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Re: New Soy Milk: Sugar-Free

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There are other possible risks aside from brain atrophy for men:

http://www.mercola.com/article/soy/avoid_soy.htm

I am not claiming these possible risks are significant, but one should

probably be aware of the issues debated regarding soy and health.

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>From: " prakasharadamba " <pk@...>

>Reply-

>

>Subject: [ ] Re: New Soy Milk: Sugar-Free

>Date: Sun, 21 Sep 2003 13:37:38 -0000

>

>Are you referring to the thread from early August re: neurotoxicity or

>is this something else?

>

>

>

>

> > No downsidess aside from the great soy health risk issue debates,

>that is.

> >

> >

> >

> >

> >

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>

>

>

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> There are other possible risks aside from brain atrophy for men:

> http://www.mercola.com/article/soy/avoid_soy.htm

> I am not claiming these possible risks are significant, but one

> should probably be aware of the issues debated regarding soy and

> health.

*****Yes, thank you for adding the point above. It is true: soy

consmption is certainly open to debate. However...

two things to keep in mind:

(1) the article on Dr. Mercola's website is not written by him

(although I'm sure he endorsed it). It's authors are the (in)famous

duo: Sally Fallon & G. Enig. One needs to keep *that* in mind

when reading any criticism of soy products.

(2) The single study (albeit well-performed) which suggested that

large soy consumption in middle age might produce dementia has been

questioned by several professionals as being weak in various areas.

For example:

a) it seems that the soy consumed by the men in question *may* have

had unusually high levels of aluminum in it and aluminum has been

implicated in dementia cases.

B) also, the study was based on the diet recollection of the

individuals involved, a notoriously UNreliable source of scientific

data.

c)in addition, the study even stated that the actual impact of the

soy on the dementia was extremely small, that there were other

factors which certainly could have contributed to the dementia of the

individuals involved.

d) furthermore, the study speculated about the causal role of

isoflavones. However, there was no direct evidence of isoflavone

producing the increase in ischemic brain disease because the study

authors did not make any direct measure of isoflavone levels, for

example from urine samples.

e) as well, the study was epidemiological in nature and these types

of studies have significant limitations (e.g., memory of

participants). As with all epidemiological studies, there are

hundreds of uncontrolled factors potentially affecting outcomes:

e.g., perhaps there was consumption of a food or foods (other than

the 26 surveyed in the study) that might have contribute to ischemic

brain aging (ex: sugar).

....there are several more of these " issues " which suggest that the

conclusions of the Hawaiian study done by White et al. are not really

conclusions but open questions meriting further study and research.

Reading the piece by Fallon and Enig in isolation slants the debate

in the opposite direction and does an injustice to the inquiry at

hand.

andy

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