found for your search term:http://tinyurl.com/srbbor /message/8567Live long & optimally! ;-)6 references to IGF-1 in this book:1. on Page 57: ". . . decrease in blood lipids (cholesterol and triglycerides ), in blood sugar and insulin, in the white blood cell count, in IGF-1 (related to growth hormone); they show an increase in a certain blood enzyme (alkaline phosphatase ),just as rodents do. The . . ."2. on Page 159: ". . . perceived physical and psychological well-being, an increase in muscle strength S"44 with n0 sig- and lean body mass, and increased IGF-i concentration, nificant adverse effects (blood glucose and insulin not increased). Possi- ble risks of long-term usage, if any, are not . . ."3. on Page 160: ". . . us any insight into HRT. Yes, I believe so. Growth hormone and its effector agent, the so-called insulin-derived growth factor (IGF-1), are decreased in CRON ani- mals. In short the adaptation consequent to calorie restriction, the only procedure known to retard . . ."4. on Page 194: ". . . evidence is not in my view sufficient to justify treatment with a powerful and potentially dangerous hormone. Growth hormone increases IGF-i, the so-called Cc insulinlike growth factor," which exerts multiple insulinlike meta- bolic effects, including increase in blood sugar, inhibition of . . ."5. on Page 195: ". . . may induce an increase in physical and psychological well-being, an increase in lean body mass, in plasma androgen and in IGF-1 concentrations. Effect on life span and disease in humans has not been established. In humans melatonin secretion from the pineal . . ."6. from Index: ". . . aging, 158 supplements, 158-6o,194-95 hotel restaurants, 242 human growth hormone, 158-59, 16o, 194 hummus, 312 Hunzas,1o6,113 hypertension, 122-25,172 Ibuprofen, 161 IGF-1. See insulin-derived growth factor immune-response capacity, 152,174 immune system age-related diseases, 52-53 autoantibodies, 40, 41, 42, 227 immunological theory of . . ."28 references to insulin in this book:1. on Page 20: ". . . I may also point out that all the changes Sears attributes to a Zone diet, e.g. low blood glucose, low insulin, low glycolsylated hemoglo- bin, were produced by the Biospherian diet '9 in which I participated, and which in its ratios . . ."2. on Page 38: ". . . body absorbs it from your intestines and your level of blood sugar rises sharply. Your pancreas then secretes the hormone insulin, which stim- ulates the utilization or storage of the sugar. With age these pathways become less effective b So-called "insulin . . ."3. on Page 44: ". . . 18% decrease Diastolic blood pressure 28% decrease Blood sugar 21% decrease Cholesterol 36% decrease White blood cell count 31% decrease Insulin 42% decrease T3 (a thyroid hormone) 19% decrease Renin gradual decrease Glycosylated hemoglobin gradual decrease Triglycerides gradual decrease . . ."4. on Page 55: ". . . biomarker tests, and there are many, follow the same positive pattern. The CRON diet prevents the usual rise in blood insulin and blood sugar that occurs with age. It prevents the insulin-resistance of the tissues that develops with age.-19 It delays . . ."5. on Page 57: ". . . of aging. Like rodents, calorie-reduced monkeys reveal a decrease in blood lipids (cholesterol and triglycerides ), in blood sugar and insulin, in the white blood cell count, in IGF-1 (related to growth hormone); they show an increase in a certain blood . . ."6. on Page 58: ". . . of the quaint terminology) increase the worm's life span by 300 percent." These genes plays a key role in the insulin-glucose metabolic pathway. There are similar genes also in mammals. Such across-species findings lend support to the glycemic theory's interpre- tation . . ."7. on Page 83: ". . . diet program. APPETITE AND ITS CONTROL Following weight loss resulting from low calorie intake, the concentra- tions of the hormones insulin and leptin (a recently discovered hor- mone released from fat tissue itself) decrease, and glucocorticoid con- centrations increase. This combination . . ."8. on Page 84: ". . . 84 BEYOND THE 120-YEAR DIET adrenal cortex antagonize the actions of the fat-derived hormone leptin and of insulin in the control of energy balance. Glucocorticoid increase (as occurs to a mild degree in people on a CRON diet) . . ."9. on Page 85: ". . . the body that result in its asking, "What's to eat?" These involve metabolic rates, the energy enzymes of the body, insulin, blood sugar, and a portion of the brain called the hypothalamus. Trying to handle all these events with a pill . . ."10. on Page 87: ". . . in humans that closely resemble some of those seen in animals on similar diets: a decrease in cholesterol, an improved insulin response, and a better regulation of blood sugar. Anorexia nervosa is a dreadful, sometimes fatal psychological dis- ease in which . . ."11. on Page 102: ". . . Bananas, watermelon, and carrots have high GIs; apples and peaches, low. (See Table 9.3.) High-GI diets generate a demand for insulin, and hyperinsuline- mia