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Re: Outcome in Children

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Thanks Claudine, I've read and sent all 3 articles about HCV and

children to Pat...she sent an email and said, " thank you! " I also

told her about this group and how it's real good for exchanging HCV

info. Actually, this is the most informative HCV e-group that I

belong to.

God bless,

> Taken from:

> Recovery, Persistence, and Sequelae in Hepatitis C

> Infection: continued...

> [sem Liver Disease 20(1):17-35,2000. © 2000 Thieme

> Medical Publishers, Inc.]

>

>

> --------------------------------------------------------------------

------------

>

> Outcome in Children

> Data from studies of infants and children are just

> beginning to emerge. Hepatitis C infection is not

> common in children because perinatal spread is

> uncommon and because needle exposures are generally

> limited to blood transfusion. The National Health and

> Nutrition Epidemiologic Survey shows that the

> prevalence of anti-HCV in children aged 6-11 is only

> 0.2% and rises to only 0.4% in those aged 12-19.[15]

> One of the most comprehensive outcome studies in

> children was conducted by Vogt et al. in Germany,[45]

> who enrolled 458 children who had cardiac surgery

> before the implementation of blood donor screening

> (Table 1). The patients had undergone cardiac surgery

> a mean of 17 years (range, 12-27 years) earlier at a

> mean age at first operation of 2.8 years. None had

> received prior or subsequent transfusions and none had

> mothers with detectable HCV infection. An age- and

> sex-matched control group from the general population

> was also studied. Anti-HCV was detected in 67 (14.6%)

> patients compared with 3 (0.7%) among the controls. At

> follow-up evaluation, 37 patients (55%) were HCV RNA

> positive and 45% appeared to have spontaneously

> cleared the infection. All but one patient was found

> to have normal ALT values, and this single patient had

> severe right-sided congestive heart failure. Of the 17

> patients who underwent liver biopsy, only 2 had

> histologic evidence of portal fibrosis, and both these

> patients had chronic congestive heart failure that

> might have accounted for the observed changes. One

> additional patient had " micronodular " cirrhosis, but

> this person was co-infected with the hepatitis B virus

> (HBV), and the relative role of HCV in the

> pathogenesis of the cirrhosis could not be

> established.

> The study of Vogt et al. is the largest reported

> outcome study in children and describes a relatively

> benign course for transfusion-associated hepatitis C

> over a period of near 20 years. Importantly, they also

> found that almost one half of the infected children

> had spontaneously cleared HCV over this interval. The

> authors concluded that the natural history of chronic

> hepatitis C in childhood is either more benign or more

> slowly progressive than in adults.

>

> Losasciulli et al.[46] reported serologic and

> molecular follow-up data on 114 children with

> childhood leukemia of whom 56 (49%) were HCV RNA

> positive at the end of chemotherapy. Seventeen year

> follow-up of the HCV RNA positive cohort revealed that

> all were asymptomatic, that ALT values were normal in

> 71%, and that 16 of the 56 (29%) had spontaneously

> cleared their viremia. No liver biopsy data was

> reported in this study. A small study by -Monzon

> et al.[47] compared the outcome in 24 HCV-infected

> children and 22 HCV-infected adults. After a mean

> follow-up of 11 years, the comparative outcomes for

> children versus adults were as follows: mean viral

> load 3.6 2105 versus 5.6 2105 copies/mL, histologic

> grade (scale 0-4) 0.6 ± 0.7 versus 3.2 ± 1.1, and

> histologic stage (scale 0-4) 0.5 ± 0.5 versus 2.6 ±

> 1.2. Thus, despite similar viral loads, both hepatic

> inflammation and fibrosis were markedly less in

> children than adults.

>

> Luban et al.[48] recently provided an interim report

> of a look-back study of 5,446 pediatric recipients of

> blood administered between 1982 and 1992. The mean age

> at transfusion was 1.0 year (range, birth to 10.7

> years). The mean age at testing was 11 years (range,

> 4-17 years). Of 1,753 recipients thus far tested, 36

> (2.0%) are confirmed anti-HCV positives compared with

> 0.3% of an age-matched nontransfused control

> population. Of the 36 HCV-positive children, all are

> asymptomatic. The range of ALT was 29-140 IU/L and 80%

> had at least one ALT value greater than 1.5 times the

> upper limit of normal. Thus far, only 7 of the 36 have

> been biopsied. After a mean interval of 13.6 years

> since exposure to blood, six patients showed only mild

> inflammation without fibrosis and one patient had mild

> inflammation with early bridging fibrosis.

>

> Thus, although data from pediatric follow-up studies

> are still sparse, the available data consistently show

> mild outcomes over the first two decades of infection

> with a high rate of spontaneous recovery (29-45%) as

> assessed by the loss of HCV RNA. Nonetheless, it is

> unclear whether hepatitis C is actually milder in

> children or just more slowly progressive. If the

> latter, then the long-anticipated lifespan of infected

> children would allow them to eventually reach the same

> levels of cirrhosis and HCC as persons infected later

> in life. It is critical that these childhood cohorts

> continue to be followed and reported and that

> additional studies be undertaken among children

> infected 10 or more years ago.

>

>

> __________________________________________________

>

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