is linked with all facets of the metabolic syndrome (insulin resist- ance, hyperlipidemia, hypertension, and visceral obesity). . . ."12. on Page 103: ". . . in food reduce glycemia by slowing empty- ing time, but glucose tolerance to the subsequent meal is impaired. And while insulin responses follow the rank order of the glycemic responses, high-protein and high-fat foods stimulate greater insulin responses than predicted by . . ."13. on Page 104: ". . . that both high-MUFA and high-carbohydrate diets lower LDL to the same degree, but high-car- bohydrate diets increase blood glucose and insulin and result in increased triglycerides and lower HDL (i.e. are atherogenic and dia- betogenic). However, reducing the GI of the . . ."14. on Page 112: ". . . diet simply does not supply enough chromium. This causes the body to use its own stores in order to help insulin handle the rise in blood sugar that follows a sugar rich meal. Part of this mobilized chromium is then excreted . . ."15. on Page 127: ". . . literally starves to death because his body is unable to derive energy from glucose, either because his ability to make insulin is faulty (type I, or "juvenile," dia- betes, accounting for about io percent of cases) or because the insulin rceptors . . ."16. on Page 128: ". . . be any of many genetic mutations. Disruption can occur anywhere in the long pathway from the synthesis and release of insulin to the complete oxidation of glucose by the target cells. There are many steps, involv- ing many enzymes, and alterations . . ."17. on Page 129: ". . . lost an average Of 24 pounds, the researchers observed an improvement in glucose metabolism and an impressive decrease in the insulin levels following a glucose meal. And CRON- diet treated, 12-month-old rats give glucose and insulin responses similar to those of . . ."18. on Page 131: ". . . for you. Studies of long-term regimes of moderate calorie reduction in humans suggest that a CRON diet should improve the insulin response and control of blood sugar not only in diabetics but in the normal elderly." OSTEOPOROSIS No one knows just . . ."19. on Page 134: ". . . in the medicine/nutrition community is that low-fat, high-carbohydrate (especially high-GI) diets cause triglycerides to go up, HDLs to go down, insulin resistance to worsen. And while total LDLs remain unchanged, there is a shift from large buoyant LDLs to small, dense . . ."20. on Page 137: ". . . blood pressure, blood cholesterol (pre-entry average of 195 becoming 123 within six to eight months), blood glucose (21 percent decline), insulin (42 percent), glycosylated hemoglobin (15 percent). Initially tryglicerides actually increased mildly, but then went gradually down (40 percent decline within . . ."21. on Page 145: ". . . is probably a mistake to think that diabetes truly accelerates aging, and that since it is caused by an out-of-control glucose/insulin metabolic system, if one could . . ."22. on Page 159: ". . . muscle strength S"44 with n0 sig- and lean body mass, and increased IGF-i concentration, nificant adverse effects (blood glucose and insulin not increased). Possi- ble risks of long-term usage, if any, are not yet identified. There is good evidence that estrogen . . ."23. on Page 160: ". . . of CRON give us any insight into HRT. Yes, I believe so. Growth hormone and its effector agent, the so-called insulin-derived growth factor (IGF-1), are decreased in CRON ani- mals. In short the adaptation consequent to calorie restriction, the only procedure . . ."24. on Page 194: ". . . this- it is indeed the only regime that works), growth hormone and IGF-1 are decreased. And of course glucose and insulin are also decreased. DHEA, an adrenal gland hormone, reaches highest concentrations in . . ."25. on Page 200: ". . . by subtracting his or her age from 220) for at least 20 minutes three times a week. Exercise increases the insulin sensitivity of the tissues and improves carbohydrate metabolism.' And the physically fit person has only about 6o percent as much . . ."26. on Page 236: ". . . simple carbohydrates like table sugar were readily digested and rapidly absorbed, producing large and rapid increases in blood sugar and insulin levels. Upon experimentation, however, investigators found the situation to be quite variable.2' The idea of complex carbohydrates has evolved into . . ."27. on Page 237: ". . . response does not closely correspond to whether a carbohydrate is simple or complex.=4* The response of blood glucose (and blood insulin) to some starches (white potatoes, for example) is nearly as great as to refined sugars, whereas other starches (most grains, . . ."28. from Index: ". . . supplements, 158-6o,194-95 hotel restaurants, 242 human growth hormone, 158-59, 16o, 194 hummus, 312 Hunzas,1o6,113 hypertension, 122-25,172 Ibuprofen, 161 IGF-1. See insulin-derived growth factor immune-response capacity, 152,174 immune system age-related diseases, 52-53 autoantibodies, 40, 41, 42, 227 immunological theory of aging, 57-58 . . .".